3.1.3.56	Muscular Dystrophies	https://pubmed.ncbi.nlm.nih.gov/33879025/	Bidirectional interconversion between PtdIns4P and PtdIns(4,5)P2 is required for autophagic lysosome reformation and protection from skeletal muscle disease.	causal interaction	4	
3.1.3.56	Muscular Dystrophies	https://pubmed.ncbi.nlm.nih.gov/33879025/	Bidirectional interconversion between PtdIns4P and PtdIns(4,5)P2 is required for autophagic lysosome reformation and protection from skeletal muscle disease.	unassigned	-	
3.1.3.56	Neoplasm Metastasis	https://pubmed.ncbi.nlm.nih.gov/33798953/	A new gene panel as a marker for ESCC poor prognosis; INPP5A, TWIST1, MMP2, and EGFR.	causal interaction	4	
3.1.3.56	Neoplasm Metastasis	https://pubmed.ncbi.nlm.nih.gov/33798953/	A new gene panel as a marker for ESCC poor prognosis; INPP5A, TWIST1, MMP2, and EGFR.	unassigned	-	
3.1.3.56	Neoplasms	https://pubmed.ncbi.nlm.nih.gov/33798953/	A new gene panel as a marker for ESCC poor prognosis; INPP5A, TWIST1, MMP2, and EGFR.	causal interaction	4	
3.1.3.56	Neoplasms	https://pubmed.ncbi.nlm.nih.gov/33798953/	A new gene panel as a marker for ESCC poor prognosis; INPP5A, TWIST1, MMP2, and EGFR.	unassigned	-	
3.1.3.56	Ataxia	https://pubmed.ncbi.nlm.nih.gov/33792664/	INPP5K and SIL1 associated pathologies with overlapping clinical phenotypes converge through dysregulation of PHGDH.	causal interaction	4	
3.1.3.56	Ataxia	https://pubmed.ncbi.nlm.nih.gov/33792664/	INPP5K and SIL1 associated pathologies with overlapping clinical phenotypes converge through dysregulation of PHGDH.	unassigned	-	
3.1.3.56	Cataract	https://pubmed.ncbi.nlm.nih.gov/33792664/	INPP5K and SIL1 associated pathologies with overlapping clinical phenotypes converge through dysregulation of PHGDH.	causal interaction	4	
3.1.3.56	Cataract	https://pubmed.ncbi.nlm.nih.gov/33792664/	INPP5K and SIL1 associated pathologies with overlapping clinical phenotypes converge through dysregulation of PHGDH.	unassigned	-