EC Number   |
General Information |
Reference  |
|---|
   2.7.1.158 | physiological function |
knock-out mutant cells are defective in nuclear mRNA export, cell morphology, polarized growth, and cell separation |
703511 |
| 2.7.1.158 | malfunction |
a functional Asp1 kinase domain abolishes invasive growth which is monopolar |
723173 |
| 2.7.1.158 | physiological function |
Asp1 kinase activity regulates cell-cell adhesion and increases resistance towards thiabendazole. The Asp1 kinase activity encoded by the N-terminal part of the protein is regulated negatively by the C-terminal domain of Asp1. Asp1 is a key regulator of the morphological switch via the cAMP protein kinase A |
723173 |
| 2.7.1.158 | malfunction |
cells lacking the enzyme display defects in dynein-dependent trafficking pathways including endosomal sorting, vesicle movement and Golgi maintenance |
-, 737654 |
| 2.7.1.158 | physiological function |
the enzyme activity is required for cytoplasmic dynein transport |
-, 737654 |
| 2.7.1.158 | malfunction |
a mutant of inositol pentakisphosphate 2-kinase displays hypersensitivity to arsenate stress and less arsenate uptake when compared to the wild type enzyme |
739275 |
| 2.7.1.158 | malfunction |
a loss-of-function mutant exhibits disturbed phosphate homeostasis and overaccumulated phosphate as a consequence of increased phosphate uptake activity and root-to-shoot phosphate translocation. The mutant also shows a phosphate deficiency-like root system architecture with reduced primary root and enhanced lateral root growth |
739312 |
| 2.7.1.158 | physiological function |
the enzyme has important roles in growth and phosphate homeostasis |
739312 |
| 2.7.1.158 | evolution |
differential spatial and temporal expression profiling of gene GmIpk1 and its two homologues Glyma06g03310 and Glyma04g03310 in Glycine max |
760240 |
| 2.7.1.158 | metabolism |
inositol 1,3,4,5,6-pentakisphosphate 2-kinase catalyzes the terminal step in the phytic acid biosynthetic pathway |
760240 |
