7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1659205&form=6&db=m Effect of partial Na pump and Na-H exchange inhibition on [Ca]i during acidosis in cardiac cells. causal interaction,ongoing research,therapeutic application,unassigned 3,4,3,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2536244&form=6&db=m Na+-dependent activation of Na+-K+ pump in human myocardium during recovery from acidosis. ongoing research,unassigned 3,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2544538&form=6&db=m Membrane damage in acute brain trauma. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3621056&form=6&db=m Regulation of intracellular pH in sheep cardiac Purkinje fibre: interactions among Na+, H+, and Ca2+1. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7629896&form=6&db=m Disturbances of potassium homeostasis in poisoning. unassigned - 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7780058&form=6&db=m Impaired cation transport in thymocytes of rats with chronic uremia includes the Na+/H+ antiporter. causal interaction,unassigned 4,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7900878&form=6&db=m Na+/H+ and Na+/Ca2+ exchange in regulation of [Na+]i and [Ca2+]i during metabolic inhibition. unassigned - 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8376992&form=6&db=m Relations between intracellular ions and energy metabolism under acidotic conditions: a study with nigericin in synaptosomes, neurons, and C6 glioma cells. causal interaction,unassigned 1,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10099866&form=6&db=m Ethyl isopropylamiloride downregulates Na,K-ATPase gene expression which confers cytotoxicity in primary proximal tubule cell cultures. causal interaction,unassigned 1,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12058884&form=6&db=m The acute effects of glycemic control on axonal excitability in human diabetic nerves. diagnostic usage,unassigned 2,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15050724&form=6&db=m Lactic acidosis progressively impairs dopamine uptake in rat striatal synaptosomes by a mechanism partially independent of the Na+/K+-ATPase dysfunction. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23577101&form=6&db=m Regulation of ion gradients across myocardial ischemic border zones: a biophysical modelling analysis. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30385152&form=6&db=m Novel anti-aging gene NM_026333 contributes to proton-induced aging via NCX1-pathway. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Acidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31806348&form=6&db=m Sublethal effects of waterborne copper and copper nanoparticles on the freshwater Neotropical teleost Prochilodus lineatus: A comparative approach. causal interaction,unassigned 1,0 7.2.2.13 Acquired Immunodeficiency Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Acromegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2196126&form=6&db=m Increased activity of digoxin-like substance in low-renin hypertension in acromegaly. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,2,0 7.2.2.13 Acromegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8317188&form=6&db=m Skeletal muscle sodium and potassium changes after successful surgery in acromegaly: relation to body composition, blood glucose, plasma insulin and blood pressure. unassigned - 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2579582&form=6&db=m Protective effect of thyroxine but not parathyroidectomy on gentamicin nephrotoxicity. causal interaction,unassigned 3,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2993017&form=6&db=m The intestinal profile of Na-K-ATPase in three rat models of acute renal failure. diagnostic usage,ongoing research,unassigned 1,4,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2998857&form=6&db=m Mechanism of intestinal adaptation in rats with acute renal failure. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6126857&form=6&db=m Renal function and Na-K-ATPase in rats after suprarenal ligation of inferior vena cava. causal interaction,unassigned 1,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6272181&form=6&db=m [Changes in Na-K-ATPase activity in renal cortex and renal medulla experimental acute renal failure] ongoing research,unassigned 3,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6288818&form=6&db=m The intestinal profile of Na-K-ATPase in two rat models of acute renal failure. diagnostic usage,ongoing research,unassigned 1,3,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7948781&form=6&db=m Experimental renal failure in the rat modulates cardiac Na,K-ATPase alpha 2 mRNA but not protein. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12631124&form=6&db=m Acute renal failure leads to dysregulation of lung salt and water channels. causal interaction,unassigned 2,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15302225&form=6&db=m Lithospermic acid B isolated from Salvia miltiorrhiza ameliorates ischemia/reperfusion-induced renal injury in rats. ongoing research,unassigned 4,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16141536&form=6&db=m Rehmannia glutinose ameliorates renal function in the ischemia/reperfusion-induced acute renal failure rats. ongoing research,unassigned 3,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16183223&form=6&db=m Yukmijihwang-tang ameliorates ischemia/reperfusion-induced renal injury in rats. causal interaction,ongoing research,unassigned 1,3,0 7.2.2.13 Acute Kidney Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32538173&form=6&db=m Carnitine, apelin and resveratrol regulate mitochondrial quality control (QC) related proteins and ameliorate acute kidney injury: role of hydrogen peroxide. ongoing research,unassigned 4,0 7.2.2.13 Acute Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1656777&form=6&db=m Upregulation of rat lung Na-K-ATPase during hyperoxic injury. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 7.2.2.13 Acute Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9124612&form=6&db=m Effects of hyperoxia on type II cell Na-K-ATPase function and expression. ongoing research,unassigned 4,0 7.2.2.13 Acute Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9458813&form=6&db=m Na(+)-K(+)-ATPase expression in alveolar epithelial cells: upregulation of active ion transport by KGF. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Acute Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17530222&form=6&db=m Alveolar epithelium and Na,K-ATPase in acute lung injury. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,1 7.2.2.13 Acute Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17972021&form=6&db=m Regulation of Na,K-ATPase during acute lung injury. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Acute Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24646745&form=6&db=m Resolvin D1 Stimulates Alveolar Fluid Clearance through Alveolar Epithelial Sodium Channel, Na,K-ATPase via ALX/cAMP/PI3K Pathway in Lipopolysaccharide-Induced Acute Lung Injury. unassigned - 7.2.2.13 Acute Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33889010&form=6&db=m Rosuvastatin Enhances Alveolar Fluid Clearance in Lipopolysaccharide-Induced Acute Lung Injury by Activating the Expression of Sodium Channel and Na,K-ATPase via the PI3K/AKT/Nedd4-2 Pathway. unassigned - 7.2.2.13 Adenocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2836053&form=6&db=m Photosensitizing effects of hematoporphyrin derivative and photofrin II on the plasma membrane enzymes 5'-nucleotidase, Na+K+-ATPase, and Mg2+-ATPase in R3230AC mammary adenocarcinomas. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 7.2.2.13 Adenocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16230337&form=6&db=m Recombinant addition of N-glycosylation sites to the basolateral Na,K-ATPase beta1 subunit results in its clustering in caveolae and apical sorting in HGT-1 cells. ongoing research,unassigned 4,0 7.2.2.13 Adenocarcinoma of Lung http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22345575&form=6&db=m Na,K-ATPase is a target of cigarette smoke and reduced expression predicts poor patient outcome of smokers with lung cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22923315&form=6&db=m Lubiprostone Targets Prostanoid Signaling and Promotes Ion Transporter Trafficking, Mucus Exocytosis, and Contractility. unassigned - 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23416519&form=6&db=m Somatic mutations in ATP1A1 and ATP2B3 lead to aldosterone-producing adenomas and secondary hypertension. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24082052&form=6&db=m Somatic ATP1A1, ATP2B3, and KCNJ5 Mutations in Aldosterone-Producing Adenomas. causal interaction,unassigned 4,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24179102&form=6&db=m Complementary somatic mutations of KCNJ5, ATP1A1, and ATP2B3 in sporadic aldosterone producing adrenal adenomas. causal interaction,unassigned 4,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24817817&form=6&db=m Overview of the genetic determinants of primary aldosteronism. causal interaction,unassigned 2,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24866132&form=6&db=m Genetic spectrum and clinical correlates of somatic mutations in aldosterone-producing adenoma. causal interaction,unassigned 2,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25496839&form=6&db=m Somatic mutations of the ATP1A1 gene and aldosterone-producing adenomas. unassigned - 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25624344&form=6&db=m Clinical characteristics of somatic mutations in chinese patients with aldosterone-producing adenoma. causal interaction,ongoing research,unassigned 4,1,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25776071&form=6&db=m DACH1, a zona glomerulosa selective gene in the human adrenal, activates transforming growth factor-? signaling and suppresses aldosterone secretion. causal interaction,unassigned 2,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25906099&form=6&db=m Prevalence and characterization of somatic mutations in Chinese aldosterone-producing adenoma patients. causal interaction,unassigned 4,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26312884&form=6&db=m Role of ANO4 in regulation of aldosterone secretion in the zona glomerulosa of the human adrenal gland. causal interaction,unassigned 1,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26351028&form=6&db=m Different Somatic Mutations in Multinodular Adrenals With Aldosterone-Producing Adenoma. causal interaction,unassigned 3,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26739091&form=6&db=m ENDOCRINE TUMOURS: The genomics of adrenocortical tumors. unassigned - 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26807823&form=6&db=m Genetics of Aldosterone-Producing Adenoma in Korean Patients. causal interaction,unassigned 4,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27485459&form=6&db=m Genetics of primary hyperaldosteronism. causal interaction,unassigned 2,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27648962&form=6&db=m Aldosterone producing adenoma with a somatic KCNJ5 mutation revealing APC dependent Familial Adenomatous Polyposis. causal interaction,unassigned 4,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27729216&form=6&db=m CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism. causal interaction,unassigned 4,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28584016&form=6&db=m Aldosterone-Producing Adenomas: Histopathology-Genotype Correlation and Identification of a Novel CACNA1D Mutation. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,1,1 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29030398&form=6&db=m On the effect of hyperaldosteronism-inducing mutations in Na/K pumps. causal interaction,unassigned 3,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30739536&form=6&db=m Genetic Characteristics of Aldosterone-Producing Adenomas in Blacks. causal interaction,diagnostic usage,unassigned 3,1,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30811176&form=6&db=m Na/K Pump Mutations Associated with Primary Hyperaldosteronism Cause Loss of Function. causal interaction,diagnostic usage,unassigned 3,3,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31207362&form=6&db=m Genomics of benign adrenocortical tumors. causal interaction,unassigned 1,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31695023&form=6&db=m Genetic causes of primary aldosteronism. unassigned - 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31705237&form=6&db=m [Primary aldosteronism : Genetics and pathology]. causal interaction,unassigned 3,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32783015&form=6&db=m Molecular Basis of Primary Aldosteronism and Adrenal Cushing Syndrome. causal interaction,unassigned 4,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32968034&form=6&db=m The landscape of molecular mechanism for aldosterone production in aldosterone-producing adenoma. causal interaction,unassigned 2,0 7.2.2.13 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33800142&form=6&db=m Unravelling the Genetic Basis of Primary Aldosteronism. causal interaction,unassigned 3,0 7.2.2.13 Adrenocortical Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26765578&form=6&db=m Molecular Heterogeneity in Aldosterone-Producing Adenomas. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,4,1 7.2.2.13 Adrenocortical Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27754862&form=6&db=m Hypomethylation of CYP11B2 in Aldosterone-Producing Adenoma. diagnostic usage,ongoing research,unassigned 4,1,0 7.2.2.13 Adrenocortical Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26739091&form=6&db=m ENDOCRINE TUMOURS: The genomics of adrenocortical tumors. unassigned - 7.2.2.13 Agenesis of Corpus Callosum http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17215889&form=6&db=m K-Cl cotransport in red blood cells from patients with KCC3 isoform mutants. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Alkalosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2851674&form=6&db=m The effect of cAMP on ion transport in the proximal tubular cells in bullfrog kidney. therapeutic application,unassigned 1,0 7.2.2.13 Alkalosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7509500&form=6&db=m Effect of low and high potassium diets on H(+)-K(+)-ATPase and Na(+)-K(+)-ATPase activities in the rat inner medullary collecting duct cells. ongoing research,unassigned 1,0 7.2.2.13 Alkalosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7629896&form=6&db=m Disturbances of potassium homeostasis in poisoning. unassigned - 7.2.2.13 Alopecia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30385152&form=6&db=m Novel anti-aging gene NM_026333 contributes to proton-induced aging via NCX1-pathway. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Altitude Sickness http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2827200&form=6&db=m Increased affinity to substrate in sarcolemmal ATPases from hearts acclimatized to high altitude hypoxia. ongoing research,unassigned 2,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2549926&form=6&db=m Ouabain binding in the human brain. Effects of Alzheimer's disease and aging. causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9481617&form=6&db=m Na,K-ATPase mRNA levels and plaque load in Alzheimer's disease. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,2,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12417319&form=6&db=m Regulation of the frontocortical sodium pump by Na+ in Alzheimer's disease: difference from the age-matched control but similarity to the rat model. ongoing research,unassigned 4,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18525130&form=6&db=m Aging, aluminium and basal forebrain lesions modify substrate kinetics of erythrocyte membrane Na,K-ATPase in the rat. unassigned - 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20401693&form=6&db=m Biogerontology in Serbia. causal interaction,unassigned 4,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24156257&form=6&db=m The effects of different antioxidants on the activity of cerebrocortical MnSOD and Na,K-ATPase from post mortem Alzheimer's disease and age-matched normal brains. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25362038&form=6&db=m Quantitative measurement of [na+] and [k+] in postmortem human brain tissue indicates disturbances in subjects with Alzheimer's disease and dementia with lewy bodies. causal interaction,diagnostic usage,unassigned 3,2,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27296892&form=6&db=m Direct interaction of beta-amyloid with Na,K-ATPase as a putative regulator of the enzyme function. causal interaction,ongoing research,unassigned 3,1,0 7.2.2.13 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28510123&form=6&db=m Alzheimer's disease A?42 peptide induces an increase in Na,K-ATPase glutathionylation. causal interaction,unassigned 1,0 7.2.2.13 Amyotrophic Lateral Sclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12514200&form=6&db=m Global loss of Na,K-ATPase and its nitric oxide-mediated regulation in a transgenic mouse model of amyotrophic lateral sclerosis. ongoing research,unassigned 3,0 7.2.2.13 Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2418897&form=6&db=m Calcium accumulated by sickle cell anemia red cells does not affect their potassium (86Rb+) flux components. causal interaction,unassigned 1,0 7.2.2.13 Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3034977&form=6&db=m Sodium-potassium pump, ion fluxes, and cellular dehydration in sickle cell anemia. ongoing research,unassigned 4,0 7.2.2.13 Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4237685&form=6&db=m [Studies on Na+-K+-ATPase and the acetylcholinesterase activity in erythrocyte membranes in saturnine anemia] ongoing research,unassigned 2,0 7.2.2.13 Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31806348&form=6&db=m Sublethal effects of waterborne copper and copper nanoparticles on the freshwater Neotropical teleost Prochilodus lineatus: A comparative approach. causal interaction,unassigned 1,0 7.2.2.13 Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32358067&form=6&db=m Cholesterol-binding protein TSPO2 coordinates maturation and proliferation of terminally differentiating erythroblasts. ongoing research,unassigned 2,0 7.2.2.13 Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33356956&form=6&db=m The potential role of Na-K-ATPase and its signaling in the development of anemia in chronic kidney disease. causal interaction,unassigned 4,0 7.2.2.13 Anemia, Sickle Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2418897&form=6&db=m Calcium accumulated by sickle cell anemia red cells does not affect their potassium (86Rb+) flux components. causal interaction,unassigned 1,0 7.2.2.13 Anemia, Sickle Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3034977&form=6&db=m Sodium-potassium pump, ion fluxes, and cellular dehydration in sickle cell anemia. ongoing research,unassigned 4,0 7.2.2.13 Angelman Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23911285&form=6&db=m Genetic Reduction of the ?1 Subunit of Na/K-ATPase Corrects Multiple Hippocampal Phenotypes in Angelman Syndrome. unassigned - 7.2.2.13 Angina, Unstable http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1649699&form=6&db=m Endogenous plasma Na,K-ATPase inhibitory activity and digoxin like immunoreactivity after acute myocardial infarction. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 7.2.2.13 Aortic Coarctation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10394073&form=6&db=m Na,K-ATPase and Ca-ATPase activities of the myocardial sarcolemma in aging rats after aorta coarctation: role of invertors. ongoing research,unassigned 2,0 7.2.2.13 Arrhythmias, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1334399&form=6&db=m Inhibition of myocardial Na(+)-K(+)-ATPase activity by bretylium: role of potassium. therapeutic application,unassigned 1,0 7.2.2.13 Arrhythmias, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2989573&form=6&db=m Effects of adenosine on transmembrane potential and sarcolemmal Na+-K+ ATPase activity of human atrial myocardium. therapeutic application,unassigned 3,0 7.2.2.13 Arrhythmias, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6307709&form=6&db=m How increased sodium influx enhances digoxin-induced arrhythmias in guinea-pig atrial muscle. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Arrhythmias, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23341448&form=6&db=m A Structural View on the Functional Importance of the Sugar Moiety and Steroid Hydroxyls of Cardiotonic Steroids in Binding to Na,K-ATPase. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Arrhythmias, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24630927&form=6&db=m Cardiac glycoside ouabain induces autophagic cell death in non-small cell lung cancer cells via a JNK-dependent decrease of Bcl-2. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Arrhythmias, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29702256&form=6&db=m Association between activities of SOD, MDA and Na+-K+-ATPase in peripheral blood of patients with acute myocardial infarction and the complication of varying degrees of arrhythmia. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,2,1 7.2.2.13 Arrhythmias, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32256664&form=6&db=m Antiarrhythmic Mechanisms of Chinese Herbal Medicine Dingji Fumai Decoction. unassigned - 7.2.2.13 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8663901&form=6&db=m Altered fatty acid, cholesterol and Na+/K+ ATPase activity in erythrocyte membrane of rheumatoid arthritis patients. diagnostic usage,unassigned 3,0 7.2.2.13 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9030200&form=6&db=m Modulation of cytokine production by human mononuclear cells following impairment of Na, K-ATPase activity. causal interaction,unassigned 3,0 7.2.2.13 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Ataxia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2426381&form=6&db=m Possible implication of an efferent neuropathy in vestibular upset. unassigned - 7.2.2.13 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11791942&form=6&db=m Modulation of Na,K-ATPase activity by immunoglobulins--II. Na,K-ATPase activity in erythrocytes from patients with various chronic disturbances. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31905689&form=6&db=m Concentration-Dependent Effects of N-3 Long-Chain Fatty Acids on Na,K-ATPase Activity in Human Endothelial Cells. unassigned - 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10809498&form=6&db=m Inhibition of Na,K-ATPase by external electrical cardioversion in a sheep model of atrial fibrillation. unassigned - 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12463096&form=6&db=m [Autoantibodies: new upstream targets of paroxysmal atrial fibrillation in patients with congestive heart failure] causal interaction,ongoing research,therapeutic application,unassigned 3,1,1,0 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12479230&form=6&db=m Sodium pump isoform expression in heart failure: implication for treatment. causal interaction,ongoing research,therapeutic application,unassigned 3,2,4,0 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12503616&form=6&db=m Remodeling of Na,K-ATPase, and membrane fluidity after atrial fibrillation in sheep. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19246813&form=6&db=m Autoimmune mechanisms underlying dilated cardiomyopathy. causal interaction,unassigned 1,0 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19299895&form=6&db=m Atrial Na,K-ATPase Increase and Potassium Dysregulation Accentuate the Risk of Postoperative Atrial Fibrillation. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,1 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21617318&form=6&db=m Contribution of acquired factors to the pathogenesis of dilated cardiomyopathy. -The cause of dilated cardiomyopathy: genetic or acquired? (Acquired-Side)-. unassigned - 7.2.2.13 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31819839&form=6&db=m Digoxin Toxicity in a Patient with Pacemaker: A Case Report. therapeutic application,unassigned 4,0 7.2.2.13 Bartter Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1329511&form=6&db=m Abnormalities of erythrocyte sodium transport systems in Bartter's syndrome. ongoing research,unassigned 3,0 7.2.2.13 Bartter Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1408334&form=6&db=m Heterogeneous derangement of cellular sodium metabolism in Bartter's syndrome. Description of two cases and review of the literature. ongoing research,unassigned 3,0 7.2.2.13 Bartter Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2741641&form=6&db=m Lymphocytic sodium and potassium pump function in Bartter's syndrome. ongoing research,unassigned 2,0 7.2.2.13 Bartter Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6090182&form=6&db=m The effect of long-term treatment with spironolactone on sodium pump abnormalities in the red blood cells of patients with Bartter's syndrome. causal interaction,ongoing research,therapeutic application,unassigned 1,4,3,0 7.2.2.13 Bartter Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19901197&form=6&db=m Physiological genomics identifies estrogen-related receptor alpha as a regulator of renal sodium and potassium homeostasis and the renin-angiotensin pathway. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Bone Resorption http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6305636&form=6&db=m Inhibition of stimulated bone resorption by vanadate. therapeutic application,unassigned 1,0 7.2.2.13 Botulism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6293618&form=6&db=m [Activity of erythrocyte membrane Na,K-ATPase in rats with experimental botulism] ongoing research,unassigned 4,0 7.2.2.13 Brain Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1325690&form=6&db=m Edema, cation content, and ATPase activity after middle cerebral artery occlusion in rats. causal interaction,ongoing research,therapeutic application,unassigned 4,4,2,0 7.2.2.13 Brain Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1982477&form=6&db=m Excitatory amino acid receptors, oxido-reductive processes and brain oedema following transient ischaemia in gerbils. unassigned - 7.2.2.13 Brain Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2544538&form=6&db=m Membrane damage in acute brain trauma. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Brain Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2662983&form=6&db=m The role of free radicals and eicosanoids in the pathogenetic mechanism underlying ischemic brain edema. causal interaction,unassigned 4,0 7.2.2.13 Brain Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8709678&form=6&db=m CDP-choline: pharmacological and clinical review. therapeutic application,unassigned 1,0 7.2.2.13 Brain Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16860980&form=6&db=m Chronic daily administration of ethyl docosahexaenoate protects against gerbil brain ischemic damage through reduction of arachidonic acid liberation and accumulation. causal interaction,diagnostic usage,ongoing research,therapeutic application 2,2,1,2 7.2.2.13 Brain Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24044899&form=6&db=m Ammonia-induced Na,K-ATPase/ouabain-mediated EGF receptor transactivation, MAPK/ERK and PI3K/AKT signaling and ROS formation cause astrocyte swelling. ongoing research,unassigned 3,0 7.2.2.13 Brain Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33054705&form=6&db=m Promising Anti-stroke Signature of Voglibose: Investigation through In- Silico Molecular Docking and Virtual Screening in In-Vivo Animal Studies. diagnostic usage,unassigned 4,0 7.2.2.13 Brain Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33608957&form=6&db=m Pre-exposure of voglibose exerts cerebroprotective effects through attenuating activation of the polyol pathway and inflammation. diagnostic usage,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Brain Injuries http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30505197&form=6&db=m Hypoxia-induced brain cell damage in male albino wistar rat. diagnostic usage,ongoing research,unassigned 4,3,0 7.2.2.13 Brain Injuries, Traumatic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9065836&form=6&db=m Inhibition of the electrogenic Na pump underlies delayed depolarization of cortical neurons after mechanical injury or glutamate. causal interaction,therapeutic application,unassigned 4,2,0 7.2.2.13 Brain Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2475924&form=6&db=m Effect of enhanced capillary activity on the blood-brain barrier during focal cerebral ischemia in cats. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,3,0 7.2.2.13 Brain Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2853824&form=6&db=m Alterations in synaptosomal membrane Na,K-ATPase of the gerbil cortex and hippocampus following reversible brain ischemia. unassigned - 7.2.2.13 Brain Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10094131&form=6&db=m Ginkgo biloba extract (EGb 761) protects Na,K-ATPase activity during cerebral ischemia in mice. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Brain Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12396078&form=6&db=m Ginkgo biloba extract (EGb 761) protects Na,K-ATPase isoenzymes during cerebral ischemia. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Brain Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15810661&form=6&db=m [Na,K-atpase activity in different brain regions in cerebral ischemia and influence of quinacrine and indomethacin administration.] causal interaction,therapeutic application,unassigned 1,2,0 7.2.2.13 Brain Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20617692&form=6&db=m [Protective effects of luteolin on neurons against oxygen-glucose deprivation/reperfusion injury via improving Na+/K+ -ATPase activity] ongoing research,unassigned 2,0 7.2.2.13 Brain Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22266555&form=6&db=m Effect of deferoxamine on Na+K+ATPase activity after cerebral ischemia in rabbits. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Brain Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6311998&form=6&db=m Na+/K+ ATPase and cell growth IV: a metabolic marker of human brain tumors? causal interaction,diagnostic usage,ongoing research,unassigned 3,3,2,0 7.2.2.13 Brain Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25976744&form=6&db=m Na/K-ATPase as a target for anticancer drugs: studies with perillyl alcohol. causal interaction,therapeutic application,unassigned 2,3,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7710638&form=6&db=m Potential suitability of Na+/K(+)-transporting ATPase in pre-screens for anti-cancer agents. ongoing research,unassigned 2,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18755149&form=6&db=m Actein inhibits the Na+-K+-ATPase and enhances the growth inhibitory effect of digitoxin on human breast cancer cells. ongoing research,unassigned 3,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19893046&form=6&db=m Down-regulation of FXYD3 expression in human lung cancers: its mechanism and potential role in carcinogenesis. therapeutic application,unassigned 1,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26631742&form=6&db=m In Vitro-In Vivo Extrapolation Scaling Factors for Intestinal P-Glycoprotein and Breast Cancer Resistance Protein: Part I: A Cross-Laboratory Comparison of Transporter-Protein Abundances and Relative Expression Factors in Human Intestine and Caco-2 Cells. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28529692&form=6&db=m Abnormal expression of ATP1A1 and ATP1A2 in breast cancer. ongoing research,unassigned 1,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29590154&form=6&db=m Na+/K+ ATPase activity promotes invasion of endocrine resistant breast cancer cells. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29777796&form=6&db=m Identification of a sodium pump Na+/K+ ATPase ?1-targeted peptide for PET imaging of breast cancer. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33456676&form=6&db=m Hyaluronic Acid Improves Hydrogen Peroxide Modulatory Effects on Calcium Channel and Sodium-Potassium Pump in 4T1 Breast Cancer Cell Line. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34180301&form=6&db=m Controllable Drug Delivery by Na+/K+ ATPase ?1 Targeting Peptide Conjugated DSPE-PEG Nanocarriers for Breast Cancer. therapeutic application,unassigned 3,0 7.2.2.13 Brugada Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8886172&form=6&db=m Hypokalemia, high erythrocyte Na+ and low erythrocyte Na,K-ATPase in relatives of patients dying from sudden unexplained death syndrome in north-east Thailand and in survivors from near-fatal attacks. diagnostic usage,unassigned 1,0 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6855027&form=6&db=m Membrane potential of rat urinary bladder epithelial cells. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17164420&form=6&db=m A pump-independent function of the Na,K-ATPase is required for epithelial junction function and tracheal tube-size control. causal interaction,unassigned 2,0 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21278788&form=6&db=m Translationally controlled tumor protein induces human breast epithelial cell transformation through the activation of Src. unassigned - 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22345575&form=6&db=m Na,K-ATPase is a target of cigarette smoke and reduced expression predicts poor patient outcome of smokers with lung cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26743443&form=6&db=m GNAS mutations in adrenal aldosterone-producing adenomas [Rapid Communication]. causal interaction,unassigned 3,0 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31691615&form=6&db=m The Na/Ca Exchange as a Target for Antitumor Effect of 4Hz Pulsing Magnetic Field. causal interaction,unassigned 4,0 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32968034&form=6&db=m The landscape of molecular mechanism for aldosterone production in aldosterone-producing adenoma. causal interaction,unassigned 2,0 7.2.2.13 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33770271&form=6&db=m Integrated analysis of miRNA and mRNA transcriptomic reveals antler growth regulatory network. causal interaction,unassigned 2,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2552715&form=6&db=m [The biological effect of sodium butyrate (NaBT) on SGC-7901 cells] diagnostic usage,ongoing research,unassigned 1,4,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2829435&form=6&db=m [Effect of UHF and ionizing radiation on the Na-K-ATPase activity of Ehrlich ascitic carcinoma cells] diagnostic usage,ongoing research,unassigned 1,3,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7710638&form=6&db=m Potential suitability of Na+/K(+)-transporting ATPase in pre-screens for anti-cancer agents. ongoing research,unassigned 2,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10411090&form=6&db=m Reduced expression of beta-subunit of Na,K-ATPase in human clear-cell renal cell carcinoma. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11179415&form=6&db=m Na,K-ATPase beta-subunit is required for epithelial polarization, suppression of invasion, and cell motility. causal interaction,ongoing research,unassigned 1,3,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12673711&form=6&db=m Analysis of the Na,K-ATPase alpha- and beta-subunit expression profiles of bladder cancer using tissue microarrays. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,2 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14699059&form=6&db=m Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma. diagnostic usage,ongoing research,therapeutic application,unassigned 1,4,1,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15616195&form=6&db=m Novel role for Na,K-ATPase in phosphatidylinositol 3-kinase signaling and suppression of cell motility. causal interaction,ongoing research,therapeutic application,unassigned 3,1,4,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17535728&form=6&db=m Membrane transporters and signal transduction. causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17535735&form=6&db=m Identification of protein kinase C as an intermediate in Na,K-ATPase Beta-subunit mediated lamellipodia formation and suppression of cell motility in carcinoma cells. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18006011&form=6&db=m Na,K-adenosine triphosphatase alpha1-subunit predicts survival of renal clear cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,3 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20501797&form=6&db=m Na,K-ATPase subunits as markers for epithelial-mesenchymal transition in cancer and fibrosis. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,3,1 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21784855&form=6&db=m Na/K-ATPase mimetic pNaKtide inhibits the growth of human cancer cells. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21812995&form=6&db=m Long-term exposure to hypoxia inhibits tumor progression of lung cancer in rats and mice. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22345575&form=6&db=m Na,K-ATPase is a target of cigarette smoke and reduced expression predicts poor patient outcome of smokers with lung cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24452105&form=6&db=m Epigenetic silencing of Na,K-ATPase ? 1 subunit gene ATP1B1 by methylation in clear cell renal cell carcinoma. unassigned - 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25275034&form=6&db=m Effects of digoxin and Na, K-ATPase immunoexpression on human oral squamous carcinomas. diagnostic usage,ongoing research,therapeutic application,unassigned 2,3,1,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25836370&form=6&db=m Glucocorticoids suppress renal cell carcinoma progression by enhancing Na,K-ATPase beta-1 subunit expression. causal interaction,unassigned 3,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26286140&form=6&db=m Na,K-ATPase ?1-subunit is a target of sonic hedgehog signaling and enhances medulloblastoma tumorigenicity. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26579519&form=6&db=m Transcriptional regulators of Na,K-ATPase subunits. causal interaction,unassigned 4,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27845894&form=6&db=m Overexpression of ATPase Na+/+ transporting alpha 1 polypeptide, ATP1A1, correlates with clinical diagnosis and progression of esophageal squamous cell carcinoma. causal interaction,diagnostic usage,unassigned 4,4,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28484360&form=6&db=m Downregulation of ATP1A1 promotes cancer development in renal cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,4 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30015972&form=6&db=m Combined assessment of low PGRMC1/positive ATP1A1 levels has enhanced prognostic value for renal cell carcinoma. causal interaction,diagnostic usage,therapeutic application,unassigned 2,4,1,0 7.2.2.13 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30127995&form=6&db=m Bufalin induced apoptosis of bladder carcinoma cells through the inactivation of Na+K+-ATPase. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Carcinoma, Ductal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29148540&form=6&db=m Single-cell heterogeneity in ductal carcinoma in situ of breast. diagnostic usage,unassigned 1,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=129155&form=6&db=m Changes in (Na+ + K+)-ATPase activity of Ehrlich ascites tumor cells produced by alteration of membrane fatty acid composition. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2294960&form=6&db=m Regulatory volume increase in Ehrlich ascites tumor cells. ongoing research,unassigned 1,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2972256&form=6&db=m Motility, heat, and lactate production in ejaculated bovine sperm. causal interaction,unassigned 1,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2986694&form=6&db=m Transport functions of the liver. Lack of correlation between hepatocellular ouabain uptake and binding to (Na+ + K+)-ATPase. diagnostic usage,unassigned 3,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6087923&form=6&db=m Studies on the relationship between glycolysis and (Na+ + K+)-ATPase in cultured cells. ongoing research,unassigned 4,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6168390&form=6&db=m A mouse homolog to the avian sarcoma virus src protein is a member of a protein kinase cascade. causal interaction,unassigned 3,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6251054&form=6&db=m Phosphorylation of the beta subunit of Na+K+-ATPase in Ehrlich ascites tumor by a membrane-bound protein kinase. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Carcinoma, Ehrlich Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6260791&form=6&db=m Regulation of phosphorylation of the beta-subunit of th Ehrlich ascites tumor Na+K+-ATPase by a protein kinase cascade. unassigned - 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=179989&form=6&db=m Isolation and characterization of the plasma membranes from rat ascites hepatomas and from normal rat livers, including newborn, regenerating, and adult livers. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1654277&form=6&db=m Tissue-specific expression of Na,K-ATPase beta-subunit. Does beta 2 expression correlate with tumorigenesis? diagnostic usage,ongoing research,unassigned 4,4,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2986694&form=6&db=m Transport functions of the liver. Lack of correlation between hepatocellular ouabain uptake and binding to (Na+ + K+)-ATPase. diagnostic usage,unassigned 3,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3015930&form=6&db=m Turnover of the catalytic subunit of Na,K-ATPase in HTC cells. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4297059&form=6&db=m Studies on plasma membranes. VI. Differences in the effect of temperature on the ATPase and (Na+-K+)-ATPase activities of plasma membranes isolated from rat liver and hepatoma. ongoing research,unassigned 4,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6133555&form=6&db=m A high-affinity (Ca2+ + Mg2+)-ATPase in plasma membranes of rat ascites hepatoma AH109A cells. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6327435&form=6&db=m Insulin regulation of Na/K pump activity in rat hepatoma cells. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27802297&form=6&db=m Identification of Tumor Antigen AF20 as Glycosylated Transferrin Receptor 1 in Complex with Heat Shock Protein 90 and/or Transporting ATPase. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32210281&form=6&db=m Inhibitors of Na+/K+ ATPase exhibit antitumor effects on multicellular tumor spheroids of hepatocellular carcinoma. