EC Number |
Disease |
PubMed ID |
Title of Publication |
Category |
Confidence Level |
---|
2.1.1.355 | Adenocarcinoma |
19345325 |
Rb Regulates DNA damage response and cellular senescence through E2F-dependent suppression of N-ras isoprenylation. |
ongoing research unassigned |
1 0 |
2.1.1.355 | Adenocarcinoma |
20573371 |
Implications of enhancer of zeste homologue 2 expression in pancreatic ductal adenocarcinoma. |
unassigned |
0 |
2.1.1.355 | Adenocarcinoma |
22536950 |
A small-molecule probe of the histone methyltransferase G9a induces cellular senescence in pancreatic adenocarcinoma. |
unassigned |
0 |
2.1.1.355 | Adenocarcinoma |
23395645 |
Enhancer of Zeste Homolog 2 Silences MicroRNA-218 in Human Pancreatic Ductal Adenocarcinoma Cells by Inducing Formation of Heterochromatin. |
causal interaction diagnostic usage ongoing research therapeutic application |
4 3 2 1 |
2.1.1.355 | Adenocarcinoma |
23807219 |
Gossypol and an HMT G9a inhibitor act in synergy to induce cell death in pancreatic cancer cells. |
causal interaction unassigned |
4 0 |
2.1.1.355 | Adenocarcinoma |
30903272 |
Robust expression of EZH2 in endocervical neoplastic lesions. |
diagnostic usage ongoing research unassigned |
1 2 0 |
2.1.1.355 | Adenocarcinoma |
31398867 |
Expression of the Major and Pro-Oncogenic H3K9 Lysine Methyltransferase SETDB1 in Non-Small Cell Lung Cancer. |
causal interaction diagnostic usage ongoing research therapeutic application |
1 3 4 1 |
2.1.1.355 | Adenocarcinoma |
32360551 |
SETDB1 Inhibits p53-Mediated Apoptosis and Is Required for Formation of Pancreatic Ductal Adenocarcinomas in Mice. |
causal interaction diagnostic usage ongoing research therapeutic application |
2 2 4 2 |
2.1.1.355 | Adenocarcinoma |
32729861 |
Histone methyltransferase G9a inhibitor-loaded redox-responsive nanoparticles for pancreatic ductal adenocarcinoma therapy. |
causal interaction therapeutic application unassigned |
4 4 0 |
2.1.1.355 | Adenocarcinoma |
33639051 |
Effects of miR-362 in Regulating the Proliferation, Invasion and Apoptosis of Gastric Cancer by Inhibiting the Expression of Tumor-Promoting Factor PRDM2. |
causal interaction diagnostic usage ongoing research unassigned |
4 1 3 0 |