Organism | UniProt | Comment | Textmining |
---|---|---|---|
Xanthomonas axonopodis | - |
pv. citri | - |
General Information | Comment | Organism |
---|---|---|
malfunction | xanthan-deficient mutants of Xanthomonas axonopodis pv. citri, the bacterium responsible for citrus canker, are generated by deletion and marker exchange of the region encoding the carboxy-terminal end of the first glycosyltransferase, GumD. Mutants of gumD do not produce xanthan and remain pathogenic in citrus plants to the same extent as wild-type bacteria. The kinetics of appearance of initial symptoms, areas of plant material affected, and growth of bacteria inside plant tissue throughout the disease process are similar for both wild-type and mutant inoculations. Exopolysaccharide deffciency does not impair the ability of the bacteria to induce hypersensitive response on non-host plants. Apart from variations in phenotypic aspects, no differences in growth or survival under diVerent stress conditions are observed between the xanthan-deficient mutant and wild-type bacteria. gumD mutants display impaired survival under oxidative stress during stationary phase as well as impaired epiphytic survival on citrus leaves | Xanthomonas axonopodis |