3.1.6.2 3(or 17)beta-hydroxysteroid dehydrogenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2146126&form=6&db=m Isolated dehydroepiandrosterone sulphate hypersecretion: a case report. causal interaction,unassigned 4,0 3.1.6.2 Abortion, Spontaneous http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9764665&form=6&db=m Unconjugated estriol as an indication for prenatal diagnosis of steroid sulfatase deficiency by in situ hybridization. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Abortion, Spontaneous http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25636695&form=6&db=m Elevated levels of dehydroepiandrosterone as a potential mechanism of dendritic cell impairment during pregnancy. causal interaction,diagnostic usage,therapeutic application,unassigned 1,3,1,0 3.1.6.2 Abscess http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9271835&form=6&db=m Serum testosterone, DHEAS, and prolactin levels in patients with a Bartholin's abscess. diagnostic usage,unassigned 4,0 3.1.6.2 Acanthosis Nigricans http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32695697&form=6&db=m Correlation of Cutaneous Manifestations with Body Mass Index, Blood Glucose, and Hormonal Levels in Patients with Polycystic Ovarian Disease. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,2,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2145810&form=6&db=m Steroid sulfatase activity in epidermis of acne-prone and non-acne-prone skin of patients with acne vulgaris. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2526307&form=6&db=m [Androgenic evaluation of women with late-onset or persistent acne] diagnostic usage,unassigned 2,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7547392&form=6&db=m Clinical, ultrasound and hormonal markers of androgenicity in acne vulgaris. diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9003848&form=6&db=m Predictors of severity of acne vulgaris in young adolescent girls: results of a five-year longitudinal study. causal interaction,diagnostic usage,therapeutic application,unassigned 2,2,1,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10856473&form=6&db=m Metformin therapy decreases hyperandrogenism and hyperinsulinemia in women with polycystic ovary syndrome. causal interaction,diagnostic usage,therapeutic application,unassigned 1,3,3,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11334899&form=6&db=m Hyperandrogenemia in patients presenting with acne. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12398233&form=6&db=m The association of serum androsterone glucuronide with inflammatory lesions in women with adult acne. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12445184&form=6&db=m Cutaneous androgen metabolism: basic research and clinical perspectives. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12748463&form=6&db=m A randomized controlled trial of second- versus third-generation oral contraceptives in the treatment of acne vulgaris. therapeutic application,unassigned 3,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15781674&form=6&db=m Correlation between serum levels of insulin-like growth factor 1, dehydroepiandrosterone sulfate, and dihydrotestosterone and acne lesion counts in adult women. causal interaction,unassigned 3,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15950641&form=6&db=m Phenotypic spectrum of polycystic ovary syndrome: clinical and biochemical characterization of the three major clinical subgroups. ongoing research,unassigned 1,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18547986&form=6&db=m [Effect on skin and hormonal hyperandrogenic manifestations of an oral estroprogestin association containing ethynilestradiol 30 mg and drospirenone 3 mg] causal interaction,therapeutic application,unassigned 2,3,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20629741&form=6&db=m Metabolic alterations of DHEA and cholesterol sulphates in the hair of patients with acne measured by liquid chromatography-mass spectrometry. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22107839&form=6&db=m CAG repeat polymorphism in androgen receptor gene is not directly associated with polycystic ovary syndrome but influences serum testosterone levels. diagnostic usage,unassigned 2,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25176476&form=6&db=m Comparison of adrenocortical steroidogenesis in women with post-adolescent severe acne and polycystic ovary syndrome. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,1,0 3.1.6.2 Acne Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32192596&form=6&db=m Androgenicity and fertility treatment in women with unexplained infertility. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,2,0 3.1.6.2 Acquired Immunodeficiency Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10843528&form=6&db=m Dehydroepiandrosterone sulfate (DHEAS) and testosterone: relation to HIV illness stage and progression over one year. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Addison Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8473406&form=6&db=m Origin and significance of plasma androsterone glucuronide levels: a parameter of adrenal androgen secretion and hepatic 5 alpha-reductase activity. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,1,0 3.1.6.2 Addison Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27112552&form=6&db=m The importance of adrenal hypoandrogenism in infertile women with low functional ovarian reserve: a case study of associated adrenal insufficiency. diagnostic usage,unassigned 4,0 3.1.6.2 Adenocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2953750&form=6&db=m In situ steroid sulfatase activity in human epithelial carcinoma cells of vaginal, ovarian, and endometrial origin. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 3.1.6.2 Adenocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7955120&form=6&db=m Inhibition of protein farnesyltransferase: a possible mechanism of tumor prevention by dehydroepiandrosterone sulfate. causal interaction,ongoing research,unassigned 4,4,0 3.1.6.2 Adenocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10443857&form=6&db=m Lifelong treatment with oral DHEA sulfate does not preserve immune function, prevent disease, or improve survival in genetically heterogeneous mice. unassigned - 3.1.6.2 Adenocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11520136&form=6&db=m Steroid sulfatase expression in ovarian clear cell adenocarcinoma: immunohistochemical study. causal interaction,diagnostic usage,ongoing research,unassigned 2,1,4,0 3.1.6.2 Adenocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34386598&form=6&db=m Dehydroepiandrosterone modulates oxidative DNA damage in pancreatic cancer: A case-control study. causal interaction,diagnostic usage,unassigned 1,4,0 3.1.6.2 Adenocarcinoma, Clear Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11520136&form=6&db=m Steroid sulfatase expression in ovarian clear cell adenocarcinoma: immunohistochemical study. causal interaction,diagnostic usage,ongoing research,unassigned 2,1,4,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6217751&form=6&db=m Adrenal and sex steroid hormone production by a virilizing adrenal adenoma and its diagnosis with computerized tomography. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8165486&form=6&db=m Laparoscopic adrenalectomy. The importance of a flank approach in the lateral decubitus position. unassigned - 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9100554&form=6&db=m Plasma dehydroepiandrosterone sulfate levels in patients with hyperfunctioning and non-hyperfunctioning adrenal tumors before and after adrenal surgery. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9167966&form=6&db=m Adrenal incidentaloma: an overview of hormonal data from the National Italian Study Group. diagnostic usage,unassigned 3,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9734186&form=6&db=m [Incidentaloma of the adrenal glands: analysis of 9 surgical cases] causal interaction,diagnostic usage,unassigned 1,4,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10494643&form=6&db=m Asymptomatic adrenal tumours: criteria for endoscopic removal. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,2,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10822224&form=6&db=m The value of dehydroepiandrosterone sulfate measurement in the differentiation between benign and malignant adrenal masses. diagnostic usage,unassigned 1,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20530985&form=6&db=m Is there an association between non-functioning adrenal adenoma and endothelial dysfunction? causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25736067&form=6&db=m A case of Cushing's syndrome due to bilateral cortisol-secreting adenomas with unilateral DHEAS oversecretion. diagnostic usage,unassigned 1,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33739426&form=6&db=m Steroid sulfation in adrenal tumors. diagnostic usage,unassigned 3,0 3.1.6.2 Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34384108&form=6&db=m DHEAS and Differential Blood Counts as Indirect Signs of Glucocorticoid Excess in Adrenal Non-Producing Adenomas. causal interaction,diagnostic usage,therapeutic application,unassigned 4,1,1,0 3.1.6.2 Adenomyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11704174&form=6&db=m Immunohistologic localization of estrone sulfatase in uterine endometrium and adenomyosis. unassigned - 3.1.6.2 Adrenal Gland Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10822224&form=6&db=m The value of dehydroepiandrosterone sulfate measurement in the differentiation between benign and malignant adrenal masses. diagnostic usage,unassigned 1,0 3.1.6.2 Adrenal Gland Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32195126&form=6&db=m Isolated DHEAS production by an adrenal neoplasm: Clinical, biochemical and pathologic characteristics. therapeutic application,unassigned 1,0 3.1.6.2 Adrenal Hyperplasia, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2146126&form=6&db=m Isolated dehydroepiandrosterone sulphate hypersecretion: a case report. causal interaction,unassigned 4,0 3.1.6.2 Adrenal Hyperplasia, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6219334&form=6&db=m Disproportionate suppression of dehydroepiandrosterone sulfate (DHEAS) in treated patients with congenital adrenal hyperplasia due to 21-hydroxylase deficiency. causal interaction,diagnostic usage,therapeutic application,unassigned 4,1,1,0 3.1.6.2 Adrenal Hyperplasia, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7621563&form=6&db=m Adrenarche does not occur in treated patients with congenital adrenal hyperplasia resulting from 21-hydroxylase deficiency. causal interaction,unassigned 1,0 3.1.6.2 Adrenal Hyperplasia, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16259799&form=6&db=m Development of a method for the measurement of dehydroepiandrosterone sulphate by liquid chromatography-tandem mass spectrometry. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,1,0 3.1.6.2 Adrenal Hyperplasia, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19492582&form=6&db=m Hydrocortisone treatment in girls with congenital adrenal hyperplasia inhibits serum dehydroepiandrosterone sulfate and affects the GH-IGF-I system. causal interaction,therapeutic application,unassigned 4,3,0 3.1.6.2 Adrenal Hyperplasia, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33045962&form=6&db=m Premature Adrenarche and its Association with Cardiovascular Risk in Females. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Adrenal Hyperplasia, Congenital http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34539912&form=6&db=m Importance of Dehydroepiandrosterone Sulfate Assessment with Special Attention for Adrenal Tumours and Arterial Hypertension. causal interaction,unassigned 1,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11158009&form=6&db=m Androgen deficiency in women with hypopituitarism. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11186254&form=6&db=m Adrenal androgen and glucocorticoid dissociation in premenopausal rheumatoid arthritis: a significant correlate or precursor to onset? causal interaction,therapeutic application,unassigned 4,3,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12007898&form=6&db=m Dehydroepiandrosterone: a springboard hormone for female sexuality. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12398784&form=6&db=m Decreased levels of dehydroepiandrosterone sulphate in severe critical illness: a sign of exhausted adrenal reserve? causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,2,1 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16362829&form=6&db=m Adrenal steroids in adrenomyeloneuropathy Dehydroepiandrosterone sulfate, androstenedione and 17alpha-hydroxyprogesterone. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,2,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17157776&form=6&db=m DHEAS deficiency during consumption of sustained-action prescribed opioids: evidence for opioid-induced inhibition of adrenal androgen production. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22420492&form=6&db=m Effects of dehydroepiandrosterone sulphate (DHEAS) replacement on insulin action and quality of life in hypopituitary females: a double blind placebo controlled study. ongoing research,unassigned 4,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25359306&form=6&db=m Central adrenal insufficiency could not be confirmed by measurement of basal serum DHEAS levels in pubertal children. diagnostic usage,unassigned 2,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25617170&form=6&db=m Is DHEA replacement beneficial in chronic adrenal failure? causal interaction,unassigned 4,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27993552&form=6&db=m New PCOS-like phenotype in older infertile women of likely autoimmune adrenal etiology with high AMH but low androgens. unassigned - 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31388512&form=6&db=m Dehydroepiandrosterone: a potential therapeutic agent in the treatment and rehabilitation of the traumatically injured patient. causal interaction,therapeutic application,unassigned 2,2,0 3.1.6.2 Adrenal Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34539912&form=6&db=m Importance of Dehydroepiandrosterone Sulfate Assessment with Special Attention for Adrenal Tumours and Arterial Hypertension. causal interaction,unassigned 1,0 3.1.6.2 Adrenocortical Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704956&form=6&db=m Reduced serum levels of dehydroepiandrosterone sulphate in adrenal incidentalomas: a marker of adrenocortical tumour. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Adrenocortical Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9100554&form=6&db=m Plasma dehydroepiandrosterone sulfate levels in patients with hyperfunctioning and non-hyperfunctioning adrenal tumors before and after adrenal surgery. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Adrenocortical Adenoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17408424&form=6&db=m Benign cortisol-secreting adrenocortical adenomas produce small amounts of androgens. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,2,0 3.1.6.2 Adrenocortical Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9100554&form=6&db=m Plasma dehydroepiandrosterone sulfate levels in patients with hyperfunctioning and non-hyperfunctioning adrenal tumors before and after adrenal surgery. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Adrenocortical Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30909017&form=6&db=m Label-free impedimetric immunosensor based on arginine-functionalized gold nanoparticles for detection of DHEAS, a biomarker of pediatric adrenocortical carcinoma. diagnostic usage,unassigned 1,0 3.1.6.2 Adrenocortical Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33875173&form=6&db=m American Association of Clinical Endocrinology Disease State Clinical Review on the Evaluation and Management of Adrenocortical Carcinoma in an Adult: a Practical Approach. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 3.1.6.2 Adrenoleukodystrophy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16362829&form=6&db=m Adrenal steroids in adrenomyeloneuropathy Dehydroepiandrosterone sulfate, androstenedione and 17alpha-hydroxyprogesterone. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,2,0 3.1.6.2 Albinism, Ocular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1674724&form=6&db=m Linkage analysis in X-linked ocular albinism. unassigned - 3.1.6.2 Albinism, Ocular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2573275&form=6&db=m An Xp22 microdeletion associated with ocular albinism and ichthyosis: approximation of breakpoints and estimation of deletion size by using cloned DNA probes and flow cytometry. causal interaction,unassigned 2,0 3.1.6.2 Albinism, Ocular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8330450&form=6&db=m Deletion analysis maps ocular albinism proximal to the steroid sulphatase locus. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Albinism, Ocular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10443071&form=6&db=m Detection of KAL-1 gene deletion with fluorescence in situ hybridization. ongoing research,unassigned 1,0 3.1.6.2 Alopecia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7926083&form=6&db=m Alterations in androgen conjugate levels in women and men with alopecia. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Alopecia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11886493&form=6&db=m Steroid sulfatase in the human hair follicle concentrates in the dermal papilla. causal interaction,therapeutic application,unassigned 2,3,0 3.1.6.2 Alopecia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32148484&form=6&db=m Increased DHEAS and Decreased Total Testosterone Serum Levels in a Subset of Men with Early-Onset Androgenetic Alopecia: Does a Male PCOS-Equivalent Exist? diagnostic usage,therapeutic application,unassigned 2,1,0 3.1.6.2 Alopecia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34539912&form=6&db=m Importance of Dehydroepiandrosterone Sulfate Assessment with Special Attention for Adrenal Tumours and Arterial Hypertension. causal interaction,unassigned 1,0 3.1.6.2 alpha-Thalassemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2344613&form=6&db=m Frequent deletions of the human X chromosome distal short arm result from recombination between low copy repetitive elements. causal interaction,unassigned 3,0 3.1.6.2 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1835658&form=6&db=m Serum dehydroepiandrosterone sulfate in Alzheimer's disease and in multi-infarct dementia. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7870929&form=6&db=m Age and sex differences of dehydroepiandrosterone sulfate (DHEAS) and cortisol (CRT) plasma levels in normal controls and Alzheimer's disease (AD). diagnostic usage,ongoing research,unassigned 2,2,0 3.1.6.2 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8010622&form=6&db=m Dehydroepiandrosterone sulphate (DHEAS) in the oldest old, aged 85 and over. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9844034&form=6&db=m Dehydroepiandrosterone sulfate decreases the interleukin-2-mediated overactivity of the natural killer cell compartment in senile dementia of the Alzheimer type. diagnostic usage,ongoing research,unassigned 2,1,0 3.1.6.2 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11744094&form=6&db=m Age-related changes of the adrenal secretory pattern: possible role in pathological brain aging. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16583455&form=6&db=m Rapid column-switching liquid chromatography/mass spectrometric assay for DHEA-sulfate in the plasma of patients with Alzheimer's disease. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,2,0 3.1.6.2 Alzheimer Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32183671&form=6&db=m Dehydroepiandrosterone (DHEA) and its Sulphate (DHEAS) in Alzheimer's Disease. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 3.1.6.2 Amnesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8580738&form=6&db=m Reversal of scopolamine induced amnesia in rats by the steroid sulfatase inhibitor estrone-3-O-sulfamate. causal interaction,diagnostic usage,unassigned 1,2,0 3.1.6.2 Amnesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9000649&form=6&db=m Memory enhancement mediated by the steroid sulfatase inhibitor (p-O-sulfamoyl)-N-tetradecanoyl tyramine. diagnostic usage,ongoing research,unassigned 1,1,0 3.1.6.2 Amnesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9399636&form=6&db=m Chronic steroid sulfatase inhibition by (p-O-sulfamoyl)-N-tetradecanoyl tyramine increases dehydroepiandrosterone sulfate in whole brain. causal interaction,unassigned 1,0 3.1.6.2 Amnesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9409701&form=6&db=m Enhanced plasma DHEAS, brain acetylcholine and memory mediated by steroid sulfatase inhibition. unassigned - 3.1.6.2 Amnesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9675286&form=6&db=m Possible role of nitric oxide in the nootropic and antiamnesic effects of neurosteroids on aging- and dizocilpine-induced learning impairment. ongoing research,therapeutic application,unassigned 2,1,0 3.1.6.2 Amnesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10821934&form=6&db=m The effect of steroid sulfatase inhibition on learning and spatial memory. unassigned - 3.1.6.2 Anemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17237323&form=6&db=m Higher serum concentrations of DHEAS predict improved nutritional status in helminth-infected children, adolescents, and young adults in Leyte, the Philippines. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,2,0 3.1.6.2 Anencephaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9764665&form=6&db=m Unconjugated estriol as an indication for prenatal diagnosis of steroid sulfatase deficiency by in situ hybridization. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Angina Pectoris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9875107&form=6&db=m von Willebrand factor, tissue plasminogen activator, and dehydroepiandrosterone sulphate predict cardiovascular death in a 10 year follow up of survivors of acute myocardial infarction. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Angiomyolipoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8165486&form=6&db=m Laparoscopic adrenalectomy. The importance of a flank approach in the lateral decubitus position. unassigned - 3.1.6.2 Anosmia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3422798&form=6&db=m More on X-linked ichthyosis, steroid sulfatase deficiency, and hypogonadism and anosmia. unassigned - 3.1.6.2 Anosmia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477126&form=6&db=m X-linked ichthyosis due to steroid sulfatase deficiency associated with hypogonadism and anosmia. causal interaction,unassigned 3,0 3.1.6.2 Anosmia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3516063&form=6&db=m A new syndrome of anosmia, ichthyosis, hypogonadism, and various neurological manifestations with deficiency of steroid sulfatase and arylsulfatase C. causal interaction,unassigned 4,0 3.1.6.2 Anosmia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704955&form=6&db=m Hypogonadotrophic hypogonadism with hyposmia, X-linked ichthyosis, and renal malformation syndrome. diagnostic usage,unassigned 1,0 3.1.6.2 Anovulation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1623996&form=6&db=m The ratio of androstenedione:11 beta-hydroxyandrostenedione is an important marker of adrenal androgen excess in women. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 Anovulation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6281086&form=6&db=m Differential response to adrenocorticotropin hormone stimulation in polycystic ovarian disease with high and low dehydroepiandrosterone sulfate levels. causal interaction,unassigned 4,0 3.1.6.2 Anovulation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19474428&form=6&db=m Inactivating PAPSS2 mutations in a patient with premature pubarche. causal interaction,ongoing research,unassigned 4,1,0 3.1.6.2 Arteriosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31347187&form=6&db=m Association of endogenous DHEA/DHEAS with coronary heart disease: A systematic review and meta-analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Arteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16783861&form=6&db=m Low serum levels of DHEAS in untreated polymyalgia rheumatica/giant cell arteritis. diagnostic usage,ongoing research,unassigned 2,3,0 3.1.6.2 Arthralgia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33592022&form=6&db=m Dehydroepiandrostenedione sulphate (DHEAS) levels predict high risk of rheumatoid arthritis (RA) in subclinical hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 Arthritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11299059&form=6&db=m Hyposecretion of the adrenal androgen dehydroepiandrosterone sulfate and its relation to clinical variables in inflammatory arthritis. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,1,0 3.1.6.2 Arthritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22170453&form=6&db=m Relation between placental growth factor-1 (PlGF-1) and vascularization, DHEAS to DHEA conversion, or aromatase expression in patients with arthritis. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,2,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7955606&form=6&db=m Blood dehydroepiandrosterone sulphate (DHEAS) levels in polymyalgia rheumatica/giant cell arteritis and primary fibromyalgia. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8299250&form=6&db=m Relationship between blood and joint tissue DHEAS levels in rheumatoid arthritis and osteoarthritis. diagnostic usage,ongoing research,unassigned 2,3,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10875318&form=6&db=m Weak androgen levels, glucocorticoid therapy, and bone mineral density in postmenopausal women with rheumatoid arthritis. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,2,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12180752&form=6&db=m Basal levels of DHEAS as a marker for disease activity in premenopausal women with recent onset rheumatoid arthritis. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12508908&form=6&db=m Hypothalamic-pituitary-adrenal axis impairment in the pathogenesis of rheumatoid arthritis and polymyalgia rheumatica. causal interaction,unassigned 3,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15635504&form=6&db=m Adrenal androgens and the immune system. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17477470&form=6&db=m Increased DHEAS levels in patients with rheumatoid arthritis after treatment with tumor necrosis factor antagonists: evidence for improved adrenal function. causal interaction,therapeutic application,unassigned 3,3,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28712091&form=6&db=m Contribution of Genetic Factors to Lower DHEAS in Patients with Rheumatoid Arthritis. diagnostic usage,unassigned 4,0 3.1.6.2 Arthritis, Rheumatoid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33592022&form=6&db=m Dehydroepiandrostenedione sulphate (DHEAS) levels predict high risk of rheumatoid arthritis (RA) in subclinical hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=887310&form=6&db=m Steroid sulfatase deficiency. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1348358&form=6&db=m Multiple sulfatase deficiency: catalytically inactive sulfatases are expressed from retrovirally introduced sulfatase cDNAs. causal interaction,ongoing research,unassigned 3,1,0 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1687673&form=6&db=m Arylsulfatases A and B in EBV-transformed lymphoid cell lines: studies on their molecular forms in cells from patients with inborn sulfatase deficiencies. Comparative diagnostic value of enzymatic assays. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,4,0 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3083810&form=6&db=m Rapid degradation of steroid sulfatase in multiple sulfatase deficiency. causal interaction,ongoing research,unassigned 2,1,0 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165268&form=6&db=m Synthesis and stability of steroid sulfatase in fibroblasts from multiple sulfatase deficiency. causal interaction,ongoing research,unassigned 4,2,0 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3531932&form=6&db=m Placental sulfatase deficiency: maternal and fetal expression of steroid sulfatase deficiency and X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9336808&form=6&db=m Ichthyosis: the skin manifestation of multiple sulfatase deficiency. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 arylsulfatase (type i) deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27003302&form=6&db=m Steroid sulfatase sulfatase deficiency and androgen activation before and after puberty. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 Asthenozoospermia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3360602&form=6&db=m [Andrologic studies in patients with X-chromosome recessive ichthyosis] unassigned - 3.1.6.2 Asthma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9427150&form=6&db=m Allergic disease as an association of steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Asthma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11560105&form=6&db=m Effects of hormone replacement therapy on endocrine and spirometric parameters in asthmatic postmenopausal women. causal interaction,therapeutic application,unassigned 2,2,0 3.1.6.2 Asthma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17381481&form=6&db=m Independent association between insulin-like growth factor-I and dehydroepiandrosterone sulphate in women in middle adulthood. diagnostic usage,ongoing research,unassigned 2,4,0 3.1.6.2 Asthma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21708057&form=6&db=m Nebulized dehydroepiandrosterone-3-sulfate improves asthma control in the moderate-to-severe asthma results of a 6-week, randomized, double-blind, placebo-controlled study. diagnostic usage,ongoing research,unassigned 2,2,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1423966&form=6&db=m Dehydroepiandrosterone sulfate, incidence of myocardial infarction, and extent of atherosclerosis in men. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2145584&form=6&db=m Changes in plasma DHEAS circadian rhythm in elderly men. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7498969&form=6&db=m Effects of dehydroepiandrosterone sulfate on cellular calcium responsiveness and vascular contractility. ongoing research,unassigned 4,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8795944&form=6&db=m Relationship between dehydroepiandrosterone sulfate and serum lipid levels in Japanese men. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9884478&form=6&db=m Distribution of dehydroepiandrosterone sulfate and relationships between its level and serum lipid levels in a rural Japanese population. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,3,3 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10764679&form=6&db=m No association between dehydroepiandrosterone sulfate and development of atherosclerosis in a prospective population study (Bruneck Study). causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11260976&form=6&db=m [Replacement therapy in adrenopause. Dehydroepiandrosterone and aging] causal interaction,unassigned 3,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11765058&form=6&db=m Serum DHEAS levels correlate with platelet cGMP in normal women. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12161487&form=6&db=m Low levels of endogenous androgens increase the risk of atherosclerosis in elderly men: the Rotterdam study. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18255343&form=6&db=m Inhibition of vascular inflammation by dehydroepiandrosterone sulfate in human aortic endothelial cells: Roles of PPARalpha and NF-kappaB. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21533767&form=6&db=m Berberine cooperates with adrenal androgen dehydroepiandrosterone sulfate to attenuate PDGF-induced proliferation of vascular smooth muscle cell A7r5 through Skp2 signaling pathway. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27510480&form=6&db=m Adrenal Hyperandrogenism and Polycystic Ovary Syndrome. therapeutic application,unassigned 1,0 3.1.6.2 Atherosclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30031631&form=6&db=m Serum Amyloid A, Paraoxonase-1 Activity, and Apolipoprotein Concentrations as Biomarkers of Subclinical Atherosclerosis Risk in Adrenal Incidentaloma Patients. causal interaction,diagnostic usage,unassigned 1,4,0 3.1.6.2 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11909687&form=6&db=m Dehydroepiandrosterone-sulfate serum levels and common age-related diseases: results from a cross-sectional Italian study of a general elderly population. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Atrial Fibrillation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23152363&form=6&db=m Dehydroepiandrosterone sulfate levels and risk of atrial fibrillation: The Rotterdam Study. causal interaction,diagnostic usage,ongoing research,unassigned 2,4,2,0 3.1.6.2 Autoimmune Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7554562&form=6&db=m Blood dehydroepiandrosterone sulphate (DHEAS) levels in pemphigoid/pemphigus and psoriasis. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 3.1.6.2 Autoimmune Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9550284&form=6&db=m Administration of dehydroepiandrosterone sulfate retards onset but not progression of autoimmune disease in NZB/W mice. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Autoimmune Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11247320&form=6&db=m Dehydroepiandrosterone sulphate serum levels in systemic sclerosis. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Autoimmune Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12073659&form=6&db=m [The role of adrenal and gonadal hormones in the pathogenesis of autoimmune polyarthritis] causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Azoospermia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27264819&form=6&db=m [Genetic analysis of a rare case with Kallman syndrome and steroid sulfatase deficiency]. unassigned - 3.1.6.2 Beckwith-Wiedemann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34395010&form=6&db=m Functional Adrenocortical Adenoma in a Child with Beckwith-Wiedemann Syndrome. causal interaction,unassigned 2,0 3.1.6.2 Bone Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11853283&form=6&db=m Serum dehydroepiandrosterone sulfate, testosterone, and biochemical markers of bone turnover in elderly Thai men. