5.6.1.4	Bardet-Biedl Syndrome	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9782090&form=6&db=m	Cloning of a novel C-terminal kinesin (KIFC3) that maps to human chromosome 16q13-q21 and thus is a candidate gene for Bardet-Biedl syndrome.	causal interaction,unassigned	1,0	
5.6.1.4	Breast Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19789344&form=6&db=m	Overexpression of kinesins mediates docetaxel resistance in breast cancer cells.	ongoing research,unassigned	3,0	
5.6.1.4	Breast Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25348260&form=6&db=m	Kinesin-14 and kinesin-5 antagonistically regulate microtubule nucleation by ?-TuRC in yeast and human cells.	therapeutic application,unassigned	1,0	
5.6.1.4	Carcinoma, Hepatocellular	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31340839&form=6&db=m	KIFC1 is activated by TCF-4 and promotes hepatocellular carcinoma pathogenesis by regulating HMGA1 transcriptional activity.	causal interaction,diagnostic usage,ongoing research,unassigned	1,1,4,0	
5.6.1.4	Influenza, Human	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11581287&form=6&db=m	KIFC3, a microtubule minus end-directed motor for the apical transport of annexin XIIIb-associated Triton-insoluble membranes.	causal interaction,therapeutic application,unassigned	1,1,0	
5.6.1.4	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23895133&form=6&db=m	Discovery and Mechanistic Study of a Small Molecule Inhibitor for Motor Protein KIFC1.	causal interaction,diagnostic usage,therapeutic application,unassigned	3,1,3,0	
5.6.1.4	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25458601&form=6&db=m	Discovery of potent KIFC1 inhibitors using a method of integrated high-throughput synthesis and screening.	causal interaction,therapeutic application,unassigned	1,4,0	
5.6.1.4	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26117688&form=6&db=m	The Aspergillus nidulans bimC4 mutation provides an excellent tool for identification of kinesin-14 inhibitors.	causal interaction,therapeutic application,unassigned	1,1,0	
5.6.1.4	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26992853&form=6&db=m	A centrosome clustering protein, KIFC1, predicts aggressive disease course in serous ovarian adenocarcinomas.	causal interaction,diagnostic usage,ongoing research,therapeutic application	4,4,3,3	
5.6.1.4	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29123223&form=6&db=m	Structural basis of small molecule ATPase inhibition of a human mitotic kinesin motor protein.	causal interaction,unassigned	1,0	
5.6.1.4	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31796052&form=6&db=m	Identification of key genes in invasive clinically non-functioning pituitary adenoma by integrating analysis of DNA methylation and mRNA expression profiles.	causal interaction,unassigned	4,0	
5.6.1.4	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33215754&form=6&db=m	Distinct retrograde microtubule motor sets drive early and late endosome transport.	therapeutic application,unassigned	1,0	
5.6.1.4	plus-end-directed kinesin atpase deficiency	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31722992&form=6&db=m	Kinesin-5 Is Dispensable for Bipolar Spindle Formation and Elongation in Candida albicans, but Simultaneous Loss of Kinesin-14 Activity Is Lethal.	unassigned	-	
5.6.1.4	Retinal Degeneration	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9782090&form=6&db=m	Cloning of a novel C-terminal kinesin (KIFC3) that maps to human chromosome 16q13-q21 and thus is a candidate gene for Bardet-Biedl syndrome.	causal interaction,unassigned	1,0	
5.6.1.4	Seminoma	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28977870&form=6&db=m	C-terminal kinesin motor KIFC1 participates in facilitating proper cell division of human seminoma.	causal interaction,diagnostic usage,ongoing research,unassigned	3,3,3,0	
5.6.1.4	Stroke	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21156132&form=6&db=m	Bidirectional power stroke by ncd kinesin.	ongoing research,unassigned	1,0	
5.6.1.4	Stroke	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22449971&form=6&db=m	The C-terminus of kinesin-14 Ncd is a crucial component of the force generating mechanism.	unassigned	-	
5.6.1.4	Stroke	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27729532&form=6&db=m	Working stroke of the kinesin-14, ncd, comprises two substeps of different direction.	therapeutic application,unassigned	1,0