EC Number   |
Inhibitors |
Structure  |
|---|
    4.1.1.19 | more |
no inhibition by 1 mM alpha-difluoromethylornithine |
 |
| 4.1.1.19 | more |
no inhibition by DL-alpha-difluoromethylornithine |
|
| 4.1.1.19 | more |
profiles of the steady-state accumulation of Synechocystis ADC transcripts and ADC specific activities under various environmental conditions, e.g. darkness and cold reduce enzyme expression, overview |
|
| 4.1.1.19 | more |
activity not affected by Mg2+ or Ca2+ at 0.1 mM |
|
| 4.1.1.19 | more |
the mRNA level of adiA is 4fold lower in the mutants deficient in both alpha-subunit and beta-subunit of major histon-like protein HU |
|
| 4.1.1.19 | more |
inhibition of ADC results in much greater levels of oxidative damage than seen in plants treated with 0.5 microM phenanthrene; treatment with DL-alpha-difluoromethylarginine suppresses the large increase in ADC activity observed when the plants are exposed to 0.1 or 0.5 microM phenanthrene in the absence of DL-alpha-difluoromethylarginine; treatment with methylglyoxal-bis(guanylhydrazone) does not significantly influence ADC activity |
|
| 4.1.1.19 | more |
1 mM phenylhydrazine does not inactivate the enzyme. No inhibition with D-arginine, L-citrulline, L-lysine, Nalpha-methyl-L-arginine, L-methionine, Nalpha-nitro-L-arginine methyl ester, or L-ornithine |
|
| 4.1.1.19 | more |
mercury compounds inhibits the enzyme activity but induce the enzyme expression in PC12 cells, molecular mechanism of mercury compounds' neurotoxicity and enzyme inhibition mechanism, molecular dynamics simulation, product-competitive displacement method, method optimization, overview |
|
| 4.1.1.19 | more |
alpha-difluoromethylornithine, DFMO, a suicide inhibitor, does not inhibit ADC activity |
|
| 4.1.1.19 | Mg2+ |
- |
  |
