EC Number |
General Information |
Reference |
---|
3.5.1.16 | malfunction |
inactivation of argE gene does not cause arginine auxotrophy or marked growth defects |
754657 |
3.5.1.16 | malfunction |
ornithine depletion is associated with altered levels of putrescine and spermine. Reduced AtNAOD expression results in alterations at the reproductive level, causing early flowering and impaired fruit setting |
-, 734884 |
3.5.1.16 | metabolism |
Sulfolobus solfataricus lacks ornithine acetyltransferase and thus forms N-acetylglutamate exclusively via the energetically less favourable reaction catalysed by N-acetylglutamate synthase, investing 1 mol of acetyl CoA per mol of N-acetyl intermediate synthesized |
-, 725629 |
3.5.1.16 | more |
three-dimensional homologic structure, molecular modeling using the X-ray crystal structure of the DapE from Haemophilus influenzae, PDB ID 3IC1, as template, overview |
735279 |
3.5.1.16 | physiological function |
involvement of N-acetylornithine deacetylase from Arabidopsis thaliana in flowering and fruit development, the enzyme is associated with ovule development, overview. The enzyme contributes to the regulation of ornithine levels in plant cells |
-, 734884 |
3.5.1.16 | physiological function |
the enzyme controls ornithine biosynthesis via a linear pathway with downstream effects on polyamine levels. The role of this enzyme is relevant for the reproductive development of Arabidopsis thaliana, probably contributing to ensure the optimal balance between cellular C and N metabolism necessary for flowering and fruit setting |
755033 |