EC Number   |
General Information |
Reference |
|---|
    1.1.1.100 | malfunction |
deletion of fabG2 reduces diffusible signal factor (DSF) family signal production |
-, 763418 |
| 1.1.1.100 | malfunction |
deletion of fabG3 does not affect growth or fatty acid composition. The enzyme is not essential for growth or fatty acid synthesis. Xcc fabG3 knockout mutant abolishes xanthomonadin production. Xcc fabG3-disrupted mutant affects virulence of Xanthomonas campestris pv. campestris in host plants. fabG3 deletion results in Xanthomonas campestris pv. campestris being sensitive to H2O2 |
763477 |
| 1.1.1.100 | malfunction |
deletion of the gene reduces the ability of Pseudomonas aeruginosa to produce several specific quorum-sensing signals. ORF mutant shows carbon utilization deficiency with D-turanose and N-acetyl-beta-D-mannosamine |
763071 |
| 1.1.1.100 | malfunction |
deletion of the gene reduces the ability of Pseudomonas aeruginosa to produce several specific quorum-sensing signals. ORF mutant shows carbon utilization deficiency with N-acetyl-beta-D-mannosamine and gelatin |
763071 |
| 1.1.1.100 | malfunction |
DELTAfabG1 cells lose the ability to accumulate poly(3-hydroxybutyrate-co-3-hydroxyvalerate). When the DELTAfabG1 strain is transformed with the fabG1 expression plasmid pWLG1, the capability of polyhydroxyalkanoate accumulation is restored. When the fabG1 gene is disrupted or knocked out, the NADPH-dependent activity of acetoacetyl-CoA reductase in the mutant, the DELTAfabG1 or fabG1-1 strain, is greatly decreased |
-, 701808 |
| 1.1.1.100 | malfunction |
loss of KCR1 function results in embryo lethality, which cannot be rescued by KCR2 expression using the KCR1 promoter. Disruption of the KCR2 gene has no obvious phenotypic effect |
700827 |
| 1.1.1.100 | malfunction |
polyhydroxyalkanoate-defective haloarchaeon |
701808 |
| 1.1.1.100 | metabolism |
FabG3 is specifically involved in xanthomonadin biosynthesis |
763477 |
| 1.1.1.100 | metabolism |
the enzyme is involved in the unsaturated fatty acid biosynthetic pathway |
-, 763086 |
| 1.1.1.100 | physiological function |
does not have key roles in fatty acid synthesis. ORF mutant shows carbon utilization deficiency with D-turanose and N-acetyl-beta-D-mannosamine |
763071 |