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Carcinoma, Intraductal, Noninfiltrating http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29148540&form=6&db=m Single-cell heterogeneity in ductal carcinoma in situ of breast. diagnostic usage,unassigned 1,0 7.2.2.13 Carcinoma, Lewis Lung http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21812995&form=6&db=m Long-term exposure to hypoxia inhibits tumor progression of lung cancer in rats and mice. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Carcinoma, Renal Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10411090&form=6&db=m Reduced expression of beta-subunit of Na,K-ATPase in human clear-cell renal cell carcinoma. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Carcinoma, Renal Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18006011&form=6&db=m Na,K-adenosine triphosphatase alpha1-subunit predicts survival of renal clear cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,3 7.2.2.13 Carcinoma, Renal Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24452105&form=6&db=m Epigenetic silencing of Na,K-ATPase ? 1 subunit gene ATP1B1 by methylation in clear cell renal cell carcinoma. unassigned - 7.2.2.13 Carcinoma, Renal Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25836370&form=6&db=m Glucocorticoids suppress renal cell carcinoma progression by enhancing Na,K-ATPase beta-1 subunit expression. causal interaction,unassigned 3,0 7.2.2.13 Carcinoma, Renal Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28484360&form=6&db=m Downregulation of ATP1A1 promotes cancer development in renal cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,4 7.2.2.13 Carcinoma, Renal Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30015972&form=6&db=m Combined assessment of low PGRMC1/positive ATP1A1 levels has enhanced prognostic value for renal cell carcinoma. causal interaction,diagnostic usage,therapeutic application,unassigned 2,4,1,0 7.2.2.13 Carcinoma, Transitional Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12673711&form=6&db=m Analysis of the Na,K-ATPase alpha- and beta-subunit expression profiles of bladder cancer using tissue microarrays. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,2 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7508043&form=6&db=m Na+, K(+)-ATPase and Na+/Ca2+ exchange isoforms: physiological and physiopathological relevance. unassigned - 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7982417&form=6&db=m Na,K-pump concentration in hypertrophied human hearts. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8288620&form=6&db=m Alteration of Na,K-ATPase subunit mRNA and protein levels in hypertrophied rat heart. causal interaction,unassigned 3,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8733174&form=6&db=m High blood pressure, oxygen radicals and antioxidants: etiological relationships. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9405189&form=6&db=m Differential regulation of Na/K-ATPase alpha-subunit isoform gene expressions in cardiac myocytes by ouabain and other hypertrophic stimuli. causal interaction,unassigned 3,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12623296&form=6&db=m Intracellular Na+ and altered Na+ transport mechanisms in cardiac hypertrophy and failure. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,4,3 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15076199&form=6&db=m Coordinated shifts in Na/K-ATPase isoforms and their endogenous ligands during cardiac hypertrophy and failure in NaCl-sensitive hypertension. causal interaction,unassigned 2,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20952666&form=6&db=m Mice expressing ouabain-sensitive {alpha}1 Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophy. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24218169&form=6&db=m Overexpression of the Na+/K+ ATPase ?2 but not ?1 isoform attenuates pathological cardiac hypertrophy and remodeling. therapeutic application,unassigned 2,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25103111&form=6&db=m Cardiac hypertrophy in mice expressing unphosphorylatable phospholemman. causal interaction,ongoing research,therapeutic application,unassigned 1,1,1,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29424237&form=6&db=m Greasing the wheels or a spanner in the works? Regulation of the cardiac sodium pump by palmitoylation. unassigned - 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32069992&form=6&db=m The Redox-Sensitive Na/K-ATPase Signaling in Uremic Cardiomyopathy. causal interaction,unassigned 3,0 7.2.2.13 Cardiomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33801629&form=6&db=m Cardiac Oxidative Signaling and Physiological Hypertrophy in the Na/K-ATPase ?1s/s?2s/s Mouse Model of High Affinity for Cardiotonic Steroids. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=132311&form=6&db=m Abnormalities in heart membranes and myofibrils during bacterial infective cardiomyopathy in the rabbit. diagnostic usage,therapeutic application,unassigned 1,1,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2851806&form=6&db=m The Na,K-pump in human myocardium: quantification in normal subjects and patients with suspected cardiomyopathy. diagnostic usage,ongoing research,unassigned 2,3,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8277820&form=6&db=m Decreased Na,K-ATPase gene expression in cardiomyopathic hamster hearts. causal interaction,unassigned 4,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17535729&form=6&db=m Molecular insights into uremic cardiomyopathy: cardiotonic steroids and na/k ATPase signaling. causal interaction,unassigned 2,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22670474&form=6&db=m Anti-apoptotic potential of gymnemic acid phospholipid complex pretreatment in Wistar rats with experimental cardiomyopathy. diagnostic usage,unassigned 4,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24404431&form=6&db=m Gender differences in the development of uremic cardiomyopathy following partial nephrectomy: Role of progesterone. unassigned - 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27694325&form=6&db=m Rapamycin Attenuates Cardiac Fibrosis in Experimental Uremic Cardiomyopathy by Reducing Marinobufagenin Levels and Inhibiting Downstream Pro-Fibrotic Signaling. causal interaction,unassigned 3,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27698370&form=6&db=m Attenuation of Na/K-ATPase Mediated Oxidant Amplification with pNaKtide Ameliorates Experimental Uremic Cardiomyopathy. causal interaction,ongoing research,therapeutic application,unassigned 2,3,1,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28338377&form=6&db=m Sodium potassium adenosine triphosphatase (Na/K-ATPase) as a therapeutic target for uremic cardiomyopathy. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29052587&form=6&db=m Corrigendum: Attenuation of Na/K-ATPase Mediated Oxidant Amplification with pNaKtide Ameliorates Experimental Uremic Cardiomyopathy. unassigned - 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29318961&form=6&db=m Targeting Na/K-ATPase Signaling: A New Approach to Control Oxidative Stress. unassigned - 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32069992&form=6&db=m The Redox-Sensitive Na/K-ATPase Signaling in Uremic Cardiomyopathy. causal interaction,unassigned 3,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32587074&form=6&db=m Central Role for Adipocyte Na,K-ATPase Oxidant Amplification Loop in the Pathogenesis of Experimental Uremic Cardiomyopathy. causal interaction,unassigned 1,0 7.2.2.13 Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32824688&form=6&db=m Oxidant-Induced Alterations in the Adipocyte Transcriptome: Role of the Na,K-ATPase Oxidant Amplification Loop. causal interaction,unassigned 2,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1323267&form=6&db=m Na,K-ATPase expression in normal and failing human left ventricle. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1338519&form=6&db=m [Correlation of intra-erythrocytic Na, K, Ca, Mg contents and ATPase activity with dilated cardiomyopathy] diagnostic usage,unassigned 2,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3376892&form=6&db=m Relation of left ventricular function and Na,K-pump concentration in suspected idiopathic dilated cardiomyopathy. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7550131&form=6&db=m Na,K-ATPase receptor subunits alpha 1, alpha 2 and alpha 3 mRNA in dilated cardiomyopathy. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10072231&form=6&db=m Myocardial adenine nucleotides, glycogen, and Na, K-ATPase in patients with idiopathic dilated cardiomyopathy requiring mechanical circulatory support. diagnostic usage,ongoing research,unassigned 2,2,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11354998&form=6&db=m Defective activity and isoform of the Na,K-ATPase in the dilated cardiomyopathic hamster. diagnostic usage,ongoing research,therapeutic application,unassigned 1,4,1,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11828798&form=6&db=m [Autoantibodies against sarcolemmal Na-K-ATPase in patients with dilated cardiomyopathy: autoimmune basis for ventricular arrhythmias in patients with congestive heart failure] causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12354443&form=6&db=m Autoantibodies produced against sarcolemmal Na-K-ATPase: possible upstream targets of arrhythmias and sudden death in patients with dilated cardiomyopathy. causal interaction,ongoing research,unassigned 2,1,0 7.2.2.13 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12463096&form=6&db=m [Autoantibodies: new upstream targets of paroxysmal atrial fibrillation in patients with congestive heart failure] causal interaction,ongoing research,therapeutic application,unassigned 3,1,1,0 7.2.2.13 Cardiotoxicity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6256069&form=6&db=m Cardiac sodium, potassium-adenosine triphosphatase as a possible site of adriamycin-induced cardiotoxicity. therapeutic application,unassigned 2,0 7.2.2.13 Cardiotoxicity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22670474&form=6&db=m Anti-apoptotic potential of gymnemic acid phospholipid complex pretreatment in Wistar rats with experimental cardiomyopathy. diagnostic usage,unassigned 4,0 7.2.2.13 Cardiotoxicity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30755704&form=6&db=m Development of Orthogonal Linear Separation Analysis (OLSA) to Decompose Drug Effects into Basic Components. therapeutic application,unassigned 2,0 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11355006&form=6&db=m Omegacoeur, a Mediterranean nutritional complement, stimulates Na,K-ATPase activity in human endothelial cells. ongoing research,unassigned 4,0 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15970564&form=6&db=m The Na/K-ATPase-mediated signal transduction as a target for new drug development. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17535728&form=6&db=m Membrane transporters and signal transduction. causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21632957&form=6&db=m Renovascular hypertension using a modified two-kidney, one-clip approach in mice is not dependent on the {alpha}1 or {alpha}2 Na,K-ATPase ouabain-binding site. causal interaction,therapeutic application,unassigned 3,3,0 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24903141&form=6&db=m Mechanisms of isoform-specific Na/K pump regulation by short- and long-term adrenergic activation in rat ventricular myocytes. unassigned - 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25101054&form=6&db=m The Trade-Off between Dietary Salt and Cardiovascular Disease; A Role for Na/K-ATPase Signaling? unassigned - 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26374826&form=6&db=m Synthesis of an Endogenous Steroidal Na Pump Inhibitor Marinobufagenin, Implicated in Human Cardiovascular Diseases, Is Initiated by CYP27A1 via Bile Acid Pathway. unassigned - 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27598118&form=6&db=m The Role of Na/K-ATPase Signaling in Oxidative Stress Related to Obesity and Cardiovascular Disease. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27643939&form=6&db=m The role of red blood cell deformability and Na,K-ATPase function in selected risk factors of cardiovascular diseases in humans: focus on hypertension, diabetes mellitus and hypercholesterolemia. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,3,1 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30041449&form=6&db=m The Role of Na/K-ATPase Signaling in Oxidative Stress Related to Aging: Implications in Obesity and Cardiovascular Disease. causal interaction,ongoing research,therapeutic application,unassigned 2,2,1,0 7.2.2.13 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32447998&form=6&db=m Allopurinol and valproic acid improve cardiac triglyceride and Na+-K+-ATPase activity independent of circulating aldosterone in female rats with glucose intolerance. causal interaction,ongoing research,therapeutic application,unassigned 1,3,1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=136953&form=6&db=m [Na-K-ATPase activity in the normal aging crystalline lens and in senile cataract] ongoing research,unassigned 1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=150777&form=6&db=m The effect of X-irradiation on the sodium-potassium-activated adenosine triphosphatase (Na-K-ATPase) activity in the epithelium of the rat lens. A histochemical and biochemical study. ongoing research,unassigned 1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1318822&form=6&db=m Na,K-ATPase and phospholipid degradation in bovine and human lenses. diagnostic usage,unassigned 3,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2558845&form=6&db=m Prefeeding of aldose reductase inhibitor and galactose cataractogenesis. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2841073&form=6&db=m The effect of near-UV light on Na-K-ATPase of the rat lens. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2854821&form=6&db=m Effect of inhibition of lens membrane Na+-K+ ATPase by ouabain pre-treatment--an in vitro study. causal interaction,unassigned 1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2988990&form=6&db=m Immunodetection of Na,K-ATPase in guinea-pig retinal layers, cornea and lens. unassigned - 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3011358&form=6&db=m Sodium-potassium-dependent-ATPase activity in Emory mouse lens. ongoing research,unassigned 4,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3011482&form=6&db=m ATP hydrolysis kinetics of Na,K-ATPase in cataract. unassigned - 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3186192&form=6&db=m Effect of glutathione depletion on cation transport and metabolism in the rabbit lens. causal interaction,unassigned 4,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4271378&form=6&db=m Myotonia induced by diazacholesterol: increased (Na+ + K+)-ATPase activity of erythrocyte ghosts and development of cataracts. unassigned - 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6139352&form=6&db=m Na,K-ATPase in simulated eye bank and cryoextracted rabbit lenses, and human eye bank lenses and cataracts. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,1,1 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6244231&form=6&db=m Sodium-potassium--dependent ATPase. II. Cytochemical localization during the reversal of galactose cataracts in rat. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6255263&form=6&db=m The Na-K-ATPase in relation to the Na, K and taurine levels in the senile cataract. diagnostic usage,unassigned 2,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6266459&form=6&db=m Lipid composition and (Na+ + K+)-ATPase activity in rat lens during triparanol-induced cataract formation. ongoing research,unassigned 3,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6297283&form=6&db=m [Metabolic abnormality of the senile cataract report II, the Na+-K+-ATPase in relation to the ATP level in the senile cataract] diagnostic usage,therapeutic application,unassigned 3,1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6391828&form=6&db=m Lens lipids. causal interaction,unassigned 4,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7698259&form=6&db=m Lenticular alterations in hypertensive rats. causal interaction,unassigned 3,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9355935&form=6&db=m Cataract development induced by repeated oral dosing with FK506 (tacrolimus) in adult rats. ongoing research,unassigned 2,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10560239&form=6&db=m Na,K-ATPase in lens epithelia from patients with senile cataracts. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,4,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11834881&form=6&db=m Pyruvate inhibits galactosemic changes in cultured cat lens epithelial cells. diagnostic usage,therapeutic application,unassigned 2,1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11980868&form=6&db=m Altered Na,K-ATPase pattern in gamma-crystallin mutant mice. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12787026&form=6&db=m The molecular chaperone alpha-crystallin incorporated into red cell ghosts protects membrane Na/K-ATPase against glycation and oxidative stress. diagnostic usage,therapeutic application,unassigned 3,1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15254964&form=6&db=m The influence of protein tyrosine phosphatase-1B on Na,K-ATPase activity in lens. causal interaction,unassigned 2,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15388076&form=6&db=m Expression, regulation and function of Na,K-ATPase in the lens. ongoing research,unassigned 1,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16797533&form=6&db=m Synergism between phospholipase D2 and sorbitol accumulation in diabetic cataract formation through modulation of Na,K-ATPase activity and osmotic stress. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19479337&form=6&db=m Over-expression of translationally controlled tumor protein in lens epithelial cells seems to be associated with cataract development. causal interaction,ongoing research,unassigned 2,4,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21190250&form=6&db=m The effect of endothelin-1 on Src-family tyrosine kinases and Na,K-ATPase activity in porcine lens epithelium. causal interaction,unassigned 3,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21792912&form=6&db=m The effect of endothelin-1 on Src-family tyrosine kinases and Na,K-ATPase activity in porcine lens epithelium. causal interaction,unassigned 3,0 7.2.2.13 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31186457&form=6&db=m Failure of Oxysterols Such as Lanosterol to Restore Lens Clarity from Cataracts. therapeutic application,unassigned 2,0 7.2.2.13 Celiac Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11321406&form=6&db=m Decreased Na+/K+ pump activity in the erythrocytes of children with treated celiac disease. unassigned - 7.2.2.13 Central Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Charcot-Marie-Tooth Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31373411&form=6&db=m ATP1A1 mutations cause intermediate Charcot-Marie-Tooth disease. causal interaction,unassigned 3,0 7.2.2.13 Charcot-Marie-Tooth Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31705535&form=6&db=m Hereditary spastic paraplegia is a novel phenotype for germline de novo ATP1A1 mutation. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Chediak-Higashi Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8612721&form=6&db=m Na,K-ATPase activity in red blood cells from patients with Chediak-Higashi syndrome. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Cholera http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=132458&form=6&db=m Prevention and reversal of cholera enterotoxin-induced intestinal secretion by methylprednisolone induction of Na+-K+-ATPase. ongoing research,unassigned 1,0 7.2.2.13 Cholera http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=218220&form=6&db=m Evidence for role of glycoprotein carbohydrates in membrane transport: specific inhibition by tunicamycin. unassigned - 7.2.2.13 Cholera http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2563204&form=6&db=m Regulation of Na+-K+-ATPase activity in kidney proximal tubules: involvement of GTP binding proteins. therapeutic application,unassigned 1,0 7.2.2.13 Cholera http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6269439&form=6&db=m Antisecretory effects of berberine in rat ileum. unassigned - 7.2.2.13 Cholera http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9671965&form=6&db=m Acute and chronic regulation of Na+/K+-ATPase transport activity in the RN22 Schwann cell line in response to stimulation of cyclic AMP production. ongoing research,therapeutic application,unassigned 1,3,0 7.2.2.13 Cholestasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2446505&form=6&db=m Effects of chlorpromazine on Na+-K+-ATPase pumping and solute transport in rat hepatocytes. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Colitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2556782&form=6&db=m Na,K-ATPase activity in rectal mucosa of children with ulcerative colitis and Crohn's disease. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Colitis, Ulcerative http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2556782&form=6&db=m Na,K-ATPase activity in rectal mucosa of children with ulcerative colitis and Crohn's disease. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Colitis, Ulcerative http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2832138&form=6&db=m Inverse relationship between colonic (Na+ + K+)-ATPase activity and degree of mucosal inflammation in inflammatory bowel disease. causal interaction,unassigned 4,0 7.2.2.13 Colitis, Ulcerative http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6088428&form=6&db=m Decreased colonic Na-K-ATPase activity in active ulcerative colitis. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,1,3 7.2.2.13 Colonic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9405883&form=6&db=m Opposite expression pattern of the human Na,K-ATPase beta 1 isoform in stomach and colon adenocarcinomas. ongoing research,unassigned 3,0 7.2.2.13 Colonic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21812995&form=6&db=m Long-term exposure to hypoxia inhibits tumor progression of lung cancer in rats and mice. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Colonic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27802297&form=6&db=m Identification of Tumor Antigen AF20 as Glycosylated Transferrin Receptor 1 in Complex with Heat Shock Protein 90 and/or Transporting ATPase. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Colorectal Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24186357&form=6&db=m Ouabain-induced alterations of the apical junctional complex involve ?1 and ?1 Na,K-ATPase downregulation and ERK1/2 activation independent of caveolae in colorectal cancer cells. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,1 7.2.2.13 Colorectal Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26858653&form=6&db=m Na,K-ATPase Isozymes in Colorectal Cancer and Liver Metastases. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Confusion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11147069&form=6&db=m [Molecular mechanisms of idiopathic nephrotic syndrome] ongoing research,unassigned 1,0 7.2.2.13 Contracture http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1726087&form=6&db=m Separate sites for the dantrolene-induced inhibition of contracture of the rat diaphragm preparation due to depolarization or to caffeine. therapeutic application,unassigned 1,0 7.2.2.13 Contracture http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2554666&form=6&db=m [Protective effects of m-nisoldipine and nisoldipine on myocardial damage in working rabbit hearts after ischemia-reperfusion] unassigned - 7.2.2.13 Contracture http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7264984&form=6&db=m Potential and tension changes induced by sodium removal in dog Purkinje fibres: role of an electrogenic sodium-calcium exchange. unassigned - 7.2.2.13 Contracture http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7900878&form=6&db=m Na+/H+ and Na+/Ca2+ exchange in regulation of [Na+]i and [Ca2+]i during metabolic inhibition. unassigned - 7.2.2.13 Contracture http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8243557&form=6&db=m Excitation-contraction uncoupling in the rat diaphragm by 2,3-butanedione monoxime. unassigned - 7.2.2.13 Contracture http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9281451&form=6&db=m Calcium induced contracture stimulates Na,K-pump rate in isolated sheep cardiac Purkinje fibers. therapeutic application,unassigned 1,0 7.2.2.13 Contracture http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11201989&form=6&db=m Pathogenesis and the role of Ca2+ overload during myocardial ischemia/reperfusion. unassigned - 7.2.2.13 Coronary Artery Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25426658&form=6&db=m Increased membrane lipid peroxidation and decreased Na+/K+-ATPase activity in erythrocytes of patients with stable coronary artery disease. diagnostic usage,ongoing research,unassigned 4,2,0 7.2.2.13 Coronary Occlusion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=130383&form=6&db=m Ischemia-induced alterations in myocardial (Na+ + K+)-ATPase and cardiac glycoside binding. diagnostic usage,ongoing research,therapeutic application,unassigned 3,1,1,0 7.2.2.13 Coronary Occlusion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2842510&form=6&db=m Alterations in heart membrane calcium transport during the development of ischemia-reperfusion injury. therapeutic application,unassigned 1,0 7.2.2.13 COVID-19 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33689516&form=6&db=m Molecular mechanisms of Na,K-ATPase dysregulation driving alveolar epithelial barrier failure in severe COVID-19. causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,1,0 7.2.2.13 Craniocerebral Trauma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29265087&form=6&db=m [An influence of cytoflavin on oxidative stress and activity of Na/K-ATPase of erythrocytes after brain trauma]. ongoing research,unassigned 3,0 7.2.2.13 Crohn Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2556782&form=6&db=m Na,K-ATPase activity in rectal mucosa of children with ulcerative colitis and Crohn's disease. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Crohn Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6816156&form=6&db=m Total body potassium depletion and the need for preoperative nutritional support in Chrohn's disease. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,1,0 7.2.2.13 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1648593&form=6&db=m Endogenous natriuretic factors: atrial natriuretic hormone and digitalis-like substance in Cushing's syndrome. diagnostic usage,ongoing research,therapeutic application,unassigned 2,2,1,0 7.2.2.13 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2996769&form=6&db=m Na-K pump activity in erythrocytes of patients with endogenous and exogenous glucocorticoid excess. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,2,0 7.2.2.13 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6248683&form=6&db=m Increased activity of the Na-K-ATPase in red cell-ghosts of patients with Cushing's syndrome: possible significance for the pathogenesis of glucocorticoid-induced hypertension. causal interaction,diagnostic usage,unassigned 3,1,0 7.2.2.13 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6256112&form=6&db=m Sodium-potassium-dependent ATPase activity in erythrocyte ghosts of patients with primary and secondary hypertension. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,2,0 7.2.2.13 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271508&form=6&db=m Na-K-ATPase in erythrocyte ghosts is not a marker for primary hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,1,0 7.2.2.13 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26743443&form=6&db=m GNAS mutations in adrenal aldosterone-producing adenomas [Rapid Communication]. causal interaction,unassigned 3,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=214742&form=6&db=m Calcium and sodium transport processes in patients with cystic fibrosis. I. A specific decrease in Mg2+-dependent, Ca2+-adenosine triphosphatase activity in erythrocyte membranes from cystic fibrosis patients. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6091022&form=6&db=m Calcium-ATPase activity in cystic fibrosis erythrocyte membranes: decreased activity in patients with pancreatic insufficiency. causal interaction,diagnostic usage,ongoing research,unassigned 2,2,3,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6947278&form=6&db=m Na and K dependence of the Na/K pump in cystic fibrosis fibroblasts. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,4,1 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8816777&form=6&db=m Immunolocalization of ion transport proteins in human autosomal dominant polycystic kidney epithelial cells. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9125202&form=6&db=m Na+/K+ ATPase in lower airway epithelium from cystic fibrosis and non-cystic-fibrosis lung. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11316265&form=6&db=m Differences in the protein-kinase-A-dependent regulation of CFTR Cl- channels and Na+-K+ pumps in guinea-pig ventricular myocytes. therapeutic application,unassigned 1,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11976939&form=6&db=m Dual regulation of cardiac Na+-K+ pumps and CFTR Cl- channels by protein kinases A and C. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12087129&form=6&db=m Lung epithelial fluid transport and the resolution of pulmonary edema. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12504282&form=6&db=m Identification of the cofilin-binding sites in the large cytoplasmic domain of Na,K-ATPase. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16793006&form=6&db=m Characterization of ion channel and transporter mRNA expressions in isolated gill chloride and pavement cells of seawater acclimating eels. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17826821&form=6&db=m Simulation analysis of intracellular Na+ and Cl- homeostasis during beta 1-adrenergic stimulation of cardiac myocyte. ongoing research,unassigned 2,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19111440&form=6&db=m Vocal Fold Surface Hydration: A Review. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22923315&form=6&db=m Lubiprostone Targets Prostanoid Signaling and Promotes Ion Transporter Trafficking, Mucus Exocytosis, and Contractility. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23319161&form=6&db=m Effects of potassium ion supplementation on survival and ion regulation in Gulf killifish Fundulus grandis larvae reared in ion deficient saline waters. diagnostic usage,unassigned 1,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24588076&form=6&db=m Knockout mice reveal key roles for claudin 18 in alveolar barrier properties and fluid homeostasis. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26245938&form=6&db=m Characterization of oncocytes in deep esophageal glands. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28458673&form=6&db=m Inflammatory Responses Regulating Alveolar Ion Transport during Pulmonary Infections. ongoing research,unassigned 2,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29664060&form=6&db=m Specialized Pro-resolving Mediators Regulate Alveolar Fluid Clearance during Acute Respiratory Distress Syndrome. causal interaction,unassigned 1,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31072021&form=6&db=m Azaspiracids Increase Mitochondrial Dehydrogenases Activity in Hepatocytes: Involvement of Potassium and Chloride Ions. unassigned - 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31344409&form=6&db=m Ursodeoxycholic acid inhibits ENaC and Na/K pump activity to restore airway surface liquid height in cystic fibrosis bronchial epithelial cells. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31493553&form=6&db=m Enhanced osmoregulatory ability marks the smoltification period in developing chum salmon (Oncorhynchus keta). diagnostic usage,unassigned 2,0 7.2.2.13 Cystic Fibrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31649543&form=6&db=m Plumbagin Prevents Secretory Diarrhea by Inhibiting CaCC and CFTR Channel Activities. ongoing research,therapeutic application,unassigned 3,2,0 7.2.2.13 Cystinosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16439594&form=6&db=m Decreased intracellular ATP content and intact mitochondrial energy generating capacity in human cystinotic fibroblasts. causal interaction,unassigned 4,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1327261&form=6&db=m Sequential tubular cell and basement membrane changes in polycystic kidney disease. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2557482&form=6&db=m In vitro modulation of tubular cyst regression in murine polycystic kidney disease. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3001068&form=6&db=m Regulation of Na,K-ATPase biosynthesis in developing Artemia salina. diagnostic usage,unassigned 3,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3029256&form=6&db=m Triiodothyronine-induced cyst formation in metanephric organ culture: the role of increased Na-K-adenosine triphosphatase activity. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,4,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7495297&form=6&db=m Analysis of the role of membrane polarity in polycystic kidney disease of transgenic SBM mice. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7622841&form=6&db=m Renal tubule Na,K-ATPase polarity in different animal models of polycystic kidney disease. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7919157&form=6&db=m Sodium pump distribution is not reversed in the DBA/2FG-pcy, polycystic kidney disease model mouse. ongoing research,therapeutic application,unassigned 4,2,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8196361&form=6&db=m Cell polarity in human renal cystic disease. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,3,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8383715&form=6&db=m Renal tubule Na,K-ATPase polarity in glucocorticoid-induced polycystic kidney disease. causal interaction,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8816777&form=6&db=m Immunolocalization of ion transport proteins in human autosomal dominant polycystic kidney epithelial cells. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9020888&form=6&db=m Polymorphism and structure of the gene coding for the alpha 1 subunit of the Artemia franciscana Na/K-ATPase. causal interaction,unassigned 2,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10450015&form=6&db=m Cystic fibrosis transmembrane conductance regulator in the kidney: clues to its role? causal interaction,unassigned 4,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12660316&form=6&db=m Na transport in autosomal recessive polycystic kidney disease (ARPKD) cyst lining epithelial cells. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12851251&form=6&db=m Histopathological analysis of renal cystic epithelia in the Pkd2WS25/- mouse model of ADPKD. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15075242&form=6&db=m Both mitogen activated protein kinase and the mammalian target of rapamycin modulate the development of functional renal proximal tubules in matrigel. unassigned - 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20926452&form=6&db=m The {beta}1 subunit of the Na,K-ATPase pump interacts with megalencephalic leucoencephalopathy with subcortical cysts protein 1 (MLC1) in brain astrocytes: new insights into MLC pathogenesis. unassigned - 7.2.2.13 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26289599&form=6&db=m Ouabain Regulates CFTR-Mediated Anion Secretion and Na,K-ATPase Transport in ADPKD Cells. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Deafness http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12062767&form=6&db=m Late-onset hearing loss in a mouse model of DFN3 non-syndromic deafness: morphologic and immunohistochemical analyses. unassigned - 7.2.2.13 Deafness http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16740909&form=6&db=m CRYM mutations cause deafness through thyroid hormone binding properties in the fibrocytes of the cochlea. causal interaction,unassigned 4,0 7.2.2.13 Death, Sudden, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11828798&form=6&db=m [Autoantibodies against sarcolemmal Na-K-ATPase in patients with dilated cardiomyopathy: autoimmune basis for ventricular arrhythmias in patients with congestive heart failure] causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Death, Sudden, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12354443&form=6&db=m Autoantibodies produced against sarcolemmal Na-K-ATPase: possible upstream targets of arrhythmias and sudden death in patients with dilated cardiomyopathy. causal interaction,ongoing research,unassigned 2,1,0 7.2.2.13 Death, Sudden, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19246813&form=6&db=m Autoimmune mechanisms underlying dilated cardiomyopathy. causal interaction,unassigned 1,0 7.2.2.13 Death, Sudden, Cardiac http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21617318&form=6&db=m Contribution of acquired factors to the pathogenesis of dilated cardiomyopathy. -The cause of dilated cardiomyopathy: genetic or acquired? (Acquired-Side)-. unassigned - 7.2.2.13 Dehydration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2824597&form=6&db=m Brain water and aging. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Dehydration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3034977&form=6&db=m Sodium-potassium pump, ion fluxes, and cellular dehydration in sickle cell anemia. ongoing research,unassigned 4,0 7.2.2.13 Dehydration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10450017&form=6&db=m Acute and chronic effects of hyperosmolality on mRNA and protein expression and the activity of Na-K-ATPase in the IMCD. unassigned - 7.2.2.13 Dehydration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15574255&form=6&db=m [Congenital xerocytosis] causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Dehydration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31691615&form=6&db=m The Na/Ca Exchange as a Target for Antitumor Effect of 4Hz Pulsing Magnetic Field. causal interaction,unassigned 4,0 7.2.2.13 Dehydration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31719660&form=6&db=m Distribution of aquaporins and sodium transporters in the gastrointestinal tract of a desert hare, Lepus yarkandensis. causal interaction,unassigned 1,0 7.2.2.13 Dehydration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33586567&form=6&db=m The 4Hz mechanical vibration-activated Na/Ca exchange as a quantum-sensitive novel target for pain therapy. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Diabetes Complications http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2824260&form=6&db=m Na+-K+-ATPase pumping activity is not directly linked to myo-inositol levels after sorbinil treatment in lenses of diabetic rats. causal interaction,unassigned 3,0 7.2.2.13 Diabetes Complications http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2834250&form=6&db=m Glomerular Na+-K+-ATPase activity in acute and chronic diabetes and with aldose reductase inhibition. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Diabetes Complications http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2838351&form=6&db=m Are disturbances of sorbitol, phosphoinositide, and Na+-K+-ATPase regulation involved in pathogenesis of diabetic neuropathy? unassigned - 7.2.2.13 Diabetes Complications http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2995180&form=6&db=m Reduced glomerular sodium/potassium adenosine triphosphatase activity in acute streptozocin diabetes and its prevention by oral sorbinil. causal interaction,therapeutic application,unassigned 4,3,0 7.2.2.13 Diabetes Complications http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14515013&form=6&db=m The relationship between red blood cell Na/K-ATPase activities and diabetic complications in patients with type 2 diabetes mellitus. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,4,0 7.2.2.13 Diabetes Complications http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17908327&form=6&db=m Association between erythrocyte Na+K+-ATPase activity and some blood lipids in type 1 diabetic patients from Lagos, Nigeria. unassigned - 7.2.2.13 Diabetes Insipidus, Nephrogenic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15153756&form=6&db=m Changes of rat kidney AQP2 and Na,K-ATPase mRNA expression in lithium-induced nephrogenic diabetes insipidus. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,3,1 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1645862&form=6&db=m Proximal tubular cell sodium concentration in early diabetic nephropathy assessed by electron microprobe analysis. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3004092&form=6&db=m Evaluation of sodium and potassium pump activity and number in diabetic erythrocytes. unassigned - 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6089093&form=6&db=m The effect of streptozotocin-induced diabetes mellitus on urinary excretion of sodium and renal Na+-K+-ATPase activity. diagnostic usage,ongoing research,unassigned 1,3,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11325071&form=6&db=m Mechanism of sodium load-induced hypertension in non-insulin dependent diabetes mellitus model rats: defective dopaminergic system to inhibit Na-K-ATPase activity in renal epithelial cells. ongoing research,therapeutic application,unassigned 3,2,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11791942&form=6&db=m Modulation of Na,K-ATPase activity by immunoglobulins--II. Na,K-ATPase activity in erythrocytes from patients with various chronic disturbances. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11900385&form=6&db=m Regulation of myocardial and skeletal muscle Na,K-ATPase in diabetes mellitus in humans and animals. causal interaction,unassigned 1,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14515013&form=6&db=m The relationship between red blood cell Na/K-ATPase activities and diabetic complications in patients with type 2 diabetes mellitus. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,4,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14690302&form=6&db=m Relation of erythrocyte Na+-K+ ATPase activity and cholesterol and oxidative stress in patients with type 2 diabetes mellitus. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,3,2 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15198371&form=6&db=m Effects of C-peptide on microvascular blood flow and blood hemorheology. unassigned - 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15970492&form=6&db=m Endogenous digitalis-like ligands and Na/K-ATPase inhibition in experimental diabetes mellitus. