EC Number   |
Natural Substrates |
|---|
   3.4.21.26 | gluten peptides + H2O |
activity in the human host gastrointestinal tract, overview |
| 3.4.21.26 | more |
cellular functions, overview |
| 3.4.21.26 | more |
cellular functions, overview, POP may regulate phosphoinositide signaling, overview |
| 3.4.21.26 | more |
cellular functions, overview, POP may regulate phosphoinositide signaling, overview, the enzyme is involved in Alzheimer's disease with enzyme inhibition leading to a reduction of beta-amyloid peptide, overview |
| 3.4.21.26 | more |
cellular functions, overview, POP may regulate phosphoinositide signaling, overview, the enzyme is involved in neuronal degeneration and Alzheimer's disease with enzyme inhibition leading to a reduction of beta-amyloid peptide, overview |
| 3.4.21.26 | more |
cleaves short proline-containing neuropeptides, and is involved in memory and learning |
| 3.4.21.26 | gluten + H2O |
degradation of gluten in the human host's intestinal tract, i.e. stomach, duodenum, jejunum, and ileum |
| 3.4.21.26 | gluten peptides + H2O |
degradation of gluten peptides in the human host's intestinal tract, i.e. stomach, duodenum, jejunum, and ileum |
| 3.4.21.26 | more |
enzyme inhibition leads to inhibition of lithium effects, the loss of enzyme activity evokes a 4fold increase in the inositol-3-phosphate concentration counteracting the Li+ ions, enzyme regulation, overview |
| 3.4.21.26 | more |
enzyme inhibitors are able to prevent the in vitro invasion of rodent muscle cells by trypomastigotes |
