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physiological function
overexpression of ESD significantly suppresses foot-and-mouth disease virus FMDV replication. Knockdown of ESD expression enhances virus replication. Sendai-virus-induced interferon signaling is enhanced by upregulation of ESD. ESD protein enhances IRF3 phosphorylation during FMDV infection. Overexpression of ESD also promotes the expression of various antiviral interferon-stimulated genes, and knockdown of ESD impairs the expression of these antiviral genes during FMDV infection
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