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16179401&form=6&db=m Effects of sprint training on extrarenal potassium regulation with intense exercise in Type 1 diabetes. ongoing research,unassigned 4,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16405108&form=6&db=m Marinobufagenin, an endogenous inhibitor of alpha-1 Na/K-ATPase, is a novel factor in pathogenesis of diabetes mellitus. causal interaction,unassigned 4,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17908327&form=6&db=m Association between erythrocyte Na+K+-ATPase activity and some blood lipids in type 1 diabetic patients from Lagos, Nigeria. unassigned - 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17942287&form=6&db=m Endogenous sodium pump inhibitors, diabetes mellitus and preeclampsia Preliminary observations and a hypothesis. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19390135&form=6&db=m Acute diabetes mellitus and its influence on renal Na,K-ATPase in both genders. causal interaction,ongoing research,unassigned 4,3,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27643939&form=6&db=m The role of red blood cell deformability and Na,K-ATPase function in selected risk factors of cardiovascular diseases in humans: focus on hypertension, diabetes mellitus and hypercholesterolemia. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,3,1 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31523248&form=6&db=m Association between a novel G94A single nucleotide polymorphism in ATP1A1 gene and type 2 diabetes mellitus among Egyptian patients. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,1,0 7.2.2.13 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=100194929&form=6&db=m Dietary lipid modulation of Na+/glucose co-transporter (SGLT1), Na+/K+ ATPase, and ornithine decarboxylase gene expression in the rat small intestine in diabetes mellitus. diagnostic usage,ongoing research,unassigned 1,3,0 7.2.2.13 Diabetes Mellitus, Experimental http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=212907&form=6&db=m [Effect of alloxan diabetes on (Na+ + K+)-activated ATPase in brush border membrane of the mucosal cell of rat small intestine] causal interaction,ongoing research,unassigned 4,4,0 7.2.2.13 Diabetes Mellitus, Experimental http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3006770&form=6&db=m Insulin stimulates renal glomerular sodium-potassium adenosine triphosphatase activity. causal interaction,therapeutic application,unassigned 4,2,0 7.2.2.13 Diabetes Mellitus, Experimental http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6303025&form=6&db=m [Increase of (Na+ + K+)-activated ATPase activity of basolateral plasma membranes from intestinal mucosa of diabetic rats] diagnostic usage,unassigned 1,0 7.2.2.13 Diabetes Mellitus, Experimental http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10063931&form=6&db=m Small intestinal Na+,K+-adenosine triphosphatase activity and gene expression in experimental diabetes mellitus. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Diabetes Mellitus, Experimental http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15970492&form=6&db=m Endogenous digitalis-like ligands and Na/K-ATPase inhibition in experimental diabetes mellitus. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1327887&form=6&db=m Hypothesis: low Na/K-ATPase activity in the red cell membrane, a potential marker of the predisposition to diabetic neuropathy. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,3,1 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2544470&form=6&db=m Membrane lipid alterations and Na+-pumping activity in erythrocytes from IDDM and NIDDM subjects. unassigned - 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8021905&form=6&db=m Erythrocyte cation transport systems in insulin-dependent diabetics: correlation with prorenin and albuminuria. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,2,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8769346&form=6&db=m Changes in Na,K-adenosine triphosphatase (ATPase) concentration and Na,K-ATPase-dependent adenosine triphosphate turnover in human erythrocytes in diabetes. diagnostic usage,ongoing research,unassigned 4,3,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9312166&form=6&db=m Glucose-specific regulation of aldose reductase in capan-1 human pancreatic duct cells In vitro. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9392498&form=6&db=m Altered platelet membrane dynamic properties in type 1 diabetes. diagnostic usage,ongoing research,unassigned 4,4,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9498637&form=6&db=m Decreased nitric oxide synthase activity in platelets from IDDM and NIDDM patients. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9748739&form=6&db=m [Erythrocyte membrane sodium transport in patients with type I diabetes mellitus (insulin-dependent diabetes mellitus)] causal interaction,unassigned 3,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16179401&form=6&db=m Effects of sprint training on extrarenal potassium regulation with intense exercise in Type 1 diabetes. ongoing research,unassigned 4,0 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17908327&form=6&db=m Association between erythrocyte Na+K+-ATPase activity and some blood lipids in type 1 diabetic patients from Lagos, Nigeria. unassigned - 7.2.2.13 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22162761&form=6&db=m C-peptide increases Na,K-ATPase expression via PKC- and MAP kinase-dependent activation of transcription factor ZEB in human renal tubular cells. causal interaction,unassigned 3,0 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2544470&form=6&db=m Membrane lipid alterations and Na+-pumping activity in erythrocytes from IDDM and NIDDM subjects. unassigned - 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8769346&form=6&db=m Changes in Na,K-adenosine triphosphatase (ATPase) concentration and Na,K-ATPase-dependent adenosine triphosphate turnover in human erythrocytes in diabetes. diagnostic usage,ongoing research,unassigned 4,3,0 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9498637&form=6&db=m Decreased nitric oxide synthase activity in platelets from IDDM and NIDDM patients. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14515013&form=6&db=m The relationship between red blood cell Na/K-ATPase activities and diabetic complications in patients with type 2 diabetes mellitus. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,4,0 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14685860&form=6&db=m Effect of resistance training on Na,K pump and Na+/H+ exchange protein densities in muscle from control and patients with type 2 diabetes. causal interaction,ongoing research,therapeutic application,unassigned 4,4,1,0 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14690302&form=6&db=m Relation of erythrocyte Na+-K+ ATPase activity and cholesterol and oxidative stress in patients with type 2 diabetes mellitus. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,3,2 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16317524&form=6&db=m Association of hypoglutathionemia with reduced Na+/K+ ATPase activity in type 2 diabetes and microangiopathy. causal interaction,unassigned 2,0 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31523248&form=6&db=m Association between a novel G94A single nucleotide polymorphism in ATP1A1 gene and type 2 diabetes mellitus among Egyptian patients. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,1,0 7.2.2.13 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33682297&form=6&db=m Impact of the metabolic syndrome on peripheral nerve structure and function in type 2 diabetes. causal interaction,unassigned 1,0 7.2.2.13 Diabetic Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9165216&form=6&db=m Alteration of Na,K-ATPase isoenzymes in diabetic cardiomyopathy: effect of dietary supplementation with fish oil (n-3 fatty acids) in rats. diagnostic usage,unassigned 1,0 7.2.2.13 Diabetic Cardiomyopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26179604&form=6&db=m ?3-Adrenoceptor, glutathionylation, and diabetic cardiomyopathy. Focus on "?3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade". causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Diabetic Nephropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2995180&form=6&db=m Reduced glomerular sodium/potassium adenosine triphosphatase activity in acute streptozocin diabetes and its prevention by oral sorbinil. causal interaction,therapeutic application,unassigned 4,3,0 7.2.2.13 Diabetic Nephropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3007926&form=6&db=m Aldose reductase, glomerular metabolism, and diabetic nephropathy. causal interaction,diagnostic usage,ongoing research,therapeutic application 2,3,2,2 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1327887&form=6&db=m Hypothesis: low Na/K-ATPase activity in the red cell membrane, a potential marker of the predisposition to diabetic neuropathy. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,3,1 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2537578&form=6&db=m In vitro correction of impaired Na+-K+-ATPase in diabetic nerve by protein kinase C agonists. causal interaction,unassigned 2,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2828823&form=6&db=m The pathogenesis and prevention of diabetic neuropathy and nephropathy. causal interaction,unassigned 2,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2956143&form=6&db=m Sciatic nerve ATPase activity is unaffected in diabetic mutant C57Bl/Ks (db/db) mice. causal interaction,unassigned 3,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3019809&form=6&db=m Glutathione redox state is not the link between polyol pathway activity and myo-inositol-related Na+-K+-ATPase defect in experimental diabetic neuropathy. unassigned - 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3307667&form=6&db=m Effect of gangliosides on nerve conduction velocity during diabetic neuropathy in the rat. unassigned - 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7589876&form=6&db=m Effect of cilostazol on experimental diabetic neuropathy in the rat. ongoing research,therapeutic application,unassigned 3,4,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8001715&form=6&db=m Effect of experimental diabetes on Na/K-ATPase activity in red blood cells, peripheral nerve and kidney. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8733942&form=6&db=m NA+/K+ ATPase impairment and experimental glycation: the role of glucose autoxidation. therapeutic application,unassigned 1,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9681464&form=6&db=m Alterations of Na,K-ATPase isoenzymes in the rat diabetic neuropathy: protective effect of dietary supplementation with n-3 fatty acids. causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9915901&form=6&db=m Fish oil supplementation prevents diabetes-induced nerve conduction velocity and neuroanatomical changes in rats. causal interaction,unassigned 3,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10377817&form=6&db=m [New trend in pathogenesis of diabetic neuropathy] causal interaction,diagnostic usage,unassigned 3,3,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10428069&form=6&db=m Localization of Na,K-ATPase alpha/beta isoforms in rat sciatic nerves: effect of diabetes and fish oil treatment. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11887161&form=6&db=m Genetic and environmental regulation of Na/K adenosine triphosphatase activity in diabetic patients. causal interaction,unassigned 3,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15758031&form=6&db=m Altered nerve excitability properties in established diabetic neuropathy. unassigned - 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18362098&form=6&db=m Activity-dependent excitability changes suggest Na+/K+ pump dysfunction in diabetic neuropathy. causal interaction,unassigned 2,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20043035&form=6&db=m Alpha-lipoic Acid and diabetic neuropathy. causal interaction,unassigned 3,0 7.2.2.13 Diabetic Neuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31523248&form=6&db=m Association between a novel G94A single nucleotide polymorphism in ATP1A1 gene and type 2 diabetes mellitus among Egyptian patients. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,1,0 7.2.2.13 Down Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Down Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12745628&form=6&db=m Cerebral chemical dominance and neural regulation of cell division, cell proliferation, neoplastic transformation, and genomic function. diagnostic usage,unassigned 4,0 7.2.2.13 Dyslipidemias http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16846594&form=6&db=m Green tea impedes dyslipidemia, lipid peroxidation, protein glycation and ameliorates Ca2+ -ATPase and Na+/K+ -ATPase activity in the heart of streptozotocin-diabetic rats. unassigned - 7.2.2.13 Dyslipidemias http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32447998&form=6&db=m Allopurinol and valproic acid improve cardiac triglyceride and Na+-K+-ATPase activity independent of circulating aldosterone in female rats with glucose intolerance. causal interaction,ongoing research,therapeutic application,unassigned 1,3,1,0 7.2.2.13 Dystocia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8929628&form=6&db=m X-ray microanalysis of myometrium in parturient women at term. causal interaction,diagnostic usage,unassigned 1,3,0 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15260948&form=6&db=m Paying the price at the pump: dystonia from mutations in a Na+/K+ -ATPase. unassigned - 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15260953&form=6&db=m Mutations in the Na+/K+ -ATPase alpha3 gene ATP1A3 are associated with rapid-onset dystonia parkinsonism. therapeutic application,unassigned 1,0 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17234593&form=6&db=m Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice. causal interaction,unassigned 4,0 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21165980&form=6&db=m Distribution of Na/K-ATPase alpha 3 isoform, a sodium-potassium P-type pump associated with rapid-onset of dystonia parkinsonism (RDP) in the adult mouse brain. unassigned - 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23652595&form=6&db=m Enhanced inhibitory neurotransmission in the cerebellar cortex of Atp1a3-deficient heterozygous mice. therapeutic application,unassigned 1,0 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24983657&form=6&db=m Heterozygous mice deficient in Atp1a3 exhibit motor deficits by chronic restraint stress. therapeutic application,unassigned 1,0 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28472154&form=6&db=m Hypothermia-induced dystonia and abnormal cerebellar activity in a mouse model with a single disease-mutation in the sodium-potassium pump. causal interaction,unassigned 1,0 7.2.2.13 Dystonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28576588&form=6&db=m [Dystonia 12: A rare and difficult diagnosis]. causal interaction,unassigned 1,0 7.2.2.13 Encephalomyelitis, Autoimmune, Experimental http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=72443&form=6&db=m [Na+-K+-ATPase activity in guinea pig brain synaptosomal fraction in experimental allergic encephalomyelitis] unassigned - 7.2.2.13 Endolymphatic Hydrops http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8075476&form=6&db=m Na-K-ATPase activity in the guinea pig stria vascularis in experimentally-induced endolymphatic hydrops. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Endolymphatic Hydrops http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8534024&form=6&db=m Na,K-ATPase in the cochlear lateral wall of human temporal bones with endolymphatic hydrops. ongoing research,unassigned 2,0 7.2.2.13 Endotoxemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12446281&form=6&db=m Endotoxemia stimulates skeletal muscle Na+-K+-ATPase and raises blood lactate under aerobic conditions in humans. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Endotoxemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22351078&form=6&db=m Inhibition of Na/K-ATPase promotes myocardial tumor necrosis factor-alpha protein expression and cardiac dysfunction via calcium/mTOR signaling in endotoxemia. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Enteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17290012&form=6&db=m Neutral Na-amino acid cotransport is differentially regulated by glucocorticoids in the normal and chronically inflamed rabbit small intestine. causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Enteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33805551&form=6&db=m Mechanism of Na-K-ATPase Inhibition by PGE2 in Intestinal Epithelial Cells. causal interaction,unassigned 4,0 7.2.2.13 Enteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34299188&form=6&db=m Molecular Mechanism of Stimulation of Na-K-ATPase by Leukotriene D4 in Intestinal Epithelial Cells. causal interaction,unassigned 4,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6095737&form=6&db=m Glial and neuronal Na+-K+ pump in epilepsy. causal interaction,unassigned 2,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6100944&form=6&db=m Epilepsy and membrane Na+K+ATPase: changes in activity using an experimental model of epilepsy. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6246771&form=6&db=m Brain synaptosomes in epilepsy: organization of ionic channels and the Na+-K+ pump. unassigned - 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8013444&form=6&db=m Na,K-ATPase is decreased in hippocampus of kainate-lesioned rats. causal interaction,unassigned 3,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14571006&form=6&db=m Changes in the isoprenoid pathway with transcendental meditation and Reiki healing practices in seizure disorder. unassigned - 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15308625&form=6&db=m Kinetic alterations due to a missense mutation in the Na,K-ATPase alpha2 subunit cause familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16037212&form=6&db=m Alterations in the alpha2 isoform of Na,K-ATPase associated with familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18191908&form=6&db=m Hypothesis on neurophysiopathological mechanisms linking epilepsy and headache. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20493076&form=6&db=m The Na,K-ATPase of nervous tissue. causal interaction,unassigned 4,0 7.2.2.13 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21731499&form=6&db=m Increased susceptibility to cortical spreading depression in the mouse model of familial hemiplegic migraine type 2. causal interaction,unassigned 3,0 7.2.2.13 Epilepsy, Absence http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7859671&form=6&db=m Abnormal cortical unit activity of the reticular formation. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Epilepsy, Generalized http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Epilepsy, Temporal Lobe http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8013444&form=6&db=m Na,K-ATPase is decreased in hippocampus of kainate-lesioned rats. causal interaction,unassigned 3,0 7.2.2.13 Esophageal Squamous Cell Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27845894&form=6&db=m Overexpression of ATPase Na+/+ transporting alpha 1 polypeptide, ATP1A1, correlates with clinical diagnosis and progression of esophageal squamous cell carcinoma. causal interaction,diagnostic usage,unassigned 4,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=146188&form=6&db=m Altered sodium permeability, calcium binding and Na-K-ATPase activity in the red blood cell membrane in essential hypertension. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1326790&form=6&db=m [Effects of saline infusion on the activity of erythrocyte sodium-potassium pump in normal subjects and in patients with essential hypertension] diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2157465&form=6&db=m The role of Na,K-ATPase inhibitor on pressor responsiveness in patients with benign essential hypertension. causal interaction,ongoing research,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2176810&form=6&db=m Diuretics and cation transport in hypertensive blacks. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2196126&form=6&db=m Increased activity of digoxin-like substance in low-renin hypertension in acromegaly. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,2,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2439745&form=6&db=m Sodium pump inhibitor in the serum of patients with essential hypertension and its partial purification from hemofiltrate. causal interaction,diagnostic usage,unassigned 3,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2440805&form=6&db=m Kinetic analysis of erythrocyte Na+-K+ pump and cotransport in essential hypertension. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2441015&form=6&db=m Measurement of circulating sodium-pump inhibitory activity in uraemia and essential hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2445677&form=6&db=m Kinetic aspects of red blood cell sodium transport systems in essential hypertension. therapeutic application,unassigned 1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2539926&form=6&db=m Digoxin-like immunoreactivity, displacement of ouabain and inhibition of Na+/K+ ATPase by four steroids known to be increased in essential hypertension. causal interaction,unassigned 4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2544279&form=6&db=m [A study on the relationship between reduced activity of red cell membrane sodium-potassium pump and essential hypertension] causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,1 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2554840&form=6&db=m [Therapeutic response to canrenone of patients with essential hypertension as a function of sodium transport anomalies and ouabain sensitivity of erythrocytes] ongoing research,unassigned 1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2667573&form=6&db=m Humoral Na+-K+ pump inhibitory activity in essential hypertension and in normotensive subjects after acute volume expansion. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,1,1 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2847190&form=6&db=m An Na+-K+-ATPase inhibitor which circulates in renal failure but not in essential hypertension. therapeutic application,unassigned 3,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3000655&form=6&db=m Abnormal relationship between dietary sodium intake and red cell sodium transport in salt-sensitive patients with essential hypertension. causal interaction,unassigned 3,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3003223&form=6&db=m Na-K-ATPase-inhibiting and glucose-6-phosphate dehydrogenase-stimulating activity of plasma and hypothalamus of the Okamoto spontaneously hypertensive rat. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3009961&form=6&db=m Blood pressure in essential hypertension correlates with the concentration of a circulating inhibitor of the sodium pump. diagnostic usage,ongoing research,therapeutic application,unassigned 2,4,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3012764&form=6&db=m Evidence for a circulating Na,K-pump inhibitor in essential hypertension. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3032788&form=6&db=m Red blood cell Na+,K+-ATPase in men with newly diagnosed or previously treated essential hypertension. causal interaction,ongoing research,therapeutic application,unassigned 3,4,3,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3033184&form=6&db=m Acute hypotensive effect of calcium antagonists and endogenous digitalis-like immunoreactivity in human essential hypertension. unassigned - 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3040305&form=6&db=m Some properties of erythrocyte Na+-K+-ATPase in essential hypertension. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,4,4 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3534847&form=6&db=m Effects of captopril and of other antihypertensive drugs on cell membrane ion transport--a preliminary report. ongoing research,therapeutic application,unassigned 3,2,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3599799&form=6&db=m Pattern of Na-K-ATPase inhibitors in plasma and urine of hypertensive patients: a preliminary report. unassigned - 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3599801&form=6&db=m The endogenous cytochemically assayable Na-K-ATPase inhibitor and its relation to hypertension. causal interaction,diagnostic usage,therapeutic application,unassigned 2,2,2,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4017267&form=6&db=m Observations on the "cascade" of Na-K-ATPase inhibitory and digoxin-like immunoreactive material in human urine: possible relevance to essential hypertension. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,2,1 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6086517&form=6&db=m Erythrocyte Na+,K+ cotransport and Na+,K+ pump in black and caucasian hypertensive patients. unassigned - 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6095682&form=6&db=m Hypothalamic sodium-transport inhibitor is a high-affinity reversible inhibitor of Na+-K+-ATPase. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6098238&form=6&db=m [Circulating inhibitor of the Na+-K+ pump in essential hypertension. Physiological and pharmacological variations] causal interaction,therapeutic application,unassigned 4,3,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6206353&form=6&db=m The role of a humoral Na+,K+-ATPase inhibitor in regulating precapillary vessel tone. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6213810&form=6&db=m [ATPase activity and sodium transport in erythrocytes of patients with essential hypertension (author's transl)] diagnostic usage,ongoing research,therapeutic application,unassigned 1,1,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271508&form=6&db=m Na-K-ATPase in erythrocyte ghosts is not a marker for primary hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271511&form=6&db=m Cation fluxes and (Na+ + K+)-activated ATPase activity in erythrocytes of patients with essential hypertension. therapeutic application,unassigned 1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6288322&form=6&db=m Raised concentration of a circulating Na+-K+-ATPase inhibitor in essential hypertension. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6303625&form=6&db=m Erythrocyte membrane sodium transport in patients with treated and untreated essential hypertension. diagnostic usage,ongoing research,unassigned 1,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6307047&form=6&db=m The role of a circulating inhibitor of Na+-K+-ATPase in essential hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6310538&form=6&db=m The relation of the natriuretic hormone to essential hypertension. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6326156&form=6&db=m Effects of rat atrial extract on sodium transport and blood pressure in the rat. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6328233&form=6&db=m Hormonal and nutritional enhancement of Na+-K+-ATPase activity may aid the prevention and treatment of essential hypertension. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6328636&form=6&db=m Effects of ouabain and potassium on rabbit arterial smooth muscle cells in culture. therapeutic application,unassigned 2,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6788127&form=6&db=m Familial abnormality of erythrocyte cation transport in essential hypertension. causal interaction,diagnostic usage,unassigned 4,2,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6826033&form=6&db=m Inhibition of the arteriolar smooth muscle NA+ -K+-pump induces an enhanced vasoconstriction in borderline but not in established essential hypertension. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9655151&form=6&db=m The role of Na,K-ATPase inhibitors in hypertension and end-stage renal disease. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9682918&form=6&db=m Simultaneous measurement of marinobufagenin, ouabain, and hypertension-associated protein in various disease states. unassigned - 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11509477&form=6&db=m Interaction of alpha(1)-Na,K-ATPase and Na,K,2Cl-cotransporter genes in human essential hypertension. causal interaction,unassigned 3,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11768735&form=6&db=m The change and significance of the Na+-K+-ATPase alpha-subunit in ouabain-hypertensive rats. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12642509&form=6&db=m Calcium antagonist treatment by lercanidipine prevents hyperpolarization in essential hypertension. unassigned - 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17446437&form=6&db=m Association of ATP1A1 and dear single-nucleotide polymorphism haplotypes with essential hypertension: sex-specific and haplotype-specific effects. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19199261&form=6&db=m [Association of the polymorphisms of sodium transport related genes with essential hypertension] causal interaction,diagnostic usage,unassigned 4,1,0 7.2.2.13 Exocrine Pancreatic Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6091022&form=6&db=m Calcium-ATPase activity in cystic fibrosis erythrocyte membranes: decreased activity in patients with pancreatic insufficiency. causal interaction,diagnostic usage,ongoing research,unassigned 2,2,3,0 7.2.2.13 Fanconi Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2159378&form=6&db=m Mutagenic response of Fanconi's anemia cells from a defined complementation group after treatment with photoactivated bifunctional psoralens. unassigned - 7.2.2.13 Fanconi Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=128760&form=6&db=m Inhibition of sodium intestinal transport and mucosal (Na+-K+)-ATPase in experimental Fanconi syndrome. ongoing research,therapeutic application,unassigned 2,3,0 7.2.2.13 Fanconi Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4249038&form=6&db=m Experimental Fanconi syndrome. I. Effect of maleic acid on renal cortical Na-K-ATPase activity and ATP levels. ongoing research,unassigned 4,0 7.2.2.13 Fanconi Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4278105&form=6&db=m Further studies on epithelial transport defect in experimental and human Fanconi syndrome. Effects of maleic acid and L-cystine on sodium transport and (Na+-K+)-ATPase of the isolated frog skin. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Fanconi Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7699953&form=6&db=m [A drug-induced transient Fanconi syndrome associated with pure red cell aplasia: pathophysiology of the electrolyte disorders] causal interaction,unassigned 3,0 7.2.2.13 Fanconi Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19106433&form=6&db=m Nephrotoxicity of cadmium & lead. causal interaction,unassigned 1,0 7.2.2.13 Fatigue Syndrome, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12745627&form=6&db=m Hypothalamic digoxin, cerebral chemical dominance and myalgic encephalomyelitis. diagnostic usage,unassigned 4,0 7.2.2.13 Feline Infectious Peritonitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25653449&form=6&db=m ATP1A1-mediated Src signaling inhibits coronavirus entry into host cells. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Fetal Growth Retardation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12788896&form=6&db=m Activity and protein expression of Na+/K+ ATPase are reduced in microvillous syncytiotrophoblast plasma membranes isolated from pregnancies complicated by intrauterine growth restriction. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Fetal Nutrition Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3013042&form=6&db=m Selective fetal malnutrition: effect of acute and chronic ethanol exposure upon rat placental Na,K-ATPase activity. causal interaction,ongoing research,unassigned 2,3,0 7.2.2.13 Gastroenteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3035940&form=6&db=m Absence of a cAMP-mediated antiabsorptive effect in an undifferentiated jejunal epithelium. diagnostic usage,unassigned 4,0 7.2.2.13 Gitelman Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30175537&form=6&db=m Renal Mg handling, FXYD2 and the central role of the Na,K-ATPase. causal interaction,unassigned 2,0 7.2.2.13 Glaucoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4227539&form=6&db=m [Digitalis glycosides as inhibitors of the Na-K-ATPase in glaucoma treatment] causal interaction,therapeutic application,unassigned 2,4,0 7.2.2.13 Glaucoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33859757&form=6&db=m Harnessing the tunable cavity of nanoceria for enhancing Y-27632-mediated alleviation of ocular hypertension. unassigned - 7.2.2.13 Glioblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12745628&form=6&db=m Cerebral chemical dominance and neural regulation of cell division, cell proliferation, neoplastic transformation, and genomic function. diagnostic usage,unassigned 4,0 7.2.2.13 Glioblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18300910&form=6&db=m Targeting the alpha 1 subunit of the sodium pump to combat glioblastoma cells. ongoing research,unassigned 3,0 7.2.2.13 Glioblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20689980&form=6&db=m The anticancer drug perillyl alcohol is a Na/K-ATPase inhibitor. ongoing research,unassigned 4,0 7.2.2.13 Glioblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29653366&form=6&db=m Bufalin inhibits glioblastoma growth by promoting proteasomal degradation of the Na+/K+-ATPase ?1 subunit. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,4,1 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2560348&form=6&db=m Determination of total (Na+ + K+)-ATPase activity of isolated or cultured cells. ongoing research,unassigned 4,0 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3034973&form=6&db=m (Na+ + K+)-ATPase in C6 glioma and rat cerebrum. diagnostic usage,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6289606&form=6&db=m Effects of methyl mercury and triethyllead on Na+K+ATPase and pyruvate dehydrogenase activities in glioma C6 cells. ongoing research,unassigned 4,0 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6289988&form=6&db=m Alterations of membrane integrity and cellular constituents by arachidonic acid in neuroblastoma and glioma cells. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7553734&form=6&db=m Mechanism of high K+ and Tl+ uptake in cultured human glioma cells. ongoing research,unassigned 1,0 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9278390&form=6&db=m Tissue-specific expression of the Na,K-ATPase beta3 subunit. The presence of beta3 in lung and liver addresses the problem of the missing subunit. ongoing research,unassigned 3,0 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30739221&form=6&db=m The sodium pump ?1 subunit regulates bufalin sensitivity of human glioblastoma cells through the p53 signaling pathway. causal interaction,ongoing research,therapeutic application,unassigned 4,3,4,0 7.2.2.13 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31114755&form=6&db=m ATP1A1 Integrates AKT and ERK Signaling via Potential Interaction With Src to Promote Growth and Survival in Glioma Stem Cells. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,4 7.2.2.13 Glucose Intolerance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32447998&form=6&db=m Allopurinol and valproic acid improve cardiac triglyceride and Na+-K+-ATPase activity independent of circulating aldosterone in female rats with glucose intolerance. causal interaction,ongoing research,therapeutic application,unassigned 1,3,1,0 7.2.2.13 Gout http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2820052&form=6&db=m Decreased NA+, K+-ATPase activity in erythrocyte membrane from rheumatoid arthritis patients. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,2,0 7.2.2.13 Hearing Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11718241&form=6&db=m Cochlear distribution of Na,K-ATPase and corticosteroid receptors in two mouse strains with congenital hearing disorders. unassigned - 7.2.2.13 Hearing Loss http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9438108&form=6&db=m Status of cochlear Na,K-ATPase in the aged SHR rat and its possible role in hearing loss. ongoing research,unassigned 3,0 7.2.2.13 Hearing Loss, Sensorineural http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21094218&form=6&db=m Simultaneously reduced NKCC1 and Na,K-ATPase expression in murine cochlear lateral wall contribute to conservation of endocochlear potential following a sensorineural hearing loss. therapeutic application,unassigned 1,0 7.2.2.13 Heart Arrest http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14649731&form=6&db=m Nitration of the striatal Na,K-ATPase alpha3 isoform occurs in normal brain development but is not increased during hypoxia-ischemia in newborn piglets. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2853742&form=6&db=m Correlations between plasma levels of an endogenous digitalis-like substance and haemodynamic parameters measured during cardiac catheterization. causal interaction,therapeutic application,unassigned 1,2,0 7.2.2.13 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8974067&form=6&db=m Human myocardial Na,K-ATPase concentration in heart failure. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 7.2.2.13 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9405890&form=6&db=m Regulation of myocardial Na,K-ATPase concentration in experimental and human heart disease. ongoing research,unassigned 4,0 7.2.2.13 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11687608&form=6&db=m Ouabain, a steroid hormone that signals with slow calcium oscillations. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15016405&form=6&db=m Interleukin-1 beta inhibits Na+-K+ ATPase activity and protein expression in cardiac myocytes. causal interaction,therapeutic application,unassigned 4,3,0 7.2.2.13 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19641704&form=6&db=m Myocardial Na,K-ATPase: Clinical aspects. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=130663&form=6&db=m Role of sarcolemmal changes in cardiac pathophysiology. diagnostic usage,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=136298&form=6&db=m Myocardial sarcolemmal ATPase in dogs with induced mitral insufficiency. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1323267&form=6&db=m Na,K-ATPase expression in normal and failing human left ventricle. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1334706&form=6&db=m [The effect of carnosine on the activity of Na,K,ATPase: prospective uses in clinical cardiology] diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1655269&form=6&db=m No upregulation of digitalis glycoside receptor (Na,K-ATPase) concentration in human heart left ventricle samples obtained at necropsy after long term digitalisation. causal interaction,unassigned 1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1849057&form=6&db=m Na,K-ATPase in isolated nephron segments in rats with experimental heart failure. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2154412&form=6&db=m The concentration of the Na,K-pump in skeletal and heart muscle in congestive heart failure. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,4,1 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2159446&form=6&db=m Intra-cellular electrolyte changes and levels of endogenous digoxin-like substance within the plasma in patients with congestive heart failure. causal interaction,diagnostic usage,unassigned 4,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7550131&form=6&db=m Na,K-ATPase receptor subunits alpha 1, alpha 2 and alpha 3 mRNA in dilated cardiomyopathy. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7796444&form=6&db=m Digoxin affects potassium homeostasis during exercise in patients with heart failure. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8147825&form=6&db=m Reduced 3H-ouabain binding site (Na,K-ATPase) concentration in ventricular myocardium of dogs with tachycardia induced heart failure. causal interaction,unassigned 2,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8281663&form=6&db=m Isoform-specific regulation of myocardial Na,K-ATPase alpha-subunit in congestive heart failure. Role of norepinephrine. unassigned - 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8285248&form=6&db=m Reductions of myocardial Na-K-ATPase activity and ouabain binding sites in heart failure: prevention by nadolol. causal interaction,diagnostic usage,unassigned 1,3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8974067&form=6&db=m Human myocardial Na,K-ATPase concentration in heart failure. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9408736&form=6&db=m Human myocardial and skeletal muscular Na,K-ATPase in relation to digoxin therapy of heart failure. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,4 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10069797&form=6&db=m Digitalis. therapeutic application,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10954051&form=6&db=m Changes in the expression of cardiac Na+-K+ ATPase subunits in the UM-X7.1 cardiomyopathic hamster. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11354998&form=6&db=m Defective activity and isoform of the Na,K-ATPase in the dilated cardiomyopathic hamster. diagnostic usage,ongoing research,therapeutic application,unassigned 1,4,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11354999&form=6&db=m RT-PCR detection of the Na,K-ATPase beta3-isoform in human heart. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11355000&form=6&db=m Expression of functional Na,K-ATPase isozymes in normal human cardiac biopsies. unassigned - 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11828798&form=6&db=m [Autoantibodies against sarcolemmal Na-K-ATPase in patients with dilated cardiomyopathy: autoimmune basis for ventricular arrhythmias in patients with congestive heart failure] causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12034663&form=6&db=m Intracellular Na(+) concentration is elevated in heart failure but Na/K pump function is unchanged. causal interaction,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12176120&form=6&db=m Myocardial Na,K-ATPase: the molecular basis for the hemodynamic effect of digoxin therapy in congestive heart failure. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12463096&form=6&db=m [Autoantibodies: new upstream targets of paroxysmal atrial fibrillation in patients with congestive heart failure] causal interaction,ongoing research,therapeutic application,unassigned 3,1,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12479230&form=6&db=m Sodium pump isoform expression in heart failure: implication for treatment. causal interaction,ongoing research,therapeutic application,unassigned 3,2,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12562669&form=6&db=m Training-induced changes in skeletal muscle Na+-K+ pump number and isoform expression in rats with chronic heart failure. causal interaction,ongoing research,therapeutic application,unassigned 4,2,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12623296&form=6&db=m Intracellular Na+ and altered Na+ transport mechanisms in cardiac hypertrophy and failure. