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EC Number Substrates Commentary Substrates Organism Products Commentary (Products) Reversibility
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec - Trypanosoma brucei ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec - Leishmania donovani ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec - Methanocaldococcus jannaschii ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec - Trypanosoma brucei brucei ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec L-phosphoseryl-tRNA is the crucial precursor for L-selenocysteinyl-tRNA formation in archaea and eukarya. Selenocysteine formation is achieved by a two-step process: O-phosphoseryl-tRNASec kinase (PSTK) phosphorylates the endogenous Ser-tRNASec to O-phosphoseryl-tRNASec, and then this misacylated amino acid-tRNA species is converted to L-selenocysteinyl-tRNASec by Sep-tRNA:Sec-tRNA synthase (SepSecS) Methanocaldococcus jannaschii ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec null mutants of PSTK abolish selenoprotein synthesis, demonstrating the essentiality of the enzyme for the formation of L-selenocysteinyl-tRNASec. Growth of the knockout strain is not impaired. Thus, unlike mammals, trypanosomes do not require selenoproteins for viability Trypanosoma brucei ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec Pstk is a limiting factor for hepatic selenoprotein biosynthesis, and its mRNA expression is most strongly affected during the lipopolysaccharide (LPS)-induced acute-phase response Mus musculus ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec about 30% of O-phospho-L-seryltRNA[Ser]Sec is converted to L-seryl-tRNA[Ser]Sec and ATP during the course of the reaction. The kinase is specific for the two major isoforms of O-phospho-L-seryl-tRNA[Ser]Sec. Seryl-tRNA1Ser does not serve as a substrate for PSTK Mus musculus ADP + O-phospho-L-seryl-tRNASec - r
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec the enzyme does not recognize free Ser as a substrate for phosphate transfer. Ser attached to tRNASec is its obligate substrate. Neither tRNASer nor L-seryl-tRNASer is a substrate for phosphorylation Methanocaldococcus jannaschii ADP + O-phospho-L-seryl-tRNASec - ?
Show all pathways known for 2.7.1.164Display the word mapDisplay the reaction diagram Show all sequences 2.7.1.164ATP + L-seryl-tRNASec - Methanocaldococcus jannaschii DSM 2661 ADP + O-phospho-L-seryl-tRNASec - ?
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