EC Number   |
Expression |
Reference |
|---|
   3.1.1.34 | down |
interferon mediates inhibition of lipoprotein lipase gene transcription in macrophages, the mechanism involves a reduction in the binding of transcription factors Sp1 and Sp3 to regulatory sequences in the LPL gene with casein kinase 2- and phosphoinositide-3-kinase-mediated regulation of transcription factors Sp1 and Sp3, overview |
708067 |
| 3.1.1.34 | down |
lipasin suppresses the activity of LPL specifically in cardiac and skeletal muscles |
-, 752181 |
| 3.1.1.34 | down |
lipoprotein lipase activity is significantly decreased in the ischemic side cortex at 2 h ischemia |
716346 |
| 3.1.1.34 | down |
stress and catecholamines reduce adipose enzyme activity. Tissue necrosis factpor alpha represses enzyme activity by downregulating transcription |
729306 |
| 3.1.1.34 | more |
effects of dietary lipid levels on the expression of LPL in the redlip mullet |
749731 |
| 3.1.1.34 | more |
there is no effect of leptin treatment (2 mg/kg body weight over 2 weeks) or pair feeding on postprandial adipose tissue lipoprotein lipase activity |
716101 |
| 3.1.1.34 | up |
during embryogenesis, the mRNA level is increased gradually |
730128 |
| 3.1.1.34 | up |
insulin, dexamethasone and cortisol increase mRNA and activity in adipose organs. Cold markedly stimulates enzyme activity in brown adipose tissue |
729306 |
| 3.1.1.34 | up |
skeletal muscle lipoprotein lipase activity is increased in leptin-treated (2 mg/kg body weight over 2 weeks) compared with pair-fed and wild type mice |
716101 |
| 3.1.1.34 | up |
the mRNA expression and activity of brain lipoprotein lipase (LPL) is increased after acute cerebral ischemia-reperfusion in rats. Increase of LPL immunopositive cells in the cerebral cortex around the infarction area is observed at 4, 6, 12 h ischemia, 2 h ischemia 2 h reperfusion, and 4 h ischemia 2 h reperfusion |
716346 |