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,4,3 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12763921&form=6&db=m Myocardial Na,K-ATPase and digoxin therapy in human heart failure. causal interaction,ongoing research,therapeutic application,unassigned 3,1,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15644955&form=6&db=m Influence of long-term treatment of imidapril on mortality, cardiac function, and gene expression in congestive heart failure due to myocardial infarction. causal interaction,ongoing research,therapeutic application,unassigned 1,2,3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15681692&form=6&db=m Modification of sarcolemmal Na+-K+-ATPase and Na+/Ca2+ exchanger expression in heart failure by blockade of renin-angiotensin system. therapeutic application,unassigned 2,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17333908&form=6&db=m [Changes of sarcolemma Na+/K+ ATPase and sarcoplasmic reticulum membrane Ca2+ ATPase activity after stem cell transplantation in chronic heart failure] causal interaction,ongoing research,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18052902&form=6&db=m "Don't flog the heart!" - development of specific drug therapies for heart failure. causal interaction,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19238540&form=6&db=m Istaroxime, a first in class new chemical entity exhibiting SERCA-2 activation and Na-K-ATPase inhibition: a new promising treatment for acute heart failure syndromes? therapeutic application,unassigned 4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19641704&form=6&db=m Myocardial Na,K-ATPase: Clinical aspects. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19705338&form=6&db=m The use of beta3-adrenergic receptor agonists in the treatment of heart failure. causal interaction,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19778013&form=6&db=m Interaction between Cardiotonic Steroids and Na,K-ATPase. Effects of pH and Ouabain-Induced Changes in Enzyme Conformation. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20952666&form=6&db=m Mice expressing ouabain-sensitive {alpha}1 Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophy. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21708939&form=6&db=m Metal Fluoride Complexes of Na,K-ATPase: CHARACTERIZATION OF FLUORIDE-STABILIZED PHOSPHOENZYME ANALOGUES AND THEIR INTERACTION WITH CARDIOTONIC STEROIDS. therapeutic application,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22451662&form=6&db=m Reduction of Na/K-ATPase potentiates marinobufagenin-induced cardiac dysfunction and myocyte apoptosis. causal interaction,unassigned 4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22546087&form=6&db=m The H?-H? domain of the ?? isoform of Na+-K+-ATPase is involved in ouabain toxicity in rat ventricular myocytes. therapeutic application,unassigned 2,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22595010&form=6&db=m Modulators of Na/K-ATPase: a patent review. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22695512&form=6&db=m Voltage-dependent K+ channels as oncotargets in malignant gliomas. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23341448&form=6&db=m A Structural View on the Functional Importance of the Sugar Moiety and Steroid Hydroxyls of Cardiotonic Steroids in Binding to Na,K-ATPase. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23549264&form=6&db=m Digoxin downregulates NDRG1 and VEGF through the inhibition of HIF-1? under hypoxic conditions in human lung adenocarcinoma A549 cells. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23674099&form=6&db=m Na/K pump regulation of cardiac repolarization: insights from a systems biology approach. causal interaction,therapeutic application,unassigned 2,2,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24630927&form=6&db=m Cardiac glycoside ouabain induces autophagic cell death in non-small cell lung cancer cells via a JNK-dependent decrease of Bcl-2. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24722410&form=6&db=m Inhibiting na+/k+ ATPase can impair mitochondrial energetics and induce abnormal Ca2+ cycling and automaticity in Guinea pig cardiomyocytes. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24793006&form=6&db=m Effects of Na/K-ATPase and its ligands on bone marrow stromal cell differentiation. causal interaction,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26708294&form=6&db=m Heterogeneous transport of digitalis-like compounds by P-glycoprotein in vesicular and cellular assays. therapeutic application,unassigned 3,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28176244&form=6&db=m Cytotoxic effects of the cardenolide convallatoxin and its Na,K-ATPase regulation. therapeutic application,unassigned 1,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29424237&form=6&db=m Greasing the wheels or a spanner in the works? Regulation of the cardiac sodium pump by palmitoylation. unassigned - 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30568725&form=6&db=m Cardiac function evaluation for a novel one-step detoxification product of Aconiti Lateralis Radix Praeparata. unassigned - 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31196146&form=6&db=m Heart failure drug proscillaridin A targets MYC overexpressing leukemia through global loss of lysine acetylation. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31360524&form=6&db=m The sodium pump and digitalis drugs: Dogmas and fallacies. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31819839&form=6&db=m Digoxin Toxicity in a Patient with Pacemaker: A Case Report. therapeutic application,unassigned 4,0 7.2.2.13 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32842758&form=6&db=m Targeting Cardiac Myocyte Na+-K+ Pump Function With ?3 Adrenergic Agonist in Rabbit Model of Severe Congestive Heart Failure. therapeutic application,unassigned 1,0 7.2.2.13 Hemiplegia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25996915&form=6&db=m Alternating Hemiplegia of Childhood: Retrospective Genetic Study and Genotype-Phenotype Correlations in 187 Subjects from the US AHCF Registry. causal interaction,unassigned 2,0 7.2.2.13 Hemiplegia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29567111&form=6&db=m Direct evidence of impaired neuronal Na/K-ATPase pump function in alternating hemiplegia of childhood. causal interaction,unassigned 1,0 7.2.2.13 Hemiplegia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32348881&form=6&db=m Neuronal modeling of alternating hemiplegia of childhood reveals transcriptional compensation and replicates a trigger-induced phenotype. causal interaction,unassigned 4,0 7.2.2.13 Hepatic Encephalopathy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6319665&form=6&db=m Effects of methanethiol on erythrocyte membrane stabilization and on Na+,K+-adenosine triphosphatase: relevance to hepatic coma. ongoing research,unassigned 4,0 7.2.2.13 Hepatic Encephalopathy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24044899&form=6&db=m Ammonia-induced Na,K-ATPase/ouabain-mediated EGF receptor transactivation, MAPK/ERK and PI3K/AKT signaling and ROS formation cause astrocyte swelling. ongoing research,unassigned 3,0 7.2.2.13 Hepatitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25653449&form=6&db=m ATP1A1-mediated Src signaling inhibits coronavirus entry into host cells. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Hepatitis B http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11791942&form=6&db=m Modulation of Na,K-ATPase activity by immunoglobulins--II. Na,K-ATPase activity in erythrocytes from patients with various chronic disturbances. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Hepatitis B, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9762418&form=6&db=m The effect of interferon therapy on erythrocyte membrane Na+,K+ ATP activity in patients with chronic hepatitis B and C virus infections. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 7.2.2.13 Hepatitis C, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9762418&form=6&db=m The effect of interferon therapy on erythrocyte membrane Na+,K+ ATP activity in patients with chronic hepatitis B and C virus infections. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 7.2.2.13 Hepatitis, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11791942&form=6&db=m Modulation of Na,K-ATPase activity by immunoglobulins--II. Na,K-ATPase activity in erythrocytes from patients with various chronic disturbances. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Hereditary Sensory and Motor Neuropathy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17215889&form=6&db=m K-Cl cotransport in red blood cells from patients with KCC3 isoform mutants. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Hernias, Diaphragmatic, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16214817&form=6&db=m Congenital diaphragmatic hernia prevents absorption of distal air space fluid in late-gestation rat fetuses. diagnostic usage,unassigned 3,0 7.2.2.13 Herpes Simplex http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8396802&form=6&db=m Role of the Na+,K+ pump in herpes simplex type 1-induced cell fusion: melittin causes specific reversion of syncytial mutants with the syn1 mutation to Syn+ (wild-type) phenotype. unassigned - 7.2.2.13 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23913004&form=6&db=m Somatic mutations in ATP1A1 and CACNA1D underlie a common subtype of adrenal hypertension. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25776071&form=6&db=m DACH1, a zona glomerulosa selective gene in the human adrenal, activates transforming growth factor-? signaling and suppresses aldosterone secretion. causal interaction,unassigned 2,0 7.2.2.13 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25958045&form=6&db=m Immunohistochemical, genetic and clinical characterization of sporadic aldosterone-producing adenomas. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,3,1 7.2.2.13 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26312884&form=6&db=m Role of ANO4 in regulation of aldosterone secretion in the zona glomerulosa of the human adrenal gland. causal interaction,unassigned 1,0 7.2.2.13 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28584012&form=6&db=m NEFM (Neurofilament Medium) Polypeptide, a Marker for Zona Glomerulosa Cells in Human Adrenal, Inhibits D1R (Dopamine D1 Receptor)-Mediated Secretion of Aldosterone. unassigned - 7.2.2.13 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33086282&form=6&db=m Y-27632 Promotes the Repair Effect of Umbilical Cord Blood-Derived Endothelial Progenitor Cells on Corneal Endothelial Wound Healing. unassigned - 7.2.2.13 Huntington Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12745628&form=6&db=m Cerebral chemical dominance and neural regulation of cell division, cell proliferation, neoplastic transformation, and genomic function. diagnostic usage,unassigned 4,0 7.2.2.13 Hydronephrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3001370&form=6&db=m Renal function and (NA+ + K+)-ATPase in chronic unilateral hydronephrosis in dogs. diagnostic usage,ongoing research,unassigned 1,2,0 7.2.2.13 Hydrothorax http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1661912&form=6&db=m Electrolyte transport across the pleura of rabbits. unassigned - 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2819817&form=6&db=m Mechanism of increased tubular Na-K-ATPase during streptozotocin-induced diabetes. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2990771&form=6&db=m The role of endogenous inhibition of Na-K-ATPase in human hypertension--sodium pump activity as a determinant of peripheral vascular resistance. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,2,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6256112&form=6&db=m Sodium-potassium-dependent ATPase activity in erythrocyte ghosts of patients with primary and secondary hypertension. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,2,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271508&form=6&db=m Na-K-ATPase in erythrocyte ghosts is not a marker for primary hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,1,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19641704&form=6&db=m Myocardial Na,K-ATPase: Clinical aspects. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,4,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25372651&form=6&db=m [Progress on genetic basis of primary aldosteronism]. causal interaction,unassigned 3,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26992195&form=6&db=m Novel genetic determinants of adrenal aldosterone regulation. causal interaction,unassigned 3,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27485459&form=6&db=m Genetics of primary hyperaldosteronism. causal interaction,unassigned 2,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27729216&form=6&db=m CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism. causal interaction,unassigned 4,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30811176&form=6&db=m Na/K Pump Mutations Associated with Primary Hyperaldosteronism Cause Loss of Function. causal interaction,diagnostic usage,unassigned 3,3,0 7.2.2.13 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32968034&form=6&db=m The landscape of molecular mechanism for aldosterone production in aldosterone-producing adenoma. causal interaction,unassigned 2,0 7.2.2.13 Hyperalgesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16460765&form=6&db=m Modulation of Na, K-ATPase activity by prostaglandin E(1) and [d-Ala(2),N-Me-Phe(4),Gly(5)-ol]-enkephalin. causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Hyperalgesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30411095&form=6&db=m Maltol, a food flavor enhancer, attenuates diabetic peripheral neuropathy in streptozotocin-induced diabetic rats. causal interaction,ongoing research,therapeutic application,unassigned 2,2,2,0 7.2.2.13 Hypercholesterolemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7848373&form=6&db=m Cholesterol enrichment inhibits Na+/K+ pump in endothelial cells. causal interaction,unassigned 4,0 7.2.2.13 Hypercholesterolemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14522815&form=6&db=m Dietary cholesterol alters Na+/K+ selectivity at intracellular Na+/K+ pump sites in cardiac myocytes. ongoing research,therapeutic application,unassigned 4,3,0 7.2.2.13 Hypercholesterolemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14690302&form=6&db=m Relation of erythrocyte Na+-K+ ATPase activity and cholesterol and oxidative stress in patients with type 2 diabetes mellitus. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,3,2 7.2.2.13 Hypercholesterolemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27643939&form=6&db=m The role of red blood cell deformability and Na,K-ATPase function in selected risk factors of cardiovascular diseases in humans: focus on hypertension, diabetes mellitus and hypercholesterolemia. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,3,1 7.2.2.13 Hyperesthesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8820823&form=6&db=m Morphophysiologic characterization of peripheral neuropathy in zinc-deficient guinea pigs. causal interaction,unassigned 4,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1337457&form=6&db=m Erythrocyte Ca, Na/K-ATPase in long-term streptozotocin diabetic rats. Effect of good glycemic control and a Ca antagonist. ongoing research,unassigned 4,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2838351&form=6&db=m Are disturbances of sorbitol, phosphoinositide, and Na+-K+-ATPase regulation involved in pathogenesis of diabetic neuropathy? unassigned - 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6093549&form=6&db=m Nerve Na+-K+-ATPase, conduction, and myo-inositol in the insulin-deficient BB rat. causal interaction,unassigned 2,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9202063&form=6&db=m Characterization of protein kinase C beta isoform activation on the gene expression of transforming growth factor-beta, extracellular matrix components, and prostanoids in the glomeruli of diabetic rats. causal interaction,ongoing research,therapeutic application,unassigned 4,2,1,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9312166&form=6&db=m Glucose-specific regulation of aldose reductase in capan-1 human pancreatic duct cells In vitro. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10377817&form=6&db=m [New trend in pathogenesis of diabetic neuropathy] causal interaction,diagnostic usage,unassigned 3,3,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11165869&form=6&db=m Pyridoxine and pyridoxamine inhibits superoxide radicals and prevents lipid peroxidation, protein glycosylation, and (Na+ + K+)-ATPase activity reduction in high glucose-treated human erythrocytes. therapeutic application,unassigned 1,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11595378&form=6&db=m Oxidative stress and eicosanoids in the kidneys of hyperglycemic rats treated with dehydroepiandrosterone. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11832341&form=6&db=m Hyperglycemia increases endothelial superoxide that impairs smooth muscle cell Na+-K+-ATPase activity. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12403306&form=6&db=m Effect of chronic hyperglycemia and vanadate treatment on erythrocyte Na/K-ATpase and Mg-ATpase in streptozotocin diabetic rats. therapeutic application,unassigned 2,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15661101&form=6&db=m Strength-duration properties and glycemic control in human diabetic motor nerves. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19682444&form=6&db=m Prophylactic role of arjunolic acid in response to streptozotocin mediated diabetic renal injury: activation of polyol pathway and oxidative stress responsive signaling cascades. causal interaction,diagnostic usage,unassigned 2,2,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26063704&form=6&db=m ?3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,1,2 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26179604&form=6&db=m ?3-Adrenoceptor, glutathionylation, and diabetic cardiomyopathy. Focus on "?3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade". causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30766472&form=6&db=m Neurons and Microglia; A Sickly-Sweet Duo in Diabetic Pain Neuropathy. unassigned - 7.2.2.13 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1438101&form=6&db=m [Theoretical aspects of the relationship between diabetic macroangiopathy and hyperinsulinism] causal interaction,unassigned 1,0 7.2.2.13 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17220688&form=6&db=m Animal and human tissue Na,K-ATPase in obesity and diabetes: A new proposed enzyme regulation. causal interaction,ongoing research,unassigned 4,4,0 7.2.2.13 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17444965&form=6&db=m Animal and human tissue Na,K-ATPase in normal and insulin-resistant states: regulation, behaviour and interpretative hypothesis on NEFA effects. causal interaction,unassigned 4,0 7.2.2.13 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17556873&form=6&db=m Hypokalemic periodic paralysis in a patient with acquired growth hormone deficiency. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6126857&form=6&db=m Renal function and Na-K-ATPase in rats after suprarenal ligation of inferior vena cava. causal interaction,unassigned 1,0 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7629896&form=6&db=m Disturbances of potassium homeostasis in poisoning. unassigned - 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8731102&form=6&db=m Studies on the mechanism of trimethoprim-induced hyperkalemia. causal interaction,unassigned 4,0 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9262462&form=6&db=m Mechanism of the exercise hyperkalemia: an alternate hypothesis. unassigned - 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10037077&form=6&db=m Regulation of Na/K-ATPase gene expression by thyroid hormone and hyperkalemia in the heart. causal interaction,ongoing research,therapeutic application,unassigned 2,2,4,0 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11798149&form=6&db=m Hydroelectrolytic equilibrium change in dialysis. ongoing research,unassigned 2,0 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20169971&form=6&db=m [Case of hyperkalemia possibly caused by gabexate mesilate] causal interaction,unassigned 1,0 7.2.2.13 Hyperkalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30175537&form=6&db=m Renal Mg handling, FXYD2 and the central role of the Na,K-ATPase. causal interaction,unassigned 2,0 7.2.2.13 Hyperlipidemias http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8391317&form=6&db=m Accelerated maximal velocity of the red blood cell Na+/K+ pump in hyperlipidemia is related to increase in 1-palmitoyl,2-arachidonoyl-plasmalogen phosphatidylethanolamine. causal interaction,unassigned 1,0 7.2.2.13 Hyperlipidemias http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21995900&form=6&db=m Gender specific influence of fish oil or atorvastatin on functional properties of renal Na,K-ATPase in healthy Wistar and hypertriglyceridemic rats. causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Hypersensitivity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6291893&form=6&db=m A serotonin agonist-antagonist reversible effect on Na+-K+-ATPase activity in the developing rat brain. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=130615&form=6&db=m Na-K-adenosine triphosphatase in the kidney of rats with renal hypertension and spontaneously hypertensive rats. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=131655&form=6&db=m Depressed function of a ouabain-sensitive sodium-potassium pump in blood vessels from renal hypertensive dogs. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=215375&form=6&db=m Altered activity of the sodium-potassium pump in arteries of rats with steroid hypertension. causal interaction,ongoing research,therapeutic application,unassigned 1,1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=384124&form=6&db=m Humoral factors and the sodium-potassium pump in volume expanded hypertension. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=387359&form=6&db=m The sodium-potassium pump in volume expanded hypertension. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1338308&form=6&db=m Sodium transport in hypertension. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1412450&form=6&db=m Roles of renal cytochrome P450-dependent arachidonic acid metabolites in hypertension. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1648593&form=6&db=m Endogenous natriuretic factors: atrial natriuretic hormone and digitalis-like substance in Cushing's syndrome. diagnostic usage,ongoing research,therapeutic application,unassigned 2,2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1656573&form=6&db=m [Na,K-ATPase in kidneys of rats with hereditary hypertension induced by stress] causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1662123&form=6&db=m Augmented Na,K-ATPase gene expression in spontaneously hypertensive rat hearts. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1662981&form=6&db=m Increased renal tubular Na-K-ATPase activity in Milan hypertensive rats in the prehypertensive period. ongoing research,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1663115&form=6&db=m Elevated endogenous Na-K-ATPase inhibitor activity in hypertensive diabetic patients with a family history of hypertension. causal interaction,diagnostic usage,unassigned 4,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1666590&form=6&db=m [The activity of the sodium-potassium pump of the mesenteric arteries and veins in normal rats and in DOCA-salt hypertension] ongoing research,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1666609&form=6&db=m [The activity and properties of Na,K-ATPase in the erythrocytes and the Na-pump inhibitor in the plasma of subjects with borderline arterial hypertension] causal interaction,ongoing research,unassigned 4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1668126&form=6&db=m [Activity of the sodium-potassium pump and values of sodium and potassium in erythrocytes in patients with non-dialyzed renal failure and arterial hypertension treated conservatively] diagnostic usage,ongoing research,therapeutic application,unassigned 3,2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1685169&form=6&db=m Erythrocyte Li-Na countertransport in hypertension: lack of atrial natriuretic peptide effect. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1848765&form=6&db=m Plasma sodium-potassium ATPase inhibition activity in low- and normal-renin hypertension. ongoing research,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1962738&form=6&db=m Endogenous digitalis-like factors: their possible pathophysiological implications with particular regard to the perinatal period. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2096434&form=6&db=m [Arterial hypertension in patients with obesity. Role of hyperinsulinism and insulin resistance] causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2157466&form=6&db=m Effects of canrenone on blood pressure in rats with reduced renal mass. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2162662&form=6&db=m Purification of a Na+/K+ ATPase inhibitor from borderline hypertensives' plasma. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2164130&form=6&db=m Impaired calcium metabolism associated with hypertension in Zucker obese rats. diagnostic usage,ongoing research,unassigned 2,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2176807&form=6&db=m Digitalislike circulating factor in hypertension: potential messenger between salt balance and intracellular sodium. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2196126&form=6&db=m Increased activity of digoxin-like substance in low-renin hypertension in acromegaly. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2225723&form=6&db=m Is the endogenous digitalis-like factor the link between hypertension and metabolic disorders as diabetes mellitus, obesity and acromegaly? causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2298467&form=6&db=m Effect of gender in centrally induced angiotensin II hypertension in dogs. ongoing research,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2322432&form=6&db=m Sodium content and sodium efflux of mononuclear leucocytes from young subjects at increased risk of developing essential hypertension. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2411605&form=6&db=m Evidence for a circulating endogenous Na+-K+ pump inhibitor in low-renin hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2413778&form=6&db=m Arteriolar responses to norepinephrine and ouabain in early 1-kidney, 1-clip hypertension in rats. therapeutic application,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2419683&form=6&db=m Differences in renal tubular Na-K-adenosine triphosphatase in spontaneously hypertensive and normotensive rats. ongoing research,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2424645&form=6&db=m A circulating Na+-K+ATPase inhibitor, erythrocyte sodium transport and hypertension in patients with chronic renal failure. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2439453&form=6&db=m Reduced sodium concentration and increased sodium-potassium pump activity of erythrocytes in human hypertension. causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2439454&form=6&db=m Reduced electrogenic sodium-potassium pump in arterioles during renovascular hypertension. causal interaction,ongoing research,therapeutic application,unassigned 2,2,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2440805&form=6&db=m Kinetic analysis of erythrocyte Na+-K+ pump and cotransport in essential hypertension. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2467109&form=6&db=m Circulating inhibitors of sodium transport at the prehypertensive stage of essential hypertension. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2471434&form=6&db=m Alterations in sodium-potassium regulation in mononuclear leucocytes from young borderline hypertensive and offspring of hypertensive patients. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2487432&form=6&db=m [Sodium transport in red blood cell membranes in essential arterial hypertension] diagnostic usage,ongoing research,unassigned 1,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2542013&form=6&db=m Chronobiological evidence for an uncoupling of the Na,K-ATPase to aldosterone in normal renin hypertension. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2544279&form=6&db=m [A study on the relationship between reduced activity of red cell membrane sodium-potassium pump and essential hypertension] causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,1 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2548471&form=6&db=m Effect of ouabain on hemodynamics in acute volume expanded hypertensive dogs. causal interaction,therapeutic application,unassigned 4,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2557224&form=6&db=m Myocardial Na+-K+-ATPase activity and [3H]ouabain binding sites in hypertensive rats. causal interaction,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2579261&form=6&db=m Studies on the role of sodium- and potassium-activated adenosine triphosphatase inhibition in the pathogenesis of human hypertension. Changes in vascular and cardiac function following inhibition of the sodium pump in normotensive subjects and effects of calcium entry blockade. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2654438&form=6&db=m Sodium chloride sensitivity, intracellular sodium concentration in erythrocytes and lymphocytes, and renin profile in essential hypertension. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,4,4 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2822824&form=6&db=m Effect of treatment with hydrochlorothiazide on the red cell Na,K-adenosine triphosphatase in men with hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2824374&form=6&db=m The vascular Na+-K+ pump in experimental hypertension. ongoing research,therapeutic application,unassigned 1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2831002&form=6&db=m Potassium, Na+-K+ pump inhibitor and low-renin hypertension. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2840392&form=6&db=m Hypertensive dog plasma inhibits the Na+-K+ pump of cultured vascular smooth muscle. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2844545&form=6&db=m Abnormal Na+-K+ ATPase kinetics in a subset of essential hypertensive patients. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,2,1 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2847194&form=6&db=m Individual variability in membrane sodium transport during salt loading--evidence against the production of a Na+-K+-ATPase inhibitor in hypertension. therapeutic application,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2848975&form=6&db=m Erythrocyte Na,K pump activity and arterial hypertension in uremic dialyzed patients. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2849538&form=6&db=m Effects of lead and natriuretic hormone on kinetics of sodium-potassium-activated adenosine triphosphatase: possible relevance to hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2849788&form=6&db=m The role of vascular Na,K-ATPase activity in salt induced hypertension in Dahl rats. causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2853745&form=6&db=m Ouabain vasoconstricts human forearm arterioles through alpha-adrenergic stimulation. causal interaction,ongoing research,therapeutic application,unassigned 1,1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2853747&form=6&db=m Pathogenesis of arterial hypertension in chronic uraemia: the role of reduced Na,K-ATPase activity. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2854370&form=6&db=m Plasma levels of digitalis-like substance in Nigerians with essential hypertension. diagnostic usage,ongoing research,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2854520&form=6&db=m [Age-related changes in Na,K-ATPase activity of the plasma membrane of kidney cells and erythrocytes in spontaneously hypertensive and normotensive rats] unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2854827&form=6&db=m Study of plasma and intraerythrocytic cations and erythrocytic membrane Na+-K+-ATPase in primary hypertension. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2895170&form=6&db=m Ouabain-induced elevation in forearm vascular resistance, calcium entry and alpha-adrenoceptor blockade, and release and removal of noradrenaline. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2989396&form=6&db=m Sodium-potassium-adenosine triphosphatase in nephron segments of spontaneously hypertensive rats. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2990769&form=6&db=m Plasma Na+-K+ ATPase inhibitory activity in normal and hypertensive subjects: relationship to intracellular electrolytes and blood pressure. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,2,2 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2990770&form=6&db=m Characteristics of hypertensive patients with increased plasma Na+-K+ pump inhibitory activity. causal interaction,diagnostic usage,unassigned 4,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2990771&form=6&db=m The role of endogenous inhibition of Na-K-ATPase in human hypertension--sodium pump activity as a determinant of peripheral vascular resistance. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3000191&form=6&db=m Na-K-ATPase in nephron segments of rats developing spontaneous hypertension. ongoing research,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3001753&form=6&db=m Endogenous sodium-potassium pump inhibitor in low renin hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,4,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3003189&form=6&db=m Decreased myocardial Na+K+ATPase activity in rats with reduced renal mass-saline hypertension. causal interaction,ongoing research,therapeutic application,unassigned 3,2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3038745&form=6&db=m Decreased intraocular pressure in dogs with one-kidney, one wrapped hypertension. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3040303&form=6&db=m Decreased Na+K+ATPase activity in the aortic endothelium and smooth muscle of the spontaneously hypertensive rats. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3280170&form=6&db=m The central effects of the renin-angiotensin system. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3289271&form=6&db=m [The role of the kidney in the pathogenesis of hypertension] causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3599798&form=6&db=m Plasma inhibitors of Na,K-ATPase: relation to salt balance and hypertension. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3599801&form=6&db=m The endogenous cytochemically assayable Na-K-ATPase inhibitor and its relation to hypertension. causal interaction,diagnostic usage,therapeutic application,unassigned 2,2,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3611769&form=6&db=m Relationship between erythrocyte volume and sodium transport in the Milan hypertensive rat and age-dependent changes. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3679448&form=6&db=m The possible role of digitalislike factors in pregnancy-induced hypertension. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3777205&form=6&db=m Effect of hypertensive rat plasma on ion transport of cultured vascular smooth muscle. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3893806&form=6&db=m Evidence for a circulating sodium-potassium pump inhibitor in low renin hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6091145&form=6&db=m Renal Na+-K+-ATPase in weanling and adult spontaneously hypertensive rats. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6095682&form=6&db=m Hypothalamic sodium-transport inhibitor is a high-affinity reversible inhibitor of Na+-K+-ATPase. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6099380&form=6&db=m Na-K-ATPase in single nephron segments of hypertension-prone rats. ongoing research,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6099840&form=6&db=m Myocardial Na,K-ATPase activity in rats with steroid and spontaneous hypertension. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6100156&form=6&db=m Cellular mechanism relating sodium and potassium to hypertension. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6132327&form=6&db=m Natriuresis and renal Na-K-ATPase activity in kidneys of normotensive and spontaneously hypertensive rats. causal interaction,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6136190&form=6&db=m Decreased myocardial Na+-K+-ATPase activity in one-kidney, one-clip hypertensive rats. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6204161&form=6&db=m The vascular Na+-K+ pump in low renin hypertension. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6206353&form=6&db=m The role of a humoral Na+,K+-ATPase inhibitor in regulating precapillary vessel tone. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6248683&form=6&db=m Increased activity of the Na-K-ATPase in red cell-ghosts of patients with Cushing's syndrome: possible significance for the pathogenesis of glucocorticoid-induced hypertension. causal interaction,diagnostic usage,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6256112&form=6&db=m Sodium-potassium-dependent ATPase activity in erythrocyte ghosts of patients with primary and secondary hypertension. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271508&form=6&db=m Na-K-ATPase in erythrocyte ghosts is not a marker for primary hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6280009&form=6&db=m Role of a humoral sodium-potassium pump inhibitor in experimental low renin hypertension. causal interaction,ongoing research,therapeutic application,unassigned 3,1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6292571&form=6&db=m Humoral factors and the sodium-potassium pump in low renin hypertension. diagnostic usage,therapeutic application,unassigned 2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6298108&form=6&db=m Sodium-potassium pump activity in reduced renal-mass hypertension. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6299628&form=6&db=m Reduced sodium-potassium dependent ATPase and its possible role in the development of hypertension in spontaneously hypertensive rats. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6303174&form=6&db=m Volume hormones and blood pressure. causal interaction,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6303176&form=6&db=m Sodium-potassium pump in low-renin hypertension. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6307048&form=6&db=m The role of sodium intake, the Na+-K+ pump and a ouabain-like humoral agent in the genesis of reduced renal mass hypertension. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6311735&form=6&db=m Sodium and potassium ion transport accelerations in erythrocytes of DOC, DOC-salt, two-kidney, one clip, and spontaneously hypertensive rats. Role of hypokalemia and cell volume. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6317363&form=6&db=m Sodium-potassium ATPase in deoxycorticosterone-salt hypertension: opposing effects of sodium load and mineralocorticoids. causal interaction,ongoing research,therapeutic application,unassigned 2,1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6329119&form=6&db=m [The sodium-potassium pump in hypertension. Mechanisms of action of potassium] therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6345202&form=6&db=m The role of a humoral sodium-potassium pump inhibitor in low-renin hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6360715&form=6&db=m Periventricular forebrain mechanisms for blood pressure regulation. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6441535&form=6&db=m [Effect of an antiglucocorticoid steroid on the arterial hypertension induced by glucocorticoids in the rat] causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6837769&form=6&db=m Some effects of aldosterone on sodium kinetics and distribution in porcine arterial wall. causal interaction,ongoing research,unassigned 4,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7298143&form=6&db=m Demonstration of a humoral inhibitor of the Na+-K+ pump in some models of experimental hypertension. ongoing research,therapeutic application,unassigned 2,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7449256&form=6&db=m Vascular sodium-potassium pump activity in various models of experimental hypertension. causal interaction,ongoing research,therapeutic application,unassigned 3,2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7508010&form=6&db=m Role of ouabain-like factors in hypertension: effects of ouabain and certain endogenous ouabain-like factors in hypertension. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7573397&form=6&db=m Reciprocal regulation of cardiac Na-K-ATPase and Na/Ca exchanger: hypertension, thyroid hormone, development. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7833833&form=6&db=m Sodium-22 uptake in erythrocytes can differentiate between the essential and the secondary hypertensive patient. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7922215&form=6&db=m Regulation of Na,K-ATPase activity. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8129535&form=6&db=m [Abnormal erythrocyte sodium transport in patients with adult polycystic kidney and hypertension] causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8282358&form=6&db=m Increased cytosolic sodium and reduced Na,K-ATPase activity in transgenic rats. causal interaction,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8567044&form=6&db=m Pathophysiological consequences of changes in the coupling ratio of Na,K-ATPase for renal sodium reabsorption and its implications for hypertension. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8772464&form=6&db=m Na-K-ATPase alpha-isoform expression in heart and vascular endothelia: cellular and developmental regulation. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8933500&form=6&db=m Direct regulation of the Na,K pump by signal transduction mechanisms. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8934358&form=6&db=m Endothelium stimulates the vascular smooth muscle cell Na/K pump of normotensive and hypertensive rats by a protein kinase C pathway. diagnostic usage,ongoing research,unassigned 2,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8952591&form=6&db=m Renal Na,K-ATPase in genetic hypertension. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9028636&form=6&db=m Short-term vs. sustained inhibition of proximal tubule Na,K-ATPase activity by dopamine: cellular mechanisms. