EC Number |
Disease |
PubMed ID |
Title of Publication |
Category |
Confidence Level |
---|
2.7.11.16 | Carcinoma, Hepatocellular |
23375037 |
GRK6 expression in patients with hepatocellular carcinoma. |
diagnostic usage ongoing research unassigned |
3 2 0 |
2.7.11.16 | Carcinoma, Hepatocellular |
28819396 |
Expression and Significances of G-Protein-Coupled Receptor Kinase 3 in Hepatocellular Carcinoma. |
causal interaction diagnostic usage ongoing research unassigned |
2 4 2 0 |
2.7.11.16 | Cardiomegaly |
22859683 |
Determining the Absolute Requirement of G Protein-Coupled Receptor Kinase 5 for Pathological Cardiac Hypertrophy. |
unassigned |
0 |
2.7.11.16 | Cardiomegaly |
23023506 |
G protein-coupled receptor kinase 5: exploring its hype in cardiac hypertrophy. |
causal interaction therapeutic application unassigned |
3 2 0 |
2.7.11.16 | Cardiomegaly |
26515328 |
Differential Role of G Protein-Coupled Receptor Kinase 5 in Physiological Versus Pathological Cardiac Hypertrophy. |
causal interaction unassigned |
3 0 |
2.7.11.16 | Cardiomegaly |
29628444 |
PH domain leucine-rich repeat protein phosphatase 2 (PHLPP2) regulates G-protein-coupled receptor kinase 5 (GRK5)-induced cardiac hypertrophy in vitro. |
causal interaction therapeutic application unassigned |
1 1 0 |
2.7.11.16 | Cardiomegaly |
32856617 |
KR-39038, a Novel GRK5 Inhibitor, Attenuates Cardiac Hypertrophy and Improves Cardiac Function in Heart Failure. |
causal interaction therapeutic application unassigned |
3 4 0 |
2.7.11.16 | Cardiomegaly |
33560342 |
GRK5 contributes to impaired cardiac function and immune cell recruitment in post-ischemic heart failure. |
ongoing research unassigned |
1 0 |
2.7.11.16 | Cardiovascular Diseases |
21036254 |
Characterization of G protein-coupled receptor kinase 4 and measuring its constitutive activity in vivo. |
causal interaction unassigned |
4 0 |
2.7.11.16 | Cardiovascular Diseases |
24210504 |
Design and synthesis of novel 3-(benzo[d]oxazol-2-yl)-5-(1-(piperidin-4-yl)-1H-pyrazol-4-yl)pyridin-2-amine derivatives as selective G-protein-coupled receptor kinase-2 and -5 inhibitors. |
causal interaction therapeutic application unassigned |
3 4 0 |