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Literature summary extracted from
- The B. subtilis MgtE magnesium transporter can functionally compensate TRPM7-deficiency in vertebrate B-cells (2012), PLoS ONE, 7, e44452.
Organism
| EC Number | Organism | UniProt | Comment | Textmining |
|---|---|---|---|---|
| 7.2.2.14 | Bacillus subtilis | - |
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Synonyms
| EC Number | Synonyms | Comment | Organism |
|---|---|---|---|
| 7.2.2.14 | MgtE | - |
Bacillus subtilis |
General Information
| EC Number | General Information | Comment | Organism |
|---|---|---|---|
| 7.2.2.14 | physiological function | overexpression of Bacillus subtilis MgtE is able to rescue the growth of DT40 B-cells lacking lacking the ion-channel kinase transient receptor potential cation channel, TRPM7. There is no detectable current development in TRPM7-deficient cells expressing MgtE. In DT40 B-cells, MgtE expression is strongly downregulated at high magnesium concentrations. The N-terminal cytoplasmic domain of MgtE is required for normal MgtE channel function | Bacillus subtilis |
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