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9555654&form=6&db=m The cytochrome P-450 inhibitor cobalt chloride prevents inhibition of renal Na,K-ATPase and redistribution of apical NHE-3 during acute hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9655151&form=6&db=m The role of Na,K-ATPase inhibitors in hypertension and end-stage renal disease. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9682906&form=6&db=m Role of ouabain-like factors and Na-K-ATPase inhibitors in hypertension--some old and recent findings. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,3,3 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9682907&form=6&db=m Role of digitalis-like substance in the hypertension of streptozotocin-induced diabetes and simulated weightlessness in rats. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9739044&form=6&db=m The alpha1 Na,K-ATPase gene is a susceptibility hypertension gene in the Dahl salt-sensitiveHSD rat. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9759982&form=6&db=m Reflux nephropathy and hypertension. causal interaction,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9830514&form=6&db=m Increased Na,K,Cl cotransporter and Na, K-ATPase activity of vascular tissue in two-kidney Goldblatt hypertension. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9877522&form=6&db=m Evidence for increased functional vascular Na+/K+ pump activity in the obese Zucker rat. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10027844&form=6&db=m PST 2238: A new antihypertensive compound that modulates Na,K-ATPase in genetic hypertension. causal interaction,ongoing research,therapeutic application,unassigned 3,1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10099845&form=6&db=m Sodium-potassium pump inhibitor in the mechanism of one-kidney, one wrap hypertension in dogs. causal interaction,therapeutic application,unassigned 2,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10628927&form=6&db=m Recent aspects in the genetic renal mechanisms involved in hypertension. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10629425&form=6&db=m Increased (Na+K+Cl) cotransport in rat arterial smooth muscle in deoxycorticosterone (DOCA)/salt-induced hypertension. diagnostic usage,therapeutic application,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10757222&form=6&db=m Ouabain-like factor: is this the natriuretic hormone? causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10919857&form=6&db=m Proximal tubule Na transporter responses are the same during acute and chronic hypertension. causal interaction,ongoing research,therapeutic application,unassigned 4,2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11325071&form=6&db=m Mechanism of sodium load-induced hypertension in non-insulin dependent diabetes mellitus model rats: defective dopaminergic system to inhibit Na-K-ATPase activity in renal epithelial cells. ongoing research,therapeutic application,unassigned 3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11355012&form=6&db=m Mechanisms and consequences of Na+,K+-pump regulation by insulin and leptin. causal interaction,ongoing research,unassigned 4,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11471547&form=6&db=m Alpha1 Na,K-ATPase and Na,K,2Cl-cotransporte/D3mit3 loci interact to increase susceptibility to salt-sensitive hypertension in Dahl S(HSD) rats. ongoing research,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11509477&form=6&db=m Interaction of alpha(1)-Na,K-ATPase and Na,K,2Cl-cotransporter genes in human essential hypertension. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11564973&form=6&db=m Non-association of the thiazide-sensitive Na,Cl-cotransporter gene with polygenic hypertension in both rats and humans. ongoing research,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11564975&form=6&db=m Inhibition of Na+,K+ pump affects nucleic acid synthesis and smooth muscle cell proliferation via elevation of the [Na+]i/[K+]i ratio: possible implication in vascular remodelling. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11641287&form=6&db=m Neither the New Zealand genetically hypertensive strain nor Dahl salt-sensitive strain has an A1079T transversion in the alpha1 isoform of the Na(+),K(+)-ATPase gene. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11675399&form=6&db=m Downstream shift in sodium pump activity along the nephron during acute hypertension. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,1,1 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11768735&form=6&db=m The change and significance of the Na+-K+-ATPase alpha-subunit in ouabain-hypertensive rats. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11882604&form=6&db=m Novel receptors for ouabain: studies in adrenocortical cells and membranes. causal interaction,therapeutic application,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11926353&form=6&db=m The alpha1-Na/K pump does not mediate the involvement of ouabain in the development of hypertension in rats. causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12187767&form=6&db=m [Fundamental and applied studies on transport and metabolism of electrolytes and glucose--aim to contact with molecular biology] causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12463096&form=6&db=m [Autoantibodies: new upstream targets of paroxysmal atrial fibrillation in patients with congestive heart failure] causal interaction,ongoing research,therapeutic application,unassigned 3,1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12470200&form=6&db=m Changes of sodium and ATP affinities of the renal Na,K-ATPase during and after nitric oxide-deficient hypertension. diagnostic usage,ongoing research,unassigned 2,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12611422&form=6&db=m Increased dietary potassium and magnesium attenuate experimental volume dependent hypertension possibly through endogenous sodium-potassium pump inhibitor. therapeutic application,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12623951&form=6&db=m Myocardial PKC beta2 and the sensitivity of Na/K-ATPase to marinobufagenin are reduced by cicletanine in Dahl hypertension. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12675854&form=6&db=m Effect of green tea extract on cardiac hypertrophy following 5/6 nephrectomy in the rat. causal interaction,therapeutic application,unassigned 1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12763917&form=6&db=m Mechanisms of pressure natriuresis: how blood pressure regulates renal sodium transport. causal interaction,ongoing research,therapeutic application,unassigned 1,1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12763920&form=6&db=m Antihypertensive compounds that modulate the Na-K pump. ongoing research,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12884521&form=6&db=m [A search for genetic loci responsible for emotional stress-induced arterial hypertension in the ISIAH rats] causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12964647&form=6&db=m Role of kidney in hypertension. therapeutic application,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15076199&form=6&db=m Coordinated shifts in Na/K-ATPase isoforms and their endogenous ligands during cardiac hypertrophy and failure in NaCl-sensitive hypertension. causal interaction,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15458945&form=6&db=m The alpha2-isoform of Na-K-ATPase mediates ouabain-induced hypertension in mice and increased vascular contractility in vitro. causal interaction,ongoing research,unassigned 2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15775778&form=6&db=m Corroboration of Dahl S Q276L alpha1Na,K-ATPase protein sequence: impact on affinities for ligands and on E1 conformation. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15970498&form=6&db=m Endogenous digitalis-like factors. An historical overview. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16243970&form=6&db=m The highly conserved cardiac glycoside binding site of Na,K-ATPase plays a role in blood pressure regulation. causal interaction,ongoing research,therapeutic application,unassigned 2,1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16247002&form=6&db=m The sodium pump and hypertension: a physiological role for the cardiac glycoside binding site of the Na,K-ATPase. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16467499&form=6&db=m Physiological role of the alpha1- and alpha2-isoforms of the Na+-K+-ATPase and biological significance of their cardiac glycoside binding site. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16755197&form=6&db=m The central role of the brain in salt-sensitive hypertension. causal interaction,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17444965&form=6&db=m Animal and human tissue Na,K-ATPase in normal and insulin-resistant states: regulation, behaviour and interpretative hypothesis on NEFA effects. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17446437&form=6&db=m Association of ATP1A1 and dear single-nucleotide polymorphism haplotypes with essential hypertension: sex-specific and haplotype-specific effects. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17518348&form=6&db=m The effects of captopril and losartan on erythrocyte membrane Na+/K(+)-ATPase activity in experimental diabetes mellitus. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17683916&form=6&db=m Endogenous bufadienolide mediates pressor response to ethanol withdrawal in rats. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17942287&form=6&db=m Endogenous sodium pump inhibitors, diabetes mellitus and preeclampsia Preliminary observations and a hypothesis. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17980562&form=6&db=m Endogenous brain Na pumps, brain ouabain-like substance and the alpha2 isoform in salt-dependent hypertension. therapeutic application,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18343354&form=6&db=m Platelet membrane fluidity and Na+/K+ ATPase activity in acute stroke. diagnostic usage,ongoing research,therapeutic application,unassigned 4,3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18524856&form=6&db=m alpha-Adducin mutations increase Na/K pump activity in renal cells by affecting constitutive endocytosis: implications for tubular Na reabsorption. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18667729&form=6&db=m Ouabain-Sensitive alpha1 Na,K-ATPase enhances natriuretic response to saline load. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19008721&form=6&db=m Monoclonal antibody to an endogenous bufadienolide, marinobufagenin, reverses preeclampsia-induced Na/K-ATPase inhibition and lowers blood pressure in NaCl-sensitive hypertension. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19474184&form=6&db=m Inhibition of natriuretic factors increases blood pressure in rats. causal interaction,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19479337&form=6&db=m Over-expression of translationally controlled tumor protein in lens epithelial cells seems to be associated with cataract development. causal interaction,ongoing research,unassigned 2,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21081803&form=6&db=m Effect of quercetin on kinetic properties of renal Na,K-ATPase in normotensive and hypertensive rats. diagnostic usage,therapeutic application,unassigned 1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21553016&form=6&db=m NKCC2 is activated in Milan hypertensive rats contributing to the maintenance of salt-sensitive hypertension. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21555512&form=6&db=m Impairment of Na/K-ATPase signaling in renal proximal tubule contributes to Dahl salt-sensitive hypertension. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21632957&form=6&db=m Renovascular hypertension using a modified two-kidney, one-clip approach in mice is not dependent on the {alpha}1 or {alpha}2 Na,K-ATPase ouabain-binding site. causal interaction,therapeutic application,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21767529&form=6&db=m Oleic and linoleic acids are active principles in Nigella sativa and stabilize an E(2)P conformation of the Na,K-ATPase. Fatty acids differentially regulate cardiac glycoside interaction with the pump. causal interaction,therapeutic application,unassigned 1,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22183064&form=6&db=m Systemic Hypertension: The Roles of Salt, Vascular Na+/K+ ATPase and The Endogenous Glycosides, Ouabain and Marinobufagenin. causal interaction,therapeutic application,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22237155&form=6&db=m Increased Expression of Na,K-ATPase and a Selective Increase in Phosphorylation at Ser-11 in the Cortex of the 2-Kidney, 1-Clip Hypertensive Rat. causal interaction,unassigned 3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22258333&form=6&db=m DOCA-Salt Hypertension Does Not Require the Ouabain-Sensitive Binding Site of the ?2 Na,K-ATPase. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22518311&form=6&db=m Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium Handling. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23416519&form=6&db=m Somatic mutations in ATP1A1 and ATP2B3 lead to aldosterone-producing adenomas and secondary hypertension. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23913004&form=6&db=m Somatic mutations in ATP1A1 and CACNA1D underlie a common subtype of adrenal hypertension. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23986517&form=6&db=m ANGIOTENSIN II-INDUCED HYPERTENSION INCREASES PLASMA MEMBRANE Na PUMP ACTIVITY BY ENHANCING Na ENTRY IN RAT THICK ASCENDING LIMBS. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24219001&form=6&db=m Cardiotonic Steroids in Adaptation to Dietary Salt Intake. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24748592&form=6&db=m Reduction of Na/K-ATPase affects cardiac remodeling and increases c-kit cell abundance in partial nephrectomized mice. causal interaction,unassigned 1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24918292&form=6&db=m Up-regulation of Rhoa/Rho kinase pathway by translationally controlled tumor protein in vascular smooth muscle cells. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25472608&form=6&db=m Paradoxical activation of the sodium chloride cotransporter (NCC) without hypertension in kidney deficient in a regulatory subunit of Na,K-ATPase, FXYD2. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25615575&form=6&db=m A functional 12T-insertion polymorphism in the ATP1A1 promoter confers decreased susceptibility to hypertension in a male Sardinian population. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,1,1 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25733244&form=6&db=m Transcriptional Regulation of Renal Dopamine D1 Receptor Function During Oxidative Stress. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26136067&form=6&db=m Marinobufagenin-induced vascular fibrosis is a likely target for mineralocorticoid antagonists. diagnostic usage,ongoing research,therapeutic application,unassigned 1,2,2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26374826&form=6&db=m Synthesis of an Endogenous Steroidal Na Pump Inhibitor Marinobufagenin, Implicated in Human Cardiovascular Diseases, Is Initiated by CYP27A1 via Bile Acid Pathway. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26603454&form=6&db=m Antioxidant resveratrol restores renal sodium transport regulation in SHR. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27643939&form=6&db=m The role of red blood cell deformability and Na,K-ATPase function in selected risk factors of cardiovascular diseases in humans: focus on hypertension, diabetes mellitus and hypercholesterolemia. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,3,1 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28089998&form=6&db=m First Clinical Experience With Targeted REnal Nerve Demodulation (TREND-1) Using a Neurotropic Agent for the Treatment of Sympathetic Hypertension. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28515088&form=6&db=m Loss of NHERF-1 expression prevents dopamine-mediated Na-K-ATPase regulation in renal proximal tubule cells from rat models of hypertension: aged F344 rats and spontaneously hypertensive rats. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30175537&form=6&db=m Renal Mg handling, FXYD2 and the central role of the Na,K-ATPase. causal interaction,unassigned 2,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30811176&form=6&db=m Na/K Pump Mutations Associated with Primary Hyperaldosteronism Cause Loss of Function. causal interaction,diagnostic usage,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31107610&form=6&db=m Inhibition of intestinal villus cell Na/K-ATPase mediates altered glucose and NaCl absorption in obesity-associated diabetes and hypertension. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31196601&form=6&db=m Acute salt loading and cardiotonic steroids in resistant hypertension. causal interaction,ongoing research,unassigned 2,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31275642&form=6&db=m A Review of the Epidemiological Evidence for Adducin Family Gene Polymorphisms and Hypertension. unassigned - 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31315267&form=6&db=m Metabolic Syndrome and Salt-Sensitive Hypertension in Polygenic Obese TALLYHO/JngJ Mice: Role of Na/K-ATPase Signaling. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31576777&form=6&db=m Monoclonal Antibody to Marinobufagenin Downregulates TGF? Profibrotic Signaling in Left Ventricle and Kidney and Reduces Tissue Remodeling in Salt-Sensitive Hypertension. causal interaction,unassigned 4,0 7.2.2.13 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34440087&form=6&db=m High Na+ Salt Diet and Remodeling of Vascular Smooth Muscle and Endothelial Cells. causal interaction,unassigned 3,0 7.2.2.13 Hypertension, Pregnancy-Induced http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2551544&form=6&db=m Endogenous inhibition of red blood cell Na,K-ATPase in essential and pregnancy-induced hypertension. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Hypertension, Pregnancy-Induced http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3030374&form=6&db=m [Preliminary study of Na/K-ATPase activity in erythrocyte ghosts in pregnancy-induced hypertension] ongoing research,unassigned 1,0 7.2.2.13 Hypertension, Pregnancy-Induced http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7920379&form=6&db=m Human hypertensive placenta contains an increased amount of Na,K-ATPase with higher affinity for cardiac glycosides. therapeutic application,unassigned 1,0 7.2.2.13 Hypertension, Pregnancy-Induced http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8054335&form=6&db=m Na,K-ATPase from placenta of women with pregnancy-induced hypertension exhibits an increased affinity for cardiac glycosides. unassigned - 7.2.2.13 Hypertension, Renal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=130615&form=6&db=m Na-K-adenosine triphosphatase in the kidney of rats with renal hypertension and spontaneously hypertensive rats. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,3,0 7.2.2.13 Hypertension, Renal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6256112&form=6&db=m Sodium-potassium-dependent ATPase activity in erythrocyte ghosts of patients with primary and secondary hypertension. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,2,0 7.2.2.13 Hypertension, Renal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271508&form=6&db=m Na-K-ATPase in erythrocyte ghosts is not a marker for primary hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,1,0 7.2.2.13 Hypertension, Renovascular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2415578&form=6&db=m Alterations in renal Na+K+ATPase activity and [3H]ouabain binding in Goldblatt hypertensive rabbits. ongoing research,unassigned 3,0 7.2.2.13 Hypertension, Renovascular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2439454&form=6&db=m Reduced electrogenic sodium-potassium pump in arterioles during renovascular hypertension. causal interaction,ongoing research,therapeutic application,unassigned 2,2,3,0 7.2.2.13 Hypertension, Renovascular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7573397&form=6&db=m Reciprocal regulation of cardiac Na-K-ATPase and Na/Ca exchanger: hypertension, thyroid hormone, development. causal interaction,unassigned 3,0 7.2.2.13 Hypertension, Renovascular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21632957&form=6&db=m Renovascular hypertension using a modified two-kidney, one-clip approach in mice is not dependent on the {alpha}1 or {alpha}2 Na,K-ATPase ouabain-binding site. causal interaction,therapeutic application,unassigned 3,3,0 7.2.2.13 Hypertension, Renovascular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22237155&form=6&db=m Increased Expression of Na,K-ATPase and a Selective Increase in Phosphorylation at Ser-11 in the Cortex of the 2-Kidney, 1-Clip Hypertensive Rat. causal interaction,unassigned 3,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1317628&form=6&db=m Increased lymphocyte thermogenesis in hyperthyroid patients. Role of Na/K pump function. Evaluation of aerobic/anaerobic metabolism. causal interaction,diagnostic usage,therapeutic application,unassigned 1,1,2,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1324264&form=6&db=m Na+K+ATPase activity and ouabain binding sites in erythrocytes in hyperthyroidism before and after treatment. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,4 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2553305&form=6&db=m Increased acylphosphatase levels in erythrocytes from hyperthyroid patients. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,3 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2987290&form=6&db=m Erythrocyte sodium/potassium adenosine triphosphatase in thyroid disease and nonthyroidal illness. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6257487&form=6&db=m Interaction of age and thyroid hormone status on Na+-K+ ATPase in rat renal cortex and liver. unassigned - 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6284135&form=6&db=m No major thermogenic role for (Na+ + K+)-dependent adenosine triphosphatase apparent in hepatocytes from hyperthyroid rats. therapeutic application,unassigned 1,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6294446&form=6&db=m Status of the red cell Na,K-pump in hyper- and hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,4,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7581995&form=6&db=m Quantity of Na/K-ATPase and glucose transporters in the plasma membrane of rat adipocytes is reduced by in vivo triiodothyronine. causal interaction,diagnostic usage,unassigned 3,1,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7738467&form=6&db=m Dietary omega 6 fatty acids and the effects of hyperthyroidism in mice. causal interaction,diagnostic usage,unassigned 4,4,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7814039&form=6&db=m Dorsolateral prostatic phosphomonoesterases and adenosine triphosphatases in hypo- and hyperthyroid rats. unassigned - 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11735082&form=6&db=m Platelet Na,K-adenosine triphosphatase as a tissue marker of hyperthyroidism. diagnostic usage,ongoing research,unassigned 4,2,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15598668&form=6&db=m Hyperthyroidism and cation pumps in human skeletal muscle. causal interaction,unassigned 4,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17032726&form=6&db=m Thyroid hormone increases mRNA and protein expression of Na+-K+-ATPase alpha2 and beta1 subunits in human skeletal muscles. diagnostic usage,ongoing research,unassigned 1,4,0 7.2.2.13 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32432016&form=6&db=m Thyrotoxic Periodic Paralysis With Features of Andersen-Tawil Syndrome: A Case Report and Literature Review. causal interaction,diagnostic usage,unassigned 4,2,0 7.2.2.13 Hypertriglyceridemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21995900&form=6&db=m Gender specific influence of fish oil or atorvastatin on functional properties of renal Na,K-ATPase in healthy Wistar and hypertriglyceridemic rats. causal interaction,ongoing research,unassigned 1,2,0 7.2.2.13 Hypertriglyceridemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22438203&form=6&db=m Antioxidant SMe1EC2 may attenuate the disbalance of sodium homeostasis in the organism induced by higher intake of cholesterol. diagnostic usage,therapeutic application,unassigned 1,1,0 7.2.2.13 Hypertrophy, Left Ventricular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12675854&form=6&db=m Effect of green tea extract on cardiac hypertrophy following 5/6 nephrectomy in the rat. causal interaction,therapeutic application,unassigned 1,2,0 7.2.2.13 Hypertrophy, Left Ventricular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16864425&form=6&db=m Effects of crocetin on the matrix metalloproteinases in cardiac hypertrophy induced by norepinephrine in rats. causal interaction,unassigned 4,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2471006&form=6&db=m Dopamine and dobutamine induce hypokalemia in anesthetized dogs. unassigned - 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3002673&form=6&db=m Effects of alterations in cell phenotype and hypokalemia on sodium-potassium pump activity in rabbit vascular smooth muscle. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6445796&form=6&db=m Effects of hypokalemia on the cardiotropic actions of digoxin in dogs. Correlation with inhibition of cardiac Na+,K+-adenosine triphosphatase. ongoing research,therapeutic application,unassigned 3,3,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7629896&form=6&db=m Disturbances of potassium homeostasis in poisoning. unassigned - 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8285977&form=6&db=m Hypokalemia after pediatric albuterol overdose: a case series. therapeutic application,unassigned 1,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8390478&form=6&db=m Regulation of collecting tubule adenosine triphosphatases by aldosterone and potassium. ongoing research,unassigned 2,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8886172&form=6&db=m Hypokalemia, high erythrocyte Na+ and low erythrocyte Na,K-ATPase in relatives of patients dying from sudden unexplained death syndrome in north-east Thailand and in survivors from near-fatal attacks. diagnostic usage,unassigned 1,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10460986&form=6&db=m Age-related changes in renal function, membrane protein metabolism, and Na,K-ATPase activity and abundance in hypokalemic F344 x BNF(1) rats. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10600933&form=6&db=m In rat tIMCD, NH4+ uptake by Na+-K+-ATPase is critical to net acid secretion during chronic hypokalemia. ongoing research,unassigned 2,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11739117&form=6&db=m In rat inner medullary collecting duct, NH uptake by the Na,K-ATPase is increased during hypokalemia. ongoing research,unassigned 3,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14656369&form=6&db=m Mechanisms of NH4+ and NH3 transport during hypokalemia. therapeutic application,unassigned 1,0 7.2.2.13 Hypokalemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17556873&form=6&db=m Hypokalemic periodic paralysis in a patient with acquired growth hormone deficiency. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypokalemic Periodic Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17556873&form=6&db=m Hypokalemic periodic paralysis in a patient with acquired growth hormone deficiency. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hyponatremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11553514&form=6&db=m Na-K-ATPase activity decreases with aging in female rat brain synaptosomes. diagnostic usage,ongoing research,unassigned 4,3,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2160889&form=6&db=m Effect of amiodarone on 3H-ouabain binding sites in human skeletal muscle. causal interaction,unassigned 1,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2847536&form=6&db=m Effects of thyroid hormone on Na+-K+ transport in resting and stimulated rat skeletal muscle. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2987290&form=6&db=m Erythrocyte sodium/potassium adenosine triphosphatase in thyroid disease and nonthyroidal illness. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3027130&form=6&db=m Triiodothyronine enhances renal response to aldosterone in the rabbit collecting tubule. ongoing research,unassigned 2,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6294446&form=6&db=m Status of the red cell Na,K-pump in hyper- and hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,4,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8071999&form=6&db=m Amiodarone decreases Na,K-ATPase alpha 2 and beta 2 expression specifically in cardiac ventricle. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8906618&form=6&db=m Differential effects of hypothyroidism on Na-K-ATPase mRNA alpha isoforms in the developing rat brain. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10076191&form=6&db=m Stimulation of Na,K-ATPase by hypothyroidism in the thyroid gland. unassigned - 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15163334&form=6&db=m Erythrocyte Na/K-ATPase is increased in subjects with subclinical hypothyroidism. diagnostic usage,unassigned 2,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16416660&form=6&db=m Selective decrease of Na+/k+ -ATPase activity in the brain of hypothyroid rats. diagnostic usage,ongoing research,therapeutic application,unassigned 1,3,3,0 7.2.2.13 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16951134&form=6&db=m Developmental acquisition of T3-sensitive Na-K-ATPase stimulation by rat alveolar epithelial cells. ongoing research,unassigned 3,0 7.2.2.13 Hypoventilation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8931839&form=6&db=m Inhibition of erythrocyte Na,K-ATPase activity during anticipatory hypoventilation in micropigs. causal interaction,diagnostic usage,unassigned 4,2,0 7.2.2.13 Ileitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9886998&form=6&db=m Corticosteroids reverse the inhibition of Na-glucose cotransport in the chronically inflamed rabbit ileum. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Immune System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30192896&form=6&db=m Comparative transcriptome analysis of Eriocheir japonica sinensis response to environmental salinity. unassigned - 7.2.2.13 Infarction, Middle Cerebral Artery http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2475924&form=6&db=m Effect of enhanced capillary activity on the blood-brain barrier during focal cerebral ischemia in cats. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,3,0 7.2.2.13 Infarction, Middle Cerebral Artery http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19463256&form=6&db=m Pre-ischemic treatment with memantine reversed the neurochemical and behavioural parameters but not energy metabolites in middle cerebral artery occluded rats. unassigned - 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2155910&form=6&db=m Evidence for poliovirus-induced cytoplasmic alkalinization in HeLa cells. therapeutic application,unassigned 1,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2530698&form=6&db=m Mitochondrial ATP-ASE as a measure of uncoupling of rat muscle mitochondria in experimental infection with Trichinella spiralis and Trichinella pseudospiralis. ongoing research,unassigned 1,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2833572&form=6&db=m Tacaribe virus infection may induce inhibition of the activity of the host cell Ca2+ and Na+/K+ pumps. ongoing research,unassigned 1,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2895174&form=6&db=m Intestinal enzyme profiles in normal and rotavirus-infected mice. ongoing research,unassigned 4,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3029278&form=6&db=m Cation content in poliovirus-infected HeLa cells. unassigned - 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8607259&form=6&db=m Inhibition of Na+K+ATPase activity in membranes of Sindbis virus-infected chick cells. causal interaction,unassigned 3,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8798517&form=6&db=m Translational regulation of Na,K-ATPase alpha1 and beta1 polypeptide expression in epithelial cells. diagnostic usage,ongoing research,unassigned 1,4,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9762418&form=6&db=m The effect of interferon therapy on erythrocyte membrane Na+,K+ ATP activity in patients with chronic hepatitis B and C virus infections. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9769335&form=6&db=m Augmentation of lung liquid clearance via adenovirus-mediated transfer of a Na,K-ATPase beta1 subunit gene. diagnostic usage,therapeutic application,unassigned 1,1,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15636635&form=6&db=m Modulation of epithelial sodium channel (ENaC) expression in mouse lung infected with Pseudomonas aeruginosa. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,2,1 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15784574&form=6&db=m DNA immunization with Na+-K+ ATPase (Sseat-6) induces protective immunity to larval Strongyloides stercoralis in mice. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16539007&form=6&db=m Inhibition of Na,K-ATPase activity reduces Babesia gibsoni infection of canine erythrocytes with inherited high K, low Na concentrations. causal interaction,unassigned 1,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17969856&form=6&db=m [Effect of extracted ZG from gardenia on Hep-2 cell membrane post infected with parainfluenza virus type 1 (PIV-1)] causal interaction,diagnostic usage,ongoing research,unassigned 1,3,3,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19286978&form=6&db=m Ubiquitination Participates in the Lysosomal Degradation of the Na,K-ATPase in Steady-State Conditions. unassigned - 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23132959&form=6&db=m Leptospira and inflammation. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25653449&form=6&db=m ATP1A1-mediated Src signaling inhibits coronavirus entry into host cells. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26999598&form=6&db=m Influenza leaves a TRAIL to pulmonary edema. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26999599&form=6&db=m Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28458673&form=6&db=m Inflammatory Responses Regulating Alveolar Ion Transport during Pulmonary Infections. ongoing research,unassigned 2,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29462184&form=6&db=m Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29532265&form=6&db=m Downregulation of Aquaporins (AQP1 and AQP5) and Na,K-ATPase in Porcine Reproductive and Respiratory Syndrome Virus-Infected Pig Lungs. causal interaction,unassigned 2,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30284628&form=6&db=m The influence of porcine epidemic diarrhea virus on pig small intestine mucosal epithelial cell function. unassigned - 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31381610&form=6&db=m The alpha-1 subunit of the Na+,K+-ATPase (ATP1A1) is required for macropinocytic entry of respiratory syncytial virus (RSV) in human respiratory epithelial cells. diagnostic usage,unassigned 3,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31697939&form=6&db=m Experimental infection by Neospora caninum in gerbil reduces activity of enzymes involved in energy metabolism. therapeutic application,unassigned 2,0 7.2.2.13 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32174134&form=6&db=m Helicobacter pylori infection impairs chaperone-assisted maturation of Na,K-ATPase in gastric epithelium. causal interaction,unassigned 2,0 7.2.2.13 Inflammatory Bowel Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2832138&form=6&db=m Inverse relationship between colonic (Na+ + K+)-ATPase activity and degree of mucosal inflammation in inflammatory bowel disease. causal interaction,unassigned 4,0 7.2.2.13 Inflammatory Bowel Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3016558&form=6&db=m Inhibition of human colonic (Na+ + K+)-ATPase by arachidonic and linoleic acid. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Inflammatory Bowel Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12959741&form=6&db=m Hypothalamic digoxin, hemispheric chemical dominance, and inflammatory bowel disease. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Inflammatory Bowel Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25462166&form=6&db=m Molecular mechanism of regulation of villus cell Na-K-ATPase in the chronically inflamed mammalian small intestine. unassigned - 7.2.2.13 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8383696&form=6&db=m Immortalization of polarized rat retinal pigment epithelium. unassigned - 7.2.2.13 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8916902&form=6&db=m Localization of cytoplasmic and extracellular domains of Na,K-ATPase by epitope tag insertion. diagnostic usage,ongoing research,unassigned 3,1,0 7.2.2.13 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26999598&form=6&db=m Influenza leaves a TRAIL to pulmonary edema. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30892074&form=6&db=m Cardiac glycosides decrease influenza virus replication by inhibiting cell protein translational machinery. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,3,0 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1438101&form=6&db=m [Theoretical aspects of the relationship between diabetic macroangiopathy and hyperinsulinism] causal interaction,unassigned 1,0 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1661300&form=6&db=m Effect of chronic renal failure on Na,K-ATPase alpha 1 and alpha 2 mRNA transcription in rat skeletal muscle. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2096434&form=6&db=m [Arterial hypertension in patients with obesity. Role of hyperinsulinism and insulin resistance] causal interaction,unassigned 4,0 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2190920&form=6&db=m Differences in blacks and whites with essential hypertension: biochemistry and endocrine. State of the art lecture. unassigned - 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2544470&form=6&db=m Membrane lipid alterations and Na+-pumping activity in erythrocytes from IDDM and NIDDM subjects. unassigned - 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2853084&form=6&db=m Lymphocyte (Na,K) ATPase-dependent 86Rb+ uptake in human obesity. unassigned - 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6328233&form=6&db=m Hormonal and nutritional enhancement of Na+-K+-ATPase activity may aid the prevention and treatment of essential hypertension. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9608544&form=6&db=m Response of insulin, glucagon, lactate, and nonesterified fatty acids to glucose in visceral obesity with and without NIDDM: relationship to hypertension. causal interaction,unassigned 4,0 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16139689&form=6&db=m Role of muscle in regulating extracellular [K+]. therapeutic application,unassigned 1,0 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26601314&form=6&db=m pNaKtide inhibits Na/K-ATPase reactive oxygen species amplification and attenuates adipogenesis. causal interaction,unassigned 4,0 7.2.2.13 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32540646&form=6&db=m Estrogen-progestin oral contraceptive and nicotine exposure synergistically confers cardio-renoprotection in female Wistar rats. causal interaction,therapeutic application,unassigned 3,3,0 7.2.2.13 Insulinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8833343&form=6&db=m Sulfonylurea binding to a low-affinity site inhibits the Na/K-ATPase and the KATP channel in insulin-secreting cells. ongoing research,unassigned 4,0 7.2.2.13 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30388404&form=6&db=m Germline De Novo Mutations in ATP1A1 Cause Renal Hypomagnesemia, Refractory Seizures, and Intellectual Disability. unassigned - 7.2.2.13 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31705535&form=6&db=m Hereditary spastic paraplegia is a novel phenotype for germline de novo ATP1A1 mutation. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Iron Overload http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26197432&form=6&db=m Effects of Iron Overload on the Activity of Na,K-ATPase and Lipid Profile of the Human Erythrocyte Membrane. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,4,0 7.2.2.13 Ischemic Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18343354&form=6&db=m Platelet membrane fluidity and Na+/K+ ATPase activity in acute stroke. diagnostic usage,ongoing research,therapeutic application,unassigned 4,3,2,0 7.2.2.13 Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16139688&form=6&db=m Multiple functions of Na,K-ATPase in epithelial cells. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29937894&form=6&db=m Changes in erythrocyte ATPase activity under different pathological conditions. unassigned - 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1329041&form=6&db=m [Effect of hemodialysis on the level of serum digoxin-like substance and sodium-potassium pump activity in erythrocytes from patients with chronic renal failure] diagnostic usage,ongoing research,unassigned 4,4,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1661300&form=6&db=m Effect of chronic renal failure on Na,K-ATPase alpha 1 and alpha 2 mRNA transcription in rat skeletal muscle. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1668126&form=6&db=m [Activity of the sodium-potassium pump and values of sodium and potassium in erythrocytes in patients with non-dialyzed renal failure and arterial hypertension treated conservatively] diagnostic usage,ongoing research,therapeutic application,unassigned 3,2,1,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1928061&form=6&db=m Extrarenal potassium tolerance in chronic renal failure: implications for the treatment of acute hyperkalemia. causal interaction,unassigned 4,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2424645&form=6&db=m A circulating Na+-K+ATPase inhibitor, erythrocyte sodium transport and hypertension in patients with chronic renal failure. causal interaction,unassigned 4,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2437805&form=6&db=m Na+-K+ pump in chronic renal failure. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,3,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2441015&form=6&db=m Measurement of circulating sodium-pump inhibitory activity in uraemia and essential hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2850986&form=6&db=m Atrial natriuretic peptide concentration and natriuretic hormone activity in plasma of patients with chronic renal failure. