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Results 1 - 8 of 8
EC Number
Amino acid exchange
Commentary
Reference
I439A/W588A
site-directed mutagenesis, the mutant shows reduced binding of serum amyloid P
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construction of antisense oligonucleotide-mediated loss-of-function and synthetic mRNA-based gain-of-function mutants. The loss-of-function phenotype is linked to cell death, evident primarily in the muscle of the somites, extensive loss of myelinated tracks, and brain edema, phenotype, overview
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gpbp-1-/- mice and Tg-hGPBP-1 animals do not display any obvious morphological phenotype, gpbp-1-/- mice also do not show a functional phenotype, whereas Tg-hGPBP-1 mice display reduced endurance that is not attributable to mitochondrial alterations
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immobilization of Goodpasture antigen-binding protein by human serum amyloid P component binding from solution and in plasma, overview. Serum amyloid P associates in the blood with a 37-kDa GPBP fragment containing the N-terminal region and the middle domain
N460A/Y461A
site-directed mutagenesis, the mutant shows unaltered binding of serum amyloid P
P500G/A501Q
site-directed mutagenesis, the mutant binding to serum amyloid P is abolished
V498A/W499A
site-directed mutagenesis, the mutant shows unaltered binding of serum amyloid P
Y461A/F462A
site-directed mutagenesis, the mutant shows reduced binding of serum amyloid P
Results 1 - 8 of 8