7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7025800&form=6&db=m	Alterations in cell function with ischemia and shock and their correction.	unassigned	-	
7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8106360&form=6&db=m	Enzymatic and functional evidence for adaptation of the vacuolar H(+)-ATPase in proximal tubule apical membranes from rats with chronic metabolic acidosis.	causal interaction,ongoing research,unassigned	2,4,0	
7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9172686&form=6&db=m	[Effect of acidosis on membrane potential and calcium transport in rat brain synaptosomes]	diagnostic usage,ongoing research,unassigned	1,4,0	
7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9273365&form=6&db=m	An immunocytochemical study of H+ ATPase in kidney transplant rejection.	causal interaction,diagnostic usage,unassigned	1,3,0	
7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10352213&form=6&db=m	Environmental distal renal tubular acidosis in Thailand: an enigma.	diagnostic usage,unassigned	2,0	
7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15050724&form=6&db=m	Lactic acidosis progressively impairs dopamine uptake in rat striatal synaptosomes by a mechanism partially independent of the Na+/K+-ATPase dysfunction.	causal interaction,therapeutic application,unassigned	3,1,0	
7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15661104&form=6&db=m	The acute effects of glycemic control on nerve conduction in human diabetics.	causal interaction,diagnostic usage,unassigned	3,2,0	
7.2.2.3	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16146060&form=6&db=m	Magnesium role in cardiovascular diseases.	ongoing research,unassigned	2,0	
7.2.2.3	Acidosis, Lactic	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15050724&form=6&db=m	Lactic acidosis progressively impairs dopamine uptake in rat striatal synaptosomes by a mechanism partially independent of the Na+/K+-ATPase dysfunction.	causal interaction,therapeutic application,unassigned	3,1,0	
7.2.2.3	Acidosis, Renal Tubular	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9273365&form=6&db=m	An immunocytochemical study of H+ ATPase in kidney transplant rejection.	causal interaction,diagnostic usage,unassigned	1,3,0