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8177473&form=6&db=m Increased cytosolic free sodium in platelets from patients with early-stage chronic renal failure. causal interaction,diagnostic usage,unassigned 3,2,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8287666&form=6&db=m Effects of haemodialysis and continuous ambulatory peritoneal dialysis on abnormalities of ion transport in vivo in patients with chronic renal failure. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,4,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8697702&form=6&db=m Increased inward passive permeability in vitro to sodium in uraemic erythrocytes. ongoing research,unassigned 1,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9655151&form=6&db=m The role of Na,K-ATPase inhibitors in hypertension and end-stage renal disease. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16516547&form=6&db=m Neuropathy, axonal Na+/K+ pump function and activity-dependent excitability changes in end-stage kidney disease. causal interaction,unassigned 1,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17535729&form=6&db=m Molecular insights into uremic cardiomyopathy: cardiotonic steroids and na/k ATPase signaling. causal interaction,unassigned 2,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21292813&form=6&db=m Endogenous cardiotonic steroids in chronic renal failure. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Kidney Failure, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30041232&form=6&db=m Dysnatremia in Renal Failure. causal interaction,unassigned 3,0 7.2.2.13 Kidney Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18006011&form=6&db=m Na,K-adenosine triphosphatase alpha1-subunit predicts survival of renal clear cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,3 7.2.2.13 Kidney Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28484360&form=6&db=m Downregulation of ATP1A1 promotes cancer development in renal cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,4 7.2.2.13 Kidney Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30120256&form=6&db=m Na/K-ATPase Y260 Phosphorylation-mediated Src Regulation in Control of Aerobic Glycolysis and Tumor Growth. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,3,0 7.2.2.13 Kwashiorkor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=150319&form=6&db=m High erythrocyte membrane (Na+ + K+)-ATPase in kwashiorkor, in vivo reversal by diuretic. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Kwashiorkor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2120031&form=6&db=m The absence of adapted sodium and potassium transport in erythrocytes of cerebral palsied children with secondary malnutrition. unassigned - 7.2.2.13 Labyrinth Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33221698&form=6&db=m Immunohistochemical location of Na+, K+-ATPase ?1 subunit in the human inner ear. diagnostic usage,ongoing research,unassigned 2,2,0 7.2.2.13 Leptospirosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7671008&form=6&db=m Inhibition of Na,K-ATPase by an endotoxin extracted from Leptospira interrogans: a possible mechanism for the physiopathology of leptospirosis. causal interaction,unassigned 4,0 7.2.2.13 Leptospirosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9239307&form=6&db=m Na,K-ATPase: a molecular target for Leptospira interrogans endotoxin. therapeutic application,unassigned 1,0 7.2.2.13 Leptospirosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15933785&form=6&db=m Leptospira interrogans activation of peripheral blood monocyte glycolipoprotein demonstrated in whole blood by the release of IL-6. unassigned - 7.2.2.13 Leptospirosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22323544&form=6&db=m Downregulation of the Na/K-ATPase Pump by Leptospiral Glycolipoprotein Activates the NLRP3 Inflammasome. unassigned - 7.2.2.13 Leptospirosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23132959&form=6&db=m Leptospira and inflammation. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Leukemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=705007&form=6&db=m New microbial secondary metabolites under preclinical development for cancer treatment. causal interaction,therapeutic application,unassigned 4,2,0 7.2.2.13 Leukemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1322833&form=6&db=m Membrane interactions of mastoparan analogues related to their differential effects on protein kinase C, Na, K-ATPase and HL60 cells. unassigned - 7.2.2.13 Leukemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1847132&form=6&db=m Membrane interactions of amphiphilic polypeptides mastoparan, melittin, polymyxin B, and cardiotoxin. Differential inhibition of protein kinase C, Ca2+/calmodulin-dependent protein kinase II and synaptosomal membrane Na,K-ATPase, and Na+ pump and differentiation of HL60 cells. causal interaction,unassigned 1,0 7.2.2.13 Leukemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2152929&form=6&db=m Inhibition of Na,K-ATPase and sodium pump by protein kinase C regulators sphingosine, lysophosphatidylcholine, and oleic acid. ongoing research,unassigned 3,0 7.2.2.13 Leukemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2159369&form=6&db=m Inhibition of protein kinase C, (sodium plus potassium)-activated adenosine triphosphatase, and sodium pump by synthetic phospholipid analogues. ongoing research,unassigned 3,0 7.2.2.13 Leukemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19442964&form=6&db=m Plasma membrane depolarization and Na,K-ATPase impairment induced by mitochondrial toxins augment leukemia cell apoptosis via a novel mitochondrial amplification mechanism. unassigned - 7.2.2.13 Leukemia, Erythroblastic, Acute http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2422034&form=6&db=m Erythroid differentiation and the Na+,K+-pump in ouabain-sensitive and ouabain-resistant Friend erythroleukemia cell lines. ongoing research,unassigned 2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1656777&form=6&db=m Upregulation of rat lung Na-K-ATPase during hyperoxic injury. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7551375&form=6&db=m Lung liquid clearance and Na,K-ATPase during acute hyperoxia and recovery in rats. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,4,2 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8730006&form=6&db=m Hyperbaric oxygenation upregulates rat lung Na,K-ATPase. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9729576&form=6&db=m Epidermal growth factor increases lung liquid clearance in rat lungs. causal interaction,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9925907&form=6&db=m Influence of inhaled nitric oxide and hyperoxia on Na,K-ATPase expression and lung edema in newborn piglets. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10424567&form=6&db=m Adenoviral-mediated overexpression of the NA,K-ATPase beta1 subunit gene increases lung edema clearance and improves survival during acute hyperoxic lung injury in rats. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10424573&form=6&db=m Ventilator-associated lung injury decreases lung ability to clear edema and downregulates alveolar epithelial cell Na,K-adenosine triphosphatase function. ongoing research,unassigned 3,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10424620&form=6&db=m Hyperoxic lung injury increases HOCl-modified lung proteins and NA,K-adenosine triphosphatase nitrotyrosine content. causal interaction,unassigned 3,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10903255&form=6&db=m beta-adrenergic stimulation restores rat lung ability to clear edema in ventilator-associated lung injury. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12682448&form=6&db=m Mechanisms of pulmonary edema clearance during acute hypoxemic respiratory failure: role of the Na,K-ATPase. causal interaction,ongoing research,therapeutic application,unassigned 3,3,4,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12947023&form=6&db=m Na,K-ATPase gene transfer increases liquid clearance during ventilation-induced lung injury. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15781423&form=6&db=m Decreased expression of both the alpha1- and alpha2-subunits of the Na-K-ATPase reduces maximal alveolar epithelial fluid clearance. unassigned - 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17185651&form=6&db=m Leptin resistance protects mice from hyperoxia-induced acute lung injury. ongoing research,therapeutic application,unassigned 3,3,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17530222&form=6&db=m Alveolar epithelium and Na,K-ATPase in acute lung injury. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,1 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17972021&form=6&db=m Regulation of Na,K-ATPase during acute lung injury. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18424620&form=6&db=m Overexpression of the Na-K-ATPase alpha2-subunit improves lung liquid clearance during ventilation-induced lung injury. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20160150&form=6&db=m Role of ubiquitination in Na,K-ATPase regulation during lung injury. ongoing research,unassigned 4,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22396561&form=6&db=m Ubiquitination and proteolysis in acute lung injury. causal interaction,therapeutic application,unassigned 1,2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24175969&form=6&db=m Oleic acid inhibits lung Na/K-ATPase in mice and induces injury with lipid body formation in leukocytes and eicosanoid production. ongoing research,unassigned 4,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25265888&form=6&db=m Murine lung injury caused by Leptospira interrogans glycolipoprotein, a specific Na/K-ATPase inhibitor. causal interaction,therapeutic application,unassigned 4,2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25399325&form=6&db=m Na/K-ATPase assay in the intact mice lung subjected to perfusion. diagnostic usage,unassigned 3,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28644844&form=6&db=m Carbonic anhydrase inhibitor attenuates ischemia-reperfusion induced acute lung injury. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,2,0 7.2.2.13 Lung Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29109255&form=6&db=m HIF and HOIL-1L-mediated PKC? degradation stabilizes plasma membrane Na,K-ATPase to protect against hypoxia-induced lung injury. therapeutic application,unassigned 1,0 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2968903&form=6&db=m [Effect of electron-acceptor compounds on the immunoregulatory mechanisms and ATPase activity of the erythrocyte membranes from patients with inoperable lung cancer] ongoing research,therapeutic application,unassigned 2,3,0 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6149925&form=6&db=m [Ca2+, Mg2+-ATPase activity of the erythrocyte membrane in patients with lung cancer] diagnostic usage,ongoing research,unassigned 2,3,0 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9579847&form=6&db=m Predicting chemotherapeutic response to small-cell lung cancer of platinum compounds by thallium-201 single-photon emission computerized tomography. causal interaction,ongoing research,therapeutic application,unassigned 3,4,4,0 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21812995&form=6&db=m Long-term exposure to hypoxia inhibits tumor progression of lung cancer in rats and mice. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22345575&form=6&db=m Na,K-ATPase is a target of cigarette smoke and reduced expression predicts poor patient outcome of smokers with lung cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24066843&form=6&db=m Steroidal Cardiac Na+/K+ ATPase Inhibitors Exhibit Strong Anti-Cancer Potential in Vitro and in Prostate and Lung Cancer Xenografts in Vivo. causal interaction,diagnostic usage,therapeutic application,unassigned 3,1,4,0 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27431571&form=6&db=m Cardiac glycosides display selective efficacy for STK11 mutant lung cancer. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28430151&form=6&db=m Na/K Pump and Beyond: Na/K-ATPase as a Modulator of Apoptosis and Autophagy. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,4 7.2.2.13 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29101813&form=6&db=m Cytotoxic and cytostatic effects of digitoxigenin monodigitoxoside (DGX) in human lung cancer cells and its link to Na,K-ATPase. ongoing research,unassigned 3,0 7.2.2.13 Lymphatic Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21812995&form=6&db=m Long-term exposure to hypoxia inhibits tumor progression of lung cancer in rats and mice. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1651446&form=6&db=m Genotoxicity of 2-amino-6-N-hydroxyadenine (AHA) to mouse lymphoma and CHO cells. unassigned - 7.2.2.13 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1651447&form=6&db=m Mutagenicity of 2-amino-N6-hydroxyadenine (AHA) at three loci in L5178Y/tk+/- mouse lymphoma cells: molecular and preliminary cytogenetic characterizations of AHA-induced tk-/- mutants. therapeutic application,unassigned 1,0 7.2.2.13 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31058149&form=6&db=m A Tool for Computation of Changes in Na+, K+, Cl- Channels and Transporters Due to Apoptosis by Data on Cell Ion and Water Content Alteration. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,2,0 7.2.2.13 Lymphoma, Non-Hodgkin http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12745628&form=6&db=m Cerebral chemical dominance and neural regulation of cell division, cell proliferation, neoplastic transformation, and genomic function. diagnostic usage,unassigned 4,0 7.2.2.13 Magnesium Deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2826728&form=6&db=m Effects of dietary magnesium on sodium-potassium pump action in the heart of rats. therapeutic application,unassigned 1,0 7.2.2.13 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=123640&form=6&db=m Development of Na+-K+-ATPase in neonatal rat brain synaptosomes after perinatal protein malnutrition. unassigned - 7.2.2.13 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=911738&form=6&db=m Effect of exercise during rehabilitation on swimming performance, metabolism and function of muscle in rats. diagnostic usage,therapeutic application,unassigned 1,1,0 7.2.2.13 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2120031&form=6&db=m The absence of adapted sodium and potassium transport in erythrocytes of cerebral palsied children with secondary malnutrition. unassigned - 7.2.2.13 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2257076&form=6&db=m Sick cell syndrome in a burned patient. causal interaction,unassigned 4,0 7.2.2.13 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6257473&form=6&db=m Effects of malnutrition and quipazine on rat cerebral cortex ATPase activity during development. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17044775&form=6&db=m Effect of nutritional status and ozone exposure on some biomarkers of oxidative stress in rat brain regions. unassigned - 7.2.2.13 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20975704&form=6&db=m Ouabain protects against adverse developmental programming of the kidney. ongoing research,unassigned 4,0 7.2.2.13 Mania http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7711160&form=6&db=m The Na,K-ATPase hypothesis for bipolar illness. unassigned - 7.2.2.13 Mania http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8539431&form=6&db=m An animal model for mania: preliminary results. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Mania http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19524007&form=6&db=m Effect of ouabain on sodium pump alpha-isoform expression in an animal model of mania. causal interaction,ongoing research,therapeutic application,unassigned 4,3,4,0 7.2.2.13 Mania http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23315013&form=6&db=m Intracerebroventricular administration of ouabain alters synaptic plasticity and dopamine release in rat medial prefrontal cortex. causal interaction,therapeutic application,unassigned 1,3,0 7.2.2.13 Mania http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23643758&form=6&db=m Intracerebroventricular administration of ouabain, a Na/K-ATPase inhibitor, activates mTOR signal pathways and protein translation in the rat frontal cortex. causal interaction,ongoing research,therapeutic application,unassigned 1,3,2,0 7.2.2.13 Mastitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22535401&form=6&db=m Polymorphisms of the ATP1A1 gene associated with mastitis in dairy cattle. causal interaction,unassigned 1,0 7.2.2.13 Mastocytoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=140377&form=6&db=m Synthesis and activation of mitotic Ca2+-adenosinetriphosphatase during the cell cycle of mouse mastocytoma cells. ongoing research,unassigned 2,0 7.2.2.13 Medulloblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25052069&form=6&db=m Inhibition of epidermal growth factor signaling by the cardiac glycoside ouabain in medulloblastoma. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Medulloblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26286140&form=6&db=m Na,K-ATPase ?1-subunit is a target of sonic hedgehog signaling and enhances medulloblastoma tumorigenicity. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 7.2.2.13 Melanoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27545456&form=6&db=m Synergistic effects of ion transporter and MAP kinase pathway inhibitors in melanoma. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Melanoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32785105&form=6&db=m Toad Venom Antiproliferative Activities on Metastatic Melanoma: Bio-Guided Fractionation and Screening of the Compounds of Two Different Venoms. causal interaction,unassigned 4,0 7.2.2.13 Memory Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15689237&form=6&db=m Dysregulation of Na+/K+ ATPase by amyloid in APP+PS1 transgenic mice. ongoing research,unassigned 4,0 7.2.2.13 Memory Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17234593&form=6&db=m Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice. causal interaction,unassigned 4,0 7.2.2.13 Meniere Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8534024&form=6&db=m Na,K-ATPase in the cochlear lateral wall of human temporal bones with endolymphatic hydrops. ongoing research,unassigned 2,0 7.2.2.13 Meniere Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33221698&form=6&db=m Immunohistochemical location of Na+, K+-ATPase ?1 subunit in the human inner ear. diagnostic usage,ongoing research,unassigned 2,2,0 7.2.2.13 Metabolic Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8070076&form=6&db=m Understanding the sodium pump and its relevance to disease. causal interaction,unassigned 3,0 7.2.2.13 Metabolic Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31315267&form=6&db=m Metabolic Syndrome and Salt-Sensitive Hypertension in Polygenic Obese TALLYHO/JngJ Mice: Role of Na/K-ATPase Signaling. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17977697&form=6&db=m Regulation of the Na,K-ATPase: Special implications for cardiovascular complications of metabolic syndrome. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26601314&form=6&db=m pNaKtide inhibits Na/K-ATPase reactive oxygen species amplification and attenuates adipogenesis. causal interaction,unassigned 4,0 7.2.2.13 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31315267&form=6&db=m Metabolic Syndrome and Salt-Sensitive Hypertension in Polygenic Obese TALLYHO/JngJ Mice: Role of Na/K-ATPase Signaling. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33682297&form=6&db=m Impact of the metabolic syndrome on peripheral nerve structure and function in type 2 diabetes. causal interaction,unassigned 1,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12539047&form=6&db=m Haploinsufficiency of ATP1A2 encoding the Na+/K+ pump alpha2 subunit associated with familial hemiplegic migraine type 2. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12915447&form=6&db=m Significant linkage to migraine with aura on chromosome 11q24. causal interaction,unassigned 1,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15133718&form=6&db=m A novel missense ATP1A2 mutation in a Finnish family with familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15308625&form=6&db=m Kinetic alterations due to a missense mutation in the Na,K-ATPase alpha2 subunit cause familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15459825&form=6&db=m A G301R Na+/K+ -ATPase mutation causes familial hemiplegic migraine type 2 with cerebellar signs. causal interaction,unassigned 3,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15495990&form=6&db=m [From gene to disease; familial hemiplegic migraine as a result of mutations in a sodium-potassium pump gene] causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15843000&form=6&db=m Na,K-ATPase mutations in familial hemiplegic migraine lead to functional inactivation. causal interaction,unassigned 4,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15970628&form=6&db=m Functional effects of Na+,K+-ATPase gene mutations. causal interaction,unassigned 3,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16037212&form=6&db=m Alterations in the alpha2 isoform of Na,K-ATPase associated with familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16538223&form=6&db=m Two de novo mutations in the Na,K-ATPase gene ATP1A2 associated with pure familial hemiplegic migraine. unassigned - 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17234593&form=6&db=m Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice. causal interaction,unassigned 4,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17473835&form=6&db=m First case of compound heterozygosity in Na,K-ATPase gene ATP1A2 in familial hemiplegic migraine. causal interaction,unassigned 1,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17877748&form=6&db=m Amino acid changes in the amino terminus of the Na,K-adenosine triphosphatase alpha-2 subunit associated to familial and sporadic hemiplegic migraine. causal interaction,unassigned 1,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21731499&form=6&db=m Increased susceptibility to cortical spreading depression in the mouse model of familial hemiplegic migraine type 2. causal interaction,unassigned 3,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22067897&form=6&db=m Migraine- and dystonia-related disease-mutations of Na+/K+-ATPases: relevance of behavioral studies in mice to disease symptoms and neurological manifestations in humans. causal interaction,unassigned 2,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23954377&form=6&db=m Familial hemiplegic migraine mutations affect Na,K-ATPase domain interactions. unassigned - 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25476251&form=6&db=m Genome-wide screen for modifiers of Na + /K + ATPase alleles identifies critical genetic loci. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29513112&form=6&db=m Smooth muscle Ca causal interaction,unassigned 3,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30768809&form=6&db=m Involvement of the Na,K-ATPase isoforms in control of cerebral perfusion. causal interaction,unassigned 2,0 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32237043&form=6&db=m Astrocytes in Atp1a2-deficient heterozygous mice exhibit hyperactivity after induction of cortical spreading depression. unassigned - 7.2.2.13 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33578253&form=6&db=m A novel ATP1A2 variant associated with severe stepwise regression, hemiplegia, epilepsy and movement disorders in two unrelated patients. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12539047&form=6&db=m Haploinsufficiency of ATP1A2 encoding the Na+/K+ pump alpha2 subunit associated with familial hemiplegic migraine type 2. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12915447&form=6&db=m Significant linkage to migraine with aura on chromosome 11q24. causal interaction,unassigned 1,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15133718&form=6&db=m A novel missense ATP1A2 mutation in a Finnish family with familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15308625&form=6&db=m Kinetic alterations due to a missense mutation in the Na,K-ATPase alpha2 subunit cause familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15459825&form=6&db=m A G301R Na+/K+ -ATPase mutation causes familial hemiplegic migraine type 2 with cerebellar signs. causal interaction,unassigned 3,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15495990&form=6&db=m [From gene to disease; familial hemiplegic migraine as a result of mutations in a sodium-potassium pump gene] causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15843000&form=6&db=m Na,K-ATPase mutations in familial hemiplegic migraine lead to functional inactivation. causal interaction,unassigned 4,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16037212&form=6&db=m Alterations in the alpha2 isoform of Na,K-ATPase associated with familial hemiplegic migraine type 2. causal interaction,unassigned 4,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16538223&form=6&db=m Two de novo mutations in the Na,K-ATPase gene ATP1A2 associated with pure familial hemiplegic migraine. unassigned - 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17234593&form=6&db=m Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice. causal interaction,unassigned 4,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17473835&form=6&db=m First case of compound heterozygosity in Na,K-ATPase gene ATP1A2 in familial hemiplegic migraine. causal interaction,unassigned 1,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21731499&form=6&db=m Increased susceptibility to cortical spreading depression in the mouse model of familial hemiplegic migraine type 2. causal interaction,unassigned 3,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22067897&form=6&db=m Migraine- and dystonia-related disease-mutations of Na+/K+-ATPases: relevance of behavioral studies in mice to disease symptoms and neurological manifestations in humans. causal interaction,unassigned 2,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23954377&form=6&db=m Familial hemiplegic migraine mutations affect Na,K-ATPase domain interactions. unassigned - 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29513112&form=6&db=m Smooth muscle Ca causal interaction,unassigned 3,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30768809&form=6&db=m Involvement of the Na,K-ATPase isoforms in control of cerebral perfusion. causal interaction,unassigned 2,0 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32237043&form=6&db=m Astrocytes in Atp1a2-deficient heterozygous mice exhibit hyperactivity after induction of cortical spreading depression. unassigned - 7.2.2.13 Migraine with Aura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33578253&form=6&db=m A novel ATP1A2 variant associated with severe stepwise regression, hemiplegia, epilepsy and movement disorders in two unrelated patients. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Mitral Valve Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=136298&form=6&db=m Myocardial sarcolemmal ATPase in dogs with induced mitral insufficiency. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 7.2.2.13 Mitral Valve Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2990714&form=6&db=m Effect of prazosin treatment on the cardiac sarcolemmal ATPase in failing heart due to mitral insufficiency in dogs. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Mitral Valve Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6095632&form=6&db=m Effects of chronic digoxin treatment on cardiac function, electrolytes, and sarcolemmal ATPase in the canine failing heart due to chronic mitral regurgitation. causal interaction,ongoing research,therapeutic application,unassigned 3,4,3,0 7.2.2.13 Mucositis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34475704&form=6&db=m Spondias pinnata bark extract- an ameliorator of inflammatory derangement in etoposide induced mucositis: An experimental approach. therapeutic application,unassigned 1,0 7.2.2.13 Multiple Sclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Multiple Sclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18438950&form=6&db=m Imaging correlates of decreased axonal Na+/K+ ATPase in chronic multiple sclerosis lesions. causal interaction,unassigned 3,0 7.2.2.13 Muscle Weakness http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11985885&form=6&db=m Biochemical changes in primary culture of skeletal muscle cells following dimethoate exposure. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,4,0 7.2.2.13 Muscle Weakness http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12074739&form=6&db=m Muscle strength, Na,K-pumps, magnesium and potassium in patients with alcoholic liver cirrhosis -- relation to spironolactone. unassigned - 7.2.2.13 Muscular Atrophy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25654120&form=6&db=m Isoform-Specific Na,K-ATPase Alterations Precede Disuse-Induced Atrophy of Rat Soleus Muscle. therapeutic application,unassigned 3,0 7.2.2.13 Muscular Atrophy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29921483&form=6&db=m Melatonin attenuates chronic immobilization stress-induced muscle atrophy in rats: Influence on lactate-to-pyruvate ratios and Na+/K+ ATPase activity. causal interaction,ongoing research,therapeutic application,unassigned 2,1,2,0 7.2.2.13 Muscular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=135834&form=6&db=m [Ouabain-sensitive (Na+-K+)-ATPase activity of skeletal muscle sarcolemmal fraction in steroid myopathy mice (author's transl)] ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Muscular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=148933&form=6&db=m [3H]-Ouabain binding to peripheral organs of cats: effect of ethanol. causal interaction,unassigned 4,0 7.2.2.13 Muscular Dystrophies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1992297&form=6&db=m Study on the erythrocytes from myotonic dystrophy with multi-nuclear NMR. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Muscular Dystrophies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10813637&form=6&db=m Loop diuretics inhibit detubulation and vacuolation in amphibian muscle fibres exposed to osmotic shock. unassigned - 7.2.2.13 Muscular Dystrophies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32293933&form=6&db=m Isoform-specific Na,K-ATPase and membrane cholesterol remodeling in motor endplates in distinct mouse models of myodystrophy. therapeutic application,unassigned 1,0 7.2.2.13 Muscular Dystrophy, Duchenne http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6247454&form=6&db=m Erythrocyte ghost Na+,K+-adenosine triphosphatase in Duchenne muscular dystrophy. unassigned - 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1334706&form=6&db=m [The effect of carnosine on the activity of Na,K,ATPase: prospective uses in clinical cardiology] diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1649699&form=6&db=m Endogenous plasma Na,K-ATPase inhibitory activity and digoxin like immunoreactivity after acute myocardial infarction. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2555931&form=6&db=m [The role of an endogenous digoxin-like factor in regulating blood circulation and in the origin of arrhythmia in myocardial ischemia] ongoing research,unassigned 1,0 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271994&form=6&db=m Studies on the Na+-K+-ATPase in myocardial infarction. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15644955&form=6&db=m Influence of long-term treatment of imidapril on mortality, cardiac function, and gene expression in congestive heart failure due to myocardial infarction. causal interaction,ongoing research,therapeutic application,unassigned 1,2,3,0 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18418421&form=6&db=m Both enalapril and losartan attenuate sarcolemmal Na+-K+-ATPase remodeling in failing rat heart due to myocardial infarction. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19787147&form=6&db=m Ventricular performance and Na+-K+ ATPase activity are reduced early and late after myocardial infarction in rats. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,3,0 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29702256&form=6&db=m Association between activities of SOD, MDA and Na+-K+-ATPase in peripheral blood of patients with acute myocardial infarction and the complication of varying degrees of arrhythmia. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,2,1 7.2.2.13 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30278458&form=6&db=m Total Saponins of Aralia Elata (Miq) Seem Alleviate Calcium Homeostasis Imbalance and Endoplasmic Reticulum Stress-Related Apoptosis Induced by Myocardial Ischemia/Reperfusion Injury. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2555947&form=6&db=m [Na,K-ATPase activity in the myocardial sarcolemma in ischemia] causal interaction,unassigned 3,0 7.2.2.13 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2989573&form=6&db=m Effects of adenosine on transmembrane potential and sarcolemmal Na+-K+ ATPase activity of human atrial myocardium. therapeutic application,unassigned 3,0 7.2.2.13 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6329291&form=6&db=m The effect of lipid intermediates on Ca2+ and Na+ permeability and (Na+ + K+)-ATPase of cardiac sarcolemma. A possible role in myocardial ischemia. ongoing research,unassigned 2,0 7.2.2.13 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12763751&form=6&db=m Preconditioning attenuates ischemia-reperfusion-induced remodeling of Na+-K+-ATPase in hearts. causal interaction,unassigned 4,0 7.2.2.13 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17873015&form=6&db=m The balance between inactivation and activation of the Na+-K+ pump underlies the triphasic accumulation of extracellular K+ during myocardial ischemia. unassigned - 7.2.2.13 Myocardial Stunning http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9457363&form=6&db=m Myocardial stunning: do we know the mechanism? causal interaction,unassigned 3,0 7.2.2.13 Myocarditis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2854115&form=6&db=m Erythrocyte Na+-K+ ATPase activity inhibition & increase in red cell fragility in experimental myocarditis produced by Indian red scorpion venom. therapeutic application,unassigned 1,0 7.2.2.13 Myotonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=123512&form=6&db=m (Na+ + K+)-ATPase activity in rats with 20, 25-diazacholesterol-induced myotonia. ongoing research,unassigned 1,0 7.2.2.13 Myotonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=129059&form=6&db=m Myotonia induced by single doses of 20, 25 diazacholesterol: increased muscle and plasma desmosterol levels, unaltered (Na+ + K+)-ATPase activity of erythrocyte ghosts. diagnostic usage,unassigned 3,0 7.2.2.13 Myotonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3966511&form=6&db=m Myotonic muscular dystrophy associated with ritodrine tocolysis. unassigned - 7.2.2.13 Myotonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4271378&form=6&db=m Myotonia induced by diazacholesterol: increased (Na+ + K+)-ATPase activity of erythrocyte ghosts and development of cataracts. unassigned - 7.2.2.13 Myotonia Congenita http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16382442&form=6&db=m Na+-K+-ATPase is not involved in the warming-up phenomenon in generalized myotonia. unassigned - 7.2.2.13 Myotonic Dystrophy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6247457&form=6&db=m Erythrocyte membrane abnormalities in human myotonic dystrophy. causal interaction,unassigned 2,0 7.2.2.13 na+/k+-exchanging atpase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15050724&form=6&db=m Lactic acidosis progressively impairs dopamine uptake in rat striatal synaptosomes by a mechanism partially independent of the Na+/K+-ATPase dysfunction. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Narcolepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7859671&form=6&db=m Abnormal cortical unit activity of the reticular formation. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2831981&form=6&db=m Local and metastatic tumor growth and membrane properties of LM fibroblasts in athymic (nude) mice. diagnostic usage,ongoing research,therapeutic application,unassigned 4,4,1,0 7.2.2.13 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21812995&form=6&db=m Long-term exposure to hypoxia inhibits tumor progression of lung cancer in rats and mice. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25622969&form=6&db=m Translationally Controlled Tumor Protein induces epithelial to mesenchymal transition and promotes cell migration, invasion and metastasis. causal interaction,unassigned 4,0 7.2.2.13 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26858653&form=6&db=m Na,K-ATPase Isozymes in Colorectal Cancer and Liver Metastases. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29309535&form=6&db=m Carbonic anhydrase 2 inhibits epithelial-mesenchymal transition and metastasis in hepatocellular carcinoma. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,4,0 7.2.2.13 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30633912&form=6&db=m Circulating Tumor Cell Clustering Shapes DNA Methylation to Enable Metastasis Seeding. unassigned - 7.2.2.13 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33566445&form=6&db=m Quantitative protein expression of blood-brain barrier transporters in the vasculature of brain metastases of patients with lung and breast cancer. diagnostic usage,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=129155&form=6&db=m Changes in (Na+ + K+)-ATPase activity of Ehrlich ascites tumor cells produced by alteration of membrane fatty acid composition. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=135642&form=6&db=m Protein synthesis in dextran sulfate-treated ascites tumor cells. ongoing research,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=139301&form=6&db=m Choroid plexus papilloma. II. Ultrastructure and ultracytochemical localization of Na-K-ATPase. ongoing research,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1965805&form=6&db=m Kidney damage in rats bearing an adrenocorticotropin and prolactin secreting tumor. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2162743&form=6&db=m The influence of Na+,K(+)-pump blockade on doxorubicin-mediated cytotoxicity and DNA strand breakage in human tumor cells. causal interaction,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2294960&form=6&db=m Regulatory volume increase in Ehrlich ascites tumor cells. ongoing research,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2484547&form=6&db=m Subchronic treatment with vanadate does not potentiate the toxicity of cardiac glycosides. diagnostic usage,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2714300&form=6&db=m The relationship between 67Ga accumulation and ATP metabolism in tumor cells in vitro. therapeutic application,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2822144&form=6&db=m [Transmembrane transfer of sodium ions in cell membranes of normal and tumor cells] causal interaction,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2831981&form=6&db=m Local and metastatic tumor growth and membrane properties of LM fibroblasts in athymic (nude) mice. diagnostic usage,ongoing research,therapeutic application,unassigned 4,4,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2836053&form=6&db=m Photosensitizing effects of hematoporphyrin derivative and photofrin II on the plasma membrane enzymes 5'-nucleotidase, Na+K+-ATPase, and Mg2+-ATPase in R3230AC mammary adenocarcinomas. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2972256&form=6&db=m Motility, heat, and lactate production in ejaculated bovine sperm. causal interaction,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2986694&form=6&db=m Transport functions of the liver. Lack of correlation between hepatocellular ouabain uptake and binding to (Na+ + K+)-ATPase. diagnostic usage,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3019397&form=6&db=m Leucine transport in relation to the activities of Na+-H+ antiporter and Na+/K+ pump stimulated by serum and a tumor promoter. diagnostic usage,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3435486&form=6&db=m Calcium-mobilizing hormones and phorbol myristate acetate mediate heterologous desensitization of the hormone-sensitive hepatic Na+/K+ pump. diagnostic usage,ongoing research,therapeutic application,unassigned 1,3,2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3955087&form=6&db=m [Changes in the microviscosity of lipid bilayer membranes of various malignant cells and tumor transplantability] unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6087923&form=6&db=m Studies on the relationship between glycolysis and (Na+ + K+)-ATPase in cultured cells. ongoing research,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6168390&form=6&db=m A mouse homolog to the avian sarcoma virus src protein is a member of a protein kinase cascade. causal interaction,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6251054&form=6&db=m Phosphorylation of the beta subunit of Na+K+-ATPase in Ehrlich ascites tumor by a membrane-bound protein kinase. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6260791&form=6&db=m Regulation of phosphorylation of the beta-subunit of th Ehrlich ascites tumor Na+K+-ATPase by a protein kinase cascade. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6303414&form=6&db=m The common occurrence of ATP diphosphohydrolase in mammalian plasma membranes. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6311998&form=6&db=m Na+/K+ ATPase and cell growth IV: a metabolic marker of human brain tumors? causal interaction,diagnostic usage,ongoing research,unassigned 3,3,2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6324839&form=6&db=m Hyperthermia, Na+K+ATPase and lactic acid production in some human tumour cells. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7809995&form=6&db=m Electrolyte levels in neoplastic and nonneoplastic human urothelium after ouabain treatment: X-ray microanalysis of bulk hydrated samples. causal interaction,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8833343&form=6&db=m Sulfonylurea binding to a low-affinity site inhibits the Na/K-ATPase and the KATP channel in insulin-secreting cells. ongoing research,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9579847&form=6&db=m Predicting chemotherapeutic response to small-cell lung cancer of platinum compounds by thallium-201 single-photon emission computerized tomography. causal interaction,ongoing research,therapeutic application,unassigned 3,4,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10334201&form=6&db=m Analysis of 8-hydroxyguanine in rat kidney genomic DNA after administration of a renal carcinogen, ferric nitrilotriacetate. ongoing research,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10481582&form=6&db=m [Binding of the antileukemia drug Escherichia coli L-asparaginase to the plasma membrane of normal human mononuclear cells] ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10687899&form=6&db=m Digitoxin is a potential anticancer agent for several types of cancer. causal interaction,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11791942&form=6&db=m Modulation of Na,K-ATPase activity by immunoglobulins--II. Na,K-ATPase activity in erythrocytes from patients with various chronic disturbances. