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2.1.1.181
malfunction
knockout of the ybiN gene leads to loss of modification at adenine1618. The modification is restored if ybiN knock-out strain has been co-transformed with a plasmid expressing the ybiN gene. Both lack of the ybiN gene and it's overexpression leads to growth retardation and loss of cell fitness comparative to the parental strain
705116
2.1.1.181
physiological function
A1618 modification could be necessary for the exit tunnel interaction with some unknown regulatory peptides
705116
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