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,4 3.1.6.2 Bone Diseases, Metabolic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8578930&form=6&db=m Dehydroepiandrosterone sulphate in the diagnosis of osteoporosis. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Bone Diseases, Metabolic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9650403&form=6&db=m [Reduced serum dehydroepiandrosterone levels in postmenopausal osteoporosis] causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,2,0 3.1.6.2 Bone Diseases, Metabolic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26813449&form=6&db=m The evaluation of hormonal and psychological parameters that affect bone mineral density in postmenopausal women. diagnostic usage,ongoing research,unassigned 2,4,0 3.1.6.2 Bone Diseases, Metabolic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30029729&form=6&db=m Dehydroepiandrosterone and Bone. diagnostic usage,therapeutic application,unassigned 2,1,0 3.1.6.2 Bone Resorption http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11853283&form=6&db=m Serum dehydroepiandrosterone sulfate, testosterone, and biochemical markers of bone turnover in elderly Thai men. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,4 3.1.6.2 Bone Resorption http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12007898&form=6&db=m Dehydroepiandrosterone: a springboard hormone for female sexuality. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Bone Resorption http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12091853&form=6&db=m Physiologic regulators of bone turnover in young women with anorexia nervosa. causal interaction,unassigned 3,0 3.1.6.2 Bone Resorption http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15589859&form=6&db=m Relationship between bone resorption and adrenal sex steroids and their derivatives in oophorectomized women. diagnostic usage,therapeutic application,unassigned 2,1,0 3.1.6.2 Brain Concussion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17669633&form=6&db=m DHEAS repeated treatment improves cognitive and behavioral deficits after mild traumatic brain injury. therapeutic application,unassigned 4,0 3.1.6.2 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22145821&form=6&db=m Dehydroepiandrosterone sulphate: action and mechanism in the brain. causal interaction,therapeutic application,unassigned 1,3,0 3.1.6.2 Brain Injuries http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16293666&form=6&db=m Dehydroepiandrosterone sulfate is neuroprotective when administered either before or after injury in a focal cortical cold lesion model. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Brain Injuries, Traumatic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30029726&form=6&db=m Astrocyte Neuroprotection and Dehydroepiandrosterone. therapeutic application,unassigned 4,0 3.1.6.2 Brain Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18249534&form=6&db=m Steroid sulfatase and sulfuryl transferase activities in human brain tumors. ongoing research,unassigned 3,0 3.1.6.2 Breast Cyst http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11958788&form=6&db=m A simple, accurate, and sensitive assay method of dehydroepiandrosterone sulfate: application for quantitative determination in human breast cyst and duct fluids. causal interaction,diagnostic usage,unassigned 2,1,0 3.1.6.2 Breast Cyst http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12163136&form=6&db=m Accumulation of 4- and 5-ene steroid sulfates in human breast cyst fluids. causal interaction,unassigned 3,0 3.1.6.2 Breast Cyst http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17467271&form=6&db=m Cytokines in human breast cyst fluid. ongoing research,unassigned 2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=141976&form=6&db=m Steroid hormones and human breast cancer. An hypothesis. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1751397&form=6&db=m In vitro effect of synthetic progestogens on estrone sulfatase activity in human breast carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,4,1 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1911438&form=6&db=m Inhibition of estrone sulfatase enzyme in human placenta and human breast carcinoma. causal interaction,ongoing research,therapeutic application,unassigned 3,4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1912612&form=6&db=m Estrone sulfatase activity and effect of antiestrogens on transformation of estrone sulfate in hormone-dependent vs. independent human breast cancer cell lines. ongoing research,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2145063&form=6&db=m Dehydroepiandrosterone sulfate and breast cancer risk. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2365501&form=6&db=m Estrone sulfate stimulates growth of nitrosomethylurea-induced breast carcinoma in vivo in the rat. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2559248&form=6&db=m Estrone sulfate and sulfatase activity in human breast cancer and endometrial cancer. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2796962&form=6&db=m [Local production of estrogen via aromatase and estrone sulfatase in breast cancer tissue] causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,2 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2942072&form=6&db=m Uptake and concentration of steroid hormones in mammary tissues. diagnostic usage,ongoing research,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4275735&form=6&db=m Steroid sulfatase activities in human breast tumors. causal interaction,ongoing research,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6581863&form=6&db=m Estrone and dehydroepiandrosterone sulfatase activities and plasma estrone sulfate levels in human breast carcinoma. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6725522&form=6&db=m In situ estrogen production via the estrone sulfatase pathway in breast tumors: relative importance versus the aromatase pathway. causal interaction,ongoing research,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7547880&form=6&db=m Inactivation of steroid sulfatase by an active site-directed inhibitor, estrone-3-O-sulfamate. ongoing research,unassigned 1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7626490&form=6&db=m The role of cytokines and sulphatase inhibitors in regulating oestrogen synthesis in breast tumours. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7696150&form=6&db=m Estrogenicity, antiestrogenicity and estrone sulfatase inhibition of estrone-3-amine and estrone-3-thiol. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7749143&form=6&db=m Dehydroepiandrosterone concentration in breast cancer tissue is related to its plasma gradient across the mammary gland. ongoing research,therapeutic application,unassigned 3,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7792836&form=6&db=m Estrone sulfate analogs as estrone sulfatase inhibitors. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,2,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8180114&form=6&db=m Inhibition of steroid sulphatase activity by steroidal methylthiophosphonates: potential therapeutic agents in breast cancer. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8180120&form=6&db=m Lack of inhibition of placental estrone sulfatase and aromatase enzymes by vitamin D3 and its analogs. causal interaction,ongoing research,therapeutic application,unassigned 1,2,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8388710&form=6&db=m Inhibition of estrone sulfatase and 17 beta-hydroxysteroid dehydrogenase by antiestrogens. causal interaction,ongoing research,therapeutic application,unassigned 2,4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8417823&form=6&db=m Inhibition of estrone sulfatase activity by estrone-3-methylthiophosphonate: a potential therapeutic agent in breast cancer. causal interaction,ongoing research,therapeutic application,unassigned 4,4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8476770&form=6&db=m Prognostic significance of aromatase and estrone sulfatase enzymes in human breast cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,4,1 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8478138&form=6&db=m Estrone sulfate promotes human breast cancer cell replication and nuclear uptake of estradiol in MCF-7 cell cultures. causal interaction,therapeutic application,unassigned 1,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8651588&form=6&db=m Regulation and inhibition of steroid sulfatase activity in breast cancer. ongoing research,therapeutic application,unassigned 3,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8664167&form=6&db=m Detection of breast cancer-associated estrone sulfatase in breast cancer biopsies and cell lines using polymerase chain reaction. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,4,4,1 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8895749&form=6&db=m In vivo activity of 4-methylcoumarin-7-O-sulfamate, a nonsteroidal, nonestrogenic steroid sulfatase inhibitor. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8918978&form=6&db=m Effect of nomegestrol acetate on estrone-sulfatase and 17beta-hydroxysteroid dehydrogenase activities in human breast cancer cells. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9009241&form=6&db=m Inhibition of placental estrone sulfatase activity and MCF-7 breast cancer cell proliferation by estrone-3-amino derivatives. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9044848&form=6&db=m Inhibition of estrone sulfatase and proliferation of human breast cancer cells by nonsteroidal (p-O-sulfamoyl)-N-alkanoyl tyramines. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9138660&form=6&db=m Relationship of serum dehydroepiandrosterone (DHEA), DHEA sulfate, and 5-androstene-3 beta, 17 beta-diol to risk of breast cancer in postmenopausal women. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9168894&form=6&db=m Daidzein sulfoconjugates are potent inhibitors of sterol sulfatase (EC 3.1.6.2). ongoing research,unassigned 2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9169912&form=6&db=m Relation of serum levels of testosterone and dehydroepiandrosterone sulfate to risk of breast cancer in postmenopausal women. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9361395&form=6&db=m Role, control and expression of estrone sulfatase and 17 beta-hydroxysteroid dehydrogenase activities in human breast cancer. ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9366497&form=6&db=m Measurement of oestrone sulphatase activity in white blood cells to monitor in vivo inhibition of steroid sulphatase activity by oestrone-3-O-sulphamate. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9449200&form=6&db=m Inhibition of estrone sulfatase in human liver microsomes by quercetin and other flavonoids. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9544207&form=6&db=m Steroidal and nonsteroidal sulfamates as potent inhibitors of steroid sulfatase. causal interaction,ongoing research,therapeutic application,unassigned 3,3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9618028&form=6&db=m The development of A-ring modified analogues of oestrone-3-O-sulphamate as potent steroid sulphatase inhibitors with reduced oestrogenicity. therapeutic application,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9654650&form=6&db=m Development of potent non-estrogenic estrone sulfatase inhibitors. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9673390&form=6&db=m Dehydroepiandrosterone sulfate estrogenic action at its physiological plasma concentration in human breast cancer cell lines. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9927050&form=6&db=m Steroid sulfatase expression is an independent predictor of recurrence in human breast cancer. diagnostic usage,ongoing research,unassigned 3,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10021916&form=6&db=m Synthesis and sulfatase inhibitory activities of (E)- and (Z)-4-hydroxytamoxifen sulfamates. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10093756&form=6&db=m Regulation of steroid sulphatase and oestradiol 17 beta-hydroxysteroid dehydrogenase in breast cancer. unassigned - 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10215035&form=6&db=m Development of (p-O-sulfamoyl)-N-alkanoyl-phenylalkyl amines as non-steroidal estrone sulfatase inhibitors. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10418996&form=6&db=m Recent advances in the development of steroid sulphatase inhibitors. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10419004&form=6&db=m Estrone sulfatase versus estrone sulfotransferase in human breast cancer: potential clinical applications. diagnostic usage,ongoing research,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10447965&form=6&db=m Synthesis and biological activity of the superestrogen (E)-17-oximino-3-O-sulfamoyl-1,3,5(10)-estratriene: x-ray crystal structure of (E)-17-oximino-3-hydroxy-1,3,5(10)-estratriene. ongoing research,unassigned 2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10767642&form=6&db=m Molecular epidemiology of breast cancer: genetic variation in steroid hormone metabolism. diagnostic usage,unassigned 2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10910045&form=6&db=m In vivo inhibition of estrone sulfatase activity and growth of nitrosomethylurea-induced mammary tumors by 667 COUMATE. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11033081&form=6&db=m Potent active site-directed inhibition of steroid sulphatase by tricyclic coumarin-based sulphamates. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11060442&form=6&db=m Differential increase of steroid sulfatase activity in XX and XY trophoblast cells from human term placenta with syncytia formation in vitro. unassigned - 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11070351&form=6&db=m Stimulation of MCF-7 breast cancer cell proliferation by estrone sulfate and dehydroepiandrosterone sulfate: inhibition by novel non-steroidal steroid sulfatase inhibitors. causal interaction,ongoing research,therapeutic application,unassigned 4,1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11091733&form=6&db=m Significance of Steroid Sulfatase Expression in Human Breast Cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,4,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11091739&form=6&db=m Localization of Estrone Sulfatase in Human Breast Carcinomas. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11250930&form=6&db=m Constitutive expression of the steroid sulfatase gene supports the growth of MCF-7 human breast cancer cells in vitro and in vivo. ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11282280&form=6&db=m Regulation of steroid sulphatase expression and activity in breast cancer. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11383450&form=6&db=m [Genetic polymorphism and variability of steroid hormone metabolism: connection with risk of developing breast neoplasms] diagnostic usage,unassigned 2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11530280&form=6&db=m Paradoxical effect of estradiol: it can block its own bioformation in human breast cancer cells. ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11832993&form=6&db=m Inhibition of oestrone sulphatase activity by tibolone and its metabolites. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11860358&form=6&db=m Review of estrone sulfatase and its inhibitors--an important new target against hormone dependent breast cancer. causal interaction,ongoing research,therapeutic application,unassigned 4,1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12161011&form=6&db=m Breast cancer: from estrogen to androgen receptor. ongoing research,unassigned 3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12163135&form=6&db=m Effect of tibolone (Org OD14) and its metabolites on aromatase and estrone sulfatase activity in human breast adipose stromal cells and in MCF-7 and T47D breast cancer cells. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12201223&form=6&db=m The influence of inflammatory cytokines on estrogen production and cell proliferation in human breast cancer cells. ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12413846&form=6&db=m Sulfamoyloxy-substituted 2-phenylindoles: antiestrogen-based inhibitors of the steroid sulfatase in human breast cancer cells. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12433043&form=6&db=m Distribution of 16alpha-[18F]fluoro-estradiol-3,17beta-disulfamate in rats, tumour-bearing mice and piglets. causal interaction,diagnostic usage,unassigned 1,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12441191&form=6&db=m Conversion of dehydroepiandrosterone sulfate at physiological plasma concentration into estrogens in MCF-7 cells. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12459709&form=6&db=m In situ production of estrogens in human breast carcinoma. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12475228&form=6&db=m Estrone 3-sulfate mimics, inhibitors of estrone sulfatase activity: homology model construction and docking studies. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12475726&form=6&db=m Steroidal oxathiazine inhibitors of estrone sulfatase. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,4,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12657638&form=6&db=m Structure of human estrone sulfatase suggests functional roles of membrane association. causal interaction,unassigned 1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12711003&form=6&db=m Norelgestromin as selective estrogen enzyme modulator in human breast cancer cell lines. Effect on sulfatase activity in comparison to medroxyprogesterone acetate. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12711019&form=6&db=m In vitro and in vivo models for the evaluation of new inhibitors of human steroid sulfatase, devoid of residual estrogenic activity. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,2,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12711020&form=6&db=m Development of a sensitive high-performance liquid chromatography method for the detection of 667 COUMATE in vivo. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12711022&form=6&db=m Inhibition of MCF-7 breast cancer cell proliferation and in vivo steroid sulphatase activity by 2-methoxyoestradiol-bis-sulphamate. ongoing research,unassigned 3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12782580&form=6&db=m Estrogen sulfotransferase and steroid sulfatase in human breast carcinoma. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12792725&form=6&db=m Immunohistochemical analysis of estrone sulfatase and aromatase in human breast cancer tissues. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12796397&form=6&db=m High expression of steroid sulfatase mRNA predicts poor prognosis in patients with estrogen receptor-positive breast cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14575357&form=6&db=m Postmenopausal breast cancer risk in relation to sex steroid hormones, prolactin and SHBG (Sweden). causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14623540&form=6&db=m Steroid sulphatase inhibitors for breast cancer therapy. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14623543&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in normal human tissue and breast carcinoma. ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14623544&form=6&db=m Role of steroid sulfatase in local formation of estrogen in post-menopausal breast cancer patients. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14637389&form=6&db=m Intracrine mechanism of estrogen synthesis in breast cancer. ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14670645&form=6&db=m Differential effects of progestins on breast tissue enzymes. unassigned - 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14672734&form=6&db=m Development of novel steroid sulfatase inhibitors. I. Synthesis and biological evaluation of biphenyl-4-O-sulfamates. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14979917&form=6&db=m Intratumoural mRNA expression of genes from the oestradiol metabolic pathway and clinical and histopathological parameters of breast cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,2,1 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15084348&form=6&db=m Development of novel steroid sulfatase inhibitors; II. TZS-8478 potently inhibits the growth of breast tumors in postmenopausal breast cancer model rats. causal interaction,ongoing research,therapeutic application,unassigned 4,4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15090207&form=6&db=m Confocal fluorescence detection expanded to UV excitation: the first continuous fluorimetric assay of human steroid sulfatase in nanoliter volume. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15145451&form=6&db=m 2-phenylindole sulfamates: inhibitors of steroid sulfatase with antiproliferative activity in MCF-7 breast cancer cells. unassigned - 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15453828&form=6&db=m Crystal structure of human carbonic anhydrase II at 1.95 A resolution in complex with 667-coumate, a novel anti-cancer agent. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15474441&form=6&db=m Understanding the biologic mechanisms responsible for breast-cancer progression during tamoxifen or fulvestrant treatment. causal interaction,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15597397&form=6&db=m Stilbene-based inhibitors of estrone sulfatase with a dual mode of action in human breast cancer cells. ongoing research,therapeutic application,unassigned 3,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15767920&form=6&db=m [Actions of a 19-norprogesterone derivative on mammary gland: nomegestrol acetate] ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15860265&form=6&db=m Recent insight on the control of enzymes involved in estrogen formation and transformation in human breast cancer. diagnostic usage,ongoing research,therapeutic application,unassigned 1,3,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15862969&form=6&db=m The effects of 2-methoxyoestrogen sulphamates on the in vitro and in vivo proliferation of breast cancer cells. causal interaction,therapeutic application,unassigned 1,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15900045&form=6&db=m Serum sex steroids in premenopausal women and breast cancer risk within the European Prospective Investigation into Cancer and Nutrition (EPIC). causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,3,3 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16078843&form=6&db=m A-ring-substituted estrogen-3-O-sulfamates: potent multitargeted anticancer agents. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16178776&form=6&db=m Steroid sulfatase inhibitors: their potential in the therapy of breast cancer. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16322318&form=6&db=m Sex steroid-producing enzymes in human breast cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,4,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16322344&form=6&db=m Postmenopausal serum androgens, oestrogens and breast cancer risk: the European prospective investigation into cancer and nutrition. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16533785&form=6&db=m Phase I Study of STX 64 (667 Coumate) in Breast Cancer Patients: The First Study of a Steroid Sulfatase Inhibitor. causal interaction,ongoing research,therapeutic application,unassigned 3,3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16541422&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in human prostate cancer. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16556483&form=6&db=m Quantitative analysis of aromatase, sulfatase and 17beta-HSD(1) mRNA expression in soft tissue metastases of breast cancer. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16702378&form=6&db=m The association of plasma DHEA and DHEA sulfate with breast cancer risk in predominantly premenopausal women. causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16864173&form=6&db=m Steroid sulfatase in breast carcinoma and change of serum estrogens levels after operation. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17000691&form=6&db=m In vivo efficacy of STX213, a second-generation steroid sulfatase inhibitor, for hormone-dependent breast cancer therapy. causal interaction,ongoing research,therapeutic application,unassigned 3,1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17061037&form=6&db=m Inhibition of steroid sulfatase activity and cell proliferation in ZR-75-1 and BT-474 human breast cancer cells by KW-2581 in vitro and in vivo. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17268815&form=6&db=m A novel steroidal selective steroid sulfatase inhibitor KW-2581 inhibits sulfated-estrogen dependent growth of breast cancer cells in vitro and in animal models. ongoing research,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17268816&form=6&db=m A novel dual-target steroid sulfatase inhibitor and antiestrogen: SR 16157, a promising agent for the therapy of breast cancer. therapeutic application,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17429562&form=6&db=m A new hormonal therapy for estrogen receptor-negative breast cancer. ongoing research,therapeutic application,unassigned 3,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17470679&form=6&db=m Steroid sulfatase: a new target for the endocrine therapy of breast cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17481887&form=6&db=m Estradiol inhibits the estrone sulfatase activity in normal and cancerous human breast tissues. causal interaction,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17596930&form=6&db=m Tissue-specific transcripts of human steroid sulfatase are under control of estrogen signaling pathways in breast carcinoma. causal interaction,ongoing research,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17604157&form=6&db=m Immunohistochemical analysis of steroid sulfatase in human tissues. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17728860&form=6&db=m Dual aromatase-sulfatase inhibitors based on the anastrozole template: synthesis, in vitro SAR, molecular modelling and in vivo activity. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17874044&form=6&db=m In situ production of sex steroids in human breast carcinoma. causal interaction,ongoing research,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17913430&form=6&db=m Chromatographic analysis and decomposition product characterization of compound SR16157 (NSC 732011). causal interaction,therapeutic application,unassigned 3,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17914670&form=6&db=m Efficacy of three potent steroid sulfatase inhibitors: pre-clinical investigations for their use in the treatment of hormone-dependent breast cancer. therapeutic application,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17943537&form=6&db=m Progestins and breast cancer. unassigned - 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17972413&form=6&db=m Dual aromatase-sulfatase inhibitors based on the anastrozole template: synthesis, in vitro SAR, molecular modelling and in vivo activity. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18288656&form=6&db=m Direct evidence for ArO-S bond cleavage upon inactivation of Pseudomonas aeruginosa arylsulfatase by aryl sulfamates. unassigned - 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18630506&form=6&db=m Inhibition of steroid sulphatase activity via the percutaneous route: a new option for breast cancer therapy. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18723489&form=6&db=m Anticancer steroid sulfatase inhibitors: synthesis of a potent fluorinated second-generation agent, in vitro and in vivo activities, molecular modeling, and protein crystallography. therapeutic application,unassigned 1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18780779&form=6&db=m Bone morphogenetic protein 6 (BMP6) and BMP7 inhibit estrogen-induced proliferation of breast cancer cells by suppressing p38 mitogen-activated protein kinase activation. causal interaction,ongoing research,therapeutic application,unassigned 1,4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18957523&form=6&db=m Association between alcohol intake and serum sex hormones and peptides differs by tamoxifen use in breast cancer survivors. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19022235&form=6&db=m Expression of 17beta-hydroxysteroid dehydrogenases and other estrogen-metabolizing enzymes in different cancer cell lines. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19308726&form=6&db=m Expression of estrogenicity genes in a lineage cell culture model of human breast cancer progression. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19419812&form=6&db=m Shikonin, an ingredient of Lithospermum erythrorhizon, down-regulates the expression of steroid sulfatase genes in breast cancer cells. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19559719&form=6&db=m Design and synthesis of carbon-11-labeled dual aromatase-steroid sulfatase inhibitors as new potential PET agents for imaging of aromatase and steroid sulfatase expression in breast cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 2,1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19747768&form=6&db=m Expression of sulfotransferases and sulfatases in human breast cancer: Impact on resveratrol metabolism. diagnostic usage,unassigned 3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20015473&form=6&db=m Shu-Gan-Liang-Xue Decoction, a Chinese herbal formula, down-regulates the expression of steroid sulfatase genes in human breast carcinoma MCF-7 cells. ongoing research,unassigned 3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20148564&form=6&db=m Highly potent first examples of dual aromatase-steroid sulfatase inhibitors based on a biphenyl template. therapeutic application,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20535958&form=6&db=m [Steroid sulfatase inhibitor] causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20937135&form=6&db=m Low salivary testosterone levels in patients with breast cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 2,3,2,1 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21342037&form=6&db=m Irosustat: a first-generation steroid sulfatase inhibitor in breast cancer. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21356310&form=6&db=m Steroid sulfatase inhibitors: Promising new tools for breast cancer therapy? causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,3,4 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21508387&form=6&db=m Effect of tamoxifen or anastrozole on steroid sulfatase activity and serum androgen concentrations in postmenopausal women with breast cancer. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21556246&form=6&db=m Expression of Estrogen Sulfotransferase 1E1 and Steroid Sulfatase in Breast Cancer: A Immunohistochemical Study. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21598196&form=6&db=m Using molecular docking to investigate the anti-breast cancer activity of low molecular weight compounds present on wild mushrooms. therapeutic application,unassigned 1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21608133&form=6&db=m Aromatase and dual aromatase-steroid sulfatase inhibitors from the letrozole and vorozole templates. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21647676&form=6&db=m Androgens and musculoskeletal symptoms among breast cancer patients on aromatase inhibitor therapy. causal interaction,ongoing research,therapeutic application,unassigned 4,2,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21925885&form=6&db=m Inhibition of steroid sulfatase with 4-substituted estrone and estradiol derivatives. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23136854&form=6&db=m Sulfatase inhibitors: a patent review. diagnostic usage,therapeutic application,unassigned 1,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23283524&form=6&db=m Postmenopausal plasma sex hormone levels and breast cancer risk over 20 years of follow-up. causal interaction,diagnostic usage,therapeutic application,unassigned 1,3,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23467843&form=6&db=m Shu-Gan-Liang-Xue Decoction Simultaneously Down-regulates Expressions of Aromatase and Steroid Sulfatase in Estrogen Receptor Positive Breast Cancer Cells. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23797179&form=6&db=m A phase I dose escalation study to determine the optimal biological dose of irosustat, an oral steroid sulfatase inhibitor, in postmenopausal women with estrogen receptor-positive breast cancer. causal interaction,ongoing research,therapeutic application,unassigned 4,4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23905452&form=6&db=m Steroid sulfatase inhibitors: promising new therapy for breast cancer. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24630694&form=6&db=m Design and synthesis of silicon-containing steroid sulfatase inhibitors possessing pro-estrogen antagonistic character. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24660681&form=6&db=m Structure Based Design, Synthesis, And Evaluation Of Potential Inhibitors Of Steroid Sulfatase. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24809288&form=6&db=m Anti-tumor effect of Shu-Gan-Liang-Xue decoction in breast cancer is related to the inhibition of aromatase and steroid sulfatase expression. therapeutic application,unassigned 1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24900302&form=6&db=m Hybrid dual aromatase-steroid sulfatase inhibitors with exquisite picomolar inhibitory activity. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26269086&form=6&db=m Synthesis and biological evaluation of thiophosphate tricyclic coumarin derivatives as steroid sulfatase inhibitors. causal interaction,therapeutic application,unassigned 3,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26974384&form=6&db=m Sulfatase inhibitors for recidivist breast cancer treatment: A chemical review. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27155470&form=6&db=m Discovery of a sulfamate-based steroid sulfatase inhibitor with intrinsic selective estrogen receptor modulator properties. therapeutic application,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28201180&form=6&db=m Constitutive Expression of the Steroid Sulfatase Gene Supports the Growth of MCF-7 Human Breast Cancer Cells in Vitroand in Vivo. ongoing research,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28612226&form=6&db=m IRIS study: a phase II study of the steroid sulfatase inhibitor Irosustat when added to an aromatase inhibitor in ER-positive breast cancer patients. ongoing research,therapeutic application,unassigned 4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28795252&form=6&db=m IPET study: an FLT-PET window study to assess the activity of the steroid sulfatase inhibitor irosustat in early breast cancer. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,3,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29071491&form=6&db=m Correction to: IRIS study: a phase II study of the steroid sulfatase inhibitor Irosustat when added to an aromatase inhibitor in ER-positive breast cancer patients. ongoing research,therapeutic application,unassigned 4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29563635&form=6&db=m In breast cancer subtypes steroid sulfatase (STS) is associated with less aggressive tumour characteristics. causal interaction,diagnostic usage,unassigned 1,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29684479&form=6&db=m Impairment of the Gn?11-controlled expression of claudin-1 and MMP-9 and collective migration of human breast cancer MCF-7 cells by DHEAS. causal interaction,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29803725&form=6&db=m Steroid sulfatase inhibition success and limitation in breast cancer clinical assays: An underlying mechanism. causal interaction,ongoing research,therapeutic application,unassigned 4,3,4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30320251&form=6&db=m C-3- and C-4-Substituted Bicyclic Coumarin Sulfamates as Potent Steroid Sulfatase Inhibitors. therapeutic application,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30604001&form=6&db=m Pre-diagnostic sex hormone levels and survival among breast cancer patients. diagnostic usage,unassigned 4,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30736188&form=6&db=m Intracrinology of breast microenvironment: hormonal status in nipple aspirate fluid and its relationship to breast cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,1,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31760088&form=6&db=m DHEAS prevents pro-metastatic and proliferative effects of 17ß-estradiol on MCF-7 breast cancer cells. ongoing research,therapeutic application,unassigned 4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31948996&form=6&db=m Inclusion of Endogenous Plasma Dehydroepiandrosterone Sulfate and Mammographic Density in Risk Prediction Models for Breast Cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,2,0 3.1.6.2 Breast Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33655862&form=6&db=m Anti-breast cancer potential of natural and synthetic coumarin derivatives. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,3,0 3.1.6.2 Campomelic Dysplasia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12364433&form=6&db=m Prenatal diagnosis of sex differentiation disorders: the role of fetal ultrasound. therapeutic application,unassigned 1,0 3.1.6.2 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24055520&form=6&db=m Induction of steroid sulfatase expression in PC-3 human prostate cancer cells by insulin-like growth factor II. causal interaction,ongoing research,therapeutic application,unassigned 3,4,3,0 3.1.6.2 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29941676&form=6&db=m Detection of a novel, primate-specific 'kill switch' tumor suppression mechanism that may fundamentally control cancer risk in humans: an unexpected twist in the basic biology of TP53. causal interaction,ongoing research,unassigned 1,2,0 3.1.6.2 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30306775&form=6&db=m Detection of a novel, primate-specific ‘kill switch’ tumor suppression mechanism that may fundamentally control cancer risk in humans: an unexpected twist in the basic biology of TP53 causal interaction,ongoing research,unassigned 1,2,0 3.1.6.2 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31195119&form=6&db=m Human steroid sulfatase enhances aerobic glycolysis through induction of HIF1? and glycolytic enzymes. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1427599&form=6&db=m [Biochemical study on steroid sulfatase and its clinical application to the obstetrics and gynecology] causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2953750&form=6&db=m In situ steroid sulfatase activity in human epithelial carcinoma cells of vaginal, ovarian, and endometrial origin. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8070131&form=6&db=m Serum levels of steroid sulfatase protein in gynecologic carcinomas. diagnostic usage,unassigned 4,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9167966&form=6&db=m Adrenal incidentaloma: an overview of hormonal data from the National Italian Study Group. diagnostic usage,unassigned 3,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11091739&form=6&db=m Localization of Estrone Sulfatase in Human Breast Carcinomas. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11321667&form=6&db=m Increased serum cortisol levels are associated with high tumour grade in patients with renal cell carcinoma. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11911290&form=6&db=m Serum dehydroepiandrosterone and dehydroepiandrosterone sulfate and risk of melanoma or squamous cell carcinoma of the skin. diagnostic usage,therapeutic application,unassigned 4,3,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12413846&form=6&db=m Sulfamoyloxy-substituted 2-phenylindoles: antiestrogen-based inhibitors of the steroid sulfatase in human breast cancer cells. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15355916&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in human endometrial carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,1 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18723074&form=6&db=m Steroid-converting enzymes in human ovarian carcinomas. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,4,3 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18843186&form=6&db=m Sulfatase Activity in Normal and Neoplastic Endometrium. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,1,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19141651&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in colon carcinoma: regulators of intratumoral estrogen concentrations and potent prognostic factors. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,2,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21073915&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in human carcinomas. diagnostic usage,ongoing research,therapeutic application,unassigned 3,1,2,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23531699&form=6&db=m Steroid sulphatase and oestrogen sulphotransferase in human non-small-cell lung carcinoma. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34257538&form=6&db=m Clinicopathological Significance of Estrogen Receptor ? and Estrogen Synthesizing/Metabolizing Enzymes in Urothelial Carcinoma of Urinary Bladder. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Carcinoma, Endometrioid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12491622&form=6&db=m [Biochemical study on steroid sulphatase in the endometrioid adenocarcinoma] causal interaction,ongoing research,unassigned 1,2,0 3.1.6.2 Carcinoma, Endometrioid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18843186&form=6&db=m Sulfatase Activity in Normal and Neoplastic Endometrium. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,1,0 3.1.6.2 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17594810&form=6&db=m [Molecular mechanisms of DHEA and DHEAs on apoptosis and cell cycle arrest via Akt pathway in hepatoma cell lines] diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Carcinoma, Intraductal, Noninfiltrating http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18310280&form=6&db=m Intratumoral concentration of sex steroids and expression of sex steroid-producing enzymes in ductal carcinoma in situ of human breast. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,1,0 3.1.6.2 Carcinoma, Non-Small-Cell Lung http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23531699&form=6&db=m Steroid sulphatase and oestrogen sulphotransferase in human non-small-cell lung carcinoma. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Carcinoma, Ovarian Epithelial http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18723074&form=6&db=m Steroid-converting enzymes in human ovarian carcinomas. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,4,3 3.1.6.2 Carcinoma, Ovarian Epithelial http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28588698&form=6&db=m Clinical significance of the estrogen-modifying enzymes steroid sulfatase and estrogen sulfotransferase in epithelial ovarian cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,1 3.1.6.2 Carcinoma, Renal Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11321667&form=6&db=m Increased serum cortisol levels are associated with high tumour grade in patients with renal cell carcinoma. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Carcinoma, Squamous Cell http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11911290&form=6&db=m Serum dehydroepiandrosterone and dehydroepiandrosterone sulfate and risk of melanoma or squamous cell carcinoma of the skin. diagnostic usage,therapeutic application,unassigned 4,3,0 3.1.6.2 Cardiomyopathy, Dilated http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18234363&form=6&db=m Dehydroepiandrosterone levels vary according as heart failure condition in patients with idiopathic dilated cardiomyopathy. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1530789&form=6&db=m Effects of age, smoking and vitamins on plasma DHEAS levels: a cross-sectional study in men. diagnostic usage,ongoing research,unassigned 4,4,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2532426&form=6&db=m Measurement of serum levels of dehydroepiandrosterone sulfate: a comparison of radioimmunoassay and enzymatic analysis. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2946952&form=6&db=m A prospective study of dehydroepiandrosterone sulfate, mortality, and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7498969&form=6&db=m Effects of dehydroepiandrosterone sulfate on cellular calcium responsiveness and vascular contractility. ongoing research,unassigned 4,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7718911&form=6&db=m [The relationship between serum dehydroepiandrosterone sulfate levels and factors associated with cardiovascular diseases: a cross-sectional study in Japan] causal interaction,diagnostic usage,ongoing research,unassigned 3,4,1,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8010622&form=6&db=m Dehydroepiandrosterone sulphate (DHEAS) in the oldest old, aged 85 and over. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597464&form=6&db=m The epidemiology of DHEAS and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,4,2,1 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8943798&form=6&db=m Dehydroepiandrosterone, dehydroepiandrosterone sulphate and cardiovascular disease. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9166121&form=6&db=m Differential effect of carbamazepine and valproate monotherapy on plasma levels of oestrone sulphate and dehydroepiandrosterone sulphate in male epileptic patients. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9884478&form=6&db=m Distribution of dehydroepiandrosterone sulfate and relationships between its level and serum lipid levels in a rural Japanese population. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,3,3 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10201649&form=6&db=m Relation of dehydroepiandrosterone and dehydroepiandrosterone sulfate with cardiovascular disease risk factors in women: longitudinal results from the Massachusetts Women's Health Study. ongoing research,therapeutic application,unassigned 3,1,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10372727&form=6&db=m Biotransformation of oral dehydroepiandrosterone in elderly men: significant increase in circulating estrogens. unassigned - 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11434668&form=6&db=m Increased visceral adipose tissue is associated with increased circulating insulin and decreased sex hormone binding globulin levels in massively obese adolescent girls. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11853289&form=6&db=m DHEA(S): the fountain of youth. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15166654&form=6&db=m Effect of moderate alcohol consumption on plasma dehydroepiandrosterone sulfate, testosterone, and estradiol levels in middle-aged men and postmenopausal women: a diet-controlled intervention study. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18255343&form=6&db=m Inhibition of vascular inflammation by dehydroepiandrosterone sulfate in human aortic endothelial cells: Roles of PPARalpha and NF-kappaB. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19298964&form=6&db=m Adrenal androgen dehydroepiandrosterone sulfate inhibits vascular remodeling following arterial injury. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20163485&form=6&db=m Cardiovascular disease is associated with greater incident dehydroepiandrosterone sulfate decline in the oldest old: the cardiovascular health study all stars study. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22640232&form=6&db=m Clinical correlates of sex steroids and gonadotropins in men over the late adulthood: the Framingham Heart Study. diagnostic usage,unassigned 1,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27771738&form=6&db=m Serum dehydroepiandrosterone levels are associated with lower risk of type 2 diabetes: the Rotterdam Study. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28476876&form=6&db=m Prognostic Value of Dehydroepiandrosterone Sulfate for Patients With Cardiovascular Disease: A Systematic Review and Meta-Analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 Cardiovascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30623167&form=6&db=m Cardiovascular Risk Factors and Dehydroepiandrosterone Sulfate Among Latinos in the Boston Puerto Rican Health Study. unassigned - 3.1.6.2 Carotid Artery Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20627295&form=6&db=m Dehydroepiandrosterone sulfate is inversely associated with sex-dependent diverse carotid atherosclerosis regardless of endothelial function. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,4,0 3.1.6.2 Cataract http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14732303&form=6&db=m Relationships between hormonal status and cataract in french postmenopausal women: the POLA study. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 Cerebral Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10064172&form=6&db=m The effect of dehydroepiandrosterone sulfate administration to patients with multi-infarct dementia. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Cholestasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29130021&form=6&db=m Transgenic Overexpression of Steroid Sulfatase Alleviates Cholestasis. unassigned - 3.1.6.2 Cholestasis, Intrahepatic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6448002&form=6&db=m Stable prolactin level after enhanced estradiol production following dehydroepiandrosterone sulfate. diagnostic usage,ongoing research,therapeutic application,unassigned 1,2,1,0 3.1.6.2 Chondrodysplasia Punctata http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1496984&form=6&db=m Role of the pseudoautosomal region in sex-chromosome pairing during male meiosis: meiotic studies in a man with a deletion of distal Xp. unassigned - 3.1.6.2 Chondrodysplasia Punctata http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1557308&form=6&db=m Prenatal diagnosis and investigation of a fetus with chondrodysplasia punctata, ichthyosis, and Kallmann syndrome due to an Xp deletion. causal interaction,unassigned 4,0 3.1.6.2 Chondrodysplasia Punctata http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1785631&form=6&db=m Deletion of the distal short arm of the X chromosome (Xp) in a patient with short stature, chondrodysplasia punctata, and X-linked ichthyosis due to steroid sulfatase deficiency. causal interaction,ongoing research,unassigned 2,2,0 3.1.6.2 Chondrodysplasia Punctata http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2339111&form=6&db=m Long-range restriction map of the terminal part of the short arm of the human X chromosome. causal interaction,unassigned 2,0 3.1.6.2 Chondrodysplasia Punctata http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2602357&form=6&db=m Contiguous gene syndromes due to deletions in the distal short arm of the human X chromosome. causal interaction,unassigned 3,0 3.1.6.2 Chondrodysplasia Punctata http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165728&form=6&db=m X/Y translocation in a family with X-linked ichthyosis, chondrodysplasia punctata, and mental retardation: DNA analysis reveals deletion of the steroid sulphatase gene and translocation of its Y pseudogene. unassigned - 3.1.6.2 Chondrodysplasia Punctata http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8832947&form=6&db=m X-linked dominant chondrodysplasia punctata with decreased dihydroxyacetone phosphate acyltransferase activity. causal interaction,ongoing research,therapeutic application,unassigned 4,1,2,0 3.1.6.2 Choriocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6452197&form=6&db=m Estrogen production as a tumor marker in patients with gonadotropin-producing neoplasms. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Choriocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12711019&form=6&db=m In vitro and in vivo models for the evaluation of new inhibitors of human steroid sulfatase, devoid of residual estrogenic activity. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,2,4 3.1.6.2 Choriocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19022235&form=6&db=m Expression of 17beta-hydroxysteroid dehydrogenases and other estrogen-metabolizing enzymes in different cancer cell lines. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Choriocarcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25919187&form=6&db=m Role of OAT4 in Uptake of Estriol Precursor 16?-Hydroxydehydroepiandrosterone Sulfate Into Human Placental Syncytiotrophoblasts From Fetus. ongoing research,unassigned 3,0 3.1.6.2 Chromosome Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24027193&form=6&db=m Are low maternal estriol levels a predictor for pro-opiomelanocortin (POMC) deficiency caused by POMC mutation during pregnancy? diagnostic usage,ongoing research,unassigned 3,1,0 3.1.6.2 Coinfection http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15139980&form=6&db=m Low plasma levels of dehydroepiandrosterone sulphate in HIV-positive patients coinfected with hepatitis C virus. diagnostic usage,ongoing research,unassigned 4,2,0 3.1.6.2 Colitis, Ulcerative http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9779307&form=6&db=m Blood and tissue dehydroepiandrosterone sulphate levels and their relationship to chronic inflammatory bowel disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,3 3.1.6.2 Colitis, Ulcerative http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9820396&form=6&db=m Association of humoral markers of inflammation and dehydroepiandrosterone sulfate or cortisol serum levels in patients with chronic inflammatory bowel disease. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,3,0 3.1.6.2 Colitis, Ulcerative http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12014422&form=6&db=m Association of dehydroepiandrosterone sulfate and testosterone deficiency with bone turnover in men with inflammatory bowel disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,1 3.1.6.2 Colonic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10815698&form=6&db=m Serum dehydroepiandrosterone and dehydroepiandrosterone sulfate and the subsequent risk of developing colon cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Colonic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17594810&form=6&db=m [Molecular mechanisms of DHEA and DHEAs on apoptosis and cell cycle arrest via Akt pathway in hepatoma cell lines] diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Colorectal Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23916543&form=6&db=m Reduced expression of steroid sulfatase in primary colorectal cancer. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Colorectal Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28326039&form=6&db=m Estrone Sulfate Transport and Steroid Sulfatase Activity in Colorectal Cancer: Implications for Hormone Replacement Therapy. diagnostic usage,therapeutic application,unassigned 1,4,0 3.1.6.2 Colorectal Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28945888&form=6&db=m Estrogen Activation by Steroid Sulfatase increases Colorectal Cancer proliferation via GPER. causal interaction,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Coma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22490610&form=6&db=m Reduced brain levels of DHEAS in hepatic coma patients: significance for increased GABAergic tone in hepatic encephalopathy. diagnostic usage,ongoing research,unassigned 4,3,0 3.1.6.2 Corneal Dystrophies, Hereditary http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23807007&form=6&db=m Pre-Descemet corneal dystrophy and X-linked ichthyosis associated with deletion of Xp22.31 containing the STS gene. unassigned - 3.1.6.2 Corneal Opacity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3162681&form=6&db=m Characterization of low-density lipoproteins from patients with recessive X-linked ichthyosis. unassigned - 3.1.6.2 Corneal Opacity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7943126&form=6&db=m Clinical pathologic correlation of superficial corneal opacities in X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Corneal Opacity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16403384&form=6&db=m X-linked recessive ichthyosis. unassigned - 3.1.6.2 Coronary Artery Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2147671&form=6&db=m Plasma levels of estradiol, testosterone, and DHEAS do not predict risk of coronary artery disease in men. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,3,1,1 3.1.6.2 Coronary Artery Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597465&form=6&db=m Dehydroepiandrosterone and coronary atherosclerosis. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 Coronary Artery Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11765058&form=6&db=m Serum DHEAS levels correlate with platelet cGMP in normal women. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 Coronary Artery Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16116338&form=6&db=m Dehydroepiandrosterone sulfate induces acute vasodilation of porcine coronary arteries in vitro and in vivo. causal interaction,unassigned 2,0 3.1.6.2 Coronary Artery Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16521435&form=6&db=m [Dehydroepiandrosterone (DHEA)--slows down the aging process?] causal interaction,ongoing research,unassigned 4,1,0 3.1.6.2 Coronary Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1423966&form=6&db=m Dehydroepiandrosterone sulfate, incidence of myocardial infarction, and extent of atherosclerosis in men. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Coronary Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7882484&form=6&db=m Dehydroepiandrosterone sulfate does not predict cardiovascular death in postmenopausal women. The Rancho Bernardo Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,2,1 3.1.6.2 Coronary Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7977274&form=6&db=m Dehydroepiandrosterone sulfate and the risk of myocardial infarction in US male physicians: a prospective study. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,2,0 3.1.6.2 Coronary Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8943798&form=6&db=m Dehydroepiandrosterone, dehydroepiandrosterone sulphate and cardiovascular disease. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Coronary Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14609305&form=6&db=m Are low dehydroepiandrosterone sulphate levels predictive for cardiovascular diseases? A review of prospective and retrospective studies. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Coronary Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17940987&form=6&db=m [Dehydroepiandrosterone therapy in men with angiographically verified coronary heart disease: the effects on plasminogen activator inhibitor-1 (PAI-1), tissue plasminogen activator (tPA) and fibrinogen plasma concentrations.] causal interaction,diagnostic usage,ongoing research,therapeutic application 2,2,2,2 3.1.6.2 Coronary Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31347187&form=6&db=m Association of endogenous DHEA/DHEAS with coronary heart disease: A systematic review and meta-analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 COVID-19 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34523302&form=6&db=m Does dehydroepiandrosterone sulfate have a role in COVID-19 prognosis and treatment? causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,1,0 3.1.6.2 Crohn Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9779307&form=6&db=m Blood and tissue dehydroepiandrosterone sulphate levels and their relationship to chronic inflammatory bowel disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,3 3.1.6.2 Crohn Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9820396&form=6&db=m Association of humoral markers of inflammation and dehydroepiandrosterone sulfate or cortisol serum levels in patients with chronic inflammatory bowel disease. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,3,0 3.1.6.2 Cryptorchidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477995&form=6&db=m [Steroid sulfatase and placental deficiency. Current data as instigators of new research] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Cryptorchidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6135610&form=6&db=m Clinical spectrum of steroid sulfatase deficiency: X-linked recessive ichthyosis, birth complications and cryptorchidism. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,4,0 3.1.6.2 Cryptorchidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12644929&form=6&db=m End-stage renal failure in a child with X-linked ichthyosis. causal interaction,unassigned 2,0 3.1.6.2 Cryptorchidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16403384&form=6&db=m X-linked recessive ichthyosis. unassigned - 3.1.6.2 Cryptorchidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24119255&form=6&db=m Nonsyndromic types of ichthyoses - an update. causal interaction,unassigned 3,0 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2146126&form=6&db=m Isolated dehydroepiandrosterone sulphate hypersecretion: a case report. causal interaction,unassigned 4,0 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704956&form=6&db=m Reduced serum levels of dehydroepiandrosterone sulphate in adrenal incidentalomas: a marker of adrenocortical tumour. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8165486&form=6&db=m Laparoscopic adrenalectomy. The importance of a flank approach in the lateral decubitus position. unassigned - 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17766720&form=6&db=m Effects of sex steroids on bone in women with subclinical or overt endogenous hypercortisolism. unassigned - 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25146553&form=6&db=m DHEAS for the prediction of subclinical Cushing's syndrome: perplexing or advantageous? diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25736067&form=6&db=m A case of Cushing's syndrome due to bilateral cortisol-secreting adenomas with unilateral DHEAS oversecretion. diagnostic usage,unassigned 1,0 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27696526&form=6&db=m Body composition in pituitary, adrenal and iatrogenic Cushing's syndrome and effects of DHEAS levels. causal interaction,ongoing research,unassigned 1,2,0 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27797672&form=6&db=m Low DHEAS: A Sensitive and Specific Test for the Detection of Subclinical Hypercortisolism in Adrenal Incidentalomas. diagnostic usage,therapeutic application,unassigned 4,1,0 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29860716&form=6&db=m A case of pheochromocytoma with negative MIBG scintigraphy, PET-CT and genetic tests (VHL included) and a rare case of post-operative erectile dysfunction. diagnostic usage,unassigned 3,0 3.1.6.2 Cushing Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30817193&form=6&db=m THE EVALUATION OF INCIDENTALLY DISCOVERED ADRENAL MASSES. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,3,0 3.1.6.2 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8165486&form=6&db=m Laparoscopic adrenalectomy. The importance of a flank approach in the lateral decubitus position. unassigned - 3.1.6.2 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9100554&form=6&db=m Plasma dehydroepiandrosterone sulfate levels in patients with hyperfunctioning and non-hyperfunctioning adrenal tumors before and after adrenal surgery. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Cysts http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12163136&form=6&db=m Accumulation of 4- and 5-ene steroid sulfates in human breast cyst fluids. causal interaction,unassigned 3,0 3.1.6.2 Deficiency Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2866054&form=6&db=m Steroid sulphatase deficiency disease. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1835658&form=6&db=m Serum dehydroepiandrosterone sulfate in Alzheimer's disease and in multi-infarct dementia. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8144828&form=6&db=m A prospective study of dehydroepiandrosterone sulfate and cognitive function in an older population: the Rancho Bernardo Study. diagnostic usage,ongoing research,therapeutic application,unassigned 2,1,1,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8917605&form=6&db=m Relationships of dehydroepiandrosterone sulfate in the elderly with functional, psychological, and mental status, and short-term mortality: a French community-based study. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,1,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10337439&form=6&db=m Dehydroepiandrosterone sulfate, diseases and mortality in a general aged population. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10796526&form=6&db=m Dehydroepiandrosterone (DHEA) supplementation for cognition and well-being. causal interaction,ongoing research,therapeutic application,unassigned 1,2,4,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10940758&form=6&db=m Hippocampal perfusion and pituitary-adrenal axis in Alzheimer's disease. causal interaction,unassigned 3,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11113605&form=6&db=m Pineal and pituitary-adrenocortical function in physiological aging and in senile dementia. diagnostic usage,unassigned 2,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11405958&form=6&db=m Dehydroepiandrosterone (DHEA) supplementation for cognitive function . ongoing research,therapeutic application,unassigned 3,3,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11909687&form=6&db=m Dehydroepiandrosterone-sulfate serum levels and common age-related diseases: results from a cross-sectional Italian study of a general elderly population. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14602750&form=6&db=m Neurosteroids: Cerebrospinal fluid levels for Alzheimer's disease and vascular dementia diagnostics. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15207411&form=6&db=m Cognitive and affective disorders in the elderly: a neuroendocrine study. unassigned - 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17435662&form=6&db=m Plasma testosterone, dehydroepiandrosterone sulfate, and cortisol in female patients with Huntington's disease. diagnostic usage,unassigned 2,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17636627&form=6&db=m WITHDRAWN: Dehydroepiandrosterone (DHEA) supplementation for cognitive function. ongoing research,therapeutic application,unassigned 3,3,0 3.1.6.2 Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19836631&form=6&db=m Growth factors decrease in subjects with mild to moderate Alzheimer's disease (AD): potential correction with dehydroepiandrosterone-sulphate (DHEAS). causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Dementia, Vascular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14602750&form=6&db=m Neurosteroids: Cerebrospinal fluid levels for Alzheimer's disease and vascular dementia diagnostics. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Dermatitis, Atopic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24974914&form=6&db=m Steroid concentrations in atopic dermatitis patients: Reduced plasma DHEAS and increased cortisone levels. diagnostic usage,unassigned 2,0 3.1.6.2 Dermatitis, Contact http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597463&form=6&db=m Relationship between humoral immunoaugmenting properties of DHEAS and IgD-receptor expression in young and aged mice. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7799892&form=6&db=m [Low levels of dehydroepiandrosterone sulfate in adult males with insulin-dependent diabetes mellitus] causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10337439&form=6&db=m Dehydroepiandrosterone sulfate, diseases and mortality in a general aged population. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15717807&form=6&db=m [A relationship between dehydroepiandrosterone sulphate and insulin resistance in obese men and women] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15877298&form=6&db=m Association of decrease in serum dehydroepiandrosterone sulfate levels with the progression to type 2 diabetes in men of a Japanese population: the Funagata Study. diagnostic usage,ongoing research,unassigned 4,4,0 3.1.6.2 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17381481&form=6&db=m Independent association between insulin-like growth factor-I and dehydroepiandrosterone sulphate in women in middle adulthood. diagnostic usage,ongoing research,unassigned 2,4,0 3.1.6.2 Diabetes Mellitus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26680487&form=6&db=m DHEA, DHEAS and prolactin correlate with glucose control parameters in women of fertile age with type-1 diabetes mellitus. diagnostic usage,unassigned 3,0 3.1.6.2 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7799892&form=6&db=m [Low levels of dehydroepiandrosterone sulfate in adult males with insulin-dependent diabetes mellitus] causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 Diabetes Mellitus, Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9678543&form=6&db=m Dehydroepiandrosterone sulfate does not prevent spontaneous and iodine-induced lymphocytic thyroiditis and diabetes mellitus in the BB/Wor rat. ongoing research,unassigned 3,0 3.1.6.2 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10509765&form=6&db=m Dietary composition affects regional body fat distribution and levels of dehydroepiandrosterone sulphate (DHEAS) in post-menopausal women with Type 2 diabetes. causal interaction,therapeutic application,unassigned 1,1,0 3.1.6.2 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15717807&form=6&db=m [A relationship between dehydroepiandrosterone sulphate and insulin resistance in obese men and women] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15877298&form=6&db=m Association of decrease in serum dehydroepiandrosterone sulfate levels with the progression to type 2 diabetes in men of a Japanese population: the Funagata Study. diagnostic usage,ongoing research,unassigned 4,4,0 3.1.6.2 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24497646&form=6&db=m Hepatic Overexpression of Steroid Sulfatase Ameliorates Mouse Models of Obesity and Type 2 Diabetes through Sex-specific Mechanisms. causal interaction,ongoing research,unassigned 1,3,0 3.1.6.2 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26923336&form=6&db=m Serum Dehydroepiandrosterone Sulfate and Risk for Type 2 Diabetes in Older Men and Women: The Pro.V.A Study. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27771738&form=6&db=m Serum dehydroepiandrosterone levels are associated with lower risk of type 2 diabetes: the Rotterdam Study. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Disorder of Sex Development, 46,XY http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12364433&form=6&db=m Prenatal diagnosis of sex differentiation disorders: the role of fetal ultrasound. therapeutic application,unassigned 1,0 3.1.6.2 Down Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10736251&form=6&db=m Placental synthesis of oestriol in Down's syndrome pregnancies. causal interaction,ongoing research,unassigned 3,2,0 3.1.6.2 Down Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12135803&form=6&db=m Advances in prenatal screening for Down syndrome: I. general principles and second trimester testing. unassigned - 3.1.6.2 Dwarfism, Pituitary http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8626841&form=6&db=m Influence of some biological indexes on sex hormone-binding globulin and androgen levels in aging or obese males. diagnostic usage,ongoing research,unassigned 2,3,0 3.1.6.2 Dyslipidemias http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33775010&form=6&db=m Dehydroepiandrosterone Sulfate (DHEAS) Levels in Polycystic Ovarian Syndrome (PCOS). diagnostic usage,ongoing research,therapeutic application,unassigned 2,1,2,0 3.1.6.2 Dysostoses http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9336808&form=6&db=m Ichthyosis: the skin manifestation of multiple sulfatase deficiency. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Dyspnea http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8917605&form=6&db=m Relationships of dehydroepiandrosterone sulfate in the elderly with functional, psychological, and mental status, and short-term mortality: a French community-based study. causal interaction,diagnostic usage,therapeutic application,unassigned 1,2,1,0 3.1.6.2 Encephalitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29796990&form=6&db=m Neuroprotective Effects of Dehydroepiandrosterone Sulfate Through Inhibiting Expression of Matrix Metalloproteinase-9 from Bradykinin-Challenged Astroglia. causal interaction,ongoing research,therapeutic application,unassigned 1,4,4,0 3.1.6.2 Endometrial Hyperplasia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6508360&form=6&db=m Estrone sulfatase activity in normal and abnormal endometrium. causal interaction,unassigned 4,0 3.1.6.2 Endometrial Hyperplasia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18467089&form=6&db=m In situ estrogen metabolism in proliferative endometria from untreated women with polycystic ovarian syndrome with and without endometrial hyperplasia. diagnostic usage,ongoing research,unassigned 1,1,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1427599&form=6&db=m [Biochemical study on steroid sulfatase and its clinical application to the obstetrics and gynecology] causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14648710&form=6&db=m Circulating levels of sex steroid hormones and risk of endometrial cancer in postmenopausal women. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,2,4 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15355916&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in human endometrial carcinoma. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,1 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18198614&form=6&db=m [Prognostic significance of assay of sex hormones, their receptors and enzymes involved in estrogen synthesis and metabolism in endometrial carcinoma] causal interaction,diagnostic usage,ongoing research,unassigned 3,4,1,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18450955&form=6&db=m The use of steroid sulfatase inhibitors as a novel therapeutic strategy against hormone-dependent endometrial cancer. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18817841&form=6&db=m Estrogen formation in endometrial and cervix cancer cell lines: Involvement of aromatase, steroid sulfatase and 17beta-hydroxysteroid dehydrogenases (types 1, 5, 7 and 12). diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19022235&form=6&db=m Expression of 17beta-hydroxysteroid dehydrogenases and other estrogen-metabolizing enzymes in different cancer cell lines. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21693170&form=6&db=m Steroid sulfatase: A pivotal player in estrogen synthesis and metabolism. causal interaction,therapeutic application,unassigned 2,1,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26996578&form=6&db=m The Role of Steroid Sulfatase as a Prognostic Factor in Patients with Endometrial Cancer. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Endometrial Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30217785&form=6&db=m The sulfatase pathway as estrogen supply in endometrial cancer. causal interaction,therapeutic application,unassigned 2,1,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15302278&form=6&db=m Gonadotropin-releasing hormone agonist inhibits estrone sulfatase expression of cystic endometriosis in the ovary. therapeutic application,unassigned 3,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16084891&form=6&db=m Strong expression of steroid sulfatase in human cumulus cells in patients with endometriosis. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,4,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17921479&form=6&db=m Estrogen metabolizing enzymes in endometrium and endometriosis. diagnostic usage,ongoing research,therapeutic application,unassigned 2,2,1,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18056119&form=6&db=m Inhibition of steroid sulphatase activity in endometriotic implants by 667 COUMATE: a potential new therapy. causal interaction,ongoing research,therapeutic application,unassigned 3,1,1,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21441545&form=6&db=m Inhibition of steroid sulfatase decreases endometriosis in an in vivo murine model. ongoing research,therapeutic application,unassigned 3,4,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23136854&form=6&db=m Sulfatase inhibitors: a patent review. diagnostic usage,therapeutic application,unassigned 1,1,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24604234&form=6&db=m Synergistic Effects of E2MATE and Norethindrone Acetate on Steroid Sulfatase Inhibition: A Randomized Phase I Proof-of-Principle Clinical Study in Women of Reproductive Age. therapeutic application,unassigned 4,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26723541&form=6&db=m Effects of steroid hormone on estrogen sulfotransferase and on steroid sulfatase expression in endometriosis tissue and stromal cells. diagnostic usage,ongoing research,unassigned 2,4,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30030390&form=6&db=m SULFATION PATHWAYS: Contribution of intracrine oestrogens to the aetiology of endometriosis. causal interaction,unassigned 4,0 3.1.6.2 Endometriosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30572523&form=6&db=m Oncological markers CA-125, CA 19-9 and endometriosis. causal interaction,unassigned 4,0 3.1.6.2 Epidermolysis Bullosa Simplex http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165358&form=6&db=m Recurrent bilateral corneal erosions due to an association of epidermolysis bullosa simplex Köbner and X-linked ichthyosis with steroid sulfatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15329076&form=6&db=m Physical growth and endocrinal disorders during pubertal maturation in girls with epilepsy. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15816945&form=6&db=m Seizure frequency and cortisol and dehydroepiandrosterone sulfate (DHEAS) levels in women with epilepsy receiving antiepileptic drug treatment. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,1,1 3.1.6.2 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20541012&form=6&db=m Effects of valproate and carbamazepine monotherapy on neuroactive steroids, their precursors and metabolites in adult men with epilepsy. causal interaction,unassigned 2,0 3.1.6.2 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29940375&form=6&db=m Effects of valproate on reproductive endocrine function in male patients with epilepsy: A systematic review and meta-analysis. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Epileptic Syndromes http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15816945&form=6&db=m Seizure frequency and cortisol and dehydroepiandrosterone sulfate (DHEAS) levels in women with epilepsy receiving antiepileptic drug treatment. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,1,1 3.1.6.2 Erectile Dysfunction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15780529&form=6&db=m Are dehydroepiandrosterone sulphate and lipids associated with erectile dysfunction? causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Erectile Dysfunction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34273785&form=6&db=m The Relationships of Dehydroepiandrosterone Sulfate, Erectile Function and General Psychological Health. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Erythema http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6592263&form=6&db=m Lipids in the pathogenesis of ichthyosis: topical cholesterol sulfate-induced scaling in hairless mice. causal interaction,ongoing research,therapeutic application,unassigned 3,1,2,0 3.1.6.2 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7982299&form=6&db=m Clinical implications of genetic and acquired defects in catecholamine synthesis and metabolism. causal interaction,unassigned 3,0 3.1.6.2 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9633788&form=6&db=m Relationship between blood pressure variability and serum dehydroepiandrosterone sulfate levels. diagnostic usage,ongoing research,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Essential Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10580744&form=6&db=m A close association between insulin resistance and dehydroepiandrosterone sulfate in subjects with essential hypertension. diagnostic usage,ongoing research,unassigned 4,3,0 3.1.6.2 Euthyroid Sick Syndromes http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7827351&form=6&db=m Altered adrenal and thyroid function in children with insulin-dependent diabetes mellitus. causal interaction,unassigned 1,0 3.1.6.2 Fatigue Syndrome, Chronic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15110921&form=6&db=m Levels of DHEA and DHEAS and responses to CRH stimulation and hydrocortisone treatment in chronic fatigue syndrome. therapeutic application,unassigned 3,0 3.1.6.2 Fatty Liver http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12828082&form=6&db=m The association of dehydroepiandrosterone, obesity, waist-hip ratio and insulin resistance with fatty liver in postmenopausal women--a hyperinsulinemic euglycemic insulin clamp study. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,3,0 3.1.6.2 Fatty Liver http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25786523&form=6&db=m Dehydroepiandrosterone sulfate and cytochrome P450 inducers alleviate fatty liver in male rats fed an orotic acid-supplemented diet. ongoing research,therapeutic application,unassigned 3,3,0 3.1.6.2 Fetal Death http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12135803&form=6&db=m Advances in prenatal screening for Down syndrome: I. general principles and second trimester testing. unassigned - 3.1.6.2 Fetal Death http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12850625&form=6&db=m Maternal serum unconjugated estriol as a predictor for Smith-Lemli-Opitz syndrome and other fetal conditions. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Fetal Growth Retardation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14764786&form=6&db=m Serum dehydroepiandrosterone sulfate levels and pubarche in short children born small for gestational age before and during growth hormone treatment. unassigned - 3.1.6.2 Fibromyalgia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12534357&form=6&db=m During a corticotropin-releasing hormone test in healthy subjects, administration of a beta-adrenergic antagonist induced secretion of cortisol and dehydroepiandrosterone sulfate and inhibited secretion of ACTH. diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Follicular Cyst http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20493737&form=6&db=m Isosexual pseudoprecocious puberty in a 2(1/2)-year-old girl presenting with intense skin pigmentation. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Fragile X Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23574351&form=6&db=m A family with fragile X syndrome, Duchenne muscular dystrophy and ichthyosis transmitted by an asymptomatic carrier. causal interaction,unassigned 1,0 3.1.6.2 Genetic Diseases, Inborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=887310&form=6&db=m Steroid sulfatase deficiency. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 Genetic Diseases, Inborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477995&form=6&db=m [Steroid sulfatase and placental deficiency. Current data as instigators of new research] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Genetic Diseases, Inborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10828635&form=6&db=m Male-pattern baldness in men with X-linked recessive ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Genetic Diseases, Inborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14571075&form=6&db=m Male-pattern baldness is common in men with X-linked recessive ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Genetic Diseases, Inborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15607112&form=6&db=m Mutations in X-linked ichthyosis disrupt the active site structure of estrone/DHEA sulfatase. causal interaction,unassigned 4,0 3.1.6.2 Genetic Diseases, Inborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23791652&form=6&db=m Xp22.3 interstitial deletion: A recognizable chromosomal abnormality encompassing VCX3A and STS genes in a patient with X-linked ichthyosis and mental retardation. causal interaction,unassigned 3,0 3.1.6.2 Genetic Diseases, Inborn http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30531180&form=6&db=m Placental steroid sulphatase deficiency: an approach to antenatal care and delivery. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Giant Cell Arteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16783861&form=6&db=m Low serum levels of DHEAS in untreated polymyalgia rheumatica/giant cell arteritis. diagnostic usage,ongoing research,unassigned 2,3,0 3.1.6.2 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=647375&form=6&db=m Drug-induced differentiation of a rat glioma in vitro: II. the expression of S-100, a glial specific protein and steroid sulfatase. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Glucose Intolerance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8550794&form=6&db=m Dehydroepiandrosterone, dehydroepiandrosterone sulfate, obesity, waist-hip ratio, and noninsulin-dependent diabetes in postmenopausal women: the Rancho Bernardo Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,1 3.1.6.2 Goiter http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34539912&form=6&db=m Importance of Dehydroepiandrosterone Sulfate Assessment with Special Attention for Adrenal Tumours and Arterial Hypertension. causal interaction,unassigned 1,0 3.1.6.2 Guillain-Barre Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8289085&form=6&db=m Neurosteroids in cerebrospinal fluid in neurologic disorders. causal interaction,diagnostic usage,unassigned 1,3,0 3.1.6.2 Gynecomastia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16355805&form=6&db=m Leptin levels in boys with pubertal gynecomastia. causal interaction,diagnostic usage,ongoing research,unassigned 1,2,4,0 3.1.6.2 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1386322&form=6&db=m Dehydroepiandrosterone (DHEA) levels in patients with prostatic cancer, heart diseases and under surgery stress. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2946952&form=6&db=m A prospective study of dehydroepiandrosterone sulfate, mortality, and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 3.1.6.2 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7882484&form=6&db=m Dehydroepiandrosterone sulfate does not predict cardiovascular death in postmenopausal women. The Rancho Bernardo Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,2,1 3.1.6.2 Heart Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9590600&form=6&db=m Low dehydroepiandrosterone sulfate and heart disease in middle-aged men: cross-sectional results from the Massachusetts Male Aging Study. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10843161&form=6&db=m The plasma levels of dehydroepiandrosterone sulfate are decreased in patients with chronic heart failure in proportion to the severity. diagnostic usage,ongoing research,unassigned 4,2,0 3.1.6.2 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12534357&form=6&db=m During a corticotropin-releasing hormone test in healthy subjects, administration of a beta-adrenergic antagonist induced secretion of cortisol and dehydroepiandrosterone sulfate and inhibited secretion of ACTH. diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16053781&form=6&db=m [A comparative survey of sex hormones levels between elderly men with chronic heart failure and normal elderly subjects] causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 Heart Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18234363&form=6&db=m Dehydroepiandrosterone levels vary according as heart failure condition in patients with idiopathic dilated cardiomyopathy. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Hepatic Encephalopathy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22490610&form=6&db=m Reduced brain levels of DHEAS in hepatic coma patients: significance for increased GABAergic tone in hepatic encephalopathy. diagnostic usage,ongoing research,unassigned 4,3,0 3.1.6.2 Hepatitis B http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10774266&form=6&db=m The effect of endogenous dehydroepiandrosterone sulphate on antibody response to hepatitis B vaccine in neonates. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,2,1 3.1.6.2 Hepatitis B http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23728318&form=6&db=m Role of serum dehydroepiandrosterone sulfate level on the clearance of chronic hepatitis B virus infection. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Hepatoblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17594810&form=6&db=m [Molecular mechanisms of DHEA and DHEAs on apoptosis and cell cycle arrest via Akt pathway in hepatoma cell lines] diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Hepatomegaly http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9043970&form=6&db=m Effects of acute administration of dehydroepiandrosterone-sulfate on adipose tissue mass and cellularity in male rats. causal interaction,diagnostic usage,unassigned 1,1,0 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7581984&form=6&db=m Selective increases in adrenal steroidogenic capacity during acute respiratory disease in infants. causal interaction,unassigned 3,0 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8964884&form=6&db=m Novel presence of luteinizing hormone/chorionic gonadotropin receptors in human adrenal glands. unassigned - 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12128283&form=6&db=m Dissecting human adrenal androgen production. causal interaction,unassigned 2,0 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15635503&form=6&db=m Adrenal androgens and aging. unassigned - 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16917887&form=6&db=m Adrenarche and the evolution of human life history. unassigned - 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18259112&form=6&db=m McCune-Albright Syndrome with Male Premature Pubarche of Unusual Origin. causal interaction,unassigned 3,0 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20649839&form=6&db=m Neurosteroid transport by the organic solute transporter OST?-OST?. unassigned - 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21622478&form=6&db=m Premature adrenarche: novel lessons from early onset androgen excess. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Herpes Zoster http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32194506&form=6&db=m DHEAS and Human Development: An Evolutionary Perspective. unassigned - 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2246674&form=6&db=m Cushing's syndrome: new variants and association with empty sella syndrome. causal interaction,unassigned 4,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2940031&form=6&db=m The effect of dehydroepiandrosterone sulfate prolactin and testosterone hormones in female fertility and hirsutism. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,1,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6261197&form=6&db=m Serum levels of DHEAS in gynecologic endocrinopathy and infertility. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8690105&form=6&db=m The efficacy of 250 mg/day flutamide in the treatment of patients with hirsutism. therapeutic application,unassigned 3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8752612&form=6&db=m Finasteride in the treatment of hirsutism: new therapeutic perspectives. causal interaction,diagnostic usage,unassigned 1,1,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9696220&form=6&db=m Idiopathic hirsutism: an uncommon cause of hirsutism in Alabama. diagnostic usage,unassigned 2,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10219882&form=6&db=m Clinical efficacy of lower dose flutamide 125 mg/day in the treatment of hirsutism. diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10376445&form=6&db=m Finasteride treatment for one year in 35 hirsute patients. diagnostic usage,therapeutic application,unassigned 1,3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10685333&form=6&db=m Total testosterone and DHEAS levels as predictors of androgen-secreting neoplasms: a populational study. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,1,2 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10856473&form=6&db=m Metformin therapy decreases hyperandrogenism and hyperinsulinemia in women with polycystic ovary syndrome. causal interaction,diagnostic usage,therapeutic application,unassigned 1,3,3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11886493&form=6&db=m Steroid sulfatase in the human hair follicle concentrates in the dermal papilla. causal interaction,therapeutic application,unassigned 2,3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12223963&form=6&db=m [Hirsutism and hypertrichosis in adults: investigations and treatment] unassigned - 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14607575&form=6&db=m Sibutramine has a positive effect on clinical and metabolic parameters in obese patients with polycystic ovary syndrome. causal interaction,diagnostic usage,unassigned 2,4,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15950641&form=6&db=m Phenotypic spectrum of polycystic ovary syndrome: clinical and biochemical characterization of the three major clinical subgroups. ongoing research,unassigned 1,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16316811&form=6&db=m Predictive value of glucose-insulin ratio in PCOS and profile of women who will benefit from metformin therapy: obese, lean, hyper or normoinsulinemic? causal interaction,diagnostic usage,therapeutic application,unassigned 3,1,1,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16543255&form=6&db=m Clomiphene citrate and dexamethazone in treatment of clomiphene citrate-resistant polycystic ovary syndrome: a prospective placebo-controlled study. causal interaction,unassigned 3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18854802&form=6&db=m Treatment of hyperandrogenism and hyperinsulinemia in PCOS patients with essential aminoacids. A pilot clinical study. diagnostic usage,unassigned 3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19352298&form=6&db=m Androgen dependence of hirsutism, acne, and alopecia in women: retrospective analysis of 228 patients investigated for hyperandrogenism. causal interaction,unassigned 2,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21041013&form=6&db=m Modified Ferriman-Gallwey hirsutism score and androgen levels in Turkish women. causal interaction,diagnostic usage,unassigned 2,4,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21856600&form=6&db=m The Hirsute Woman: Challenges in the Evaluation and Management. diagnostic usage,therapeutic application,unassigned 1,3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22107839&form=6&db=m CAG repeat polymorphism in androgen receptor gene is not directly associated with polycystic ovary syndrome but influences serum testosterone levels. diagnostic usage,unassigned 2,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22296561&form=6&db=m Impact of adrenal versus ovarian androgen ratio on signs and symptoms of polycystic ovarian syndrome. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28575537&form=6&db=m Association between biochemical hyperandrogenism parameters and Ferriman-Gallwey score in patients with polycystic ovary syndrome: A systematic review and meta-regression analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 2,2,3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28924479&form=6&db=m Marked hyperandrogenicity in a 60-year-old woman. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31256251&form=6&db=m The effect of probiotics, prebiotics, and synbiotics on hormonal and inflammatory indices in women with polycystic ovary syndrome: a systematic review and meta-analysis. causal interaction,unassigned 3,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31346998&form=6&db=m Effects of Metformin Treatment on Soluble Leptin Receptor Levels in Women with Polycystic Ovary Syndrome. diagnostic usage,ongoing research,therapeutic application,unassigned 2,3,4,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31582418&form=6&db=m First Report of Retroperitoneal Mucinous Cystadenoma in a Patient with Hirsutism. unassigned - 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32442282&form=6&db=m Screening for Androgen Excess in Women: Accuracy of Self-Reported Excess Body Hair Growth and Menstrual Dysfunction. diagnostic usage,unassigned 1,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32695697&form=6&db=m Correlation of Cutaneous Manifestations with Body Mass Index, Blood Glucose, and Hormonal Levels in Patients with Polycystic Ovarian Disease. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,2,0 3.1.6.2 Hirsutism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32892549&form=6&db=m The role and importance of auxiliary tests in differential diagnosis in patients with mildly high basal 17-oh-progesterone levels in the evaluation of hirsutism. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,1,0 3.1.6.2 HIV Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8740936&form=6&db=m Relationship between sex steroid hormone levels and CD4 lymphocytes in HIV infected men. causal interaction,unassigned 4,0 3.1.6.2 HIV Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9093780&form=6&db=m Blood cortisol and dehydroepiandrosterone sulphate (DHEAS) levels and CD4 T cell counts in HIV infection. causal interaction,diagnostic usage,unassigned 1,1,0 3.1.6.2 HIV Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18003813&form=6&db=m Usefulness of alternate prognostic serum and plasma markers for antiretroviral therapy for human immunodeficiency virus type 1 infection. diagnostic usage,ongoing research,unassigned 4,4,0 3.1.6.2 HIV Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19576195&form=6&db=m HIV-1 clade C infection and progressive disruption in the relationship between cortisol, DHEAS and CD4 cell numbers: a two-year follow-up study. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,4,4 3.1.6.2 Hydrocephalus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7521964&form=6&db=m Second-trimester maternal serum screening using alpha-fetoprotein, human chorionic gonadotrophin, and unconjugated oestriol: experience of a regional programme. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,1,0 3.1.6.2 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8165486&form=6&db=m Laparoscopic adrenalectomy. The importance of a flank approach in the lateral decubitus position. unassigned - 3.1.6.2 Hyperaldosteronism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10494643&form=6&db=m Asymptomatic adrenal tumours: criteria for endoscopic removal. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,2,0 3.1.6.2 Hyperalgesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19422393&form=6&db=m Intrathecal injection of the neurosteroid, DHEAS, produces mechanical allodynia in mice: involvement of spinal sigma-1 and GABA(A) receptors. ongoing research,therapeutic application,unassigned 1,1,0 3.1.6.2 Hyperalgesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26876754&form=6&db=m Peripheral neurosteroids enhance P2X receptor-induced mechanical allodynia via a sigma-1 receptor-mediated mechanism. causal interaction,therapeutic application,unassigned 1,1,0 3.1.6.2 Hyperalgesia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31030988&form=6&db=m Neurosteroid dehydroepiandrosterone sulphate enhances pain transmission in rat spinal cord dorsal horn. diagnostic usage,ongoing research,therapeutic application,unassigned 1,2,1,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6281086&form=6&db=m Differential response to adrenocorticotropin hormone stimulation in polycystic ovarian disease with high and low dehydroepiandrosterone sulfate levels. causal interaction,unassigned 4,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8062947&form=6&db=m Ovarian suppression with triptorelin and adrenal stimulation with adrenocorticotropin in functional hyperadrogenism: role of adrenal and ovarian cytochrome P450c17 alpha. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,2,1 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8884544&form=6&db=m Clinical features and hormonal characteristics in a case of ovarian arrhenoblastoma. unassigned - 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9093190&form=6&db=m Lack of an ovarian function influence on the increased adrenal androgen secretion present in women with functional ovarian hyperandrogenism. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,1,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10887449&form=6&db=m Hirsutism in women. Effective therapy that is safe for long-term use. diagnostic usage,unassigned 3,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12387582&form=6&db=m Diurnal rhythm and effects of oral contraceptives on serum dehydroepiandrosterone sulfate (DHEAS) are related to alterations in serum albumin rather than to changes in adrenocortical steroid secretion. causal interaction,unassigned 3,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17336975&form=6&db=m Investigation of hypothalamo-pituitary-gonadal axis and glucose intolerance among the first-degree female relatives of women with polycystic ovary syndrome. causal interaction,unassigned 3,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17392600&form=6&db=m Prevalence and metabolic characteristics of adrenal androgen excess in hyperandrogenic women with different phenotypes. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18377903&form=6&db=m Clinical, endocrine, and metabolic effects of acarbose, a alpha-glucosidase inhibitor, in overweight and nonoverweight patients with polycystic ovarian syndrome. diagnostic usage,ongoing research,therapeutic application,unassigned 3,2,3,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18547986&form=6&db=m [Effect on skin and hormonal hyperandrogenic manifestations of an oral estroprogestin association containing ethynilestradiol 30 mg and drospirenone 3 mg] causal interaction,therapeutic application,unassigned 2,3,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21041013&form=6&db=m Modified Ferriman-Gallwey hirsutism score and androgen levels in Turkish women. causal interaction,diagnostic usage,unassigned 2,4,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22835278&form=6&db=m Leydig cell hyperplasia and Leydig cell tumour in postmenopausal women: report of two cases. therapeutic application,unassigned 2,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25565293&form=6&db=m Adrenal androgen excess and body mass index in polycystic ovary syndrome. