EC Number   |
Natural Substrates |
|---|
   2.7.10.1 | ATP + a protein |
- |
| 2.7.10.1 | ATP + a [macrophage-stimulating protein]-L-tyrosine |
- |
| 2.7.10.1 | ATP + a [protein]-L-tyrosine |
- |
| 2.7.10.1 | ATP + a [protein]-L-tyrosine |
FLT-3 is involved in signal transduction, mechanisms |
| 2.7.10.1 | ATP + a [protein]-L-tyrosine |
phosphorylation of downstream signalling proteins |
| 2.7.10.1 | ATP + a [protein]-L-tyrosine |
regulation mechanisms and ligand binding, ligand binding induces a conformational change, overview |
| 2.7.10.1 | ATP + a [protein]-L-tyrosine |
stem cell factor and Kit receptor are essential playing important roles in e.g. gametogenesis, hematopoiesis, mast cell development and function, and melanogenesis, complete absence is lethal, enzyme deficiencies lead to defects in white and red blood cell production, to hypopigmentation, and to sterility, Kit receptor signaling regulation by autophosphorylation and dephosphorylation via a phosphatase, regulation mechanism, overview |
| 2.7.10.1 | ATP + a [protein]-L-tyrosine |
stem cell factor and Kit receptor are essential playing important roles in gametogenesis, hematopoiesis, mast cell development and function, and melanogenesis, complete absence is lethal, enzyme deficiencies lead to defects in white and red blood cell production, to hypopigmentation, and to sterility |
| 2.7.10.1 | ATP + a [protein]-L-tyrosine |
stem cell factor and Kit receptor are essential playing important roles in gametogenesis, hematopoiesis, mast cell development and function, and melanogenesis, complete absence is lethal, enzyme deficiencies lead to defects in white and red blood cell production, to hypopigmentation, and to sterility, Kit receptor signaling regulation by autophosphorylation and dephosphorylation via a phosphatase |
| 2.7.10.1 | ATP + cortactin-L-tyrosine |
- |
