4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20797690&form=6&db=m	Mutations in DHDPSL are responsible for primary hyperoxaluria type III.	causal interaction,unassigned	3,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22115483&form=6&db=m	Re: Mutations in DHDPSL are Responsible for Primary Hyperoxaluria Type III.	causal interaction,unassigned	4,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22391140&form=6&db=m	The enzyme 4-hydroxy-2-oxoglutarate aldolase is deficient in primary hyperoxaluria type 3.	causal interaction,diagnostic usage,ongoing research,unassigned	4,3,2,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26340091&form=6&db=m	Two Novel HOGA1 Splicing Mutations Identified in a Chinese Patient with Primary Hyperoxaluria Type 3.	causal interaction,diagnostic usage,unassigned	3,1,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27096395&form=6&db=m	Cellular degradation of 4-hydroxy-2-oxoglutarate aldolase leads to absolute deficiency in primary hyperoxaluria type 3.	causal interaction,unassigned	4,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32003182&form=6&db=m	[Genetic aspects of primary hyperoxaluria: epidemiology, ethiology, pathogenesis, and clinical signs of the disorder].	diagnostic usage,unassigned	1,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31123811&form=6&db=m	Nine novel HOGA1 gene mutations identified in primary hyperoxaluria type 3 and distinct clinical and biochemical characteristics in Chinese children.	causal interaction,ongoing research,unassigned	3,1,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31696211&form=6&db=m	Regulation of human 4-hydroxy-2-oxoglutarate aldolase by pyruvate and ?-ketoglutarate: implications for primary hyperoxaluria type-3.	ongoing research,therapeutic application,unassigned	2,1,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33350326&form=6&db=m	Mutations in HOGA1 do Not Confer a Dominant Phenotype Manifesting as Kidney Stone Disease.	causal interaction,ongoing research,unassigned	4,3,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22781098&form=6&db=m	Novel findings in patients with primary hyperoxaluria type III and implications for advanced molecular testing strategies.	causal interaction,therapeutic application,unassigned	4,3,0