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25585504&form=6&db=m Influence of adrenal hyperandrogenism on the clinical and metabolic phenotype of women with polycystic ovary syndrome. diagnostic usage,ongoing research,unassigned 2,1,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28620546&form=6&db=m Ovarian morphology is associated with insulin resistance in women with polycystic ovary syndrome: a cross sectional study. diagnostic usage,ongoing research,unassigned 2,2,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30820213&form=6&db=m Comparison of Two Statistical Methods to Determine Normal Range of Androgen Hormones: K-Means Cluster Analysis and Receiver Operating Characteristic Curve. diagnostic usage,therapeutic application,unassigned 4,1,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30958034&form=6&db=m Association of Luteinizing hormone and LH receptor gene polymorphism with susceptibility of Polycystic ovary syndrome. unassigned - 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31461705&form=6&db=m Low DHEAS Concentration in a Girl Presenting with Short Stature and Premature Pubarche: A Novel PAPSS2 Gene Mutation. causal interaction,unassigned 4,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32259819&form=6&db=m High DHEAS Level in Girls Is Associated with Earlier Pubertal Maturation and Mild Increase in Androgens throughout Puberty without Affecting Postmenarche Ovarian Morphology. diagnostic usage,ongoing research,unassigned 4,2,0 3.1.6.2 Hyperandrogenism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34155264&form=6&db=m Impact of rs2414096 polymorphism of CYP19 gene on susceptibility of polycystic ovary syndrome and hyperandrogenism in Kashmiri women. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,2,1 3.1.6.2 Hypercholesterolemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6940862&form=6&db=m Increased cholesterol sulfate in plasma and red blood cell membranes of steroid sulfatase deficient patients. unassigned - 3.1.6.2 Hypercholesterolemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597464&form=6&db=m The epidemiology of DHEAS and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,4,2,1 3.1.6.2 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1835937&form=6&db=m No acute effect of physiological insulin increase on dehydroepiandrosterone sulfate in women with obesity and/or polycystic ovarian disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,1,1 3.1.6.2 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11118002&form=6&db=m Dehydroepiandrosterone sulfate and beta-cell function: enhanced glucose-induced insulin secretion and altered gene expression in rodent pancreatic beta-cells. ongoing research,unassigned 3,0 3.1.6.2 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18692843&form=6&db=m Impact of glycemic variations on the regulation of androgen metabolism in obese women with polycystic ovary syndrome. unassigned - 3.1.6.2 Hyperglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28179626&form=6&db=m Hypothalamic-pituitary-adrenal-axis dysregulation and double product increases potentiate ischemic heart disease risk in a Black male cohort: the SABPA study. causal interaction,unassigned 2,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1826009&form=6&db=m Effect of acute physiological elevations of insulin on circulating androgen levels in nonobese women. causal interaction,unassigned 2,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1835937&form=6&db=m No acute effect of physiological insulin increase on dehydroepiandrosterone sulfate in women with obesity and/or polycystic ovarian disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,1,1 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2550341&form=6&db=m Lack of effect of insulin or insulin-like growth factor I on the steroid sulfatase activity of human placental cytotrophoblasts. unassigned - 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7561622&form=6&db=m Acute hyperinsulinemia, androgen homeostasis and insulin sensitivity in healthy man. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7799892&form=6&db=m [Low levels of dehydroepiandrosterone sulfate in adult males with insulin-dependent diabetes mellitus] causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8028500&form=6&db=m Hyperinsulinemia is associated with menstrual irregularity and altered serum androgens in Pima Indian women. causal interaction,unassigned 4,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8949487&form=6&db=m [Polycystic ovary and diazoxide. In vivo study] unassigned - 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8966389&form=6&db=m [Androgen response in women with polycystic ovary syndrome and hyperinsulinemia during stimulation with corticotrophin and inhibition with dexamethasone] unassigned - 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11008070&form=6&db=m Growth and pubertal development following pediatric heart transplantation: a 15-year experience at Ste-Justine Hospital. unassigned - 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12223963&form=6&db=m [Hirsutism and hypertrichosis in adults: investigations and treatment] unassigned - 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12749433&form=6&db=m Troglitazone decreases adrenal androgen levels in women with polycystic ovary syndrome. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,1 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15302613&form=6&db=m Vanadyl sulfate ameliorates insulin resistance and restores plasma dehydroepiandrosterone-sulfate levels in fructose-fed, insulin-resistant rats. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16117191&form=6&db=m Role of dehydroepiandrosterone sulfate levels on body composition after laparoscopic adjustable gastric banding in pre-menopausal morbidly obese women. causal interaction,unassigned 2,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16579998&form=6&db=m Serum soluble leptin receptor levels and free leptin index in women with polycystic ovary syndrome: relationship to insulin resistance and androgens. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,3,1 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18439591&form=6&db=m Dehydroepiandrosterone sulfate and insulin resistance in patients with polycystic ovary syndrome. diagnostic usage,ongoing research,therapeutic application,unassigned 4,2,2,0 3.1.6.2 Hyperinsulinism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21713743&form=6&db=m Prevention of high fructose-induced metabolic syndrome in male wistar rats by aqueous extract of Tamarindus indica seed. causal interaction,unassigned 3,0 3.1.6.2 Hyperlipoproteinemia Type II http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26079405&form=6&db=m Gonadal steroids, gonadotropins and DHEAS in young adults with familial hypercholesterolemia who had initiated statin therapy in childhood. causal interaction,therapeutic application,unassigned 2,4,0 3.1.6.2 Hyperprolactinemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1459258&form=6&db=m Effect of hyperprolactinemia on the androgen response to an oral glucose load. causal interaction,unassigned 3,0 3.1.6.2 Hyperprolactinemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2246674&form=6&db=m Cushing's syndrome: new variants and association with empty sella syndrome. causal interaction,unassigned 4,0 3.1.6.2 Hypersensitivity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31030988&form=6&db=m Neurosteroid dehydroepiandrosterone sulphate enhances pain transmission in rat spinal cord dorsal horn. diagnostic usage,ongoing research,therapeutic application,unassigned 1,2,1,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8586291&form=6&db=m Dehydroepiandrosterone sulfate in a long-term care aged population. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597464&form=6&db=m The epidemiology of DHEAS and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,4,2,1 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9590600&form=6&db=m Low dehydroepiandrosterone sulfate and heart disease in middle-aged men: cross-sectional results from the Massachusetts Male Aging Study. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9633788&form=6&db=m Relationship between blood pressure variability and serum dehydroepiandrosterone sulfate levels. diagnostic usage,ongoing research,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9821661&form=6&db=m Activation of 11 beta-hydroxysteroid dehydrogenase by dehydroepiandrosterone sulphate as an anti-hypertensive agent in spontaneously hypertensive rats. causal interaction,diagnostic usage,unassigned 1,2,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9875107&form=6&db=m von Willebrand factor, tissue plasminogen activator, and dehydroepiandrosterone sulphate predict cardiovascular death in a 10 year follow up of survivors of acute myocardial infarction. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10604493&form=6&db=m Relation between dehydroepiandrosterone sulfate and blood pressure levels in a population-based sample. unassigned - 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10925210&form=6&db=m Steroid sulfatase inhibitor alters blood pressure and steroid profiles in hypertensive rats. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11159150&form=6&db=m Low dehydroepiandrosterone and ischemic heart disease in middle-aged men: prospective results from the Massachusetts Male Aging Study. unassigned - 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12807512&form=6&db=m Left ventricular mass index and ventricular septum thickness are associated with serum dehydroepiandrosterone-sulphate levels in hypertensive women. diagnostic usage,ongoing research,unassigned 2,1,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20163485&form=6&db=m Cardiovascular disease is associated with greater incident dehydroepiandrosterone sulfate decline in the oldest old: the cardiovascular health study all stars study. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21307886&form=6&db=m Cortisol, dehydroepiandrosterone sulphate, their ratio and hypertension: evidence of associations in male veterans from the Vietnam Experience Study. causal interaction,diagnostic usage,ongoing research,unassigned 1,1,2,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26375874&form=6&db=m Analysis of relationships between the concentrations of total testosterone and dehydroepiandrosterone sulfate and the occurrence of selected metabolic disorders in aging men. causal interaction,diagnostic usage,ongoing research,therapeutic application 2,2,2,1 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27771738&form=6&db=m Serum dehydroepiandrosterone levels are associated with lower risk of type 2 diabetes: the Rotterdam Study. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28179626&form=6&db=m Hypothalamic-pituitary-adrenal-axis dysregulation and double product increases potentiate ischemic heart disease risk in a Black male cohort: the SABPA study. causal interaction,unassigned 2,0 3.1.6.2 Hypertension http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34539912&form=6&db=m Importance of Dehydroepiandrosterone Sulfate Assessment with Special Attention for Adrenal Tumours and Arterial Hypertension. causal interaction,unassigned 1,0 3.1.6.2 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9100552&form=6&db=m Decreased serum IGF-I and dehydroepiandrosterone sulphate may be risk factors for the development of reduced bone mass in postmenopausal women with endogenous subclinical hyperthyroidism. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,1,1 3.1.6.2 Hyperthyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11523906&form=6&db=m Serum concentration of androstenediol and androstenediol sulfate in patients with hyperthyroidism and hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,3,0 3.1.6.2 Hypoglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18692843&form=6&db=m Impact of glycemic variations on the regulation of androgen metabolism in obese women with polycystic ovary syndrome. unassigned - 3.1.6.2 Hypoglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23490274&form=6&db=m Cortisol hyporesponsiveness to the low dose ACTH test is a frequent finding in a pediatric population with type 1 diabetes mellitus. diagnostic usage,unassigned 1,0 3.1.6.2 Hypoglycemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28201960&form=6&db=m Hypoglycemia and Glucocorticoid-free Immunosuppression are Related With Low Dehydroepiandrosterone Sulfate Level. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3007328&form=6&db=m X-linked ichthyosis, due to steroid sulphatase deficiency, associated with Kallmann syndrome (hypogonadotropic hypogonadism and anosmia): linkage relationships with Xg and cloned DNA sequences from the distal short arm of the X chromosome. causal interaction,unassigned 3,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3422798&form=6&db=m More on X-linked ichthyosis, steroid sulfatase deficiency, and hypogonadism and anosmia. unassigned - 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477126&form=6&db=m X-linked ichthyosis due to steroid sulfatase deficiency associated with hypogonadism and anosmia. causal interaction,unassigned 3,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477995&form=6&db=m [Steroid sulfatase and placental deficiency. Current data as instigators of new research] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3516063&form=6&db=m A new syndrome of anosmia, ichthyosis, hypogonadism, and various neurological manifestations with deficiency of steroid sulfatase and arylsulfatase C. causal interaction,unassigned 4,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6489382&form=6&db=m Steroid sulphatase deficiency and hypogonadism. causal interaction,unassigned 4,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6584254&form=6&db=m Ichthyosis vulgaris with hypogenitalism and hypogonadism: evidence for different genotypes by lipoprotein electrophoresis and steroid sulfatase testing. causal interaction,unassigned 4,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704955&form=6&db=m Hypogonadotrophic hypogonadism with hyposmia, X-linked ichthyosis, and renal malformation syndrome. diagnostic usage,unassigned 1,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8552218&form=6&db=m Primary hypogonadism in females with infantile onset spinocerebellar ataxia. diagnostic usage,ongoing research,unassigned 4,2,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9499614&form=6&db=m X-linked ichthyosis with hypogonadism: not always Kallmann's syndrome. causal interaction,unassigned 3,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11158009&form=6&db=m Androgen deficiency in women with hypopituitarism. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15309306&form=6&db=m [Hormone therapy in the aging male. Estrogen, DHEA, melatonin, somatotropin] causal interaction,diagnostic usage,unassigned 2,1,0 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26266675&form=6&db=m Inhibin B, AMH, but not INSL3, IGF1 or DHEAS support differentiation between constitutional delay of growth and puberty and hypogonadotropic hypogonadism. unassigned - 3.1.6.2 Hypogonadism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27910716&form=6&db=m Persistent amenorrhea and decreased DHEAS to cortisol ratio after recovery from anorexia nervosa. causal interaction,unassigned 4,0 3.1.6.2 Hypophosphatemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1679744&form=6&db=m Mapping of the glycine receptor alpha 2-subunit gene and the GABAA alpha 3-subunit gene on the mouse X chromosome. unassigned - 3.1.6.2 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6449317&form=6&db=m Plasma dehydroepiandrosterone sulphate in hypothyroid premenopausal women. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 3.1.6.2 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11523906&form=6&db=m Serum concentration of androstenediol and androstenediol sulfate in patients with hyperthyroidism and hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,3,0 3.1.6.2 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11909687&form=6&db=m Dehydroepiandrosterone-sulfate serum levels and common age-related diseases: results from a cross-sectional Italian study of a general elderly population. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Hypothyroidism http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33592022&form=6&db=m Dehydroepiandrostenedione sulphate (DHEAS) levels predict high risk of rheumatoid arthritis (RA) in subclinical hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=80684&form=6&db=m Enzymatic basis of typical X-linked icthyosis. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=160423&form=6&db=m X-linked ichthyosis: a metabolic disease. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=294955&form=6&db=m Steroid sulfatase deficiency and X-linked ichthyosis. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=575348&form=6&db=m Deficiency of arylsulfatase C in cultured skin fibroblasts of X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1427599&form=6&db=m [Biochemical study on steroid sulfatase and its clinical application to the obstetrics and gynecology] causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1496984&form=6&db=m Role of the pseudoautosomal region in sex-chromosome pairing during male meiosis: meiotic studies in a man with a deletion of distal Xp. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1539590&form=6&db=m Identification of point mutations in the steroid sulfatase gene of three patients with X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1785631&form=6&db=m Deletion of the distal short arm of the X chromosome (Xp) in a patient with short stature, chondrodysplasia punctata, and X-linked ichthyosis due to steroid sulfatase deficiency. causal interaction,ongoing research,unassigned 2,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1830629&form=6&db=m Increased levels of DHEAS in serum of patients with X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1893078&form=6&db=m Keratinocyte lipid fluidity under the influence of cholesterols, hydrocortisones, "active lipid", tocopherol and retinoic acid--a fluorescence polarization study with regard to physiological and pathophysiological epidermopoiesis and its therapeutic accessibility. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1898710&form=6&db=m Testis cancer. Ichthyosis constitutes a significant risk factor. therapeutic application,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1951426&form=6&db=m Two cases of steroid sulfatase deficiency with complex phenotype due to contiguous gene deletions. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1983039&form=6&db=m Physical mapping of deletion breakpoints in patients with X-linked ichthyosis: evidence for clustering of distal and proximal breakpoints. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2080119&form=6&db=m Steroid sulfatase activity in nails: screening for X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2095187&form=6&db=m Commercial assay for steroid sulphatase activity in X-linked ichthyosis. diagnostic usage,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2158060&form=6&db=m [Ichthyosis and steroid sulfatase: study of enzymatic activity in leukocytes and fibroblasts according to the sex and type of ichthyosis] causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2253421&form=6&db=m [Two cases of X-linked ichthyosis associated with myopathies] causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2366000&form=6&db=m Detection of carriers for X-linked ichthyosis by Southern blot analysis and identification of one family with a de novo mutation. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2424217&form=6&db=m Lipoprotein electrophoresis in recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2424236&form=6&db=m Carrier identification in steroid sulphatase deficiency and recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2448986&form=6&db=m Substrate specific sulfatase activity from hair follicles in recessive X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2573275&form=6&db=m An Xp22 microdeletion associated with ocular albinism and ichthyosis: approximation of breakpoints and estimation of deletion size by using cloned DNA probes and flow cytometry. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2644167&form=6&db=m Molecular heterogeneity of steroid sulfatase deficiency: a multicenter study on 57 unrelated patients, at DNA and protein levels. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2659403&form=6&db=m [The genetics and molecular genetics of X-chromosomal recessive ichthyosis] causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2722337&form=6&db=m Ichthyosis. Reliability of clinical signs in the differentiation between autosomal dominant and sex-linked forms. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2743959&form=6&db=m X-linked recessive ichthyosis. Enzymatic diagnosis of affected males and female carriers. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2813406&form=6&db=m Molecular studies of deletions at the human steroid sulfatase locus. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2865141&form=6&db=m Absence of testicular steroid sulphatase activity in a boy with recessive X-linked ichthyosis and testicular maldescent. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2866054&form=6&db=m Steroid sulphatase deficiency disease. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2884621&form=6&db=m Deletion of a DNA sequence in eight of nine families with X-linked ichthyosis (steroid sulphatase deficiency). causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2942400&form=6&db=m Steroid sulfatase. Biosynthesis and processing in normal and mutant fibroblasts. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2977418&form=6&db=m Metabolism of 3H-dehydroepiandrosterone sulphate by subjects with steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3007328&form=6&db=m X-linked ichthyosis, due to steroid sulphatase deficiency, associated with Kallmann syndrome (hypogonadotropic hypogonadism and anosmia): linkage relationships with Xg and cloned DNA sequences from the distal short arm of the X chromosome. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3034252&form=6&db=m Genetic heterogeneity of steroid sulfatase deficiency revealed with cDNA for human steroid sulfatase. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3091922&form=6&db=m Partial lyonisation of steroid sulphatase gene in single hair roots. ongoing research,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3162681&form=6&db=m Characterization of low-density lipoproteins from patients with recessive X-linked ichthyosis. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165358&form=6&db=m Recurrent bilateral corneal erosions due to an association of epidermolysis bullosa simplex Köbner and X-linked ichthyosis with steroid sulfatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165728&form=6&db=m X/Y translocation in a family with X-linked ichthyosis, chondrodysplasia punctata, and mental retardation: DNA analysis reveals deletion of the steroid sulphatase gene and translocation of its Y pseudogene. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3203072&form=6&db=m X-linked ichthyosis and ichthyosis vulgaris: comparison of their clinical features based on biochemical analysis. diagnostic usage,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3422798&form=6&db=m More on X-linked ichthyosis, steroid sulfatase deficiency, and hypogonadism and anosmia. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3457782&form=6&db=m [Biochemical diagnosis of X chromosomal ichthyosis] diagnostic usage,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3460334&form=6&db=m Fine mapping of the distal short arm of the human X chromosome using X/Y translocations. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3468016&form=6&db=m [Arylsulfatase C deficiency in leukocytes in patients and carrier of X-chromosome recessive ichthyosis] causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3470217&form=6&db=m [X-chromosomal recessive ichthyosis. Detection of heterozygote status in genetically possible carriers by determination of arylsulfatase C activity] diagnostic usage,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3474618&form=6&db=m Isolation and characterization of a steroid sulfatase cDNA clone: genomic deletions in patients with X-chromosome-linked ichthyosis. causal interaction,ongoing research,therapeutic application,unassigned 2,1,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477126&form=6&db=m X-linked ichthyosis due to steroid sulfatase deficiency associated with hypogonadism and anosmia. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3479355&form=6&db=m X-linked recessive ichthyosis. Reinvestigation of a family first described in 1928. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3480263&form=6&db=m Deletions of the steroid sulphatase gene in "classical" X-linked ichthyosis and in X-linked ichthyosis associated with Kallmann syndrome. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3480541&form=6&db=m Cloning of a cDNA for steroid sulfatase: frequent occurrence of gene deletions in patients with recessive X chromosome-linked ichthyosis. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3516063&form=6&db=m A new syndrome of anosmia, ichthyosis, hypogonadism, and various neurological manifestations with deficiency of steroid sulfatase and arylsulfatase C. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3531932&form=6&db=m Placental sulfatase deficiency: maternal and fetal expression of steroid sulfatase deficiency and X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3709904&form=6&db=m Ichthyosis of steroid sulphatase deficiency. Clinical study of 76 cases. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3857937&form=6&db=m Steroid sulphatase deficiency in patients initially diagnosed as ichthyosis vulgaris or recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3858674&form=6&db=m X-linkage of steroid sulphatase in the mouse is evidence for a functional Y-linked allele. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3860470&form=6&db=m Genetic complementation of steroid sulphatase after somatic cell hybridization of X-linked ichthyosis and multiple sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864397&form=6&db=m Familial X-linked ichthyosis, steroid sulfatase deficiency, mental retardation, and nullisomy for Xp223-pter. causal interaction,therapeutic application,unassigned 2,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864567&form=6&db=m Abnormal androgen and oestrogen metabolism in men with steroid sulphatase deficiency and recessive X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864728&form=6&db=m Recessive X-linked ichthyosis: lack of immunologically detectable steroid sulfatase enzyme protein. causal interaction,ongoing research,unassigned 2,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3868421&form=6&db=m The effect of steroid sulphatase on stratum corneum shedding in patients with X-linked ichthyosis. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3868422&form=6&db=m Detection of heterozygotes of X-linked ichthyosis by measuring steroid sulphatase activity of lymphocytes. Mode of inheritance in three families. diagnostic usage,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3914597&form=6&db=m [Ichthyosis X. Clinical review and rapid identification using lipid electrophoresis] causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6135610&form=6&db=m Clinical spectrum of steroid sulfatase deficiency: X-linked recessive ichthyosis, birth complications and cryptorchidism. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6140547&form=6&db=m Steroid sulphatase deficiency associated with testis cancer. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6238102&form=6&db=m Cholesterol sulfotransferase of newborn mouse epidermis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6290359&form=6&db=m [Progress in dermatology: new biochemical aspects] unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6304199&form=6&db=m Failure of steroid sulfatase to desulfate vitamin D3 sulfate. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6422158&form=6&db=m Hair root analysis in X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6434831&form=6&db=m Cholesterol sulphate in the microsomal sulphatase deficient placenta. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6448625&form=6&db=m Serum steroid sulphates in ichthyosis. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6461439&form=6&db=m Prenatal diagnosis of X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6573909&form=6&db=m A new method for the determination of steroid sulphatase activity in leukocytes in X-linked recessive ichthyosis. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6580240&form=6&db=m [The enzyme arylsulfatase C and steroid sulfatase as biochemical markers and pathogenetic factors in X-chromosome recessively inherited ichthyosis] diagnostic usage,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6582028&form=6&db=m Steroid sulphatase in man: a non inactivated X-locus with partial gene dosage compensation. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6584254&form=6&db=m Ichthyosis vulgaris with hypogenitalism and hypogonadism: evidence for different genotypes by lipoprotein electrophoresis and steroid sulfatase testing. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6592175&form=6&db=m Stratum corneum lipids in disorders of cornification. Steroid sulfatase and cholesterol sulfate in normal desquamation and the pathogenesis of recessive X-linked ichthyosis. causal interaction,ongoing research,unassigned 3,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6592263&form=6&db=m Lipids in the pathogenesis of ichthyosis: topical cholesterol sulfate-induced scaling in hairless mice. causal interaction,ongoing research,therapeutic application,unassigned 3,1,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6594286&form=6&db=m Steroid sulfatase = aryl sulfatase C? Chromatographic and electrophoretic properties in extracts from placental microsomes and skin fibroblasts. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6596909&form=6&db=m Placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6652948&form=6&db=m A familial X/Y translocation in a boy with ichthyosis, hypogonadism and mental retardation. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6820437&form=6&db=m Review: genetics of steroid sulphatase deficiency and X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6929654&form=6&db=m X-linked ichthyosis and X-linked placental sulfatase deficiency: a disease entity. Histochemical observations. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6930361&form=6&db=m Assignment by deletion mapping of the steroid sulfatase X-linked ichthyosis locus to Xp223. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6932268&form=6&db=m Autosomal dominant and X-linked ichthyosis in the same family. Biochemical analysis of steroid sulfatase activity. diagnostic usage,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6935987&form=6&db=m Arylsulfatase C activities in skin preparations of X-linked and autosomal dominant ichthyosis. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6939689&form=6&db=m Steroid sulfatase of human leukocytes and epidermis and the diagnosis of recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6940862&form=6&db=m Increased cholesterol sulfate in plasma and red blood cell membranes of steroid sulfatase deficient patients. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6944097&form=6&db=m Steroid sulphatase activity in the skin biopsies of various types of ichthyosis. diagnostic usage,ongoing research,unassigned 4,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6945674&form=6&db=m X-linked ichthyosis: increased blood cholesterol sulfate and electrophoretic mobility of low-density lipoprotein. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6947719&form=6&db=m Steroid sulfatase, X-linked ichthyosis, and stratum corneum cell cohesion. ongoing research,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6947980&form=6&db=m Stratum corneum lipids in disorders of cornification: increased cholesterol sulfate content of stratum corneum in recessive x-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6948769&form=6&db=m Further evidence for the assignment of the steroid sulfatase X-linked ichthyosis locus to the telomer of Xp. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6953038&form=6&db=m Rapid laboratory diagnostic of X-linked ichthyosis. causal interaction,diagnostic usage,therapeutic application,unassigned 1,3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6985463&form=6&db=m Regional assignment of the gene locus for steroid sulfatase. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7138038&form=6&db=m Measuring scale steroid sulfatase and beta-lipoprotein mobility in ichthyosis. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7208152&form=6&db=m Sulfatase activity of keratinizing tissues in X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7426407&form=6&db=m Sex-linked ichthyosis and placental sulphatase C deficiency. causal interaction,ongoing research,unassigned 4,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7546451&form=6&db=m The biochemical identification of carrier state in mothers of sporadic cases of X-linked recessive ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7577600&form=6&db=m Palmoplantar keratoderma with an unusual composition of stratum corneum and serum sterol derivatives: a new entity? causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704955&form=6&db=m Hypogonadotrophic hypogonadism with hyposmia, X-linked ichthyosis, and renal malformation syndrome. diagnostic usage,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7726134&form=6&db=m Low maternal serum unconjugated estriol during prenatal screening as an indication of placental steroid sulfatase deficiency and X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7857850&form=6&db=m Oral fluconazole treatment of fungating candidiasis in the keratitis, ichthyosis and deafness (KID) syndrome. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7943126&form=6&db=m Clinical pathologic correlation of superficial corneal opacities in X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7951667&form=6&db=m Genetic skin diseases. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7999591&form=6&db=m A clinical and genetic study of X-linked recessive ichthyosis and contiguous gene defects. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8001874&form=6&db=m Steroid sulfatase activity in human leukocytes. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8052574&form=6&db=m Prenatal detection of X-linked ichthyosis by maternal serum screening for Down syndrome. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8186420&form=6&db=m Steroid sulfatase deficiency in Japanese patients: characterization of X-linked ichthyosis by using polymerase chain reaction. