EC Number   |
General Information |
Reference |
|---|
   2.7.1.166 | evolution |
adaptive strategies adopted by the organism during persistent infection: genome sequencing of three isolates from patients with chronic obstructive lung disease reveals sequential acquisition of single-nucleotide variants in the AMP permease sapC, the heme acquisition systems hgpB, hgpC, hup and hxuC, the 3-deoxy-D-manno-octulosonic acid kinase kdkA, the long-chain fatty acid transporter ompP1, and the phosphoribosylamine glycine ligase purD |
739514 |
| 2.7.1.166 | malfunction |
adaptive strategies adopted by the organism during persistent infection: genome sequencing of three isolates from patients with chronic obstructive lung disease reveals sequential acquisition of single-nucleotide variants in the AMP permease sapC, the heme acquisition systems hgpB, hgpC, hup and hxuC, the 3-deoxy-D-manno-octulosonic acid kinase kdkA, the long-chain fatty acid transporter ompP1, and the phosphoribosylamine glycine ligase purD |
739514 |
| 2.7.1.166 | malfunction |
Kdo kinase mutant produces only glycoform B (containing two unphosphorylated Kdo residues) and not glycoform A (containing a single phosphorylated 3-deoxy-D-manno-octulosonic acid molecule that is frequently substituted with a phosphoethanolamine residue attached to the phosphate group) |
698240 |
