EC Number   |
Disease |
PubMed ID |
Title of Publication |
Category |
Confidence Level |
|---|
    1.1.1.2 | Acidosis |
9028626 |
Ethanol metabolism, cirrhosis and alcoholism. |
therapeutic application
unassigned |
1
0 |
| 1.1.1.2 | Acute Kidney Injury |
6223803 |
Changes in 25-hydroxyvitamin D3 alpha- and 24-hydroxylase activities of kidney cells isolated from rats with either unilateral kidney damage or acute renal insufficiency. |
ongoing research
unassigned |
4
0 |
| 1.1.1.2 | Adenocarcinoma |
19028477 |
Cancer biomarker AKR1B10 and carbonyl metabolism. |
causal interaction
diagnostic usage
ongoing research
therapeutic application |
3
3
1
4 |
| 1.1.1.2 | Adenocarcinoma of Lung |
17597105 |
Aldo-keto reductase family 1 B10 gene silencing results in growth inhibition of colorectal cancer cells: Implication for cancer intervention. |
causal interaction
diagnostic usage
ongoing research
therapeutic application |
4
3
4
4 |
| 1.1.1.2 | Adenocarcinoma of Lung |
19236911 |
Structure and promoter characterization of aldo-keto reductase family 1 B10 gene. |
causal interaction
diagnostic usage
ongoing research
unassigned |
2
3
3
0 |
| 1.1.1.2 | Adrenocortical Carcinoma |
34027794 |
Homeobox A5 activates p53 pathway to inhibit proliferation and promote apoptosis of adrenocortical carcinoma cells by inducing Aldo-Keto reductase family 1 member B10 expression. |
diagnostic usage
unassigned |
1
0 |
| 1.1.1.2 | Arthritis |
454491 |
Effect of adjuvant polyarthritis on liver alcohol dehydrogenase in the rat. |
ongoing research
unassigned |
4
0 |
| 1.1.1.2 | Ascorbic Acid Deficiency |
17222174 |
Vitamin C. Biosynthesis, recycling and degradation in mammals. |
causal interaction
unassigned |
2
0 |
| 1.1.1.2 | Astrocytoma |
23643085 |
[Effect of AKR1A1 knock-down on H2;O2; and 4-hydroxynonenal-induced cytotoxicity in human 1321N1 astrocytoma cells]. |
causal interaction
ongoing research
therapeutic application
unassigned |
1
4
1
0 |
| 1.1.1.2 | Breast Neoplasms |
16048908 |
Proteomic study reveals that proteins involved in metabolic and detoxification pathways are highly expressed in HER-2/neu-positive breast cancer. |
causal interaction
unassigned |
4
0 |