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8262159&form=6&db=m Correction of steroid sulfatase deficiency by gene transfer into basal cells of tissue-cultured epidermis from patients with recessive X-linked ichthyosis. causal interaction,ongoing research,unassigned 1,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8330450&form=6&db=m Deletion analysis maps ocular albinism proximal to the steroid sulphatase locus. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8362907&form=6&db=m Prenatal in situ hybridization test for deleted steroid sulfatase gene. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8473391&form=6&db=m Xp22.3 deletions in isolated familial Kallmann's syndrome. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8588575&form=6&db=m X-linked ichthyosis without STS deficiency: clinical, genetical, and molecular studies. causal interaction,ongoing research,unassigned 2,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8615047&form=6&db=m A study of the steroid sulfatase gene in families with X-linked ichthyosis using polymerase chain reaction. ongoing research,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8791985&form=6&db=m Steroid sulphatase deficiency: identification of heterozygotes using hydrolysis of dehydroepiandrosterone sulphate by peripheral leucocytes. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8832947&form=6&db=m X-linked dominant chondrodysplasia punctata with decreased dihydroxyacetone phosphate acyltransferase activity. causal interaction,ongoing research,therapeutic application,unassigned 4,1,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9101220&form=6&db=m Prenatal diagnosis of X-linked ichthyosis using molecular cytogenetics. diagnostic usage,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9175741&form=6&db=m A model of corrective gene transfer in X-linked ichthyosis. ongoing research,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9242515&form=6&db=m A novel point mutation in the steroid sulfatase gene in X-linked ichthyosis. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9247725&form=6&db=m Diagnosis of a deletion of steroid sulfatase by polymerase chain reaction and high-performance liquid chromatography. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9252398&form=6&db=m Characterization of point mutations in patients with X-linked ichthyosis. Effects on the structure and function of the steroid sulfatase protein. causal interaction,ongoing research,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9336808&form=6&db=m Ichthyosis: the skin manifestation of multiple sulfatase deficiency. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9375723&form=6&db=m X-linked ichthyosis in Mexico: high frequency of deletions in the steroid sulfatase encoding gene. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9499614&form=6&db=m X-linked ichthyosis with hypogonadism: not always Kallmann's syndrome. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9640374&form=6&db=m X-linked ichthyosis: relation between cholesterol sulphate, dehydroepiandrosterone sulphate and patient's age. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9764155&form=6&db=m Deletion pattern of the steroid sulphatase gene in Japanese patients with X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10228635&form=6&db=m Most "sporadic" cases of X-linked ichthyosis are not de novo mutations. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10393443&form=6&db=m Absence of male-pattern baldness in men with X-linked recessive ichthyosis? A hypothesis to be challenged. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10533035&form=6&db=m Submicroscopic Xpter deletion in a boy with growth and mental retardation caused by a familial t(X;14). unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10607842&form=6&db=m A human sex-chromosomal gene family expressed in male germ cells and encoding variably charged proteins. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10679952&form=6&db=m PCR diagnosis of X-linked ichthyosis: identification of a novel mutation (E560P) of the steroid sulfatase gene. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10692123&form=6&db=m Mutation report: a novel partial deletion of exons 2-10 of the STS gene in recessive X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10759956&form=6&db=m Deletion patterns of the STS gene and flanking sequences in Israeli X-linked ichthyosis patients and carriers: analysis by polymerase chain reaction and fluorescence in situ hybridization techniques. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10828635&form=6&db=m Male-pattern baldness in men with X-linked recessive ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10844566&form=6&db=m Novel point mutations in the steroid sulfatase gene in patients with X-linked ichthyosis: transfection analysis using the mutated genes. causal interaction,ongoing research,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10905086&form=6&db=m [Occurrence of X-linked ichthyosis along with atopy] causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11079322&form=6&db=m [Hope for new therapies of severe genetic skin diseases] causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11231321&form=6&db=m Deletion of exons 1-5 of the STS gene causing X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11477606&form=6&db=m Carrier identification by FISH analysis in isolated cases of X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11844872&form=6&db=m Deletion pattern of the STS gene in X-linked ichthyosis in a Mexican population. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11910205&form=6&db=m Molecular analysis of X-linked ichthyosis in Japan. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12174111&form=6&db=m Mild ichthyosis in a 4-year-old boy with multiple sulphatase deficiency. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12406347&form=6&db=m Somatic and germinal mosaicism for the steroid sulfatase gene deletion in a steroid sulfatase deficiency carrier. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,1 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12553849&form=6&db=m Ichthyosis: etiology, diagnosis, and management. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12850625&form=6&db=m Maternal serum unconjugated estriol as a predictor for Smith-Lemli-Opitz syndrome and other fetal conditions. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14567193&form=6&db=m [Functional consequences of cutaneous lipid perturbation] causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14571075&form=6&db=m Male-pattern baldness is common in men with X-linked recessive ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15009711&form=6&db=m Basis for abnormal desquamation and permeability barrier dysfunction in RXLI. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15607112&form=6&db=m Mutations in X-linked ichthyosis disrupt the active site structure of estrone/DHEA sulfatase. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16109567&form=6&db=m [Investigation of steroid sulfatase gene in two pedigrees with X-linked ichthyosis] unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16390921&form=6&db=m Low estriol levels in the maternal triple-marker screen as a predictor of isolated adrenocorticotropic hormone deficiency caused by a new mutation in the TPIT gene. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16720460&form=6&db=m X-chromosome inactivation: role in skin disease expression. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17113756&form=6&db=m Deletion of distal promoter of VCXA in a patient with X-linked ichthyosis associated with borderline mental retardation. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17603054&form=6&db=m Renal dysgenesis and KAL1 gene defects in patients with sporadic Kallmann syndrome. ongoing research,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18076704&form=6&db=m Analysis of the VCX3A, VCX2 and VCX3B genes shows that VCX3A gene deletion is not sufficient to result in mental retardation in X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18341575&form=6&db=m An update on molecular aspects of the non-syndromic ichthyoses. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18413370&form=6&db=m X-linked ichthyosis (steroid sulfatase deficiency) is associated with increased risk of attention deficit hyperactivity disorder, autism and social communication deficits. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19200188&form=6&db=m Atypical X-linked ichthyosis in a patient with a large deletion involving the steroid sulfatase (STS) gene. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20523225&form=6&db=m Burn injury and wound healing in X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20941942&form=6&db=m The detection of steroid sulfatase gene deletion (STS) in Egyptian males with X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21692775&form=6&db=m Novel filaggrin mutation but no other loss-of-function variants found in ethiopian atopic-dermatitis patients. causal interaction,diagnostic usage,unassigned 2,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21928690&form=6&db=m Updated molecular genetics and pathogenesis of ichthiyoses. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21931015&form=6&db=m Banding pattern on polarized hair microscopic examination and unilateral polymicrogyria in a patient with steroid sulfatase deficiency. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21945601&form=6&db=m Exacerbation of X-linked ichthyosis phenotype in a female by inheritance of filaggrin and steroid sulfatase mutations. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22314765&form=6&db=m [X-chromosome-linked ichthyosis associated to epilepsy, hyperactivity, autism and mental retardation, due to the Xp22.31 microdeletion]. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22419362&form=6&db=m Steroid-resistant nephrotic syndrome associated with steroid sulfatase deficiency-x-linked recessive ichthyosis: a case report and review of literature. causal interaction,therapeutic application,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22486194&form=6&db=m X-linked recessive ichthyosis: an impaired barrier function evokes limited gene responses before and after moisturizing treatments. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22629078&form=6&db=m X-linked ichthyosis along with epidermolysis bullosa. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23340970&form=6&db=m [X-linked recessive ichthyosis (XRI), cerebellar ataxia and neuropsychiatric symptoms]. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23574351&form=6&db=m A family with fragile X syndrome, Duchenne muscular dystrophy and ichthyosis transmitted by an asymptomatic carrier. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23791652&form=6&db=m Xp22.3 interstitial deletion: A recognizable chromosomal abnormality encompassing VCX3A and STS genes in a patient with X-linked ichthyosis and mental retardation. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23807007&form=6&db=m Pre-Descemet corneal dystrophy and X-linked ichthyosis associated with deletion of Xp22.31 containing the STS gene. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24291327&form=6&db=m Role of cholesterol sulfate in epidermal structure and function: lessons from X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25502769&form=6&db=m High levels of oxysterol sulfates in serum of patients with steroid sulfatase deficiency. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25659225&form=6&db=m The phenotype spectrum of X-linked ichthyosis identified by chromosomal microarray. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26676689&form=6&db=m X?linked ichthyosis and Crigler?Najjar syndrome ?: Coexistence in a male patient with two copy number variable regions of 2q37.1 and Xp22.3. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27192889&form=6&db=m CO-OCCURRENCE OF PRIMARY MICROCEPHALY CAUSED BY A NOVEL HOMOZYGOUS ASPM MUTATION ALONG WITH X-LINKED ICHTHYOSIS IN THE SAME PATIENT. unassigned - 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27711218&form=6&db=m Behavioural and Psychiatric Phenotypes in Men and Boys with X-Linked Ichthyosis: Evidence from a Worldwide Online Survey. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27817780&form=6&db=m [Identification of gene mutation and prenatal diagnosis in a family with X-linked ichthyosis]. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27919139&form=6&db=m [A Case of Steroid Sulfatase Deficiency Complicated by Bilateral Undescended Testis]. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28302098&form=6&db=m In vivo confocal microscopy of pre-Descemet corneal dystrophy associated with X-linked ichthyosis: a case report. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28710038&form=6&db=m Steroid sulfatase and filaggrin mutations in a boy with severe ichthyosis, elevated serum IgE level and moyamoya syndrome. causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28934990&form=6&db=m X-linked ichthyosis associated with psychosis and behavioral abnormalities: a case report. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29672931&form=6&db=m X-linked ichthyosis: Clinical and molecular findings in 35 Italian patients. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29901853&form=6&db=m Evidence of the high prevalence of neurological disorders in nonsyndromic X-linked recessive ichthyosis: a retrospective case series. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30021537&form=6&db=m Exacerbation of ichthyosis vulgaris phenotype by co-inheritance of STS and FLG mutations in a Chinese family with ichthyosis: a case report. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30352392&form=6&db=m Kallmann syndrome and ichthyosis: a case of contiguous gene deletion syndrome. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30531180&form=6&db=m Placental steroid sulphatase deficiency: an approach to antenatal care and delivery. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30768640&form=6&db=m Behavioural and psychiatric phenotypes in female carriers of genetic mutations associated with X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32139392&form=6&db=m Medical and neurobehavioural phenotypes in carriers of X-linked ichthyosis-associated genetic deletions in the UK Biobank. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33026262&form=6&db=m Clinical features and genetic analysis of two Chinese families with X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3203072&form=6&db=m X-linked ichthyosis and ichthyosis vulgaris: comparison of their clinical features based on biochemical analysis. diagnostic usage,unassigned 4,0 3.1.6.2 Ichthyosis Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3857937&form=6&db=m Steroid sulphatase deficiency in patients initially diagnosed as ichthyosis vulgaris or recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6584254&form=6&db=m Ichthyosis vulgaris with hypogenitalism and hypogonadism: evidence for different genotypes by lipoprotein electrophoresis and steroid sulfatase testing. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis Vulgaris http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6951797&form=6&db=m Elevated levels of arylsulfatase C activity in cultured skin fibroblasts of patients with autosomal dominant ichthyosis vulgaris. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=136200&form=6&db=m Prenatal diagnosis of placental steroid sulfatase deficiency. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=294955&form=6&db=m Steroid sulfatase deficiency and X-linked ichthyosis. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=887310&form=6&db=m Steroid sulfatase deficiency. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1427599&form=6&db=m [Biochemical study on steroid sulfatase and its clinical application to the obstetrics and gynecology] causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1496984&form=6&db=m Role of the pseudoautosomal region in sex-chromosome pairing during male meiosis: meiotic studies in a man with a deletion of distal Xp. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1549475&form=6&db=m Characterization of a low copy repetitive element S232 involved in the generation of frequent deletions of the distal short arm of the human X chromosome. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1639422&form=6&db=m Sequence analysis of a partial deletion of the human steroid sulfatase gene reveals 3 bp of homology at deletion breakpoints. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1690506&form=6&db=m The human arylsulfatase-C isoenzymes: two distinct genes that escape from X inactivation. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1785631&form=6&db=m Deletion of the distal short arm of the X chromosome (Xp) in a patient with short stature, chondrodysplasia punctata, and X-linked ichthyosis due to steroid sulfatase deficiency. causal interaction,ongoing research,unassigned 2,2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1923309&form=6&db=m Ocular findings in X-linked ichthyosis: a survey on 38 cases. causal interaction,diagnostic usage,ongoing research,unassigned 2,1,2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1951426&form=6&db=m Two cases of steroid sulfatase deficiency with complex phenotype due to contiguous gene deletions. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2253421&form=6&db=m [Two cases of X-linked ichthyosis associated with myopathies] causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2344613&form=6&db=m Frequent deletions of the human X chromosome distal short arm result from recombination between low copy repetitive elements. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2602357&form=6&db=m Contiguous gene syndromes due to deletions in the distal short arm of the human X chromosome. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2644167&form=6&db=m Molecular heterogeneity of steroid sulfatase deficiency: a multicenter study on 57 unrelated patients, at DNA and protein levels. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2743959&form=6&db=m X-linked recessive ichthyosis. Enzymatic diagnosis of affected males and female carriers. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2859745&form=6&db=m Steroid sulfatase deficiency and the genetics of the short arm of the human X chromosome. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3034252&form=6&db=m Genetic heterogeneity of steroid sulfatase deficiency revealed with cDNA for human steroid sulfatase. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3162681&form=6&db=m Characterization of low-density lipoproteins from patients with recessive X-linked ichthyosis. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165358&form=6&db=m Recurrent bilateral corneal erosions due to an association of epidermolysis bullosa simplex Köbner and X-linked ichthyosis with steroid sulfatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3422798&form=6&db=m More on X-linked ichthyosis, steroid sulfatase deficiency, and hypogonadism and anosmia. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3462435&form=6&db=m Regulation of serum testosterone in men with steroid sulfatase deficiency: response to human chorionic gonadotropin. causal interaction,diagnostic usage,ongoing research,unassigned 2,2,4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3464202&form=6&db=m Dosage of the Sts gene in the mouse. ongoing research,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3469950&form=6&db=m Steroid sulfatase deficiency: enzymatic studies using placenta and leucocytes in one family. diagnostic usage,ongoing research,therapeutic application,unassigned 3,2,2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477126&form=6&db=m X-linked ichthyosis due to steroid sulfatase deficiency associated with hypogonadism and anosmia. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477995&form=6&db=m [Steroid sulfatase and placental deficiency. Current data as instigators of new research] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3478297&form=6&db=m Multipoint linkage analysis of steroid sulfatase (X-linked ichthyosis) and distal Xp markers. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3531932&form=6&db=m Placental sulfatase deficiency: maternal and fetal expression of steroid sulfatase deficiency and X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864079&form=6&db=m Placental steroid sulfatase deficiency. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864397&form=6&db=m Familial X-linked ichthyosis, steroid sulfatase deficiency, mental retardation, and nullisomy for Xp223-pter. causal interaction,therapeutic application,unassigned 2,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3865454&form=6&db=m [Placental steroid sulfatase deficiency. Reduced estriol production] unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6123259&form=6&db=m Placental steroid deficiency: association with arylsulfatase A deficiency. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6135610&form=6&db=m Clinical spectrum of steroid sulfatase deficiency: X-linked recessive ichthyosis, birth complications and cryptorchidism. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6234482&form=6&db=m Placental steroid sulfatase deficiency: biochemical diagnosis and clinical review. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6461439&form=6&db=m Prenatal diagnosis of X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6929654&form=6&db=m X-linked ichthyosis and X-linked placental sulfatase deficiency: a disease entity. Histochemical observations. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6940862&form=6&db=m Increased cholesterol sulfate in plasma and red blood cell membranes of steroid sulfatase deficient patients. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6985463&form=6&db=m Regional assignment of the gene locus for steroid sulfatase. causal interaction,unassigned 1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7726134&form=6&db=m Low maternal serum unconjugated estriol during prenatal screening as an indication of placental steroid sulfatase deficiency and X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7794518&form=6&db=m Very low maternal serum unconjugated estriol and prenatal diagnosis of steroid sulfatase deficiency. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7943126&form=6&db=m Clinical pathologic correlation of superficial corneal opacities in X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7951263&form=6&db=m Characterization of the deletion breakpoints in a patient with steroid sulfatase deficiency. ongoing research,unassigned 2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8001874&form=6&db=m Steroid sulfatase activity in human leukocytes. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8059846&form=6&db=m Maternal steroid sulfatase deficiency--cause of high-risk pregnancy? unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8186420&form=6&db=m Steroid sulfatase deficiency in Japanese patients: characterization of X-linked ichthyosis by using polymerase chain reaction. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8262159&form=6&db=m Correction of steroid sulfatase deficiency by gene transfer into basal cells of tissue-cultured epidermis from patients with recessive X-linked ichthyosis. causal interaction,ongoing research,unassigned 1,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8403433&form=6&db=m Enzymatic diagnosis of steroid sulfatase deficiency by high performance liquid chromatography. diagnostic usage,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9101220&form=6&db=m Prenatal diagnosis of X-linked ichthyosis using molecular cytogenetics. diagnostic usage,unassigned 1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9336808&form=6&db=m Ichthyosis: the skin manifestation of multiple sulfatase deficiency. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9583203&form=6&db=m Frequency of steroid sulfatase deficiency in Hiroshima. diagnostic usage,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9764665&form=6&db=m Unconjugated estriol as an indication for prenatal diagnosis of steroid sulfatase deficiency by in situ hybridization. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10092953&form=6&db=m Steroid sulfatase activity in leukocytes: a comparative study in 45,X; 46,Xi(Xq) and carriers of steroid sulfatase deficiency. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10692123&form=6&db=m Mutation report: a novel partial deletion of exons 2-10 of the STS gene in recessive X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10844566&form=6&db=m Novel point mutations in the steroid sulfatase gene in patients with X-linked ichthyosis: transfection analysis using the mutated genes. causal interaction,ongoing research,unassigned 4,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11231321&form=6&db=m Deletion of exons 1-5 of the STS gene causing X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11676848&form=6&db=m Maternal transmission of the 3 bp deletion within exon 7 of the STS gene in steroid sulfatase deficiency. causal interaction,therapeutic application,unassigned 1,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11844872&form=6&db=m Deletion pattern of the STS gene in X-linked ichthyosis in a Mexican population. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12135803&form=6&db=m Advances in prenatal screening for Down syndrome: I. general principles and second trimester testing. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12364433&form=6&db=m Prenatal diagnosis of sex differentiation disorders: the role of fetal ultrasound. therapeutic application,unassigned 1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12406347&form=6&db=m Somatic and germinal mosaicism for the steroid sulfatase gene deletion in a steroid sulfatase deficiency carrier. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,1 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12424769&form=6&db=m Low or absent unconjugated estriol in pregnancy: an indicator for steroid sulfatase deficiency detectable by fluorescence in situ hybridization and biochemical analysis. diagnostic usage,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12850625&form=6&db=m Maternal serum unconjugated estriol as a predictor for Smith-Lemli-Opitz syndrome and other fetal conditions. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12970278&form=6&db=m Congenital hypopituitarism as a cause of undetectable estriol levels in the maternal triple-marker screen. causal interaction,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14646417&form=6&db=m Association of undetectable unconjugated estriol on multiple marker screening with steroid sulfatase deficiency. causal interaction,diagnostic usage,unassigned 1,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16504797&form=6&db=m Steroid Sulfatase Deficiency With Bilateral Periventricular Nodular Heterotopia. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19418464&form=6&db=m Maternal urine and serum steroid measurements to identify steroid sulfatase deficiency (STSD) in second trimester pregnancies. diagnostic usage,unassigned 2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19609942&form=6&db=m Steroid sulfatase deficiency and contiguous gene deletion syndrome amongst pregnant patients with low serum unconjugated estriols. diagnostic usage,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20715120&form=6&db=m Prevalence of steroid sulfatase deficiency in California according to race and ethnicity. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21723668&form=6&db=m Steroid sulfatase-deficient mice exhibit endophenotypes relevant to Attention Deficit Hyperactivity Disorder. ongoing research,unassigned 1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21931015&form=6&db=m Banding pattern on polarized hair microscopic examination and unilateral polymicrogyria in a patient with steroid sulfatase deficiency. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22419362&form=6&db=m Steroid-resistant nephrotic syndrome associated with steroid sulfatase deficiency-x-linked recessive ichthyosis: a case report and review of literature. causal interaction,therapeutic application,unassigned 4,3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22475435&form=6&db=m Does steroid sulfatase deficiency influence postpartum psychosis risk? unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24027193&form=6&db=m Are low maternal estriol levels a predictor for pro-opiomelanocortin (POMC) deficiency caused by POMC mutation during pregnancy? diagnostic usage,ongoing research,unassigned 3,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24119255&form=6&db=m Nonsyndromic types of ichthyoses - an update. causal interaction,unassigned 3,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25502769&form=6&db=m High levels of oxysterol sulfates in serum of patients with steroid sulfatase deficiency. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26239050&form=6&db=m Simultaneous quantification of cholesterol sulfate, androgen sulfates, and progestagen sulfates in human serum by LC-MS/MS. diagnostic usage,unassigned 4,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27264819&form=6&db=m [Genetic analysis of a rare case with Kallman syndrome and steroid sulfatase deficiency]. unassigned - 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27531568&form=6&db=m Role of steroid sulfatase in steroid homeostasis and characterization of the sulfated steroid pathway: Evidence from steroid sulfatase deficiency. causal interaction,unassigned 2,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27817780&form=6&db=m [Identification of gene mutation and prenatal diagnosis in a family with X-linked ichthyosis]. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Ichthyosis, X-Linked http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27919139&form=6&db=m [A Case of Steroid Sulfatase Deficiency Complicated by Bilateral Undescended Testis]. causal interaction,unassigned 3,0 3.1.6.2 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9402143&form=6&db=m Cytokine dysregulation and increased oxidation is prevented by dehydroepiandrosterone in mice infected with murine leukemia retrovirus. causal interaction,unassigned 1,0 3.1.6.2 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12716389&form=6&db=m The relationships between dehydroepiandrosterone sulphate (DHEAS), the intensity of Schistosoma mansoni infection and parasite-specific antibody responses. A cross-sectional study in residents of endemic communities in north-east Ethiopia. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,2,0 3.1.6.2 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15315002&form=6&db=m Dehydroepiandrosterone sulphate and androstenedione concentration in the serum of male patients with chronic hepatitis C. diagnostic usage,ongoing research,unassigned 2,4,0 3.1.6.2 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16300484&form=6&db=m Raised cortisol:DHEAS ratios in the elderly after injury: potential impact upon neutrophil function and immunity. causal interaction,unassigned 2,0 3.1.6.2 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17237323&form=6&db=m Higher serum concentrations of DHEAS predict improved nutritional status in helminth-infected children, adolescents, and young adults in Leyte, the Philippines. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,2,0 3.1.6.2 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19576195&form=6&db=m HIV-1 clade C infection and progressive disruption in the relationship between cortisol, DHEAS and CD4 cell numbers: a two-year follow-up study. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,4,4 3.1.6.2 Infertility http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1672669&form=6&db=m Peritoneal fluid from patients with unexplained infertility inhibits growth of 2-cell mouse embryos. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Infertility http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2940031&form=6&db=m The effect of dehydroepiandrosterone sulfate prolactin and testosterone hormones in female fertility and hirsutism. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,1,0 3.1.6.2 Infertility http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6261197&form=6&db=m Serum levels of DHEAS in gynecologic endocrinopathy and infertility. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Infertility http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11561833&form=6&db=m Plasma homocysteine, fasting insulin, and androgen patterns among women with polycystic ovaries and infertility. diagnostic usage,ongoing research,therapeutic application,unassigned 2,2,1,0 3.1.6.2 Infertility http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16543255&form=6&db=m Clomiphene citrate and dexamethazone in treatment of clomiphene citrate-resistant polycystic ovary syndrome: a prospective placebo-controlled study. causal interaction,unassigned 3,0 3.1.6.2 Infertility http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27737330&form=6&db=m Age related endocrine patterns observed in polycystic ovary syndrome patients vs. ovulatory controls: descriptive data from a university based infertility center. diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Infertility, Female http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2940031&form=6&db=m The effect of dehydroepiandrosterone sulfate prolactin and testosterone hormones in female fertility and hirsutism. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,1,0 3.1.6.2 Infertility, Male http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28292621&form=6&db=m Serum reproductive hormone levels and ultrasound findings in female offspring after intracytoplasmic sperm injection: first results. causal interaction,diagnostic usage,unassigned 1,2,0 3.1.6.2 Inflammatory Bowel Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9820396&form=6&db=m Association of humoral markers of inflammation and dehydroepiandrosterone sulfate or cortisol serum levels in patients with chronic inflammatory bowel disease. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,3,0 3.1.6.2 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8080168&form=6&db=m The development of effective vaccine adjuvants employing natural regulators of T-cell lymphokine production in vivo. therapeutic application,unassigned 4,0 3.1.6.2 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597462&form=6&db=m DHEAS as an effective vaccine adjuvant in elderly humans. Proof-of-principle studies. