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Literature summary extracted from

  • Michielse, C.B.; Reijnen, L.; Olivain, C.; Alabouvette, C.; Rep, M.
    Degradation of aromatic compounds through the alpha-ketoadipate pathway is required for pathogenicity of the tomato wilt pathogen Fusarium oxysporum f. sp. lycopersici (2012), Mol. Plant Pathol., 13, 1089-1100.
    View publication on PubMedView publication on EuropePMC

Organism

EC Number Organism UniProt Comment Textmining
5.5.1.2 Fusarium oxysporum
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5.5.1.5 Fusarium oxysporum
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Synonyms

EC Number Synonyms Comment Organism
5.5.1.2 2-chloromuconate cycloisomerase
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 Fusarium oxysporum
5.5.1.2 3-Carboxy-cis,cis-muconate lactonizing enzyme
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 Fusarium oxysporum
5.5.1.2 CMLE
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 Fusarium oxysporum
5.5.1.5 3-Carboxy-cis,cis-muconate lactonizing enzyme
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 Fusarium oxysporum
5.5.1.5 CMLE
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 Fusarium oxysporum

General Information

EC Number General Information Comment Organism
5.5.1.2 malfunction inactivation of CMLE does not affect superficial tomato root colonization, but does inhibit in planta growth, as well as growth and conidial germination in the presence of certain phenolic compounds Fusarium oxysporum
5.5.1.2 physiological function CMLE is required for pathogenicity Fusarium oxysporum
5.5.1.5 malfunction deletion mutant cannot catabolize phenolic compounds known to be degraded via the beta-ketoadipate pathway. Mutant is impaired in root invasion and is nonpathogenic, even though it shows normal superficial root colonization Fusarium oxysporum