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Literature summary extracted from
- Single neuron transcriptomics identify SRSF/SR protein B52 as a regulator of axon growth and choline acetyltransferase splicing (2016), Sci. Rep., 6, 34952 .
Organism
| EC Number | Organism | UniProt | Comment | Textmining |
|---|---|---|---|---|
| 2.3.1.6 | Drosophila melanogaster | P07668 | - |
- |
General Information
| EC Number | General Information | Comment | Organism |
|---|---|---|---|
| 2.3.1.6 | physiological function | SRSF/SR protein B52 function is required for ChATsplicing. At the end of embryogenesis, loss of B52 function impedes splicing of ChAT, reduces acetylcholine synthesis, and extends the period of uncoordinated muscle twitches during larval hatching | Drosophila melanogaster |
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Release 2023.1 (January 2023)
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