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9014295&form=6&db=m The use of oral dehydroepiandrosterone sulfate as an adjuvant in tetanus and influenza vaccination of the elderly. ongoing research,therapeutic application,unassigned 1,3,0 3.1.6.2 Influenza, Human http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9180672&form=6&db=m The effect of DHEAS on influenza vaccination in aging adults. causal interaction,ongoing research,therapeutic application,unassigned 3,2,3,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1459258&form=6&db=m Effect of hyperprolactinemia on the androgen response to an oral glucose load. causal interaction,unassigned 3,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7561622&form=6&db=m Acute hyperinsulinemia, androgen homeostasis and insulin sensitivity in healthy man. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7927194&form=6&db=m Effects of exercise training on plasma androgens in men. diagnostic usage,unassigned 2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8548945&form=6&db=m Androgens and insulin resistance in type 1 diabetic men. unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8612854&form=6&db=m Can metformin reduce insulin resistance in polycystic ovary syndrome? unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10580744&form=6&db=m A close association between insulin resistance and dehydroepiandrosterone sulfate in subjects with essential hypertension. diagnostic usage,ongoing research,unassigned 4,3,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11889181&form=6&db=m Relationship between the GH/IGF-I axis, insulin sensitivity, and adrenal androgens in normal prepubertal and pubertal boys. diagnostic usage,unassigned 2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12749433&form=6&db=m Troglitazone decreases adrenal androgen levels in women with polycystic ovary syndrome. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,1 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12828082&form=6&db=m The association of dehydroepiandrosterone, obesity, waist-hip ratio and insulin resistance with fatty liver in postmenopausal women--a hyperinsulinemic euglycemic insulin clamp study. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,3,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15053331&form=6&db=m Obesity is the major factor determining an insulin sensitivity and androgen production in women with anovulary cycles. diagnostic usage,ongoing research,therapeutic application,unassigned 2,4,1,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15717807&form=6&db=m [A relationship between dehydroepiandrosterone sulphate and insulin resistance in obese men and women] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16579998&form=6&db=m Serum soluble leptin receptor levels and free leptin index in women with polycystic ovary syndrome: relationship to insulin resistance and androgens. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,3,1 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16580388&form=6&db=m Hyperinsulinemia and circulating dehydroepiandrosterone sulfate in white and Mexican American women with polycystic ovary syndrome. diagnostic usage,unassigned 2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18439591&form=6&db=m Dehydroepiandrosterone sulfate and insulin resistance in patients with polycystic ovary syndrome. diagnostic usage,ongoing research,therapeutic application,unassigned 4,2,2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18475046&form=6&db=m Relation between adiponectin and bone mineral density in elderly post-menopausal women: role of body composition, leptin, insulin resistance, and dehydroepiandrosterone sulfate. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18554599&form=6&db=m Polycystic ovary syndrome is associated with elevated plasma soluble CD40 ligand, a marker of coronary artery disease. unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19111298&form=6&db=m Effect of metformin on serum visfatin levels in patients with polycystic ovary syndrome. diagnostic usage,ongoing research,unassigned 4,4,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19214546&form=6&db=m Serum 25-hydroxyvitamin D concentrations in obese and non-obese women with polycystic ovary syndrome. unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19342030&form=6&db=m In polycystic ovary syndrome, adrenal steroids are regulated differently in the morning versus in response to nutrient intake. causal interaction,unassigned 3,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19368913&form=6&db=m Increased plasma thrombin-activatable fibrinolysis inhibitor levels in young obese women with polycystic ovary syndrome. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,4,1 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19552904&form=6&db=m Short-term metformin treatment for clomiphene citrate-resistant women with polycystic ovary syndrome. unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20096825&form=6&db=m Recommendations for investigation of hyperandrogenism. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22693174&form=6&db=m Myostatin and its association with abdominal obesity, androgen and follistatin levels in women with polycystic ovary syndrome. diagnostic usage,unassigned 4,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22835450&form=6&db=m Opposing effects of dehydroepiandrosterone sulfate and free testosterone on metabolic phenotype in women with polycystic ovary syndrome. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23704104&form=6&db=m Low dehydroepiandrosterone sulfate is associated with increased risk of ischemic stroke among women. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,1,2 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24092831&form=6&db=m The severity of menstrual dysfunction as a predictor of insulin resistance in PCOS. diagnostic usage,ongoing research,unassigned 2,2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25381943&form=6&db=m Is there a relationship between cardiovascular risk factors and dehydroepiandrosterone sulfate levels in childhood obesity? causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25555552&form=6&db=m A Twin-Sibling Study on Early Growth and Hormone Levels in Adolescents. unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27511825&form=6&db=m The link between metabolic features and TSH levels in polycystic ovary syndrome is modulated by the body weight: an euglycemic-hyperinsulinemic clamp study. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28160242&form=6&db=m Serum Zinc and Adiponectin Levels in Patients with Polycystic Ovary Syndrome, Adjusted for Anthropometric, Biochemical, Dietary Intake, and Physical Activity Measures. diagnostic usage,unassigned 4,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28595797&form=6&db=m Chromium supplementation and polycystic ovary syndrome: A systematic review and meta-analysis. ongoing research,unassigned 2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28751368&form=6&db=m Functional correlates of detailed body composition in healthy elderly subjects. diagnostic usage,unassigned 2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29068242&form=6&db=m Heterogenous origins of hyperandrogenism in the polycystic ovary syndrome in relation to body mass index and insulin resistance. causal interaction,diagnostic usage,unassigned 1,2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30056577&form=6&db=m Body mass index at the presentation of premature adrenarche is associated with components of metabolic syndrome at puberty. ongoing research,unassigned 1,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30352391&form=6&db=m Higher steroid sulfation is linked to successful weight loss in obese children. ongoing research,unassigned 1,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30727783&form=6&db=m Familial clustering of metabolic phenotype in brothers of women with polycystic ovary syndrome. causal interaction,unassigned 2,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30958034&form=6&db=m Association of Luteinizing hormone and LH receptor gene polymorphism with susceptibility of Polycystic ovary syndrome. unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31070948&form=6&db=m MIFEPRISTONE TREATMENT FOR MILD AUTONOMOUS CORTISOL SECRETION DUE TO ADRENAL ADENOMAS: A PILOT STUDY. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,3,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32148484&form=6&db=m Increased DHEAS and Decreased Total Testosterone Serum Levels in a Subset of Men with Early-Onset Androgenetic Alopecia: Does a Male PCOS-Equivalent Exist? diagnostic usage,therapeutic application,unassigned 2,1,0 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33530089&form=6&db=m The Unique Role of 11-Oxygenated C19 Steroids in Both Premature Adrenarche and Premature Pubarche. unassigned - 3.1.6.2 Insulin Resistance http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33775010&form=6&db=m Dehydroepiandrosterone Sulfate (DHEAS) Levels in Polycystic Ovarian Syndrome (PCOS). diagnostic usage,ongoing research,therapeutic application,unassigned 2,1,2,0 3.1.6.2 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1496984&form=6&db=m Role of the pseudoautosomal region in sex-chromosome pairing during male meiosis: meiotic studies in a man with a deletion of distal Xp. unassigned - 3.1.6.2 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2602357&form=6&db=m Contiguous gene syndromes due to deletions in the distal short arm of the human X chromosome. causal interaction,unassigned 3,0 3.1.6.2 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165728&form=6&db=m X/Y translocation in a family with X-linked ichthyosis, chondrodysplasia punctata, and mental retardation: DNA analysis reveals deletion of the steroid sulphatase gene and translocation of its Y pseudogene. unassigned - 3.1.6.2 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864397&form=6&db=m Familial X-linked ichthyosis, steroid sulfatase deficiency, mental retardation, and nullisomy for Xp223-pter. causal interaction,therapeutic application,unassigned 2,1,0 3.1.6.2 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9727739&form=6&db=m Analysis of an interstitial deletion in a patient with Kallmann syndrome, X-linked ichthyosis and mental retardation. unassigned - 3.1.6.2 Intellectual Disability http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23565415&form=6&db=m Unusual presentation of Kallmannn syndrome with contiguous gene deletion in three siblings of a family. causal interaction,unassigned 2,0 3.1.6.2 Ischemic Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23704104&form=6&db=m Low dehydroepiandrosterone sulfate is associated with increased risk of ischemic stroke among women. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,1,2 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1557308&form=6&db=m Prenatal diagnosis and investigation of a fetus with chondrodysplasia punctata, ichthyosis, and Kallmann syndrome due to an Xp deletion. causal interaction,unassigned 4,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2328992&form=6&db=m Long-range physical mapping around the human steroid sulfatase locus. diagnostic usage,unassigned 2,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2339111&form=6&db=m Long-range restriction map of the terminal part of the short arm of the human X chromosome. causal interaction,unassigned 2,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2602357&form=6&db=m Contiguous gene syndromes due to deletions in the distal short arm of the human X chromosome. causal interaction,unassigned 3,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3007328&form=6&db=m X-linked ichthyosis, due to steroid sulphatase deficiency, associated with Kallmann syndrome (hypogonadotropic hypogonadism and anosmia): linkage relationships with Xg and cloned DNA sequences from the distal short arm of the X chromosome. causal interaction,unassigned 3,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3480263&form=6&db=m Deletions of the steroid sulphatase gene in "classical" X-linked ichthyosis and in X-linked ichthyosis associated with Kallmann syndrome. causal interaction,unassigned 3,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704955&form=6&db=m Hypogonadotrophic hypogonadism with hyposmia, X-linked ichthyosis, and renal malformation syndrome. diagnostic usage,unassigned 1,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7806223&form=6&db=m Isolation of a new gene GS2 (DXS1283E) from a CpG island between STS and KAL1 on Xp22.3. ongoing research,unassigned 2,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8276392&form=6&db=m A yeast artificial chromosome contig linking the steroid sulfatase and Kallmann syndrome loci on the human X chromosome short arm. ongoing research,unassigned 1,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8330450&form=6&db=m Deletion analysis maps ocular albinism proximal to the steroid sulphatase locus. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9713559&form=6&db=m Contiguous gene syndrome due to deletion of the first three exons of the Kallmann gene and complete deletion of the steroid sulphatase gene. unassigned - 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9764665&form=6&db=m Unconjugated estriol as an indication for prenatal diagnosis of steroid sulfatase deficiency by in situ hybridization. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Kallmann Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30352392&form=6&db=m Kallmann syndrome and ichthyosis: a case of contiguous gene deletion syndrome. causal interaction,unassigned 4,0 3.1.6.2 Leiomyoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2212823&form=6&db=m [Study on the local estrogen biosynthesis in human uterine leiomyoma] causal interaction,diagnostic usage,ongoing research,therapeutic application 2,1,4,2 3.1.6.2 Leiomyoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2351313&form=6&db=m Estrone sulfatase activity in human uterine leiomyoma. diagnostic usage,ongoing research,unassigned 1,3,0 3.1.6.2 Leukemia, Myeloid, Acute http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16178010&form=6&db=m Retinoid-mediated stimulation of steroid sulfatase activity in myeloid leukemic cell lines requires RARalpha and RXR and involves the phosphoinositide 3-kinase and ERK-MAP kinase pathways. diagnostic usage,unassigned 3,0 3.1.6.2 Leukodystrophy, Metachromatic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12249&form=6&db=m Steroid sulfatase in brain: comparison of sulfohydrolase activities for various steroid sulfates in normal and pathological brains, including the various forms of metachromatic leukodystrophy. unassigned - 3.1.6.2 Leukodystrophy, Metachromatic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23508&form=6&db=m Enzymic detection of metachromatic leukodystrophy patients and heterozygotes. diagnostic usage,therapeutic application,unassigned 3,3,0 3.1.6.2 Leukodystrophy, Metachromatic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4422475&form=6&db=m Juvenile metachromatic leukodystrophy: deficiency of an arylsufatase A component. causal interaction,unassigned 4,0 3.1.6.2 Leukodystrophy, Metachromatic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9336808&form=6&db=m Ichthyosis: the skin manifestation of multiple sulfatase deficiency. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Li-Fraumeni Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33875173&form=6&db=m American Association of Clinical Endocrinology Disease State Clinical Review on the Evaluation and Management of Adrenocortical Carcinoma in an Adult: a Practical Approach. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 3.1.6.2 Liver Cirrhosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28810889&form=6&db=m Dehydroepiandrosterone sulfate and dehydroepiandrosterone sulfate/cortisol ratio in cirrhotic patients with septic shock: another sign of hepatoadrenal syndrome? causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 3.1.6.2 Liver Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22490610&form=6&db=m Reduced brain levels of DHEAS in hepatic coma patients: significance for increased GABAergic tone in hepatic encephalopathy. diagnostic usage,ongoing research,unassigned 4,3,0 3.1.6.2 Liver Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26220752&form=6&db=m Inflammatory regulation of steroid sulfatase: A novel mechanism to control estrogen homeostasis and inflammation in chronic liver disease. causal interaction,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Liver Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19594690&form=6&db=m Dehydroepiandrosterone sulphate improves cholestasis-associated fatigue in bile duct ligated rats. causal interaction,therapeutic application,unassigned 2,2,0 3.1.6.2 Liver Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19022235&form=6&db=m Expression of 17beta-hydroxysteroid dehydrogenases and other estrogen-metabolizing enzymes in different cancer cell lines. diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Lung Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20163485&form=6&db=m Cardiovascular disease is associated with greater incident dehydroepiandrosterone sulfate decline in the oldest old: the cardiovascular health study all stars study. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Lupus Erythematosus, Systemic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7955606&form=6&db=m Blood dehydroepiandrosterone sulphate (DHEAS) levels in polymyalgia rheumatica/giant cell arteritis and primary fibromyalgia. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Lupus Erythematosus, Systemic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15635504&form=6&db=m Adrenal androgens and the immune system. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Lymphopenia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34384108&form=6&db=m DHEAS and Differential Blood Counts as Indirect Signs of Glucocorticoid Excess in Adrenal Non-Producing Adenomas. causal interaction,diagnostic usage,therapeutic application,unassigned 4,1,1,0 3.1.6.2 Malnutrition http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17237323&form=6&db=m Higher serum concentrations of DHEAS predict improved nutritional status in helminth-infected children, adolescents, and young adults in Leyte, the Philippines. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,2,0 3.1.6.2 Mania http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22356824&form=6&db=m Polymorphisms in AKR1C4 and HSD3B2 and differences in serum DHEAS and progesterone are associated with paranoid ideation during mania or hypomania in bipolar disorder. causal interaction,unassigned 3,0 3.1.6.2 Melanoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11911290&form=6&db=m Serum dehydroepiandrosterone and dehydroepiandrosterone sulfate and risk of melanoma or squamous cell carcinoma of the skin. diagnostic usage,therapeutic application,unassigned 4,3,0 3.1.6.2 Memory Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2952220&form=6&db=m Effects of dehydroepiandrosterone and its sulfate on brain tissue in culture and on memory in mice. causal interaction,therapeutic application,unassigned 3,3,0 3.1.6.2 Memory Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10854906&form=6&db=m Effects of sigma(1) receptor agonist SA4503 and neuroactive steroids on performance in a radial arm maze task in rats. ongoing research,unassigned 2,0 3.1.6.2 Memory Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15464829&form=6&db=m DHEAS improves learning and memory in aged SAMP8 mice but not in diabetic mice. causal interaction,diagnostic usage,unassigned 1,2,0 3.1.6.2 Meningeal Carcinomatosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8289085&form=6&db=m Neurosteroids in cerebrospinal fluid in neurologic disorders. causal interaction,diagnostic usage,unassigned 1,3,0 3.1.6.2 Metabolic Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3162681&form=6&db=m Characterization of low-density lipoproteins from patients with recessive X-linked ichthyosis. unassigned - 3.1.6.2 Metabolic Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27817780&form=6&db=m [Identification of gene mutation and prenatal diagnosis in a family with X-linked ichthyosis]. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19401371&form=6&db=m The role of genetic variation in the Lamin A/C gene in the etiology of Polycystic Ovary Syndrome. ongoing research,unassigned 2,0 3.1.6.2 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19827728&form=6&db=m [Could serum DHEA and DHEAS levels be good risk predictors of metabolic syndrome and osteoporosis in the population of ageing men?] diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20164211&form=6&db=m Cortisol, DHEAS, their ratio and the metabolic syndrome: evidence from the Vietnam Experience Study. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,2,0 3.1.6.2 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20176080&form=6&db=m Dehydroepiandrosterone (DHEA) treatment in vitro inhibits adipogenesis in human omental but not subcutaneous adipose tissue. causal interaction,unassigned 3,0 3.1.6.2 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26591589&form=6&db=m [ROLE OF CAPSAICIN-SENSITIVE NERVES IN THE REGULATION OF DEHYDROEPIANDROSTERONE SULFATE BLOOD CONTENT UNDER NORMAL AND FRUCTOSE-INDUCED METABOLIC SYNDROME]. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32368067&form=6&db=m Cortisol and DHEAS Related to Metabolic Syndrome in Patients with Schizophrenia. unassigned - 3.1.6.2 Metabolism, Inborn Errors http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2813406&form=6&db=m Molecular studies of deletions at the human steroid sulfatase locus. causal interaction,unassigned 2,0 3.1.6.2 Migraine Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20851729&form=6&db=m Sugar alters the level of serum insulin and plasma glucose and the serum cortisol:DHEAS ratio in female migraine sufferers. causal interaction,diagnostic usage,unassigned 2,4,0 3.1.6.2 Moyamoya Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28710038&form=6&db=m Steroid sulfatase and filaggrin mutations in a boy with severe ichthyosis, elevated serum IgE level and moyamoya syndrome. causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 Mucocele http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30811473&form=6&db=m Investigation of adrenal and thyroid gland dysfunction in dogs with ultrasonographic diagnosis of gallbladder mucocele formation. causal interaction,therapeutic application,unassigned 4,3,0 3.1.6.2 Multiple Endocrine Neoplasia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33875173&form=6&db=m American Association of Clinical Endocrinology Disease State Clinical Review on the Evaluation and Management of Adrenocortical Carcinoma in an Adult: a Practical Approach. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 3.1.6.2 Multiple Endocrine Neoplasia Type 1 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33875173&form=6&db=m American Association of Clinical Endocrinology Disease State Clinical Review on the Evaluation and Management of Adrenocortical Carcinoma in an Adult: a Practical Approach. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 3.1.6.2 Multiple Sulfatase Deficiency Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1687673&form=6&db=m Arylsulfatases A and B in EBV-transformed lymphoid cell lines: studies on their molecular forms in cells from patients with inborn sulfatase deficiencies. Comparative diagnostic value of enzymatic assays. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,4,0 3.1.6.2 Multiple Sulfatase Deficiency Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3083810&form=6&db=m Rapid degradation of steroid sulfatase in multiple sulfatase deficiency. causal interaction,ongoing research,unassigned 2,1,0 3.1.6.2 Multiple Sulfatase Deficiency Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165268&form=6&db=m Synthesis and stability of steroid sulfatase in fibroblasts from multiple sulfatase deficiency. causal interaction,ongoing research,unassigned 4,2,0 3.1.6.2 Multiple Sulfatase Deficiency Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9336808&form=6&db=m Ichthyosis: the skin manifestation of multiple sulfatase deficiency. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Murine Acquired Immunodeficiency Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10371406&form=6&db=m Immune dysfunction during alcohol consumption and murine AIDS: the protective role of dehydroepiandrosterone sulfate. ongoing research,unassigned 2,0 3.1.6.2 Muscle Weakness http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16415544&form=6&db=m The effect of dehydroepiandrosterone sulfate (DHEAS) on myotonia: intracellular studies. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,4,1 3.1.6.2 Muscular Dystrophies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23574351&form=6&db=m A family with fragile X syndrome, Duchenne muscular dystrophy and ichthyosis transmitted by an asymptomatic carrier. causal interaction,unassigned 1,0 3.1.6.2 Muscular Dystrophy, Duchenne http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2344613&form=6&db=m Frequent deletions of the human X chromosome distal short arm result from recombination between low copy repetitive elements. causal interaction,unassigned 3,0 3.1.6.2 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1423966&form=6&db=m Dehydroepiandrosterone sulfate, incidence of myocardial infarction, and extent of atherosclerosis in men. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7977274&form=6&db=m Dehydroepiandrosterone sulfate and the risk of myocardial infarction in US male physicians: a prospective study. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,2,0 3.1.6.2 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8943798&form=6&db=m Dehydroepiandrosterone, dehydroepiandrosterone sulphate and cardiovascular disease. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Myocardial Infarction http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9875107&form=6&db=m von Willebrand factor, tissue plasminogen activator, and dehydroepiandrosterone sulphate predict cardiovascular death in a 10 year follow up of survivors of acute myocardial infarction. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2946952&form=6&db=m A prospective study of dehydroepiandrosterone sulfate, mortality, and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 3.1.6.2 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7882484&form=6&db=m Dehydroepiandrosterone sulfate does not predict cardiovascular death in postmenopausal women. The Rancho Bernardo Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,2,1 3.1.6.2 Myocardial Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597464&form=6&db=m The epidemiology of DHEAS and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,4,2,1 3.1.6.2 Myotonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9710041&form=6&db=m A pilot study of dehydroepiandrosterone sulfate in myotonic dystrophy. unassigned - 3.1.6.2 Myotonia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16415544&form=6&db=m The effect of dehydroepiandrosterone sulfate (DHEAS) on myotonia: intracellular studies. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,4,1 3.1.6.2 Myotonic Dystrophy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16415544&form=6&db=m The effect of dehydroepiandrosterone sulfate (DHEAS) on myotonia: intracellular studies. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,4,1 3.1.6.2 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10079390&form=6&db=m Dehydroepiandrosterone sulfate (DHEAS) secretion in early and advanced solid neoplasms: selective deficiency in metastatic disease. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 3.1.6.2 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10494643&form=6&db=m Asymptomatic adrenal tumours: criteria for endoscopic removal. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,2,0 3.1.6.2 Neoplasm Metastasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19306108&form=6&db=m Arylsulfatase B regulates colonic epithelial cell migration by effects on MMP9 expression and RhoA activation. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Neoplasm, Residual http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8282139&form=6&db=m Hormonal factors and prognosis in epithelial ovarian cancer: a multivariate analysis. diagnostic usage,therapeutic application,unassigned 4,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=141976&form=6&db=m Steroid hormones and human breast cancer. An hypothesis. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1423966&form=6&db=m Dehydroepiandrosterone sulfate, incidence of myocardial infarction, and extent of atherosclerosis in men. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2146126&form=6&db=m Isolated dehydroepiandrosterone sulphate hypersecretion: a case report. causal interaction,unassigned 4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2181598&form=6&db=m [Hyperandrogenism in premenopausal women] causal interaction,unassigned 3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2344367&form=6&db=m Sulfate derivatives of 2-phenylindoles as novel steroid sulfataseinhibitors. An in vitro study on structure-activity-relationship. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2519212&form=6&db=m [Tissue culture and estrogen, to clarify the roles of estrone sulfate] causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2532426&form=6&db=m Measurement of serum levels of dehydroepiandrosterone sulfate: a comparison of radioimmunoassay and enzymatic analysis. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2953750&form=6&db=m In situ steroid sulfatase activity in human epithelial carcinoma cells of vaginal, ovarian, and endometrial origin. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2956173&form=6&db=m Decreased serum level of dehydroepiandrosterone sulfate in postmenopausal women with ovarian cancer. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2969800&form=6&db=m The hydrolysis of estrone sulfate and dehydroepiandrosterone sulfate by MCF-7 human breast cancer cells. ongoing research,therapeutic application,unassigned 3,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2974852&form=6&db=m The treatment of a hyperandrogenic and virilizing state in an elderly female with a synthetic LHRH agonist. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3224612&form=6&db=m Preneoplastic lesions and prognostic factors of breast cancer. unassigned - 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3732908&form=6&db=m Peripheral and ovarian venous concentrations of steroid and gonadotropin hormones in postmenopausal women with epithelial ovarian tumors. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6217751&form=6&db=m Adrenal and sex steroid hormone production by a virilizing adrenal adenoma and its diagnosis with computerized tomography. causal interaction,diagnostic usage,unassigned 3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6309640&form=6&db=m Strategic approach of hyperandrogenism in women. unassigned - 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6508360&form=6&db=m Estrone sulfatase activity in normal and abnormal endometrium. causal interaction,unassigned 4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7626490&form=6&db=m The role of cytokines and sulphatase inhibitors in regulating oestrogen synthesis in breast tumours. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7696150&form=6&db=m Estrogenicity, antiestrogenicity and estrone sulfatase inhibition of estrone-3-amine and estrone-3-thiol. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7749143&form=6&db=m Dehydroepiandrosterone concentration in breast cancer tissue is related to its plasma gradient across the mammary gland. ongoing research,therapeutic application,unassigned 3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8013393&form=6&db=m Regulation of macrophage dehydroepiandrosterone sulfate metabolism by inflammatory cytokines. therapeutic application,unassigned 1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8180114&form=6&db=m Inhibition of steroid sulphatase activity by steroidal methylthiophosphonates: potential therapeutic agents in breast cancer. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8417823&form=6&db=m Inhibition of estrone sulfatase activity by estrone-3-methylthiophosphonate: a potential therapeutic agent in breast cancer. causal interaction,ongoing research,therapeutic application,unassigned 4,4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8476770&form=6&db=m Prognostic significance of aromatase and estrone sulfatase enzymes in human breast cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,4,1 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8664167&form=6&db=m Detection of breast cancer-associated estrone sulfatase in breast cancer biopsies and cell lines using polymerase chain reaction. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,4,4,1 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8903427&form=6&db=m Evidence of in situ estrogen synthesis in nitrosomethylurea-induced rat mammary tumors via the enzyme estrone sulfatase. causal interaction,ongoing research,unassigned 1,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9009236&form=6&db=m Synthesis and sulfatase inhibitory activities of non-steroidal estrone sulfatase inhibitors. causal interaction,therapeutic application,unassigned 3,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9009241&form=6&db=m Inhibition of placental estrone sulfatase activity and MCF-7 breast cancer cell proliferation by estrone-3-amino derivatives. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9044848&form=6&db=m Inhibition of estrone sulfatase and proliferation of human breast cancer cells by nonsteroidal (p-O-sulfamoyl)-N-alkanoyl tyramines. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9100554&form=6&db=m Plasma dehydroepiandrosterone sulfate levels in patients with hyperfunctioning and non-hyperfunctioning adrenal tumors before and after adrenal surgery. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9116318&form=6&db=m Influence of estrogen metabolism on proliferation of human breast cancer. diagnostic usage,ongoing research,unassigned 2,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9166121&form=6&db=m Differential effect of carbamazepine and valproate monotherapy on plasma levels of oestrone sulphate and dehydroepiandrosterone sulphate in male epileptic patients. diagnostic usage,ongoing research,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9215119&form=6&db=m Girls with virilisation in childhood: a diagnostic protocol for investigation. diagnostic usage,ongoing research,therapeutic application,unassigned 2,1,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9654650&form=6&db=m Development of potent non-estrogenic estrone sulfatase inhibitors. