3.5.4.B9	Acquired Immunodeficiency Syndrome	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15609224&form=6&db=m	Exhaustive genotyping of the CEM15 (APOBEC3G) gene and absence of association with AIDS progression in a French cohort.	causal interaction,diagnostic usage,ongoing research,therapeutic application	1,3,2,1	
3.5.4.B9	Breast Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23852168&form=6&db=m	Evidence for APOBEC3B mutagenesis in multiple human cancers.	causal interaction,ongoing research,therapeutic application,unassigned	3,2,1,0	
3.5.4.B9	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28077648&form=6&db=m	Human Papillomavirus 16 E6 Upregulates APOBEC3B via the TEAD Transcription Factor.	causal interaction,diagnostic usage,ongoing research,unassigned	4,1,2,0	
3.5.4.B9	Infections	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18675436&form=6&db=m	APOBEC3G-independent reduction in virion infectivity during long-term HIV-1 replication in terminally differentiated macrophages.	therapeutic application,unassigned	1,0	
3.5.4.B9	Influenza, Human	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18999897&form=6&db=m	Apobec 3F and apobec 3G have no inhibition and hypermutation effect on the human influenza A virus.	ongoing research,therapeutic application,unassigned	2,1,0	
3.5.4.B9	Leukemia	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15254195&form=6&db=m	APOBEC3G targets specific virus species.	unassigned	-	
3.5.4.B9	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28077648&form=6&db=m	Human Papillomavirus 16 E6 Upregulates APOBEC3B via the TEAD Transcription Factor.	causal interaction,diagnostic usage,ongoing research,unassigned	4,1,2,0	
3.5.4.B9	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28646470&form=6&db=m	APOBEC3B and IL-6 form a positive feedback loop in hepatocellular carcinoma cells.	unassigned	-