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EC Number
Source Tissue
Commentary
Reference
3.4.17.23
aorta thoracica
chronic treatment with the AT1R antagonist almesartan induces a fivefold increase in ACE2 mRNA in the aorta which leads to a significant increase in aortic angiotensin(1-7) protein expression
667145
3.4.17.23
arterial endothelial cell
-
762787
3.4.17.23
arterial smooth muscle
-
762787
3.4.17.23
artery
non-diseased mammary arteries and atherosclerotic carotid arteries. Total vessel wall expression of ACE and ACE2 is similar during all stages of atherosclerosis. The observed ACE2 protein is enzymatically active and activity is lower in the stable advanced atherosclerotic lesions, compared to early and ruptured atherosclerotic lesions
693806
3.4.17.23
astrocyte
transcriptional regulation of ACE2 mRNA in astrocytes is dependent on the relative concentrations of both angiotensin II and angiotensin(17) as well as on interaction with their respective receptors
-
,
667146
3.4.17.23
atherosclerotic plaque
-
681180
3.4.17.23
atherosclerotic plaque
cells in atherosclerotic plaques co-express ACE2, Oct-4, and CD34
679704
3.4.17.23
bile duct
-
690356
3.4.17.23
blood
-
732780
3.4.17.23
blood
coronary sinus blood, evidence against a major role for angiotensin converting enzyme-related carboxypeptidase in angiotensin peptide metabolism in the human coronary circulation
669704
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