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Results 1 - 10 of 101

EC Number	Substrates	Commentary Substrates	Organism	Products	Commentary (Products)	Reversibility
1.6.1.2	more	diabetes is potentially linked to a defective transhydrogenase gene	Mus musculus	?	-	?
1.6.1.2	more	the glucose intolerance and impaired insulin secretion of the C57BL/6J mouse strain results from oxidative stress due to a mutated nicotinamide nucleotide transhydrogenase. Mutation of this gene in a mouse strain with normal insulin secretion results in strong glucose intolerance	Mus musculus	?	-	?
1.6.1.2	more	insulin hypersecretion is associated with increased Nnt expression. It can be suggest that nicotinamide nucleotide transhydrogenase must play an important role in beta cell function	Mus musculus	?	-	?
1.6.1.2	more	with ongoing NADPH and NAD+ generation, the proton-translocating, mitochondrial transhydrogenase can serve as an additional anaerobic phosphorylation site	Hymenolepis diminuta	?	-	?
1.6.1.2	NADH + NADP+	-	Mus musculus	NAD+ + NADPH	-	r
1.6.1.2	NADH + NADP+	-	Escherichia coli	NAD+ + NADPH	-	?
1.6.1.2	NADH + NADP+	-	Escherichia coli	NAD+ + NADPH	-	r
1.6.1.2	NADH + NADP+	-	Homo sapiens	NAD+ + NADPH	-	r
1.6.1.2	NADH + NADP+	-	Bos taurus	NAD+ + NADPH	-	?
1.6.1.2	NADH + NADP+	-	Bos taurus	NAD+ + NADPH	-	r

Results 1 - 10 of 101

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Release 2023.1 (January 2023)