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12673711&form=6&db=m Analysis of the Na,K-ATPase alpha- and beta-subunit expression profiles of bladder cancer using tissue microarrays. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,2 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12890662&form=6&db=m Role of Na-K-ATPase in the assembly of tight junctions. therapeutic application,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15383549&form=6&db=m Translationally controlled tumor protein interacts with the third cytoplasmic domain of Na,K-ATPase alpha subunit and inhibits the pump activity in HeLa cells. ongoing research,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15970485&form=6&db=m Na/K-ATPase, endogenous digitalis like compounds and cancer development -- a hypothesis. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16139688&form=6&db=m Multiple functions of Na,K-ATPase in epithelial cells. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16488623&form=6&db=m Tumor necrosis factor alpha down-regulates the Na+-K+ ATPase and the Na+-K+2Cl- cotransporter in the kidney cortex and medulla. causal interaction,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17028035&form=6&db=m Tumor necrosis factor alpha alters Na+-K+ ATPase activity in rat cardiac myocytes: involvement of NF-kappaB, AP-1 and PGE2. therapeutic application,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17249331&form=6&db=m Biomarkers of induced electromagnetic field and cancer. causal interaction,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18006011&form=6&db=m Na,K-adenosine triphosphatase alpha1-subunit predicts survival of renal clear cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,3 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18228203&form=6&db=m Evidence for a potential tumor suppressor role for the Na,K-ATPase beta1-subunit. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19109419&form=6&db=m A link between FXYD3 (Mat-8)-mediated Na,K-ATPase regulation and differentiation of Caco-2 intestinal epithelial cells. causal interaction,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19128250&form=6&db=m S163 is critical for FXYD5 modulation of wound healing in airway epithelial cells. ongoing research,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19329430&form=6&db=m Changes in sodium pump expression dictate the effects of ouabain on cell growth. ongoing research,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19388149&form=6&db=m Roles of ERK, PI3 kinase, and PLC-gamma pathways induced by overexpression of translationally controlled tumor protein in HeLa cells. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19479337&form=6&db=m Over-expression of translationally controlled tumor protein in lens epithelial cells seems to be associated with cataract development. causal interaction,ongoing research,unassigned 2,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19671733&form=6&db=m Oleandrin-mediated inhibition of human tumor cell proliferation: importance of Na,K-ATPase alpha subunits as drug targets. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20501797&form=6&db=m Na,K-ATPase subunits as markers for epithelial-mesenchymal transition in cancer and fibrosis. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,3,1 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21784855&form=6&db=m Na/K-ATPase mimetic pNaKtide inhibits the growth of human cancer cells. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21812995&form=6&db=m Long-term exposure to hypoxia inhibits tumor progression of lung cancer in rats and mice. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21859451&form=6&db=m Potassium bicarbonate and D-ribose effects on A72 canine and HTB-126 human cancer cell line proliferation in vitro. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22006678&form=6&db=m Influence of N-methyl-D-aspartate receptors on ouabain activation of nuclear factor-?B in the rat hippocampus. causal interaction,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22345575&form=6&db=m Na,K-ATPase is a target of cigarette smoke and reduced expression predicts poor patient outcome of smokers with lung cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22351078&form=6&db=m Inhibition of Na/K-ATPase promotes myocardial tumor necrosis factor-alpha protein expression and cardiac dysfunction via calcium/mTOR signaling in endotoxemia. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22438768&form=6&db=m "The Lower Threshold" phenomenon in tumor cells toward endogenous digitalis-like compounds: Responsible for tumorigenesis? ongoing research,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22595010&form=6&db=m Modulators of Na/K-ATPase: a patent review. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24066843&form=6&db=m Steroidal Cardiac Na+/K+ ATPase Inhibitors Exhibit Strong Anti-Cancer Potential in Vitro and in Prostate and Lung Cancer Xenografts in Vivo. causal interaction,diagnostic usage,therapeutic application,unassigned 3,1,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24186357&form=6&db=m Ouabain-induced alterations of the apical junctional complex involve ?1 and ?1 Na,K-ATPase downregulation and ERK1/2 activation independent of caveolae in colorectal cancer cells. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,1 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24918292&form=6&db=m Up-regulation of Rhoa/Rho kinase pathway by translationally controlled tumor protein in vascular smooth muscle cells. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24922661&form=6&db=m The steroidal Na+/K+ ATPase inhibitor 3-[(R)-3-pyrrolidinyl]oxime derivative (3-R-POD) induces potent pro-apoptotic responses in colonic tumor cells. causal interaction,ongoing research,therapeutic application,unassigned 2,3,3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24942548&form=6&db=m A Steroidal Na+/K+ ATPase Inhibitor Triggers Pro-apoptotic Signaling and Induces Apoptosis in Prostate and Lung Tumor Cells. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25052069&form=6&db=m Inhibition of epidermal growth factor signaling by the cardiac glycoside ouabain in medulloblastoma. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25198786&form=6&db=m Na+/K+ ATPase inhibitors in cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25275034&form=6&db=m Effects of digoxin and Na, K-ATPase immunoexpression on human oral squamous carcinomas. diagnostic usage,ongoing research,therapeutic application,unassigned 2,3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25290152&form=6&db=m 21-Benzylidene digoxin: a proapoptotic cardenolide of cancer cells that up-regulates Na,K-ATPase and epithelial tight junctions. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,1,3,1 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25598355&form=6&db=m The Neuro-Immune Pathophysiology of Central and Peripheral Fatigue in Systemic Immune-Inflammatory and Neuro-Immune Diseases. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25622969&form=6&db=m Translationally Controlled Tumor Protein induces epithelial to mesenchymal transition and promotes cell migration, invasion and metastasis. causal interaction,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25770213&form=6&db=m Sodium-calcium Exchanger 1 Regulates Epithelial Cell Migration via Calcium-Dependent Extracellular-Signal-Regulated Kinase Signaling. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25796981&form=6&db=m Application of an LC-MS/MS method for the simultaneous quantification of human intestinal transporter proteins absolute abundance using a QconCAT technique. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25836370&form=6&db=m Glucocorticoids suppress renal cell carcinoma progression by enhancing Na,K-ATPase beta-1 subunit expression. causal interaction,unassigned 3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25854427&form=6&db=m Insulin induces phosphorylation of serine residues of translationally controlled tumor protein in 293T cells. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25976744&form=6&db=m Na/K-ATPase as a target for anticancer drugs: studies with perillyl alcohol. causal interaction,therapeutic application,unassigned 2,3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26122772&form=6&db=m ?-Benzylidene digoxin derivatives synthesis and molecular modeling: Evaluation of anticancer and the Na,K-ATPase activity effect. ongoing research,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26285814&form=6&db=m Novel somatic mutations and distinct molecular signature in aldosterone-producing adenomas. causal interaction,diagnostic usage,unassigned 2,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26286140&form=6&db=m Na,K-ATPase ?1-subunit is a target of sonic hedgehog signaling and enhances medulloblastoma tumorigenicity. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26342025&form=6&db=m Tumor interstitial fluid promotes malignant phenotypes of lung cancer independently of angiogenesis. causal interaction,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26494691&form=6&db=m Improved Brain Tumor Classification by Sodium MR Imaging: Prediction of IDH Mutation Status and Tumor Progression. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26579519&form=6&db=m Transcriptional regulators of Na,K-ATPase subunits. causal interaction,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26739091&form=6&db=m ENDOCRINE TUMOURS: The genomics of adrenocortical tumors. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26815163&form=6&db=m Activating mutations in CTNNB1 in aldosterone producing adenomas. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27027435&form=6&db=m Functional characterization and anti-cancer action of the clinical phase II cardiac Na+/K+ ATPase inhibitor istaroxime: in vitro and in vivo properties and cross talk with the membrane androgen receptor. therapeutic application,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27240166&form=6&db=m Tumor suppression in basal keratinocytes via dual non-cell-autonomous functions of a Na,K-ATPase beta subunit. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27845894&form=6&db=m Overexpression of ATPase Na+/+ transporting alpha 1 polypeptide, ATP1A1, correlates with clinical diagnosis and progression of esophageal squamous cell carcinoma. causal interaction,diagnostic usage,unassigned 4,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28160550&form=6&db=m Dysadherin specific drug conjugates for the treatment of thyroid cancers with aggressive phenotypes. ongoing research,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28430151&form=6&db=m Na/K Pump and Beyond: Na/K-ATPase as a Modulator of Apoptosis and Autophagy. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,4 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28484360&form=6&db=m Downregulation of ATP1A1 promotes cancer development in renal cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,4 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28720385&form=6&db=m Differences of lipid membrane modulation and oxidative stress by digoxin and 21-benzylidene digoxin. diagnostic usage,ongoing research,therapeutic application,unassigned 1,1,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29117117&form=6&db=m Anticancer and Immunogenic Properties of Cardiac Glycosides. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29653366&form=6&db=m Bufalin inhibits glioblastoma growth by promoting proteasomal degradation of the Na+/K+-ATPase ?1 subunit. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,4,1 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29777796&form=6&db=m Identification of a sodium pump Na+/K+ ATPase ?1-targeted peptide for PET imaging of breast cancer. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29867020&form=6&db=m Some Biological Consequences of the Inhibition of Na,K-ATPase by Translationally Controlled Tumor Protein (TCTP). causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30015972&form=6&db=m Combined assessment of low PGRMC1/positive ATP1A1 levels has enhanced prognostic value for renal cell carcinoma. causal interaction,diagnostic usage,therapeutic application,unassigned 2,4,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30120256&form=6&db=m Na/K-ATPase Y260 Phosphorylation-mediated Src Regulation in Control of Aerobic Glycolysis and Tumor Growth. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,3,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30216173&form=6&db=m Protective effects of St. John's wort in the hepatic ischemia/reperfusion injury in rats. diagnostic usage,ongoing research,unassigned 4,2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30372816&form=6&db=m Long-circulating and fusogenic liposomes loaded with a glucoevatromonoside derivative induce potent antitumor response. causal interaction,therapeutic application,unassigned 4,1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31000732&form=6&db=m RNA Sequencing Provides Novel Insights into the Transcriptome of Aldosterone Producing Adenomas. causal interaction,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31046873&form=6&db=m FCY-302, a Novel Small Molecule, Induces Apoptosis in Leukemia and Myeloma Cells by Attenuating Key Antioxidant and Mitochondrial Enzymes. unassigned - 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31207362&form=6&db=m Genomics of benign adrenocortical tumors. causal interaction,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31691615&form=6&db=m The Na/Ca Exchange as a Target for Antitumor Effect of 4Hz Pulsing Magnetic Field. causal interaction,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31803429&form=6&db=m Discovery of selective, antimetastatic and anti-cancer stem cell metallohelices via post-assembly modification. therapeutic application,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31972293&form=6&db=m Effects of dietary monoterpene, myrcene, administration on immune- and health-related genes expression in common carp gill following exposure to copper sulfate. therapeutic application,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32210281&form=6&db=m Inhibitors of Na+/K+ ATPase exhibit antitumor effects on multicellular tumor spheroids of hepatocellular carcinoma. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32480062&form=6&db=m Bufadienolide glycosides and bufadienolides from the whole plants of Helleborus lividus, and their cytotoxic activity. diagnostic usage,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32507359&form=6&db=m Adrenocortical tumorigenesis: Lessons from genetics. causal interaction,unassigned 2,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32684846&form=6&db=m Comprehensive analysis of the expression of sodium/potassium-ATPase ? subunits and prognosis of ovarian serous cystadenocarcinoma. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33713729&form=6&db=m Na/K-ATPase: Their role in cell adhesion and migration in cancer. ongoing research,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33776926&form=6&db=m Molecular Genetic and Genomic Alterations in Cushing's Syndrome and Primary Aldosteronism. causal interaction,unassigned 4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33900205&form=6&db=m GENETICS IN ENDOCRINOLOGY: Impact of race and sex on genetic causes of aldosterone-producing adenomas. causal interaction,unassigned 1,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33956349&form=6&db=m Inverse agonism at the Na/K-ATPase receptor reverses EMT in prostate cancer cells. causal interaction,diagnostic usage,unassigned 4,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34251542&form=6&db=m The expression of the alpha1 subunit of Na+/K+-ATPase is related to tumor development and clinical outcomes in gastric cancer. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,4,0 7.2.2.13 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34277634&form=6&db=m TRAF2 Is a Novel Ubiquitin E3 Ligase for the Na,K-ATPase ?-Subunit That Drives Alveolar Epithelial Dysfunction in Hypercapnia. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,3,0 7.2.2.13 Nephritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1666908&form=6&db=m [Na,K-Atpase in renal tissue of rats in the dynamics of experimental nephritis] ongoing research,unassigned 4,0 7.2.2.13 Nephrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11675400&form=6&db=m Increased synthesis and avp unresponsiveness of Na,K-ATPase in collecting duct from nephrotic rats. diagnostic usage,unassigned 3,0 7.2.2.13 Nephrosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31566427&form=6&db=m Nephrotic syndrome is associated with increased plasma K+ concentration, intestinal K+ losses, and attenuated urinary K+ excretion: a study in rats and humans. unassigned - 7.2.2.13 Nephrotic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7984251&form=6&db=m [Role of tubular Na-K-ATPase in nephrotic syndrome induced by puromycin in the rat] causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Nephrotic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8386183&form=6&db=m Mechanism of enhanced Na-K-ATPase activity in cortical collecting duct from rats with nephrotic syndrome. ongoing research,therapeutic application,unassigned 4,3,0 7.2.2.13 Nephrotic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10592934&form=6&db=m [Renal K-ATPases: structure, function and dysfunction] therapeutic application,unassigned 1,0 7.2.2.13 Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7859671&form=6&db=m Abnormal cortical unit activity of the reticular formation. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31425744&form=6&db=m Factors in the disease severity of ATP1A3 mutations: Impairment, misfolding, and allele competition. unassigned - 7.2.2.13 Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32301109&form=6&db=m Region- and neuronal-subtype-specific expression of Na,K-ATPase alpha and beta subunit isoforms in the mouse brain. diagnostic usage,unassigned 1,0 7.2.2.13 Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34114062&form=6&db=m Maturation of the Na,K-ATPase in the Endoplasmic Reticulum in Health and Disease. causal interaction,therapeutic application,unassigned 4,3,0 7.2.2.13 Neuroacanthocytosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32439941&form=6&db=m Decreased Na+/K+ ATPase Expression and Depolarized Cell Membrane in Neurons Differentiated from Chorea-Acanthocytosis Patients. ongoing research,unassigned 2,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=153571&form=6&db=m Weak A.C. magnetic field effects: changes in cell sodium pump activity following whole animal exposure. unassigned - 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1654277&form=6&db=m Tissue-specific expression of Na,K-ATPase beta-subunit. Does beta 2 expression correlate with tumorigenesis? diagnostic usage,ongoing research,unassigned 4,4,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1847297&form=6&db=m Resting membrane potential in 41A3 mouse neuroblastoma cells. Effect of increased glucose and galactose concentrations. causal interaction,therapeutic application,unassigned 3,3,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2546844&form=6&db=m Effect of galactose and glucose levels and sorbinil treatment on myo-inositol metabolism and Na+-K+ pump activity in cultured neuroblastoma cells. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2560348&form=6&db=m Determination of total (Na+ + K+)-ATPase activity of isolated or cultured cells. ongoing research,unassigned 4,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2839622&form=6&db=m Effect of increased glucose levels on Na+/K+-pump activity in cultured neuroblastoma cells. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,3,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6289988&form=6&db=m Alterations of membrane integrity and cellular constituents by arachidonic acid in neuroblastoma and glioma cells. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8177372&form=6&db=m Reduced Na+/K+ ATPase transport activity, resting membrane potential, and bradykinin-stimulated phosphatidylinositol synthesis by polyol accumulation in cultured neuroblastoma cells. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8391430&form=6&db=m Regulation of Na-K-Cl cotransport, Na,K-adenosine triphosphatase, and Na/H exchanger in human neuroblastoma NB-OK-1 cells by atrial natriuretic peptide. ongoing research,unassigned 2,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8412773&form=6&db=m Reversal of hyperglycemic-induced defects in myo-inositol metabolism and Na+/K+ pump activity in cultured neuroblastoma cells by normalizing glucose levels. ongoing research,unassigned 3,0 7.2.2.13 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27296892&form=6&db=m Direct interaction of beta-amyloid with Na,K-ATPase as a putative regulator of the enzyme function. causal interaction,ongoing research,unassigned 3,1,0 7.2.2.13 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9065836&form=6&db=m Inhibition of the electrogenic Na pump underlies delayed depolarization of cortical neurons after mechanical injury or glutamate. causal interaction,therapeutic application,unassigned 4,2,0 7.2.2.13 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9135086&form=6&db=m Na,K-ATPase: increases in alpha1-messenger RNA and decreases in alpha3-messenger RNA levels in aging rat cerebral cortex. causal interaction,unassigned 3,0 7.2.2.13 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30316912&form=6&db=m Repeated Restraint Stress Decreases Na,K-ATPase Activity via Oxidative and Nitrosative Damage in the Frontal Cortex of Rats. therapeutic application,unassigned 1,0 7.2.2.13 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31642819&form=6&db=m Localization dependent sensitivity of cerebral Na,K-ATPase to irradiation induced oxidative imbalance in rats. causal interaction,unassigned 2,0 7.2.2.13 Non-alcoholic Fatty Liver Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30588181&form=6&db=m Phenotypic Alteration of Hepatocytes in Non-Alcoholic Fatty Liver Disease. causal interaction,unassigned 2,0 7.2.2.13 Non-alcoholic Fatty Liver Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31877680&form=6&db=m Mechanistic Insight of Na/K-ATPase Signaling and HO-1 into Models of Obesity and Nonalcoholic Steatohepatitis. causal interaction,ongoing research,unassigned 2,1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=149887&form=6&db=m Activity of (Na+ + K+)-ATPase in the liver of animals with experimental obesity. ongoing research,unassigned 3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1334239&form=6&db=m Catecholamine-stimulated potassium transport in erythrocytes from normal and obese subjects. causal interaction,therapeutic application,unassigned 4,2,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2164130&form=6&db=m Impaired calcium metabolism associated with hypertension in Zucker obese rats. diagnostic usage,ongoing research,unassigned 2,3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2416703&form=6&db=m Red-cell sodium-potassium pump and sodium-lithium countertransport in human obesity. Re-evaluation of the methods and association in a Finnish population. ongoing research,unassigned 2,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2826967&form=6&db=m Erythrocyte Na-K-ATPase membrane activity in obese patients fed over a long-term period with a very-low-calorie diet. causal interaction,ongoing research,unassigned 1,3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2827939&form=6&db=m Abnormalities of sodium transport by sodium, potassium-activated adenosine triphosphatase in erythrocytes from obese children. unassigned - 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2831162&form=6&db=m Erythrocyte Na+-K+ ATPase activity in childhood: regulation by genetic factors independent of body weight. causal interaction,unassigned 1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2993783&form=6&db=m Heterogeneity of the erythrocyte Na-K pump status in human obesity. ongoing research,unassigned 4,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3969017&form=6&db=m Erythrocyte heat production in human obesity: microcalorimetric investigation of sodium-potassium pump and cell metabolism. ongoing research,unassigned 2,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6252462&form=6&db=m Reduced activity of the red-cell sodium-potassium pump in human obesity. causal interaction,ongoing research,unassigned 4,2,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6268982&form=6&db=m Hepatic sodium-potassium pump in obesity. ongoing research,unassigned 2,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6280694&form=6&db=m The Na+-K+-pump, energy metabolism, and obesity. therapeutic application,unassigned 1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6302395&form=6&db=m Erythrocyte sodium-potassium-stimulated adenosine triphosphatase activity is not related to obesity. unassigned - 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6323914&form=6&db=m Sodium-potassium pump in cultured fibroblasts from obese donors; no evidence for an inherent decrease of basal or insulin-stimulated activity. diagnostic usage,ongoing research,unassigned 1,1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6330493&form=6&db=m Changes in cell membrane Na,K-ATPase activity associated with induction of dietary obesity in the rat. ongoing research,unassigned 4,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7132741&form=6&db=m Altered erythrocyte Na+ + K+ pump in adolescent obesity. causal interaction,ongoing research,therapeutic application,unassigned 4,2,3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7453787&form=6&db=m Red-cell sodium-potassium pump in obesity. unassigned - 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11355012&form=6&db=m Mechanisms and consequences of Na+,K+-pump regulation by insulin and leptin. causal interaction,ongoing research,unassigned 4,3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16286513&form=6&db=m Hyperphagia and obesity in Na,K-ATPase alpha2 subunit-defective mice. causal interaction,ongoing research,therapeutic application,unassigned 2,2,3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16350567&form=6&db=m Altered brown adipose tissue and Na,K pump activities during diet-induced obesity and weight loss in rats. causal interaction,ongoing research,unassigned 2,3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17045221&form=6&db=m The role of obesity and its bioclinical correlates in the progression of chronic kidney disease. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17220688&form=6&db=m Animal and human tissue Na,K-ATPase in obesity and diabetes: A new proposed enzyme regulation. causal interaction,ongoing research,unassigned 4,4,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17444965&form=6&db=m Animal and human tissue Na,K-ATPase in normal and insulin-resistant states: regulation, behaviour and interpretative hypothesis on NEFA effects. causal interaction,unassigned 4,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26601314&form=6&db=m pNaKtide inhibits Na/K-ATPase reactive oxygen species amplification and attenuates adipogenesis. causal interaction,unassigned 4,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27598118&form=6&db=m The Role of Na/K-ATPase Signaling in Oxidative Stress Related to Obesity and Cardiovascular Disease. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30041449&form=6&db=m The Role of Na/K-ATPase Signaling in Oxidative Stress Related to Aging: Implications in Obesity and Cardiovascular Disease. causal interaction,ongoing research,therapeutic application,unassigned 2,2,1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31107610&form=6&db=m Inhibition of intestinal villus cell Na/K-ATPase mediates altered glucose and NaCl absorption in obesity-associated diabetes and hypertension. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31138824&form=6&db=m The Adipocyte Na/K-ATPase Oxidant Amplification Loop is the Central Regulator of Western Diet-Induced Obesity and Associated Comorbidities. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31315267&form=6&db=m Metabolic Syndrome and Salt-Sensitive Hypertension in Polygenic Obese TALLYHO/JngJ Mice: Role of Na/K-ATPase Signaling. causal interaction,ongoing research,therapeutic application,unassigned 4,3,1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31877680&form=6&db=m Mechanistic Insight of Na/K-ATPase Signaling and HO-1 into Models of Obesity and Nonalcoholic Steatohepatitis. causal interaction,ongoing research,unassigned 2,1,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33154488&form=6&db=m Author Correction: The Adipocyte Na/K-ATPase Oxidant Amplification Loop is the Central Regulator of Western Diet-Induced Obesity and Associated Comorbidities. causal interaction,unassigned 2,0 7.2.2.13 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33202598&form=6&db=m The Pivotal Role of Adipocyte-Na K peptide in Reversing Systemic Inflammation in Obesity and COVID-19 in the Development of Heart Failure. causal interaction,unassigned 3,0 7.2.2.13 Olfaction Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25646894&form=6&db=m Dietary sodium protects fish against copper-induced olfactory impairment. diagnostic usage,unassigned 1,0 7.2.2.13 Osteoarthritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2820052&form=6&db=m Decreased NA+, K+-ATPase activity in erythrocyte membrane from rheumatoid arthritis patients. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,2,0 7.2.2.13 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30134193&form=6&db=m Immunization with Na+/K+ ATPase DR peptide prevents bone loss in an ovariectomized rat osteoporosis model. therapeutic application,unassigned 4,0 7.2.2.13 Osteosarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2431089&form=6&db=m Parathyroid hormone stimulation of the Na+/K+ pump in rat clonal osteosarcoma cells. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Osteosarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2446938&form=6&db=m Insulin stimulation of Na+-K+ pump in clonal rat osteosarcoma cells. ongoing research,unassigned 4,0 7.2.2.13 Osteosarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31822650&form=6&db=m The Na+/K+ ATPase Inhibitor Ouabain Attenuates Stemness and Chemoresistance of Osteosarcoma Cells. causal interaction,ongoing research,therapeutic application,unassigned 2,4,1,0 7.2.2.13 Otitis Media http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10651433&form=6&db=m Labeling of the glucocorticoid receptor and Na,K-ATPase in a rat otitis media model. ongoing research,therapeutic application,unassigned 4,1,0 7.2.2.13 Otosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15241221&form=6&db=m Spiral ligament and stria vascularis changes in cochlear otosclerosis: effect on hearing level. ongoing research,unassigned 2,0 7.2.2.13 Ototoxicity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3032880&form=6&db=m Effect of cisplatin on hair cell morphology and lateral wall Na,K-ATPase activity. therapeutic application,unassigned 1,0 7.2.2.13 Ovarian Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31499071&form=6&db=m New bufadienolides extracted from Rhinella marina inhibit Na,K-ATPase and induce apoptosis by activating caspases 3 and 9 in human breast and ovarian cancer cells. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6328636&form=6&db=m Effects of ouabain and potassium on rabbit arterial smooth muscle cells in culture. therapeutic application,unassigned 2,0 7.2.2.13 Pancreatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19671733&form=6&db=m Oleandrin-mediated inhibition of human tumor cell proliferation: importance of Na,K-ATPase alpha subunits as drug targets. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 7.2.2.13 Pancreatitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20467345&form=6&db=m Montelukast, a selective cysteinyl leukotriene receptor 1 antagonist, reduces cerulein-induced pancreatic injury in rats. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,2,0 7.2.2.13 Pancreatitis, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11791942&form=6&db=m Modulation of Na,K-ATPase activity by immunoglobulins--II. Na,K-ATPase activity in erythrocytes from patients with various chronic disturbances. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Papilloma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=139301&form=6&db=m Choroid plexus papilloma. II. Ultrastructure and ultracytochemical localization of Na-K-ATPase. ongoing research,unassigned 1,0 7.2.2.13 Papilloma, Choroid Plexus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=139301&form=6&db=m Choroid plexus papilloma. II. Ultrastructure and ultracytochemical localization of Na-K-ATPase. ongoing research,unassigned 1,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2500620&form=6&db=m Potassium uptake in muscle during paramyotonic weakness. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2548465&form=6&db=m Erythrocyte sodium-potassium pump in thyrotoxic periodic paralysis. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,1,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6287910&form=6&db=m Periodic paralysis and the sodium-potassium pump. unassigned - 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7740232&form=6&db=m [Thyrotoxic periodic paralysis. Discussion of the role of Na-K-ATPase, apropos of a case] unassigned - 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8392857&form=6&db=m Thyrotoxicosis presenting as a life threatening hypokalaemic paralysis: investigation of the Na/K pump in isolated leucocytes. diagnostic usage,therapeutic application,unassigned 1,2,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9793040&form=6&db=m [Periodic hypokalemic paralysis disclosing thyrotoxicosis] unassigned - 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17606467&form=6&db=m A novel family of transmembrane proteins interacting with {beta} subunits of the Na,K-ATPase. causal interaction,unassigned 2,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20624687&form=6&db=m Altered axonal excitability properties in juvenile muscular atrophy of distal upper extremity (Hirayama disease). diagnostic usage,ongoing research,unassigned 2,1,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21708955&form=6&db=m Na+,K+-pump stimulation improves contractility in isolated muscles of mice with hyperkalemic periodic paralysis. ongoing research,therapeutic application,unassigned 4,3,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25476251&form=6&db=m Genome-wide screen for modifiers of Na + /K + ATPase alleles identifies critical genetic loci. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32432016&form=6&db=m Thyrotoxic Periodic Paralysis With Features of Andersen-Tawil Syndrome: A Case Report and Literature Review. causal interaction,diagnostic usage,unassigned 4,2,0 7.2.2.13 Paralysis, Hyperkalemic Periodic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2500620&form=6&db=m Potassium uptake in muscle during paramyotonic weakness. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Paralysis, Hyperkalemic Periodic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21708955&form=6&db=m Na+,K+-pump stimulation improves contractility in isolated muscles of mice with hyperkalemic periodic paralysis. ongoing research,therapeutic application,unassigned 4,3,0 7.2.2.13 Paraneoplastic Syndromes, Nervous System http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25809299&form=6&db=m Neuronal Na+/K+ ATPase is an autoantibody target in paraneoplastic neurologic syndrome. causal interaction,unassigned 3,0 7.2.2.13 Parkinson Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Parkinsonian Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15260953&form=6&db=m Mutations in the Na+/K+ -ATPase alpha3 gene ATP1A3 are associated with rapid-onset dystonia parkinsonism. therapeutic application,unassigned 1,0 7.2.2.13 Parkinsonian Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17234593&form=6&db=m Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice. causal interaction,unassigned 4,0 7.2.2.13 Parkinsonian Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21165980&form=6&db=m Distribution of Na/K-ATPase alpha 3 isoform, a sodium-potassium P-type pump associated with rapid-onset of dystonia parkinsonism (RDP) in the adult mouse brain. unassigned - 7.2.2.13 Parkinsonian Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23652595&form=6&db=m Enhanced inhibitory neurotransmission in the cerebellar cortex of Atp1a3-deficient heterozygous mice. therapeutic application,unassigned 1,0 7.2.2.13 Parkinsonian Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24983657&form=6&db=m Heterozygous mice deficient in Atp1a3 exhibit motor deficits by chronic restraint stress. therapeutic application,unassigned 1,0 7.2.2.13 Pediatric Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3025397&form=6&db=m Childhood obesity: plasma potassium and Na-K-ATPase indicate that the electrolyte content of food may be important. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Peripheral Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2421135&form=6&db=m A sodium-pump defect in diabetic peripheral nerve corrected by sorbinil administration: relationship to myo-inositol metabolism and nerve conduction slowing. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 7.2.2.13 Peripheral Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3019804&form=6&db=m Decreased myo-inositol content and Na+-K+-ATPase activity in superior cervical ganglion of STZ-diabetic rat and prevention by aldose reductase inhibition. causal interaction,unassigned 3,0 7.2.2.13 Peripheral Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8820823&form=6&db=m Morphophysiologic characterization of peripheral neuropathy in zinc-deficient guinea pigs. causal interaction,unassigned 4,0 7.2.2.13 Peripheral Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32843829&form=6&db=m Sciatic Nerve Structural and Functional Recovery with Extract of Phyllanthus amarus and Esculetin in STZ-Induced Hyperglycemic Rats. causal interaction,unassigned 1,0 7.2.2.13 Peritonitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18323749&form=6&db=m Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states. ongoing research,unassigned 3,0 7.2.2.13 Peritonitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25653449&form=6&db=m ATP1A1-mediated Src signaling inhibits coronavirus entry into host cells. causal interaction,ongoing research,unassigned 3,4,0 7.2.2.13 Pheochromocytoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2560348&form=6&db=m Determination of total (Na+ + K+)-ATPase activity of isolated or cultured cells. ongoing research,unassigned 4,0 7.2.2.13 Pheochromocytoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2843606&form=6&db=m Differential response of adenylate cyclase and ATPase activities after chronic ethanol exposure of PC12 cells. ongoing research,unassigned 4,0 7.2.2.13 Plasmacytoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6293566&form=6&db=m Characterization of a beta-actinin-like protein in purified non-muscle cell membranes. Its activity on (Na+ + K+)-ATPase. ongoing research,unassigned 2,0 7.2.2.13 Pneumonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23209347&form=6&db=m Oleic acid induces lung injury in mice through activation of the ERK pathway. causal interaction,therapeutic application,unassigned 1,4,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1327261&form=6&db=m Sequential tubular cell and basement membrane changes in polycystic kidney disease. causal interaction,ongoing research,unassigned 1,4,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7622841&form=6&db=m Renal tubule Na,K-ATPase polarity in different animal models of polycystic kidney disease. diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7919157&form=6&db=m Sodium pump distribution is not reversed in the DBA/2FG-pcy, polycystic kidney disease model mouse. ongoing research,therapeutic application,unassigned 4,2,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8196361&form=6&db=m Cell polarity in human renal cystic disease. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,3,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8383715&form=6&db=m Renal tubule Na,K-ATPase polarity in glucocorticoid-induced polycystic kidney disease. causal interaction,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10450015&form=6&db=m Cystic fibrosis transmembrane conductance regulator in the kidney: clues to its role? causal interaction,unassigned 4,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12890662&form=6&db=m Role of Na-K-ATPase in the assembly of tight junctions. therapeutic application,unassigned 2,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17151336&form=6&db=m Ouabain binds with high affinity to the Na,K-ATPase in human polycystic kidney cells and induces extracellular signal-regulated kinase activation and cell proliferation. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17344187&form=6&db=m Selective basolateral localization of overexpressed Na-K-ATPase beta1- and beta2- subunits is disrupted by butryate treatment of MDCK cells. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21697238&form=6&db=m Ouabain activates the Na,K-ATPase signalosome to induce autosomal dominant polycystic kidney disease cell proliferation. ongoing research,unassigned 4,0 7.2.2.13 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23761677&form=6&db=m Novel role of ouabain as a cystogenic factor in autosomal dominant polycystic kidney disease. causal interaction,ongoing research,therapeutic application,unassigned 1,1,4,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7919157&form=6&db=m Sodium pump distribution is not reversed in the DBA/2FG-pcy, polycystic kidney disease model mouse. ongoing research,therapeutic application,unassigned 4,2,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8129535&form=6&db=m [Abnormal erythrocyte sodium transport in patients with adult polycystic kidney and hypertension] causal interaction,unassigned 3,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9140043&form=6&db=m Epithelial cell polarity and disease. causal interaction,unassigned 2,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10450015&form=6&db=m Cystic fibrosis transmembrane conductance regulator in the kidney: clues to its role? causal interaction,unassigned 4,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17151336&form=6&db=m Ouabain binds with high affinity to the Na,K-ATPase in human polycystic kidney cells and induces extracellular signal-regulated kinase activation and cell proliferation. