EC Number   |
Disease |
PubMed ID |
Title of Publication |
Category |
Confidence Level |
|---|
   3.4.24.29 | Abscess |
23801604 |
Staphylokinase Promotes the Establishment of Staphylococcus aureus Skin Infections While Decreasing Disease Severity. |
causal interaction
diagnostic usage
ongoing research
therapeutic application |
3
3
2
1 |
| 3.4.24.29 | Anaphylaxis |
10496894 |
Complement-dependent accumulation and degradation of platelets in the lung and liver induced by injection of lipopolysaccharides. |
causal interaction
unassigned |
1
0 |
| 3.4.24.29 | Anemia, Dyserythropoietic, Congenital |
7506081 |
Aberrant regulation of complement by the erythrocytes of hereditary erythroblastic multinuclearity with a positive acidified serum lysis test (HEMPAS). |
unassigned |
0 |
| 3.4.24.29 | Anti-Glomerular Basement Membrane Disease |
19906361 |
Autoimmune kidney diseases. |
diagnostic usage
unassigned |
2
0 |
| 3.4.24.29 | Arthritis, Experimental |
12902517 |
Prevention of collagen-induced arthritis in mice transgenic for the complement inhibitor complement receptor 1-related gene/protein y. |
ongoing research
unassigned |
3
0 |
| 3.4.24.29 | Arthritis, Infectious |
14998519 |
Impact of staphylococcal protease expression on the outcome of infectious arthritis. |
diagnostic usage
ongoing research
therapeutic application
unassigned |
3
4
1
0 |
| 3.4.24.29 | Atypical Hemolytic Uremic Syndrome |
19584399 |
Hyperfunctional C3 convertase leads to complement deposition on endothelial cells and contributes to atypical hemolytic uremic syndrome. |
ongoing research
unassigned |
3
0 |
| 3.4.24.29 | Atypical Hemolytic Uremic Syndrome |
22250080 |
A novel C3 mutation causing increased formation of the C3 convertase in familial atypical hemolytic uremic syndrome. |
causal interaction
unassigned |
4
0 |
| 3.4.24.29 | aureolysin deficiency |
28729648 |
A haplotype in CFH family genes confers high risk of rare glomerular nephropathies. |
causal interaction
unassigned |
4
0 |
| 3.4.24.29 | Autoimmune Diseases |
14512320 |
Intravenously applied IgG stimulates complement attenuation in a complement-dependent autoimmune disease at the amplifying C3 convertase level. |
unassigned |
0 |
