EC Number   |
General Information  |
Reference |
|---|
   2.7.1.86 | malfunction |
deletion of the POS5 gene results in respiratory deficiency, anti-oxidative dysfunction and arginine auxotrophy of cells, and the respiratory deficiency is fatal for mitochondrial function |
-, 721139 |
| 2.7.1.86 | malfunction |
DELTApos5 mitochondria cannot efficiently synthesize Fe-S clusters on endogenous aconitase or imported ferredoxin |
-, 722693 |
| 2.7.1.86 | malfunction |
yeast cells lacking the mitochondrial NADH kinase encoded by POS5 display increased sensitivity to hydrogen peroxide, a slow-growth phenotype, reduced mitochondrial function and increased levels of mitochondrial protein oxidation and mitochondrial DNA mutations |
-, 705593 |
| 2.7.1.86 | physiological function |
a triple mutant lacking the activities of ATP-NAD/NADH kinase Utr1, ATP-NADH kinase Pos5 and ATP-NADH kinase Yel041wp, i.e. Yef1, is viable and does not show growth defects. Presence of Utr1 protein is critical for growth in low iron medium |
661696 |
| 2.7.1.86 | physiological function |
the enzyme contributes to the maintenance of respiratory chain complex activities |
-, 721139 |
| 2.7.1.86 | physiological function |
the enzyme is required for efficient iron-sulfur cluster biogenesis in wild type mitochondria |
-, 722693 |
| 2.7.1.86 | physiological function |
the enzyme might have a role in waterlogging tolerance in legumes |
759974 |
