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Results 1 - 10 of 30 > >>
EC Number Protein Variants Commentary Reference
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23D167N dominant-negative mutant of CDK9, inhibits transcriptional activation by EBNA 2 682208
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23D173A CDK8 mutant, shows no effect on basal or EBNA 2-activated transcription 682208
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23D324N inactive Ctk1 mutant 661832
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23D324N with catalytically dead allele of CTK1 -, 681926
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23K41M CDK7 mutant, both EBNA 2-activated and basal transcription from Cp reporter construct are not particularly dependent on CDK7 kinase activity with only small effects observed in the presence of high levels of mutant K41M 682208
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23K85A kinase-dead mutant of isoform GSK-3beta 762057
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23more a temperature-sensitive mutant and the analog-sensitive mutant of Kin28 is that upon heat inactivation, Kin28ts alleles are unable to retain protein interactions with other subunits of the TFIIH complex 689778
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23more construction of an enzyme-deficient ctk1DELTA mutant strain which shows selective abolishment of histone H3 Lys36 methylation -, 661828
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23more construction of ctk-disruption mutant strains displaying defects in nucleolar structure, the RNA polymerase I functions less effective in the mutant cells, the in vitro transcription is impaired 662973
Display the word mapDisplay the reaction diagram Show all sequences 2.7.11.23more construction of several enzyme-deficient disruption mutant strains, overview, CTK1 can rescue the growth defects of an Ess1-deficient mutant at low concentration, but enhances the defects if overexpressed 661832
Results 1 - 10 of 30 > >>