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Literature summary for 1.13.99.1 extracted from

  • Tominaga, T.; Sharma, I.; Fujita, Y.; Doi, T.; Wallner, A.K.; Kanwar, Y.S.
    Myo-inositol oxygenase accentuates renal tubular injury initiated by endoplasmic reticulum stress (2019), Am. J. Physiol. Renal Physiol., 316, F301-F315 .
    View publication on PubMedView publication on EuropePMC
Search for more literature for 1.13.99.1

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
myo-inositol + O2 Homo sapiens
-
 D-glucuronate + H2O
-
 ?
myo-inositol + O2 Sus scrofa
-
 D-glucuronate + H2O
-
 ?
myo-inositol + O2 Mus musculus
-
 D-glucuronate + H2O
-
 ?

Organism

Organism UniProt Comment Textmining
Homo sapiens
-
-
-
Mus musculus Q9QXN5
-
-
Sus scrofa
-
-
-

Source Tissue

Source Tissue Comment Organism Textmining
HK-2 cell
-
 Homo sapiens
-
LLC-PK1 cell
-
 Sus scrofa
-
renal proximal tubule cell
-
 Homo sapiens
-
renal proximal tubule cell
-
 Sus scrofa
-
renal proximal tubule cell
-
 Mus musculus
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
myo-inositol + O2
-
 Homo sapiens D-glucuronate + H2O
-
 ?
myo-inositol + O2
-
 Sus scrofa D-glucuronate + H2O
-
 ?
myo-inositol + O2
-
 Mus musculus D-glucuronate + H2O
-
 ?

Subunits

Subunits Comment Organism
? x * 33000, SDS-PAGE Homo sapiens
? x * 33000, SDS-PAGE Sus scrofa
? x * 33000, SDS-PAGE Mus musculus

Synonyms

Synonyms Comment Organism
MIOX
-
 Homo sapiens
MIOX
-
 Sus scrofa
MIOX
-
 Mus musculus
Myo-inositol oxygenase
-
 Homo sapiens
Myo-inositol oxygenase
-
 Sus scrofa
Myo-inositol oxygenase
-
 Mus musculus

General Information

General Information Comment Organism
physiological function enzyme overexpression accentuates the cellular injury related to endoplasmic reticulum stress and accentuates tunicamycin-induced generation of reactive oxygen species Homo sapiens
physiological function enzyme overexpression accentuates the cellular injury related to endoplasmic reticulum stress and accentuates tunicamycin-induced generation of reactive oxygen species Sus scrofa
physiological function enzyme overexpression accentuates the cellular injury related to endoplasmic reticulum stress and accentuates tunicamycin-induced generation of reactive oxygen species Mus musculus