EC Number   |
General Information |
Reference |
|---|
   2.4.2.58 | malfunction |
complete loss of enzyme function results in a shared defect in gametophytic tip cell growth. Knockout mutants suffer from a strong male sterility defect as a consequence of pollen tubes that fail to fully elongate following pollination |
739321 |
| 2.4.2.58 | malfunction |
complete loss of enzyme function results in a shared defect in gametophytic tip cell growth. Knockout mutation results in larger multicellular filamentous networks due to increased elongation of protonemal tip cells. The mutants also lack cell-wall associated hydroxyproline arabinosides |
739321 |
| 2.4.2.58 | malfunction |
enzyme mutants display low male fertility and pollen tube growth defects, leading to reduced male transmission and low seed set |
759958 |
| 2.4.2.58 | malfunction |
enzyme mutants have enlarged meristems |
739152 |
| 2.4.2.58 | malfunction |
loss-of-function mutations in hydroxyproline O-arabinosyltransferase-encoding genes cause pleiotropic phenotypes that include enhanced hypocotyl elongation, defects in cell wall thickening, early flowering, early senescence and impaired pollen tube growth |
736862 |
| 2.4.2.58 | physiological function |
essential role of Hyp O-arabinosylation in both vegetative and reproductive growth (hypocotyl elongation, cell wall thickening, flowering, senescence and pollen tube growth) |
736862 |
| 2.4.2.58 | physiological function |
isoforms HPAT1 and HPAT3 are redundantly required for full pollen fertility |
759958 |
| 2.4.2.58 | physiological function |
the enzyme negatively regulates root nodule symbiosis |
759570 |
| 2.4.2.58 | physiological function |
the enzyme plays a major role in influencing cell elongation during tip growth |
739321 |
