Literature summary for 6.3.4.5 extracted from

Bizzoco, E.; Vannucchi, M.G.; Faussone-Pellegrini, M.S.
Transient ischemia increases neuronal nitric oxide synthase, argininosuccinate synthetase and argininosuccinate lyase co-expression in rat striatal neurons (2007), Exp. Neurol., 204, 252-259.

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Application

Application	Comment	Organism
medicine	transient ischemia induces changes in nitric oxide synthase, argininosuccinate synthetase and argininsuccinate lyase co-expression in the rat striatal neurons. At 24 h after reperfusion in the animals with a longer survival and better motor-sensory performances there is an increase in these neuronal populations. It is hypothesized that the elevated expression of argininosuccinate synthetase and argininsuccinate lyase protects ischemic striatal neurons from tissue damages by maintaining a correct NO production	Rattus norvegicus

Organism

Organism	UniProt	Comment	Textmining
Rattus norvegicus	-	-	-

Source Tissue

Source Tissue	Comment	Organism	Textmining
corpus striatum	in a rat ischemia model the striatum is examined at 24, 72 and 144 h after reperfusion and compared with rats in normal conditions. At 24 h after reperfusion, the number of the nitric oxide synthase-positive neurons and the percentage of those co-expressing argininosuccinate synthetase and argininsuccinate lyase and nitric oxide synthase are significantly increased in the animals with a longer survival	Rattus norvegicus	-

Synonyms

Synonyms	Comment	Organism
Argininosuccinate synthetase	-	Rattus norvegicus
ASS	-	Rattus norvegicus

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Release 2023.1 (January 2023)