EC Number |
General Information |
Reference |
---|
3.1.1.103 | malfunction |
enzyme mutants have increased susceptibilities to beta-lactams in the presence or absence of 0.02% Triton X-100 compared with the susceptibilities of the parent strains, while the susceptibilities of the mutants to other antibiotics are not altered |
-, 744046 |
3.1.1.103 | malfunction |
the inactivation of the enzyme in two methicillin-resistant Staphylococcus aureus strains does not cause a reduction in methicillin resistance |
-, 744928 |
3.1.1.103 | physiological function |
the enzyme affects the methicillin resistance level in methicillin-resistant Staphylococcus aureus |
-, 744047 |
3.1.1.103 | physiological function |
the enzyme has weak D-Ala-D-Ala-carboxypeptidase activity and is capable of covalently incorporating C14-Gly into cell walls |
-, 744048 |
3.1.1.103 | physiological function |
the enzyme is involved in cell wall synthesis |
-, 744046 |
3.1.1.103 | physiological function |
the enzyme is part of the core cell wall stimulon and is involved in methicillin resistance in Staphylococcus aureus |
-, 746240 |
3.1.1.103 | physiological function |
the enzyme modulates D-alanylation of teichoic acids and is involved in cell division, biofilm formation, autolysis, and colonization |
745677 |
3.1.1.103 | physiological function |
the penicillin-binding protein may compensate for suppressed peptidoglycan biosynthesis underbeta-lactam induced cell wall stress conditions |
-, 745281 |