6.2.1.4	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18392745&form=6&db=m	Disorders caused by deficiency of succinate-CoA ligase.	causal interaction,unassigned	4,0	
6.2.1.4	Acidosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20227526&form=6&db=m	Marked mitochondrial DNA depletion associated with a novel SUCLG1 gene mutation resulting in lethal neonatal acidosis, multi-organ failure, and interrupted aortic arch.	causal interaction,unassigned	4,0	
6.2.1.4	Acidosis, Lactic	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19526370&form=6&db=m	A novel missense mutation in SUCLG1 associated with mitochondrial DNA depletion, encephalomyopathic form, with methylmalonic aciduria.	causal interaction,unassigned	3,0	
6.2.1.4	Acidosis, Lactic	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21093335&form=6&db=m	Fatal infantile lactic acidosis and a novel homozygous mutation in the SUCLG1 gene: a mitochondrial DNA depletion disorder.	causal interaction,ongoing research,unassigned	4,2,0	
6.2.1.4	Acidosis, Lactic	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21639866&form=6&db=m	Neonatal lactic acidosis with methylmalonic aciduria by novel mutations in the SUCLG1 gene.	causal interaction,unassigned	4,0	
6.2.1.4	Adenocarcinoma, Mucinous	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34174878&form=6&db=m	Multi-omics analyses of human colorectal cancer revealed three mitochondrial genes potentially associated with poor outcomes of patients.	unassigned	-	
6.2.1.4	Adenoma	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28709933&form=6&db=m	Identification of novel biomarker and therapeutic target candidates for diagnosis and treatment of follicular carcinoma.	diagnostic usage,therapeutic application,unassigned	3,1,0	
6.2.1.4	Adenoma	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34174878&form=6&db=m	Multi-omics analyses of human colorectal cancer revealed three mitochondrial genes potentially associated with poor outcomes of patients.	unassigned	-	
6.2.1.4	Alzheimer Disease	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25027320&form=6&db=m	SUCLG2 identified as both a determinator of CSF A?1-42 levels and an attenuator of cognitive decline in Alzheimer's disease.	causal interaction,unassigned	2,0	
6.2.1.4	Anemia, Sideroblastic	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10727444&form=6&db=m	Interaction between succinyl CoA synthetase and the heme-biosynthetic enzyme ALAS-E is disrupted in sideroblastic anemia.	causal interaction,unassigned	2,0