4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22771891&form=6&db=m	4-Hydroxy-2-oxoglutarate aldolase inactivity in primary hyperoxaluria type 3 and glyoxylate reductase inhibition.	diagnostic usage,unassigned	2,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27096395&form=6&db=m	Cellular degradation of 4-hydroxy-2-oxoglutarate aldolase leads to absolute deficiency in primary hyperoxaluria type 3.	causal interaction,unassigned	4,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33543760&form=6&db=m	Clinical characterization of primary hyperoxaluria type 3 in comparison to types 1 and 2: a retrospective cohort study.	causal interaction,unassigned	4,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28271480&form=6&db=m	Dihydrodipicolinate Synthase: Structure, Dynamics, Function, and Evolution.	therapeutic application,unassigned	3,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29071511&form=6&db=m	Folding Defects Leading to Primary Hyperoxaluria.	therapeutic application,unassigned	1,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27561601&form=6&db=m	HOGA1 Gene Mutations of Primary Hyperoxaluria Type 3 in Tunisian Patients.	causal interaction,diagnostic usage,unassigned	4,1,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26428388&form=6&db=m	Hydroxyproline metabolism in a mouse model of Primary Hyperoxaluria Type 3.	causal interaction,therapeutic application,unassigned	4,1,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31401635&form=6&db=m	Mutation Hot Spot Region in the HOGA1 Gene Associated with Primary Hyperoxaluria Type 3 in the Chinese Population.	unassigned	-	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20797690&form=6&db=m	Mutations in DHDPSL are responsible for primary hyperoxaluria type III.	causal interaction,unassigned	3,0	
4.1.3.16	Hyperoxaluria	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33350326&form=6&db=m	Mutations in HOGA1 do Not Confer a Dominant Phenotype Manifesting as Kidney Stone Disease.	causal interaction,ongoing research,unassigned	4,3,0