EC Number   |
Protein Variants |
Reference |
|---|
   2.8.2.15 | A679G |
naturally occuring polymorphisms of SULT2A1, genotyping, SULT2A1 variant allele frequencies in African-American populations, overview |
676256 |
| 2.8.2.15 | G187C |
naturally occuring polymorphisms of SULT2A1, genotyping, SULT2A1 variant allele frequencies in African-American populations, overview |
676256 |
| 2.8.2.15 | G781A |
naturally occuring polymorphisms of SULT2A1, genotyping, SULT2A1 variant allele frequencies in African-American populations, overview |
676256 |
| 2.8.2.15 | H99A |
site-directed mutagenesis, 50% reduced sulfating activity toward 4-androstene-3,17-dione compared to wild-type |
760621 |
| 2.8.2.15 | H99S |
site-directed mutagenesis, mutant H99S shows complete loss of the sulfating activity toward 4-androstene-3,17-dione |
760621 |
| 2.8.2.15 | more |
construction of a SULT2B1b truncation mutant lacking the proline- and serine-rich carboxyl end of about 53 amino acids, removal of the PSC extension significantly decreases the thermostability of the expressed enzyme and the rate of dehydroepiandrosterone sulfation, truncated SULT2B1b is incapable of translocation to nuclei in transfected human BeWo choriocarcinoma cells |
673295 |
| 2.8.2.15 | more |
construction of truncation mutants of SULT2A1 for analysis of SF1 binding sites and promoter activity, overview |
673350 |
| 2.8.2.15 | more |
disruption of the DR4-type motif and the IR2-type motif prevents in vitro binding of nuclear receptors pregnane X receptor and constitutive androstane receptor to their specific binding sites, overview |
676007 |
| 2.8.2.15 | more |
farnesoid X receptor FXR-null mice express a constitutively high level of SULT2A |
677268 |
| 2.8.2.15 | W77A |
site-directed mutagenesis, 50% reduced sulfating activity toward 4-androstene-3,17-dione compared to wild-type |
760621 |
