EC Number   |
Disease |
PubMed ID |
Title of Publication |
Category |
Confidence Level |
|---|
   1.8.5.1 | Acatalasia |
28782990 |
Glutathione oxidation in response to intracellular H2O2: Key but overlapping roles for dehydroascorbate reductases. |
causal interaction
unassigned |
3
0 |
| 1.8.5.1 | Acidosis |
9553959 |
Acidosis-induced metallothionein (MT) mRNA expression in neonatal rat primary astrocyte cultures. |
diagnostic usage
ongoing research
unassigned |
3
1
0 |
| 1.8.5.1 | Acidosis |
9622659 |
Dimethyl sulfoxide, but not acidosis-induced metallothionein mRNA expression in neonatal rat primary astrocyte cultures is inhibited by the bioflavonoid, quercetin. |
unassigned |
0 |
| 1.8.5.1 | Adenocarcinoma |
8692980 |
Antisense RNA to the type I insulin-like growth factor receptor suppresses tumor growth and prevents invasion by rat prostate cancer cells in vivo. |
causal interaction
diagnostic usage
ongoing research
therapeutic application |
4
1
4
4 |
| 1.8.5.1 | Alzheimer Disease |
26836194 |
Overexpression of Metallothionein-1 Modulates the Phenotype of the Tg2576 Mouse Model of Alzheimer's Disease. |
causal interaction
ongoing research
unassigned |
1
1
0 |
| 1.8.5.1 | Alzheimer Disease |
26993125 |
Structure, function and disease relevance of Omega-class glutathione transferases. |
causal interaction
therapeutic application
unassigned |
4
4
0 |
| 1.8.5.1 | Alzheimer Disease |
28134760 |
Influence of Transgenic Metallothionein-1 on Gliosis, CA1 Neuronal Loss, and Brain Metal Levels of the Tg2576 Mouse Model of Alzheimer's Disease. |
ongoing research
unassigned |
1
0 |
| 1.8.5.1 | Amyotrophic Lateral Sclerosis |
26993125 |
Structure, function and disease relevance of Omega-class glutathione transferases. |
causal interaction
therapeutic application
unassigned |
4
4
0 |
| 1.8.5.1 | Arthritis |
28344594 |
Articular inflammation induced by an enzymatically-inactive Lys49 phospholipase A |
ongoing research
therapeutic application
unassigned |
3
4
0 |
| 1.8.5.1 | Arthritis, Rheumatoid |
30055006 |
Metallothionein-1 suppresses rheumatoid arthritis pathogenesis by shifting the Th17/Treg balance. |
causal interaction
therapeutic application
unassigned |
2
1
0 |
