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EC Number
BRENDA No.
Title
Journal
Volume
Pages
Year
Organism
PubMed ID
3.5.1.109
712591
A possible mechanism underlying the ceramide deficiency in atopic dermatitis: expression of a deacylase enzyme that cleaves the N-acyl linkage of sphingomyelin and glucosylceramide
J. Dermatol. Sci.
55
1-9
2009
Homo sapiens
19443184
3.5.1.109
712912
Abnormal expression of the novel epidermal enzyme, glucosylceramide deacylase, and the accumulation of its enzymatic reaction product, glucosylsphingosine, in the skin of patients with atopic dermatitis
Lab. Invest.
83
397-408
2003
Homo sapiens
12649340
3.5.1.109
712668
High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis
J. Invest. Dermatol.
115
406-413
2000
Homo sapiens
10951276
3.5.1.109
711127
The skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide
Biochem. J.
350
747-756
2000
Homo sapiens
10970788
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