1.5.3.16	Adenocarcinoma	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12827295&form=6&db=m	Induction of the PAOh1/SMO polyamine oxidase by polyamine analogues in human lung carcinoma cells.	causal interaction,diagnostic usage,ongoing research,therapeutic application	3,1,4,1	
1.5.3.16	Brain Infarction	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27037020&form=6&db=m	Aggravation of brain infarction through an increase in acrolein production and a decrease in glutathione with aging.	unassigned	-	
1.5.3.16	Brain Injuries	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26530396&form=6&db=m	Astrocyte-Dependent Vulnerability to Excitotoxicity in Spermine Oxidase-Overexpressing Mouse.	causal interaction,unassigned	1,0	
1.5.3.16	Breast Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16207710&form=6&db=m	Spermine oxidase SMO(PAOh1), Not N1-acetylpolyamine oxidase PAO, is the primary source of cytotoxic H2O2 in polyamine analogue-treated human breast cancer cell lines.	causal interaction,diagnostic usage,ongoing research,unassigned	4,1,3,0	
1.5.3.16	Breast Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20946629&form=6&db=m	Spermine oxidase (SMO) activity in breast tumor tissues and biochemical analysis of the anticancer spermine analogues BENSpm and CPENSpm.	therapeutic application,unassigned	1,0	
1.5.3.16	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17145855&form=6&db=m	Tumor necrosis factor-alpha increases reactive oxygen species by inducing spermine oxidase in human lung epithelial cells: a potential mechanism for inflammation-induced carcinogenesis.	ongoing research,unassigned	3,0	
1.5.3.16	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18302221&form=6&db=m	Increased spermine oxidase expression in human prostate cancer and prostatic intraepithelial neoplasia tissues.	causal interaction,diagnostic usage,ongoing research,unassigned	4,3,3,0	
1.5.3.16	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18422650&form=6&db=m	Nuclear localization of human spermine oxidase isoforms - possible implications in drug response and disease etiology.	causal interaction,therapeutic application,unassigned	4,3,0	
1.5.3.16	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27041578&form=6&db=m	Epigenetic silencing of miR-124 prevents spermine oxidase regulation: implications for Helicobacter pylori-induced gastric cancer.	causal interaction,therapeutic application,unassigned	4,1,0	
1.5.3.16	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32863742&form=6&db=m	The Mechanism of Bacteroides fragilis Toxin Contributes to Colon Cancer Formation.	causal interaction,diagnostic usage,ongoing research,unassigned	1,2,1,0