EC Number |
Application |
Reference |
---|
2.7.11.17 | medicine |
arrhythmia is favored by CamKII activation during acidosis |
690363 |
2.7.11.17 | medicine |
blocking CaMKII or CaMKII-mediated signaling may offer a novel therapeutic target for the treatment of neuropathic pain |
705313, 705318 |
2.7.11.17 | medicine |
Ca2+/calmodulin-dependent protein kinases may be involved in cancer, cardiac hypertrophy, heart failure, arrhythmia, cardiomyocyte apoptosis, the regulation of higher order neuronal functions such as memory, learning disorder, Angelman's syndrome, Parkinson's disease, Alzheimer's disease, delayed neuronal death, diabetes, and osteoporosis |
691539 |
2.7.11.17 | medicine |
CaM-kinases act as potential targets in cancer therapy, strategies, overview |
664865 |
2.7.11.17 | medicine |
CaMKII activity is significantly increased in heart failure |
693128 |
2.7.11.17 | medicine |
CaMKII inhibition may be a promising therapeutic target for the treatment of arrhythmias and contractile dysfunction |
740328 |
2.7.11.17 | medicine |
CaMKII is an essential component of intracellular signaling pathways regulating chondrocyte maturation |
691978 |
2.7.11.17 | medicine |
CaMKII is associated with diabetes-induced renal and vascular dysfunction |
691668 |
2.7.11.17 | medicine |
CaMKIIcelta expression and activation state are increased in heart failure |
691837 |
2.7.11.17 | medicine |
enzyme-dependent alterations in Na+ channel gating may cause arrhythmogenesis in many millions of people carrying the clinical diagnosis of heart failure |
722760 |