EC Number |
General Information |
Reference |
---|
5.5.1.24 | malfunction |
a mutant deficient in tocopherol cyclase is totally devoid of tocopherol. Tocopherol deficiency results in the increase in ascorbate and glutathione. The simultaneous loss of tocopherol and glutathione in a tocopherol cyclase/cad2 double deletion mutant results in moderate oxidative stress that affects the stability and the efficiency of the photosynthetic apparatus (reduced growth, chlorophyll content, and photosynthetic quantum) |
728508 |
5.5.1.24 | malfunction |
deletion of the enzyme leads to tocopherol deficiency and 2,3-dimethyl-6-phytylbenzene-1,4-diol accumulation |
728506 |
5.5.1.24 | malfunction |
the inactivation of vte1 genes results in increased guaiacol peroxidase activity and 1.4fold higher concentrations of total and oxidized glutathione and 1.2-1.3fold higher [GSSG]/[total GSH] ratio. Ascorbate peroxidase activity is lower in the tocopherol-deficient mutant vte1 than that in wild type plants |
728496 |
5.5.1.24 | malfunction |
transgenic plants with VTE1 RNA interference are more sensitive to salt stress (treatment with 100 mM NaCl for 10 days) |
728794 |
5.5.1.24 | metabolism |
key enzyme of tocopherol (vitamin E) synthesis |
727854 |
5.5.1.24 | metabolism |
the enzyme catalyzes the penultimate step of tocopherol synthesis |
727218 |
5.5.1.24 | metabolism |
the enzyme is involved in prenylquinone metabolism |
747864 |
5.5.1.24 | metabolism |
tocopherol cyclase catalyzes the penultimate step of tocopherol synthesis |
728508 |
5.5.1.24 | physiological function |
enzyme overexpression can increase the tolerance to drought stress. The increase in tocopherol affects cellular signaling and antioxidant defense of plants in response to drought |
748909 |
5.5.1.24 | physiological function |
overexpression of the enzyme increases the antioxidant capacity of rice seedlings. Overexpression of the enzyme is able to efficiently eliminate H2O2 produced by salt stress |
728794 |