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17344187&form=6&db=m Selective basolateral localization of overexpressed Na-K-ATPase beta1- and beta2- subunits is disrupted by butryate treatment of MDCK cells. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21697238&form=6&db=m Ouabain activates the Na,K-ATPase signalosome to induce autosomal dominant polycystic kidney disease cell proliferation. ongoing research,unassigned 4,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23761677&form=6&db=m Novel role of ouabain as a cystogenic factor in autosomal dominant polycystic kidney disease. causal interaction,ongoing research,therapeutic application,unassigned 1,1,4,0 7.2.2.13 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26289599&form=6&db=m Ouabain Regulates CFTR-Mediated Anion Secretion and Na,K-ATPase Transport in ADPKD Cells. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Polycystic Kidney, Autosomal Recessive http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7919157&form=6&db=m Sodium pump distribution is not reversed in the DBA/2FG-pcy, polycystic kidney disease model mouse. ongoing research,therapeutic application,unassigned 4,2,0 7.2.2.13 Polycystic Kidney, Autosomal Recessive http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12660316&form=6&db=m Na transport in autosomal recessive polycystic kidney disease (ARPKD) cyst lining epithelial cells. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 7.2.2.13 Polyneuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1327887&form=6&db=m Hypothesis: low Na/K-ATPase activity in the red cell membrane, a potential marker of the predisposition to diabetic neuropathy. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,3,1 7.2.2.13 Polyneuropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2561396&form=6&db=m Effect of aldose reductase inhibitor (Ponalrestat) on erythrocyte Na,K-ATPase activity in non-insulin-dependent diabetic patients with polyneuropathy. ongoing research,therapeutic application,unassigned 2,2,0 7.2.2.13 Polyuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2537474&form=6&db=m Effect of potassium depletion on ischemic renal failure. causal interaction,unassigned 2,0 7.2.2.13 Polyuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10966935&form=6&db=m Altered expression of renal AQPs and Na(+) transporters in rats with lithium-induced NDI. unassigned - 7.2.2.13 Polyuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12187767&form=6&db=m [Fundamental and applied studies on transport and metabolism of electrolytes and glucose--aim to contact with molecular biology] causal interaction,unassigned 3,0 7.2.2.13 Porcine Reproductive and Respiratory Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29532265&form=6&db=m Downregulation of Aquaporins (AQP1 and AQP5) and Na,K-ATPase in Porcine Reproductive and Respiratory Syndrome Virus-Infected Pig Lungs. causal interaction,unassigned 2,0 7.2.2.13 Potassium Deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=132355&form=6&db=m Increase in the (Na+ + K+)-ATPase activity in heart muscle after chronic treatment with digitoxin or potassium deficient diet. causal interaction,unassigned 3,0 7.2.2.13 Potassium Deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6289258&form=6&db=m Effect of low potassium-diet on Na-K-ATPase in rat nephron segments. unassigned - 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10466474&form=6&db=m Circulating bufodienolide and cardenolide sodium pump inhibitors in preeclampsia. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,2,3 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11150823&form=6&db=m Preeclampsia and Na,K-ATPase activity of red blood cell ghosts from neonatal and maternal blood. diagnostic usage,ongoing research,unassigned 1,4,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17535731&form=6&db=m Endogenous Na/K-ATPase inhibitors in patients with preeclampsia. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17942287&form=6&db=m Endogenous sodium pump inhibitors, diabetes mellitus and preeclampsia Preliminary observations and a hypothesis. causal interaction,therapeutic application,unassigned 4,4,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19008721&form=6&db=m Monoclonal antibody to an endogenous bufadienolide, marinobufagenin, reverses preeclampsia-induced Na/K-ATPase inhibition and lowers blood pressure in NaCl-sensitive hypertension. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19158804&form=6&db=m Erythrocyte sodium/potassium ATPase activity in severe preeclampsia. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,2,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19927009&form=6&db=m Interaction of Digibind with endogenous cardiotonic steroids from preeclamptic placentae. causal interaction,therapeutic application,unassigned 1,2,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22649120&form=6&db=m DigiFab Interacts With Endogenous Cardiotonic Steroids and Reverses Preeclampsia-Induced Na/K-ATPase Inhibition. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23878005&form=6&db=m Magnesium Sulfate Potentiates Effect of Digifab on Marinobufagenin-Induced Na/K-ATPase Inhibition. causal interaction,therapeutic application,unassigned 3,4,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26374826&form=6&db=m Synthesis of an Endogenous Steroidal Na Pump Inhibitor Marinobufagenin, Implicated in Human Cardiovascular Diseases, Is Initiated by CYP27A1 via Bile Acid Pathway. unassigned - 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30104471&form=6&db=m Antibody to Marinobufagenin Reverses Placenta-Induced Fibrosis of Umbilical Arteries in Preeclampsia. causal interaction,unassigned 3,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31713584&form=6&db=m Antibody against Na/K-ATPase inhibitor lowers blood pressure and increases vascular Fli1 in experimental preeclampsia. causal interaction,ongoing research,therapeutic application,unassigned 3,3,4,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31915545&form=6&db=m Endogenous Bufadienolides, Fibrosis and Preeclampsia. therapeutic application,unassigned 3,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33382878&form=6&db=m S-glutathionylation of the Na+-K+ Pump: A Novel Redox Mechanism in Preeclampsia. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,1 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33616648&form=6&db=m Uncovering the molecular cause of preeclampsia - The role of glutathionylation of the Na+/K+ ATPase transporter. causal interaction,unassigned 1,0 7.2.2.13 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33669287&form=6&db=m Preeclampsia: Cardiotonic Steroids, Fibrosis, Fli1 and Hint to Carcinogenesis. causal interaction,unassigned 3,0 7.2.2.13 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22695512&form=6&db=m Voltage-dependent K+ channels as oncotargets in malignant gliomas. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,4,0 7.2.2.13 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28514771&form=6&db=m Istaroxime, a potential anticancer drug in prostate cancer, exerts beneficial functional effects in healthy and diseased human myocardium. causal interaction,therapeutic application,unassigned 2,4,0 7.2.2.13 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33956349&form=6&db=m Inverse agonism at the Na/K-ATPase receptor reverses EMT in prostate cancer cells. causal interaction,diagnostic usage,unassigned 4,4,0 7.2.2.13 Protein Deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2851113&form=6&db=m Interaction of alcohol and protein deficiency on rat brain synaptosomal (Na+-K+)-ATPase. ongoing research,unassigned 1,0 7.2.2.13 Protein-Energy Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2120031&form=6&db=m The absence of adapted sodium and potassium transport in erythrocytes of cerebral palsied children with secondary malnutrition. unassigned - 7.2.2.13 Proteinuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10069978&form=6&db=m Genetic and biochemical determinants of abnormal monovalent ion transport in primary hypertension. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Proteinuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11147069&form=6&db=m [Molecular mechanisms of idiopathic nephrotic syndrome] ongoing research,unassigned 1,0 7.2.2.13 Proteinuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25187430&form=6&db=m Rostafuroxin protects from podocyte injury and proteinuria induced by adducin genetic variants and ouabain. therapeutic application,unassigned 4,0 7.2.2.13 Proteinuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32119995&form=6&db=m Ageing reduces angiotensin II type 1 receptor antagonism mediated pre-conditioning effects in ischemic kidneys by inducing oxidative and inflammatory stress. causal interaction,diagnostic usage,ongoing research,unassigned 2,4,2,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1656777&form=6&db=m Upregulation of rat lung Na-K-ATPase during hyperoxic injury. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8131602&form=6&db=m Alveolar type II cell Na,K-ATPase is upregulated during mechanical ventilation-induced pulmonary edema. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9309983&form=6&db=m Dopamine stimulates sodium transport and liquid clearance in rat lung epithelium. unassigned - 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9925907&form=6&db=m Influence of inhaled nitric oxide and hyperoxia on Na,K-ATPase expression and lung edema in newborn piglets. causal interaction,therapeutic application,unassigned 3,2,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11355011&form=6&db=m Role and regulation of lung Na,K-ATPase. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11815434&form=6&db=m Na,K-ATPase overexpression improves alveolar fluid clearance in a rat model of elevated left atrial pressure. causal interaction,unassigned 3,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12038043&form=6&db=m [Respiratory distress. New Perspectives to lung edema treatment] causal interaction,unassigned 3,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12592959&form=6&db=m Regulation of lung edema clearance by dopamine. diagnostic usage,therapeutic application,unassigned 2,3,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12682448&form=6&db=m Mechanisms of pulmonary edema clearance during acute hypoxemic respiratory failure: role of the Na,K-ATPase. causal interaction,ongoing research,therapeutic application,unassigned 3,3,4,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15781423&form=6&db=m Decreased expression of both the alpha1- and alpha2-subunits of the Na-K-ATPase reduces maximal alveolar epithelial fluid clearance. unassigned - 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17530222&form=6&db=m Alveolar epithelium and Na,K-ATPase in acute lung injury. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,2,1 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23470626&form=6&db=m Lipoxin A4 Activates Alveolar Epithelial ENaC, Na,K-ATPase and Increases Alveolar Fluid Clearance. causal interaction,unassigned 4,0 7.2.2.13 Pulmonary Edema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26999598&form=6&db=m Influenza leaves a TRAIL to pulmonary edema. causal interaction,ongoing research,unassigned 3,3,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1668126&form=6&db=m [Activity of the sodium-potassium pump and values of sodium and potassium in erythrocytes in patients with non-dialyzed renal failure and arterial hypertension treated conservatively] diagnostic usage,ongoing research,therapeutic application,unassigned 3,2,1,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2441015&form=6&db=m Measurement of circulating sodium-pump inhibitory activity in uraemia and essential hypertension. diagnostic usage,ongoing research,therapeutic application,unassigned 3,3,1,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2537474&form=6&db=m Effect of potassium depletion on ischemic renal failure. causal interaction,unassigned 2,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2554164&form=6&db=m Effects of dialysis and transplantation on red cell Na pump function in renal failure. ongoing research,unassigned 4,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2847190&form=6&db=m An Na+-K+-ATPase inhibitor which circulates in renal failure but not in essential hypertension. therapeutic application,unassigned 3,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2983540&form=6&db=m Sodium potassium ATPase activity in human rectal mucosa with and without renal insufficiency. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,4,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2984308&form=6&db=m Altered distribution pattern of Na+-K+-ATPase and succinate dehydrogenase activities along the nephron in human acute post-transplant renal failure. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,3,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2987715&form=6&db=m Effect of hemodialysis on red blood cell Na+-K+-ATPase activity in terminal renal failure. ongoing research,unassigned 4,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6126857&form=6&db=m Renal function and Na-K-ATPase in rats after suprarenal ligation of inferior vena cava. causal interaction,unassigned 1,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7629896&form=6&db=m Disturbances of potassium homeostasis in poisoning. unassigned - 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7833833&form=6&db=m Sodium-22 uptake in erythrocytes can differentiate between the essential and the secondary hypertensive patient. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7948781&form=6&db=m Experimental renal failure in the rat modulates cardiac Na,K-ATPase alpha 2 mRNA but not protein. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 7.2.2.13 Renal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15808437&form=6&db=m [Physiological effects of the connecting peptide] causal interaction,ongoing research,therapeutic application,unassigned 4,2,1,0 7.2.2.13 Renal Insufficiency, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2983540&form=6&db=m Sodium potassium ATPase activity in human rectal mucosa with and without renal insufficiency. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,4,0 7.2.2.13 Renal Insufficiency, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7051849&form=6&db=m Mechanism of potassium adaptation. causal interaction,diagnostic usage,unassigned 1,1,0 7.2.2.13 Renal Insufficiency, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16509935&form=6&db=m Jejunal dopamine and Na,K-ATPase activity in early chronic renal insufficiency. causal interaction,diagnostic usage,therapeutic application,unassigned 3,1,1,0 7.2.2.13 Renal Insufficiency, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26374826&form=6&db=m Synthesis of an Endogenous Steroidal Na Pump Inhibitor Marinobufagenin, Implicated in Human Cardiovascular Diseases, Is Initiated by CYP27A1 via Bile Acid Pathway. unassigned - 7.2.2.13 Renal Insufficiency, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29775473&form=6&db=m Na/K-ATPase signaling mediates miR-29b-3p regulation and cardiac fibrosis formation in mice with chronic kidney disease. causal interaction,ongoing research,unassigned 4,3,0 7.2.2.13 Renal Insufficiency, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33356956&form=6&db=m The potential role of Na-K-ATPase and its signaling in the development of anemia in chronic kidney disease. causal interaction,unassigned 4,0 7.2.2.13 Reperfusion Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3028176&form=6&db=m O2 free radicals: cause of ischemia-reperfusion injury to cardiac Na+-K+-ATPase. therapeutic application,unassigned 1,0 7.2.2.13 Reperfusion Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16609151&form=6&db=m Sex differences in heat shock protein 72 expression and localization in rats following renal ischemia-reperfusion injury. causal interaction,ongoing research,unassigned 1,3,0 7.2.2.13 Reperfusion Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18050619&form=6&db=m [Effect of electroacupuncture of "Neiguan" (PC 6) on Na+-K+-ATPase activity and its gene expression in cardiocyte membrane in rats with myocardial ischemia-reperfusion injury] ongoing research,unassigned 2,0 7.2.2.13 Reperfusion Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20622333&form=6&db=m [Effect of cardiomyopeptidin for injection on energy metabolism in isolated hearts of young rats after ischemia-reperfusion injury] diagnostic usage,ongoing research,unassigned 3,2,0 7.2.2.13 Reperfusion Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21441313&form=6&db=m Enhencement of Na/K pump activity by chronic intermittent hypobaric hypoxia protected against reperfusion injury. therapeutic application,unassigned 1,0 7.2.2.13 Reperfusion Injury http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29702141&form=6&db=m DRm217 attenuates myocardial ischemia-reperfusion injury via stabilizing plasma membrane Na+-K+-ATPase, inhibiting Na+-K+-ATPase/ROS pathway and activating PI3K/Akt and ERK1/2. therapeutic application,unassigned 1,0 7.2.2.13 Respiratory Distress Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14578215&form=6&db=m Leukotriene D4 activates alveolar epithelial Na,K-ATPase and increases alveolar fluid clearance. causal interaction,unassigned 4,0 7.2.2.13 Respiratory Distress Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25399325&form=6&db=m Na/K-ATPase assay in the intact mice lung subjected to perfusion. diagnostic usage,unassigned 3,0 7.2.2.13 Respiratory Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12592959&form=6&db=m Regulation of lung edema clearance by dopamine. diagnostic usage,therapeutic application,unassigned 2,3,0 7.2.2.13 Respiratory Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12682448&form=6&db=m Mechanisms of pulmonary edema clearance during acute hypoxemic respiratory failure: role of the Na,K-ATPase. causal interaction,ongoing research,therapeutic application,unassigned 3,3,4,0 7.2.2.13 Respiratory Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34277634&form=6&db=m TRAF2 Is a Novel Ubiquitin E3 Ligase for the Na,K-ATPase ?-Subunit That Drives Alveolar Epithelial Dysfunction in Hypercapnia. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,3,0 7.2.2.13 Retinal Degeneration http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6271302&form=6&db=m [Early changes in retinal 5'-nucleotidase activity in hereditary retinal degeneration] diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Retinal Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2551574&form=6&db=m (Na+ + K+)-ATPase and opsin in retinas of RCS dystrophic rats: time course study. ongoing research,unassigned 2,0 7.2.2.13 Retinoschisis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21196491&form=6&db=m The Na/K-ATPase is obligatory for membrane anchorage of retinoschisin, the protein involved in the pathogenesis of X-linked juvenile retinoschisis. causal interaction,unassigned 2,0 7.2.2.13 Rhabdomyolysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7629896&form=6&db=m Disturbances of potassium homeostasis in poisoning. unassigned - 7.2.2.13 Sarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11179415&form=6&db=m Na,K-ATPase beta-subunit is required for epithelial polarization, suppression of invasion, and cell motility. causal interaction,ongoing research,unassigned 1,3,0 7.2.2.13 Sarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15133131&form=6&db=m Na,K-ATPase beta1-subunit increases the translation efficiency of the alpha1-subunit in MSV-MDCK cells. unassigned - 7.2.2.13 Sarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15616195&form=6&db=m Novel role for Na,K-ATPase in phosphatidylinositol 3-kinase signaling and suppression of cell motility. causal interaction,ongoing research,therapeutic application,unassigned 3,1,4,0 7.2.2.13 Sarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15972700&form=6&db=m A moving new role for the sodium pump in epithelial cells and carcinomas. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,3,0 7.2.2.13 Sarcoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17535735&form=6&db=m Identification of protein kinase C as an intermediate in Na,K-ATPase Beta-subunit mediated lamellipodia formation and suppression of cell motility in carcinoma cells. ongoing research,therapeutic application,unassigned 3,1,0 7.2.2.13 Sarcoma, Yoshida http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=124601&form=6&db=m Inactivation of (Na-++K-+)-stimulated ATPase by a cytotoxic protein from cobra venom in relation to its lytic effects on cells. ongoing research,unassigned 3,0 7.2.2.13 Sarcoma, Yoshida http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=233302&form=6&db=m Influence of charge on the inactivation of membrane bound (Na+ + K+)-ATPase of Yoshida sarcoma cells by inhibitor proteins from cobra venom. diagnostic usage,ongoing research,unassigned 3,3,0 7.2.2.13 Schistosomiasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8304977&form=6&db=m Heterogeneity of ouabain binding sites in Schistosoma mansoni. First evidence for the presence of two (Na+ + K+)-ATPase isoforms in platyhelminths. diagnostic usage,ongoing research,unassigned 1,3,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=129335&form=6&db=m The (Na+ + K+)-ATPase activity in brain of Quaking mice. ongoing research,unassigned 2,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2423926&form=6&db=m Sodium pathway markers in normal and kindled frog brains. diagnostic usage,ongoing research,unassigned 3,1,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6248893&form=6&db=m Membrane Na+K+ATPase activity: changes using an experimental model of alcohol dependence and withdrawal. causal interaction,diagnostic usage,unassigned 3,2,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6405441&form=6&db=m Possible mechanism of digoxin-induced convulsions. therapeutic application,unassigned 2,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7859671&form=6&db=m Abnormal cortical unit activity of the reticular formation. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8740210&form=6&db=m Extracellular K+ accumulations and synchronous GABA-mediated potentials evoked by 4-aminopyridine in the adult rat hippocampus. causal interaction,unassigned 3,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15082235&form=6&db=m Altered Na+-K+ ATPase immunoreactivity within GABAergic neurons in the gerbil hippocampal complex induced by spontaneous seizure and vigabatrin treatment. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,2,3 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17588504&form=6&db=m Evolving in thin air-Lessons from the llama fetus in the altiplano. therapeutic application,unassigned 1,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18245348&form=6&db=m Neuropathology in Drosophila mutants with increased seizure susceptibility. unassigned - 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18827752&form=6&db=m NA+, K+-ATPase Activity in the Brain of the Rats with Kainic Acid-Induced Seizures: Influence of Lamotrigine. diagnostic usage,ongoing research,unassigned 1,4,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25476251&form=6&db=m Genome-wide screen for modifiers of Na + /K + ATPase alleles identifies critical genetic loci. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29225049&form=6&db=m Oxidative stress and Na,K-ATPase activity differential regulation in brainstem and forebrain of Wistar Audiogenic rats may lead to increased seizure susceptibility. diagnostic usage,ongoing research,unassigned 3,4,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30388404&form=6&db=m Germline De Novo Mutations in ATP1A1 Cause Renal Hypomagnesemia, Refractory Seizures, and Intellectual Disability. unassigned - 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31705535&form=6&db=m Hereditary spastic paraplegia is a novel phenotype for germline de novo ATP1A1 mutation. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32584666&form=6&db=m Perinatal cardiopulmonary adaptation to the thin air of the Alto Andino by a native Altiplano dweller, the llama. unassigned - 7.2.2.13 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33206576&form=6&db=m Na+-K+-ATPase functions in the developing hippocampus: regional differences in CA1 and CA3 neuronal excitability and role in epileptiform network bursting. unassigned - 7.2.2.13 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2257076&form=6&db=m Sick cell syndrome in a burned patient. causal interaction,unassigned 4,0 7.2.2.13 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3040290&form=6&db=m Alterations of Na+-K+-ATPase, Ca2+-ATPase, and Mg2+-ATPase activities in erythrocyte, muscle, and liver of traumatic and septic patients. causal interaction,unassigned 1,0 7.2.2.13 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8403243&form=6&db=m Externalization and internalization of (Na+ + K+)-ATPase in rat heart during different phases of sepsis. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,4,1 7.2.2.13 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12005173&form=6&db=m Role of skeletal muscle Na+-K+ ATPase activity in increased lactate production in sub-acute sepsis. causal interaction,ongoing research,unassigned 3,1,0 7.2.2.13 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16810041&form=6&db=m Lactate and shock state: the metabolic view. causal interaction,ongoing research,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21478253&form=6&db=m Sepsis Impairs Alveolar Epithelial Function by Down-regulating Na,K-ATPase Pump. causal interaction,ongoing research,unassigned 1,1,0 7.2.2.13 Shock, Septic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16810041&form=6&db=m Lactate and shock state: the metabolic view. causal interaction,ongoing research,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Shoulder Impingement Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8156065&form=6&db=m Changes in muscle fiber cross-sectional area and concentrations of Na,K-ATPase in deltoid muscle in patients with impingement syndrome of the shoulder. ongoing research,unassigned 4,0 7.2.2.13 Sinusitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2177995&form=6&db=m Na/K-ATPase in rabbit paranasal sinus mucosa during induced sinusitis. causal interaction,diagnostic usage,unassigned 1,4,0 7.2.2.13 Sleep Deprivation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18571330&form=6&db=m Stimulatory role of calcium in rapid eye movement sleep deprivation-induced noradrenaline-mediated increase in Na-K-ATPase activity in rat brain. unassigned - 7.2.2.13 Sleep Deprivation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32707223&form=6&db=m Ouabain, a Na-K-ATPase inhibitor, enhances wakefulness in rats. unassigned - 7.2.2.13 Spastic Paraplegia, Hereditary http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31705535&form=6&db=m Hereditary spastic paraplegia is a novel phenotype for germline de novo ATP1A1 mutation. causal interaction,ongoing research,unassigned 3,2,0 7.2.2.13 Spinal Cord Injuries http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2985770&form=6&db=m Role of monoamines in experimental spinal cord injury in rats. Relationship between Na+-K+-ATPase and lipid peroxidation. ongoing research,unassigned 3,0 7.2.2.13 Spinal Cord Injuries http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15862959&form=6&db=m Progesterone neuroprotection in spinal cord trauma involves up-regulation of brain-derived neurotrophic factor in motoneurons. diagnostic usage,unassigned 1,0 7.2.2.13 Spinal Cord Injuries http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19318112&form=6&db=m Progesterone neuroprotection in traumatic CNS injury and motoneuron degeneration. diagnostic usage,ongoing research,unassigned 3,1,0 7.2.2.13 Squamous Cell Carcinoma of Head and Neck http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25275034&form=6&db=m Effects of digoxin and Na, K-ATPase immunoexpression on human oral squamous carcinomas. diagnostic usage,ongoing research,therapeutic application,unassigned 2,3,1,0 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=220995&form=6&db=m Effect of starvation, alloxan diabetes and adrenalectomy on Na+ K+-ATPase of the mucosa of the small intestine of rat. causal interaction,unassigned 4,0 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3001056&form=6&db=m Alterations in amino acid transport in Na,K-ATPase amplified HeLa cells. ongoing research,unassigned 1,0 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3418420&form=6&db=m Reduced ion transport in erythrocytes of male Sprague-Dawley rats during starvation. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6617164&form=6&db=m Influence of nutrition on biochemical sea water adaptation of the rainbow trout (Salmo gairdneri richardson). causal interaction,unassigned 2,0 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16707566&form=6&db=m Low doses of ouabain protect from serum deprivation-triggered apoptosis and stimulate kidney cell proliferation via activation of NF-kappaB. unassigned - 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23401964&form=6&db=m [Effect of stress actions on some hematologic and biochemical parameters of rat blood and on energetic intermolecular interactions in lipid extract under effect of light radiation]. diagnostic usage,ongoing research,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23789400&form=6&db=m [Na,K-ATPase activity of erythrocytes of rats during prolonged starvation]. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,2,0 7.2.2.13 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25916122&form=6&db=m [The ability of cells to adjust to the low oxigen content associated with Na,K-ATPase glutationilation]. unassigned - 7.2.2.13 Stomach Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12127692&form=6&db=m Identification of genes differentially expressed between gastric cancers and normal gastric mucosa with cDNA microarrays. causal interaction,unassigned 3,0 7.2.2.13 Stomach Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34251542&form=6&db=m The expression of the alpha1 subunit of Na+/K+-ATPase is related to tumor development and clinical outcomes in gastric cancer. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,4,0 7.2.2.13 Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10069978&form=6&db=m Genetic and biochemical determinants of abnormal monovalent ion transport in primary hypertension. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15456803&form=6&db=m Blocking the anoxic depolarization protects without functional compromise following simulated stroke in cortical brain slices. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18343354&form=6&db=m Platelet membrane fluidity and Na+/K+ ATPase activity in acute stroke. diagnostic usage,ongoing research,therapeutic application,unassigned 4,3,2,0 7.2.2.13 Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30584244&form=6&db=m DR-region of Na+/K+ ATPase is a target to treat excitotoxicity and stroke. causal interaction,therapeutic application,unassigned 2,2,0 7.2.2.13 Subacute Sclerosing Panencephalitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11922232&form=6&db=m Inhibition of membrane Na+-K+ ATPase activity: a common pathway in central nervous system disorders. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,1,3 7.2.2.13 Tachycardia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8147825&form=6&db=m Reduced 3H-ouabain binding site (Na,K-ATPase) concentration in ventricular myocardium of dogs with tachycardia induced heart failure. causal interaction,unassigned 2,0 7.2.2.13 Tachycardia, Ventricular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11828798&form=6&db=m [Autoantibodies against sarcolemmal Na-K-ATPase in patients with dilated cardiomyopathy: autoimmune basis for ventricular arrhythmias in patients with congestive heart failure] causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Tachycardia, Ventricular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12354443&form=6&db=m Autoantibodies produced against sarcolemmal Na-K-ATPase: possible upstream targets of arrhythmias and sudden death in patients with dilated cardiomyopathy. causal interaction,ongoing research,unassigned 2,1,0 7.2.2.13 Tachypnea http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27659026&form=6&db=m Hypothyroxinemia and Risk for Transient Tachypnea of Newborn. unassigned - 7.2.2.13 Thymoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3012384&form=6&db=m Properties of Na+/K+ ATPase and alkaline phosphatase alter during spontaneous and radiation-induced leukemogenesis in mice. ongoing research,unassigned 3,0 7.2.2.13 Thyroid Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6132023&form=6&db=m Changes in localization of ouabain-sensitive, potassium-dependent p-nitrophenylphosphatase activity in human thyroid carcinoma cells. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,4,0 7.2.2.13 Thyrotoxicosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8392857&form=6&db=m Thyrotoxicosis presenting as a life threatening hypokalaemic paralysis: investigation of the Na/K pump in isolated leucocytes. diagnostic usage,therapeutic application,unassigned 1,2,0 7.2.2.13 Toxemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2560866&form=6&db=m [Na,K-ATPase activity as one of criteria in identifying the risk group for development of late toxemia in pregnancy screening] causal interaction,diagnostic usage,unassigned 1,4,0 7.2.2.13 Transient Tachypnea of the Newborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27659026&form=6&db=m Hypothyroxinemia and Risk for Transient Tachypnea of Newborn. unassigned - 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=218125&form=6&db=m Post-obstructive nephropathy in the rat: relationship between NA-K-ATPase activity and renal function. causal interaction,unassigned 3,0 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1661300&form=6&db=m Effect of chronic renal failure on Na,K-ATPase alpha 1 and alpha 2 mRNA transcription in rat skeletal muscle. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2832446&form=6&db=m Abnormal cation transport in uremia. Mechanisms in adipocytes and skeletal muscle from uremic rats. causal interaction,ongoing research,therapeutic application,unassigned 1,2,1,0 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4010843&form=6&db=m Digoxin-like immunoreacting substance(s) in the serum of patients with chronic uremia. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,3,2 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6086716&form=6&db=m Erythrocyte Na,K pump in uremia. Acute correction of a transport defect by hemodialysis. causal interaction,therapeutic application,unassigned 2,1,0 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6304546&form=6&db=m Red blood cell sodium transport and phosphate release in uremia. ongoing research,therapeutic application,unassigned 2,1,0 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7780058&form=6&db=m Impaired cation transport in thymocytes of rats with chronic uremia includes the Na+/H+ antiporter. causal interaction,unassigned 4,0 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7948781&form=6&db=m Experimental renal failure in the rat modulates cardiac Na,K-ATPase alpha 2 mRNA but not protein. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 7.2.2.13 Uremia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8184885&form=6&db=m Na pump defects in chronic uremia cannot be attributed to changes in Na-K-ATPase mRNA or protein. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,4,0 7.2.2.13 Ureteral Obstruction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=144549&form=6&db=m Effect of osmolality on renal medullary Na-K-ATPase activity in the postobstructive kidney. unassigned - 7.2.2.13 Ureteral Obstruction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8380811&form=6&db=m Transport defects of rabbit medullary thick ascending limb cells in obstructive nephropathy. causal interaction,therapeutic application,unassigned 1,1,0 7.2.2.13 Ureteral Obstruction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16189288&form=6&db=m alpha-MSH prevents impairment in renal function and dysregulation of AQPs and Na-K-ATPase in rats with bilateral ureteral obstruction. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 7.2.2.13 Ureteral Obstruction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25997532&form=6&db=m Involvement of NOX in the regulation of renal tubular expression of Na/K-ATPase in acute unilateral ureteral obstruction rats. causal interaction,ongoing research,unassigned 2,2,0 7.2.2.13 Ureteral Obstruction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29788096&form=6&db=m pNaKtide ameliorates renal interstitial fibrosis through inhibition of sodium-potassium adenosine triphosphatase-mediated signaling pathways in unilateral ureteral obstruction mice. causal interaction,unassigned 1,0 7.2.2.13 Urinary Bladder Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12673711&form=6&db=m Analysis of the Na,K-ATPase alpha- and beta-subunit expression profiles of bladder cancer using tissue microarrays. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,2 7.2.2.13 Urinary Bladder Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18006011&form=6&db=m Na,K-adenosine triphosphatase alpha1-subunit predicts survival of renal clear cell carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,3 7.2.2.13 Vaccinia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8798517&form=6&db=m Translational regulation of Na,K-ATPase alpha1 and beta1 polypeptide expression in epithelial cells. diagnostic usage,ongoing research,unassigned 1,4,0 7.2.2.13 Vascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16431390&form=6&db=m Electrolytes and sodium transport mechanism in diabetes mellitus. causal interaction,unassigned 3,0 7.2.2.13 Ventricular Dysfunction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10069797&form=6&db=m Digitalis. therapeutic application,unassigned 3,0 7.2.2.13 Ventricular Dysfunction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19787147&form=6&db=m Ventricular performance and Na+-K+ ATPase activity are reduced early and late after myocardial infarction in rats. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,3,0 7.2.2.13 Ventricular Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1338220&form=6&db=m Mechanisms of protective effects of berbamine on ischemia/reperfusion injury in isolated rat heart. therapeutic application,unassigned 2,0 7.2.2.13 Ventricular Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1649699&form=6&db=m Endogenous plasma Na,K-ATPase inhibitory activity and digoxin like immunoreactivity after acute myocardial infarction. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 7.2.2.13 Ventricular Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2555931&form=6&db=m [The role of an endogenous digoxin-like factor in regulating blood circulation and in the origin of arrhythmia in myocardial ischemia] ongoing research,unassigned 1,0 7.2.2.13 Ventricular Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10809497&form=6&db=m Specific up-regulation of mitochondrial F0F1-ATPase activity after short episodes of atrial fibrillation in sheep. therapeutic application,unassigned 2,0 7.2.2.13 Vesicular Stomatitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8383696&form=6&db=m Immortalization of polarized rat retinal pigment epithelium. unassigned - 7.2.2.13 Vesicular Stomatitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19620635&form=6&db=m Membrane proteins follow multiple pathways to the basolateral cell surface in polarized epithelial cells. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Virus Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8607259&form=6&db=m Inhibition of Na+K+ATPase activity in membranes of Sindbis virus-infected chick cells. causal interaction,unassigned 3,0 7.2.2.13 Virus Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11791942&form=6&db=m Modulation of Na,K-ATPase activity by immunoglobulins--II. Na,K-ATPase activity in erythrocytes from patients with various chronic disturbances. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 7.2.2.13 Virus Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27239254&form=6&db=m Basal Glutathionylation of Na,K-ATPase ?-Subunit Depends on Redox Status of Cells during the Enzyme Biosynthesis. causal interaction,unassigned 1,0 7.2.2.13 Vitamin B 6 Deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2854768&form=6&db=m Effects of salinity on vitamin B6 deficiency in rainbow trout (Salmo gairdneri Richardson). unassigned - 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1326337&form=6&db=m [The role of a GTP-binding protein in coupling of a muscarinic cholinergic receptor and Na,K-ATPase in myocardial sarcolemma] causal interaction,diagnostic usage,therapeutic application,unassigned 1,3,1,0 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1674758&form=6&db=m Beta adrenergic modulation of cardiac rhythm in a rat model of altered sympathetic neural development. unassigned - 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2174907&form=6&db=m Short-term regulation of Na+/K+ adenosine triphosphatase by recombinant human serotonin 5-HT1A receptor expressed in HeLa cells. therapeutic application,unassigned 1,0 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2563204&form=6&db=m Regulation of Na+-K+-ATPase activity in kidney proximal tubules: involvement of GTP binding proteins. therapeutic application,unassigned 1,0 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2850027&form=6&db=m Stimulation of cardiac alpha receptors increases Na/K pump current and decreases gK via a pertussis toxin-sensitive pathway. causal interaction,unassigned 2,0 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2851813&form=6&db=m Pertussis toxin modulates dopamine inhibition of Na,K-ATPase activity in rat proximal convoluted tubule segments. therapeutic application,unassigned 1,0 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21618533&form=6&db=m Hyposmotic stress causes ATP release and stimulates Na, K-ATPase activity in porcine lens. unassigned - 7.2.2.13 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23698121&form=6&db=m Human kidney-2 cells harbor functional dopamine D1 receptors that require Gi? for Gq/11? signaling. ongoing research,therapeutic application,unassigned 1,1,0 7.2.2.13 Wolfram Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17947299&form=6&db=m Sodium-potassium ATPase 1 subunit is a molecular partner of Wolframin, an endoplasmic reticulum protein involved in ER stress. causal interaction,therapeutic application,unassigned 3,1,0 7.2.2.13 Zika Virus Infection http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32669655&form=6&db=m Inhibition of Na+/K+ ATPase blocks Zika virus infection in mice. causal interaction,therapeutic application,unassigned 4,4,0