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Literature summary extracted from
- The response regulator Slr1588 regulates spsA but is not crucial for salt acclimation of Synechocystis sp. PCC 6803 (2017), Front. Microbiol., 8, 1176 .
Organism
| EC Number | Organism | UniProt | Comment | Textmining |
|---|---|---|---|---|
| 3.1.3.69 | Synechocystis sp. PCC 6803 | Q55034 | - |
- |
Synonyms
| EC Number | Synonyms | Comment | Organism |
|---|---|---|---|
| 3.1.3.69 | Slr0746 | - |
Synechocystis sp. PCC 6803 |
General Information
| EC Number | General Information | Comment | Organism |
|---|---|---|---|
| 3.1.3.69 | physiological function | deletion of the entire coding sequence of response regulator slr1588 compromises the expression of the downstream situated StpA gene, which encodes glucosylglycerol-phosphate phosphatase. Mutation of slr1588 by deleting the N-terminal part of this protein (1slr1588-F976) does not affect StpA expression, glucosylglycerol accumulation as well as salt tolerance, while the mutation of StpA results in the a salt-sensitive phenotype. In the 1slr1588-F976 mutant, SpsA is up-regulated but sucrose content is lowered due to increased invertase activity | Synechocystis sp. PCC 6803 |
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