EC Number   |
General Information |
Reference |
|---|
   2.4.1.241 | malfunction |
in etiolated dgd1 (knockout mutant) seedlings, digalactosyldiacylglycerol content decreases to 20% of the wild-type level, the lattice structure of prolamellar bodies is disordered, and the development of prothylakoids is impaired. Membrane-associated processes of protochlorophyllide biosynthesis, formation of the protochlorophyllide-NADPH:protochlorophyllide oxidoreductase-NADPH complex, and dissociation of the complex after the photoconversion of protochlorophyllide to chlorophyllide were impaired in dgd1, although the photoconversion reaction by NADPH:protochlorophyllide oxidoreductase is not affected by the DGDG deficiency. Total carotenoid content also decreases in etiolated dgd1 seedlings, but the carotenoid composition is unchanged |
759980 |
| 2.4.1.241 | malfunction |
MtDGD1-RNA interference transgenic plants exhibit significant decreases in nodule number, symbiotic nitrogen fixation activity, and digalactosyldiacylglycerol abundance in the nodules, as well as abnormal nodule and symbiosome development |
759791 |
| 2.4.1.241 | physiological function |
Dgd1 mutants have a greater than 90% reduction in digalactosyldiacylglycerol content, reduced photosynthesis, and altered chloroplast morphology. Mutant plants show an extremely short inflorescence stem. Phloem cap cells are lignified and jasmonic acid-responsive genes are highly upregulated under normal growth conditions. The coronative insensitive1 Dgd1 and allene oxide synthase Dgd1 double mutants no longer exhibit the short inflorescence stem and lignification phenotypes but still have the same lipid profile and reduced photosynthesis as Dgd1 single mutants. Dgd1 mutants display increased levels of jasmonic acid, jasmonic acid-isoleucine, 12-oxo-phytodienoic acid, and arabidopsides. Jasmonic acid biosynthesis in Dgd1 mutants is initially activated through the increased expression of genes encoding 13-lipoxygenases and phospholipase A-Ig3 (At1g51440), and is sustained by further increases in 13-lipoxygenase and allene oxide cyclase mRNA and protein levels |
736994 |
| 2.4.1.241 | physiological function |
gene 7942dgdA encodiung digalactosyldiacylglycerol synthase is essential. 7942dgdA can be knocked out only when genes for cyanobacterial or plant digalactosyldiacylglycerol synthases are expressed. Lack of digalactosyldiacylglycerol cannot be compensated by the other membrane lipids in Synechococcus elongatus PCC 7942 or by glucosylgalactosyldiacylglycerol synthesized by the beta-GlcT gene of Chloroflexus aurantiacus. Digalactosyldiacylglycerol has an indispensable role and the second galactose molecule is the key |
-, 735719 |
| 2.4.1.241 | physiological function |
the enzyme is involved in the formation of the internal membrane structures in etioplasts as well as in membrane-associated processes of pigment biosynthesis and pigment-protein complex organization |
759980 |
| 2.4.1.241 | physiological function |
the enzyme plays an essential role in nodule development and nitrogen fixation. It contributes to effective nodule organogenesis and nitrogen fixation by affecting the synthesis and content of digalactosyldiacylglycerol during symbiosis |
759791 |
| 2.4.1.241 | physiological function |
the N-terminal sequence of DGD1 is required for galactolipid transfer between the outer and the inner envelope of chloroplast |
760062 |
