EC Number |
Expression |
Reference |
---|
2.4.1.38 | down |
anti-TNFR1 antibody suppresses the expression of beta-1,4-GalT I, and TNF-alpha autocrine |
720285 |
2.4.1.38 | down |
estrogen antagonists ICI 182,780 and ER-a-ERE binding blocker TPBM inhibit the expression of estrogen-induced B4GALT1 |
718763 |
2.4.1.38 | down |
inhibition of MEK/ERK, PI3K/AKT and NF-kappaB suppresses recombinant human osteopontin-induced beta1,4-GalT-I expression |
719575 |
2.4.1.38 | up |
beta 1, 4-GalT-I iss highly expressed in endometrium during implantation window |
719575 |
2.4.1.38 | up |
beta-1,4-GalT-I mRNA is significant increased 2 h after lipopolysaccharide stimulation with 0.001 mg/ml |
704010 |
2.4.1.38 | up |
CDK11p58 and beta-1,4-GalT-I are induced in lipopolysaccharide-challenged rat primary astrocytes in a affinis dose- and time-dependent manner. CDK11p58 regulates the expression of beta-1,4-GalT-I by interacting with it. The expression of beta-1,4-GalT-I increases, and astrocyte activation enhances due to the overexpression of CDK11p58 |
719592 |
2.4.1.38 | up |
estrogen induces beta-1,4-galactosyltransferase 1 expression |
718763 |
2.4.1.38 | up |
expression of beta1,4-GalT-I and galactose-containing glycans increases in synovial tissue of rheumatoid arthritis patients compared with osteoarthritis patients and healthy controls. Most galactose-containing glycans and beta1,4-GalT-I are expressed in inflammatory cells |
719591 |
2.4.1.38 | up |
expression of beta1,4-GalT-I increases in the cartilage and synovial tissue of osteoarthritis patients compared with healthy controls |
719593 |
2.4.1.38 | up |
mRNA and protein expression of beta1,4-GalT-I increases in synovial tissue of a collagen-induced rat model of rheumatoid arthritis compared with the normal group at 10d and 15d after collagen-induced |
719310 |