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Results 1 - 6 of 6
EC Number
General Information
Commentary
Reference
malfunction
deletion of the POS5 gene results in respiratory deficiency, anti-oxidative dysfunction and arginine auxotrophy of cells, and the respiratory deficiency is fatal for mitochondrial function
malfunction
DELTApos5 mitochondria cannot efficiently synthesize Fe-S clusters on endogenous aconitase or imported ferredoxin
malfunction
yeast cells lacking the mitochondrial NADH kinase encoded by POS5 display increased sensitivity to hydrogen peroxide, a slow-growth phenotype, reduced mitochondrial function and increased levels of mitochondrial protein oxidation and mitochondrial DNA mutations
physiological function
a triple mutant lacking the activities of ATP-NAD/NADH kinase Utr1, ATP-NADH kinase Pos5 and ATP-NADH kinase Yel041wp, i.e. Yef1, is viable and does not show growth defects. Presence of Utr1 protein is critical for growth in low iron medium
physiological function
the enzyme contributes to the maintenance of respiratory chain complex activities
physiological function
the enzyme is required for efficient iron-sulfur cluster biogenesis in wild type mitochondria
Results 1 - 6 of 6