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9797847&form=6&db=m Study of glucose tolerance in consecutive patients harbouring incidental adrenal tumours. Study Group of Incidental Adrenal Adenoma. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10079390&form=6&db=m Dehydroepiandrosterone sulfate (DHEAS) secretion in early and advanced solid neoplasms: selective deficiency in metastatic disease. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10197641&form=6&db=m Dietary intervention at middle age: caloric restriction but not dehydroepiandrosterone sulfate increases lifespan and lifetime cancer incidence in mice. diagnostic usage,ongoing research,unassigned 1,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10215035&form=6&db=m Development of (p-O-sulfamoyl)-N-alkanoyl-phenylalkyl amines as non-steroidal estrone sulfatase inhibitors. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,4 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10337439&form=6&db=m Dehydroepiandrosterone sulfate, diseases and mortality in a general aged population. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10545798&form=6&db=m Aromatase and intracrinology of estrogen in hormone-dependent tumors. causal interaction,therapeutic application,unassigned 1,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10685333&form=6&db=m Total testosterone and DHEAS levels as predictors of androgen-secreting neoplasms: a populational study. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,1,2 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10843528&form=6&db=m Dehydroepiandrosterone sulfate (DHEAS) and testosterone: relation to HIV illness stage and progression over one year. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10888707&form=6&db=m The adrenal steroid status in relation to inflammatory cytokines (interleukin-6 and tumour necrosis factor) in polymyalgia rheumatica. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10910045&form=6&db=m In vivo inhibition of estrone sulfatase activity and growth of nitrosomethylurea-induced mammary tumors by 667 COUMATE. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10925213&form=6&db=m Elevated steroid sulfatase expression in breast cancers. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11091733&form=6&db=m Significance of Steroid Sulfatase Expression in Human Breast Cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,4,4 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11091739&form=6&db=m Localization of Estrone Sulfatase in Human Breast Carcinomas. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11115773&form=6&db=m Human granulosa cells are a site of sulphatase activity and are able to utilize dehydroepiandrosterone sulphate as a precursor for oestradiol production. therapeutic application,unassigned 4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11260976&form=6&db=m [Replacement therapy in adrenopause. Dehydroepiandrosterone and aging] causal interaction,unassigned 3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11321667&form=6&db=m Increased serum cortisol levels are associated with high tumour grade in patients with renal cell carcinoma. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11592593&form=6&db=m Early shifts of adrenal steroid synthesis before and after relief of short-term cholestasis. diagnostic usage,unassigned 4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11909687&form=6&db=m Dehydroepiandrosterone-sulfate serum levels and common age-related diseases: results from a cross-sectional Italian study of a general elderly population. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12398784&form=6&db=m Decreased levels of dehydroepiandrosterone sulphate in severe critical illness: a sign of exhausted adrenal reserve? causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,2,1 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12534357&form=6&db=m During a corticotropin-releasing hormone test in healthy subjects, administration of a beta-adrenergic antagonist induced secretion of cortisol and dehydroepiandrosterone sulfate and inhibited secretion of ACTH. diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12747791&form=6&db=m Carbonic anhydrase inhibitors. Inhibition of cytosolic isozymes I and II and transmembrane, tumor-associated isozyme IX with sulfamates including EMATE also acting as steroid sulfatase inhibitors. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12792725&form=6&db=m Immunohistochemical analysis of estrone sulfatase and aromatase in human breast cancer tissues. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12814632&form=6&db=m Microtiter plate cellular assay for human steroid sulfatase with fluorescence readout. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12852749&form=6&db=m First dual aromatase-steroid sulfatase inhibitors. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15084348&form=6&db=m Development of novel steroid sulfatase inhibitors; II. TZS-8478 potently inhibits the growth of breast tumors in postmenopausal breast cancer model rats. causal interaction,ongoing research,therapeutic application,unassigned 4,4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15106362&form=6&db=m Breast cancer size in postmenopausal women is correlated with body mass index and androgen serum levels. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15149660&form=6&db=m 2-Alkylsulfanyl estrogen derivatives: synthesis of a novel class of multi-targeted anti-tumour agents. therapeutic application,unassigned 1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15328524&form=6&db=m Pharmacokinetics of the nonsteroidal steroid sulphatase inhibitor 667 COUMATE and its sequestration into red blood cells in rats. ongoing research,unassigned 2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15637992&form=6&db=m [Sertoli-Leydig cell tumour (arrhenoblastoma) in a patient with polycystic ovary syndrome: clinical, ultrasonographic, hormonal and histopathological evaluation] causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15862971&form=6&db=m Anti-cancer activities of novel D-ring modified 2-substituted estrogen-3-O-sulfamates. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15865431&form=6&db=m First crystal structures of human carbonic anhydrase II in complex with dual aromatase-steroid sulfatase inhibitors. therapeutic application,unassigned 3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15879355&form=6&db=m Defect of a subpopulation of natural killer immune cells in Graves' disease and Hashimoto's thyroiditis: normalizing effect of dehydroepiandrosterone sulfate. ongoing research,unassigned 2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15989553&form=6&db=m Aromatase inhibitors and other novel agents in breast cancer treatment. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16132094&form=6&db=m Synthesis of a non-hydrolyzable estrone sulfate analogue bearing the difluoromethanesulfonamide group and its evaluation as a steroid sulfatase inhibitor. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16166425&form=6&db=m Estrogen inhibits cell proliferation through in situ production in human thymoma. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16303840&form=6&db=m Estrogen-metabolizing enzymes in breast cancers from women over the age of 80 years. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16541422&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in human prostate cancer. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16556483&form=6&db=m Quantitative analysis of aromatase, sulfatase and 17beta-HSD(1) mRNA expression in soft tissue metastases of breast cancer. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16618615&form=6&db=m Unusual Visualization of an Adrenal Carcinoma on NP-59 Scintiscan. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16960027&form=6&db=m DHEAS levels and mortality in disabled older women: the Women's Health and Aging Study I. causal interaction,unassigned 3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17470679&form=6&db=m Steroid sulfatase: a new target for the endocrine therapy of breast cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17477470&form=6&db=m Increased DHEAS levels in patients with rheumatoid arthritis after treatment with tumor necrosis factor antagonists: evidence for improved adrenal function. causal interaction,therapeutic application,unassigned 3,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17594810&form=6&db=m [Molecular mechanisms of DHEA and DHEAs on apoptosis and cell cycle arrest via Akt pathway in hepatoma cell lines] diagnostic usage,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17604157&form=6&db=m Immunohistochemical analysis of steroid sulfatase in human tissues. causal interaction,diagnostic usage,ongoing research,unassigned 4,3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17690503&form=6&db=m Relation of Serum Levels of Estrogen and Dehydroepiandrosterone Sulfate to Hormone Receptor Status among Postmenopausal Women with Breast Cancer. diagnostic usage,unassigned 4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17696419&form=6&db=m 3,17-disubstituted 2-alkylestra-1,3,5(10)-trien-3-ol derivatives: synthesis, in vitro and in vivo anticancer activity. causal interaction,ongoing research,unassigned 1,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17708571&form=6&db=m ANXA7 expression represents hormone-relevant tumor suppression in different cancers. unassigned - 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18268111&form=6&db=m V89L polymorphism of the 5alpha-reductase Type II gene (SRD5A2), endogenous sex hormones, and prostate cancer risk. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,4,1 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18450955&form=6&db=m The use of steroid sulfatase inhibitors as a novel therapeutic strategy against hormone-dependent endometrial cancer. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18843186&form=6&db=m Sulfatase Activity in Normal and Neoplastic Endometrium. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19159687&form=6&db=m Expression level of enzymes related to in situ estrogen synthesis and clinicopathological parameters in breast cancer patients. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19250195&form=6&db=m The development of steroid sulfatase inhibitors for hormone-dependent cancer therapy. causal interaction,unassigned 4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19559719&form=6&db=m Design and synthesis of carbon-11-labeled dual aromatase-steroid sulfatase inhibitors as new potential PET agents for imaging of aromatase and steroid sulfatase expression in breast cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 2,1,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19747768&form=6&db=m Expression of sulfotransferases and sulfatases in human breast cancer: Impact on resveratrol metabolism. diagnostic usage,unassigned 3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20096825&form=6&db=m Recommendations for investigation of hyperandrogenism. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20120745&form=6&db=m Early development of osteoporosis in male smokers with hypoandrogenism due to fascioliasis with or without schistosomiasis added by life style. unassigned - 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20297840&form=6&db=m Structures of human carbonic anhydrase II/inhibitor complexes reveal a second binding site for steroidal and nonsteroidal inhibitors. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20498139&form=6&db=m Cortisol, DHEA sulphate, their ratio, and all-cause and cause-specific mortality in the Vietnam Experience Study. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21238537&form=6&db=m Development of steroid sulfatase inhibitors. unassigned - 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21464173&form=6&db=m In Vitro Metabolism of Irosustat, a Novel Steroid Sulphatase Inhibitor: Inter-Species Comparison, Metabolite Identification and Metabolic Enzyme Identification. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21598196&form=6&db=m Using molecular docking to investigate the anti-breast cancer activity of low molecular weight compounds present on wild mushrooms. therapeutic application,unassigned 1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21859802&form=6&db=m Steroid sulfatase inhibitors for estrogen- and androgen-dependent cancers. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21904110&form=6&db=m Induction of steroid sulfatase expression by tumor necrosis factor-? through phosphatidylinositol 3-kinase/Akt signaling pathway in PC-3 human prostate cancer cells. ongoing research,unassigned 3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21990014&form=6&db=m Structure-activity relationship for the first-in-class clinical steroid sulfatase inhibitor Irosustat (STX64, BN83495). diagnostic usage,ongoing research,therapeutic application,unassigned 3,1,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22264754&form=6&db=m 17?-Arylsulfonamides of 17?-aminoestra-1,3,5(10)-trien-3-ol as highly potent inhibitors of steroid sulfatase. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22451700&form=6&db=m In vitro evaluation of the interaction potential of irosustat with drug-metabolizing enzymes. causal interaction,therapeutic application,unassigned 3,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22542502&form=6&db=m Synthesis of 2-[11C]methoxy-3,17?-O,O-bis(sulfamoyl)estradiol as a new potential PET agent for imaging of steroid sulfatase (STS) in cancers. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23326806&form=6&db=m Evaluation of cytotoxic effects of several novel tetralin derivatives against Hela, MDA-MB-468, and MCF-7 cancer cells. therapeutic application,unassigned 2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23476785&form=6&db=m The sulfatase pathway for estrogen formation: targets for the treatment and diagnosis of hormone-associated tumors. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,4 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23916543&form=6&db=m Reduced expression of steroid sulfatase in primary colorectal cancer. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24009850&form=6&db=m Bacterial Lipopolysaccharides Induce Steroid Sulfatase Expression and Cell Migration through IL-6 Pathway in Human Prostate Cancer Cells. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25146553&form=6&db=m DHEAS for the prediction of subclinical Cushing's syndrome: perplexing or advantageous? diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25992880&form=6&db=m Discovery and Development of the Aryl O-Sulfamate Pharmacophore for Oncology and Women's Health. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26044867&form=6&db=m In vitro interactions between mammary fibroblasts (Hs 578Bst) and cancer epithelial cells (MCF-7) modulate aromatase, steroid sulfatase and 17?-hydroxysteroid dehydrogenases. causal interaction,ongoing research,unassigned 3,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26181260&form=6&db=m Statin Use at the Time of Initiation of Androgen Deprivation Therapy and Time to Progression in Patients With Hormone-Sensitive Prostate Cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 2,3,4,1 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26861448&form=6&db=m Simultaneous robotic-assisted adrenalectomy and partial nephrectomy: technical aspects. diagnostic usage,therapeutic application,unassigned 2,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26974384&form=6&db=m Sulfatase inhibitors for recidivist breast cancer treatment: A chemical review. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27645128&form=6&db=m Role of OATP transporters in steroid uptake by prostate cancer cells in vivo. causal interaction,diagnostic usage,therapeutic application,unassigned 2,2,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28032462&form=6&db=m Synthesis and biological evaluation of N-acylated tyramine sulfamates containing C-F bonds as steroid sulfatase inhibitors. causal interaction,unassigned 2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28118677&form=6&db=m Predicting malignancy in adrenal incidentaloma and evaluation of a novel risk stratification algorithm. causal interaction,diagnostic usage,unassigned 1,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28924479&form=6&db=m Marked hyperandrogenicity in a 60-year-old woman. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29563635&form=6&db=m In breast cancer subtypes steroid sulfatase (STS) is associated with less aggressive tumour characteristics. causal interaction,diagnostic usage,unassigned 1,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29803725&form=6&db=m Steroid sulfatase inhibition success and limitation in breast cancer clinical assays: An underlying mechanism. causal interaction,ongoing research,therapeutic application,unassigned 4,3,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29941676&form=6&db=m Detection of a novel, primate-specific 'kill switch' tumor suppression mechanism that may fundamentally control cancer risk in humans: an unexpected twist in the basic biology of TP53. causal interaction,ongoing research,unassigned 1,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30186172&form=6&db=m Estrone-3-Sulfate Stimulates the Proliferation of T47D Breast Cancer Cells Stably Transfected With the Sodium-Dependent Organic Anion Transporter SOAT (SLC10A6). causal interaction,diagnostic usage,ongoing research,therapeutic application 3,1,4,2 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30306775&form=6&db=m Detection of a novel, primate-specific ‘kill switch’ tumor suppression mechanism that may fundamentally control cancer risk in humans: an unexpected twist in the basic biology of TP53 causal interaction,ongoing research,unassigned 1,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30572523&form=6&db=m Oncological markers CA-125, CA 19-9 and endometriosis. causal interaction,unassigned 4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31149308&form=6&db=m THE RARE CASE OF MIXED GONADAL DYSGENESIS, MOSAIC KARYOTYPE, PETROCLIVAL MENINGIOMA AND IDIOPATHIC HYPERDEHYDROEPIANDROSTERONISM. unassigned - 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31195119&form=6&db=m Human steroid sulfatase enhances aerobic glycolysis through induction of HIF1? and glycolytic enzymes. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31499503&form=6&db=m Hyperandrogenism and Adrenocortical Tumors. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31904042&form=6&db=m Achieving the ratiometric imaging of steroid sulfatase in living cells and tissues with a two-photon fluorescent probe. diagnostic usage,unassigned 3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32173116&form=6&db=m A new series of aryl sulfamate derivatives: Design, synthesis, and biological evaluation. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32435230&form=6&db=m Very High Dehydroepiandrosterone Sulfate (DHEAS) in Serum of an Overweight Female Adolescent Without a Tumor. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33112833&form=6&db=m From adrenarche to aging of adrenal zona reticularis: precocious female adrenopause onset. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,2,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33655862&form=6&db=m Anti-breast cancer potential of natural and synthetic coumarin derivatives. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33875173&form=6&db=m American Association of Clinical Endocrinology Disease State Clinical Review on the Evaluation and Management of Adrenocortical Carcinoma in an Adult: a Practical Approach. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34321053&form=6&db=m Simultaneous expression of steroid sulfatase and androgen receptor reduced overall survival of patients with epithelial ovarian tumors. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34539912&form=6&db=m Importance of Dehydroepiandrosterone Sulfate Assessment with Special Attention for Adrenal Tumours and Arterial Hypertension. causal interaction,unassigned 1,0 3.1.6.2 Nephrotic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22419362&form=6&db=m Steroid-resistant nephrotic syndrome associated with steroid sulfatase deficiency-x-linked recessive ichthyosis: a case report and review of literature. causal interaction,therapeutic application,unassigned 4,3,0 3.1.6.2 Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10064172&form=6&db=m The effect of dehydroepiandrosterone sulfate administration to patients with multi-infarct dementia. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Nervous System Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29017935&form=6&db=m Physiological implications of DHEAS-induced non-classical steroid hormone signaling. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Neural Tube Defects http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7521964&form=6&db=m Second-trimester maternal serum screening using alpha-fetoprotein, human chorionic gonadotrophin, and unconjugated oestriol: experience of a regional programme. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,1,0 3.1.6.2 Neuralgia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31495080&form=6&db=m Spinal Nitric Oxide Synthase Type II Increases Neurosteroid-metabolizing Cytochrome P450c17 Expression in a Rodent Model of Neuropathic Pain. causal interaction,ongoing research,therapeutic application,unassigned 1,4,1,0 3.1.6.2 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=4474185&form=6&db=m Expression of the neuron-specific protein, 14-3-2, and steroid sulfatase in neuroblastoma cell hybrids. unassigned - 3.1.6.2 Neuroblastoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11574977&form=6&db=m Effect of dehydroepiandrosterone and its sulfate metabolite on neuronal cell viability in culture. diagnostic usage,ongoing research,unassigned 1,2,0 3.1.6.2 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10567728&form=6&db=m Actions of dehydroepiandrosterone and its sulfate in the central nervous system: effects on cognition and emotion in animals and humans. causal interaction,unassigned 2,0 3.1.6.2 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11599307&form=6&db=m Preclinical development of neurosteroids as neuroprotective agents for the treatment of neurodegenerative diseases. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15545016&form=6&db=m Studies on homocysteine and dehydroepiandrosterone sulphate plasma levels in Alzheimer's disease patients and in Parkinson's disease patients. causal interaction,unassigned 3,0 3.1.6.2 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30029726&form=6&db=m Astrocyte Neuroprotection and Dehydroepiandrosterone. therapeutic application,unassigned 4,0 3.1.6.2 Neuroinflammatory Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29796990&form=6&db=m Neuroprotective Effects of Dehydroepiandrosterone Sulfate Through Inhibiting Expression of Matrix Metalloproteinase-9 from Bradykinin-Challenged Astroglia. causal interaction,ongoing research,therapeutic application,unassigned 1,4,4,0 3.1.6.2 Neurologic Manifestations http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3516063&form=6&db=m A new syndrome of anosmia, ichthyosis, hypogonadism, and various neurological manifestations with deficiency of steroid sulfatase and arylsulfatase C. causal interaction,unassigned 4,0 3.1.6.2 Non-alcoholic Fatty Liver Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19701841&form=6&db=m Endocrine factors associated with non-alcoholic fatty liver disease in women with polycystic ovary syndrome: Do androgens play a role? diagnostic usage,ongoing research,unassigned 2,1,0 3.1.6.2 Non-alcoholic Fatty Liver Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20001571&form=6&db=m Endocrine factors associated with non-alcoholic fatty liver disease in women with polycystic ovary syndrome: Do androgens play a role? diagnostic usage,ongoing research,unassigned 2,1,0 3.1.6.2 Non-alcoholic Fatty Liver Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21841322&form=6&db=m Serum dehydroepiandrosterone sulphate levels in patients with non-alcoholic Fatty liver disease. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1835937&form=6&db=m No acute effect of physiological insulin increase on dehydroepiandrosterone sulfate in women with obesity and/or polycystic ovarian disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,1,1 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2946952&form=6&db=m A prospective study of dehydroepiandrosterone sulfate, mortality, and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7718911&form=6&db=m [The relationship between serum dehydroepiandrosterone sulfate levels and factors associated with cardiovascular diseases: a cross-sectional study in Japan] causal interaction,diagnostic usage,ongoing research,unassigned 3,4,1,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7882484&form=6&db=m Dehydroepiandrosterone sulfate does not predict cardiovascular death in postmenopausal women. The Rancho Bernardo Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,2,2,1 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7955458&form=6&db=m The independent effects of polycystic ovary syndrome and obesity on serum concentrations of gonadotrophins and sex steroids in premenopausal women. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8348161&form=6&db=m Obesity affects circulating estradiol levels of premenopausal hirsute women receiving leuprolide acetate depot. ongoing research,therapeutic application,unassigned 2,1,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8452124&form=6&db=m A prospective study of dehydroepiandrosterone sulfate (DHEAS) and bone mineral density in older men and women. causal interaction,therapeutic application,unassigned 1,2,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8550794&form=6&db=m Dehydroepiandrosterone, dehydroepiandrosterone sulfate, obesity, waist-hip ratio, and noninsulin-dependent diabetes in postmenopausal women: the Rancho Bernardo Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,1 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9590600&form=6&db=m Low dehydroepiandrosterone sulfate and heart disease in middle-aged men: cross-sectional results from the Massachusetts Male Aging Study. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9703954&form=6&db=m Dehydroepiandrosterone reduces proliferation and differentiation of 3T3-L1 preadipocytes. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9762876&form=6&db=m Predictors of sex hormone levels among the elderly: a study in Greece. diagnostic usage,unassigned 2,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10325597&form=6&db=m [The influence of obesity on sexual development in pubertal children] causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11159150&form=6&db=m Low dehydroepiandrosterone and ischemic heart disease in middle-aged men: prospective results from the Massachusetts Male Aging Study. unassigned - 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11341297&form=6&db=m GG-genotype in the promotor region of uncoupling-protein-1 gene is associated with lower level of dehydroepiandrosterone in type 2 diabetes. diagnostic usage,unassigned 4,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11573555&form=6&db=m A study of insulin resistance in subjects with acanthosis nigricans. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12828082&form=6&db=m The association of dehydroepiandrosterone, obesity, waist-hip ratio and insulin resistance with fatty liver in postmenopausal women--a hyperinsulinemic euglycemic insulin clamp study. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,3,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19276095&form=6&db=m The association between dehydroepiandosterone and frailty in older men and women. causal interaction,unassigned 1,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19342030&form=6&db=m In polycystic ovary syndrome, adrenal steroids are regulated differently in the morning versus in response to nutrient intake. causal interaction,unassigned 3,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23530294&form=6&db=m Age-related decline in salivary dehydroepiandrosterone sulfate and associated health risks among African Americans. causal interaction,diagnostic usage,ongoing research,unassigned 4,2,2,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24497646&form=6&db=m Hepatic Overexpression of Steroid Sulfatase Ameliorates Mouse Models of Obesity and Type 2 Diabetes through Sex-specific Mechanisms. causal interaction,ongoing research,unassigned 1,3,0 3.1.6.2 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33878788&form=6&db=m Differential Effects of Various Androgens on Polycystic Ovary Syndrome. ongoing research,therapeutic application,unassigned 1,2,0 3.1.6.2 Obesity, Abdominal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8550794&form=6&db=m Dehydroepiandrosterone, dehydroepiandrosterone sulfate, obesity, waist-hip ratio, and noninsulin-dependent diabetes in postmenopausal women: the Rancho Bernardo Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,2,4,1 3.1.6.2 Obesity, Abdominal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8862983&form=6&db=m Dehydroepiandrosterone sulfate and testosterone are independently associated with body fat distribution in premenopausal women. causal interaction,unassigned 3,0 3.1.6.2 Obesity, Abdominal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22693174&form=6&db=m Myostatin and its association with abdominal obesity, androgen and follistatin levels in women with polycystic ovary syndrome. diagnostic usage,unassigned 4,0 3.1.6.2 Obesity, Abdominal http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31641727&form=6&db=m Allostatic Load as a Biological Substrate to Intrinsic Capacity: A Secondary Analysis of CRELES. therapeutic application,unassigned 1,0 3.1.6.2 Obesity, Morbid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12647279&form=6&db=m Serum concentrations of dehydroepiandrosterone sulfate and leptin in obese patients with normal serum cholesterol. unassigned - 3.1.6.2 Obesity, Morbid http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16117191&form=6&db=m Role of dehydroepiandrosterone sulfate levels on body composition after laparoscopic adjustable gastric banding in pre-menopausal morbidly obese women. causal interaction,unassigned 2,0 3.1.6.2 Opportunistic Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10843528&form=6&db=m Dehydroepiandrosterone sulfate (DHEAS) and testosterone: relation to HIV illness stage and progression over one year. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Osteoarthritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7554562&form=6&db=m Blood dehydroepiandrosterone sulphate (DHEAS) levels in pemphigoid/pemphigus and psoriasis. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 3.1.6.2 Osteoarthritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7955606&form=6&db=m Blood dehydroepiandrosterone sulphate (DHEAS) levels in polymyalgia rheumatica/giant cell arteritis and primary fibromyalgia. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Osteoarthritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8299250&form=6&db=m Relationship between blood and joint tissue DHEAS levels in rheumatoid arthritis and osteoarthritis. diagnostic usage,ongoing research,unassigned 2,3,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2970263&form=6&db=m Sex hormone status and osteoporosis in postmenopausal women with rheumatoid arthritis. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8452124&form=6&db=m A prospective study of dehydroepiandrosterone sulfate (DHEAS) and bone mineral density in older men and women. causal interaction,therapeutic application,unassigned 1,2,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9650403&form=6&db=m [Reduced serum dehydroepiandrosterone levels in postmenopausal osteoporosis] causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,2,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10341026&form=6&db=m Carnitine and dehydroepiandrosterone sulfate induce protein synthesis in porcine primary osteoblast-like cells. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10821876&form=6&db=m Effects of age on serum dehydroepiandrosterone sulfate, IGF-I, and IL-6 levels in women. unassigned - 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11155790&form=6&db=m Replacement therapy with DHEA plus corticosteroids in patients with chronic inflammatory diseases--substitutes of adrenal and sex hormones. causal interaction,unassigned 4,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11260976&form=6&db=m [Replacement therapy in adrenopause. Dehydroepiandrosterone and aging] causal interaction,unassigned 3,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11853283&form=6&db=m Serum dehydroepiandrosterone sulfate, testosterone, and biochemical markers of bone turnover in elderly Thai men. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,4 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11909687&form=6&db=m Dehydroepiandrosterone-sulfate serum levels and common age-related diseases: results from a cross-sectional Italian study of a general elderly population. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11940375&form=6&db=m Treatment of osteoporosis in men using dehydroepiandrosterone sulfate. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16521435&form=6&db=m [Dehydroepiandrosterone (DHEA)--slows down the aging process?] causal interaction,ongoing research,unassigned 4,1,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17334880&form=6&db=m CYP3A7*1C polymorphism, serum dehydroepiandrosterone sulfate level, and bone mineral density in postmenopausal women. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19827728&form=6&db=m [Could serum DHEA and DHEAS levels be good risk predictors of metabolic syndrome and osteoporosis in the population of ageing men?] diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28875157&form=6&db=m Sex hormones and quantitative ultrasound parameters at the heel in men and women from the general population. diagnostic usage,unassigned 3,0 3.1.6.2 Osteoporosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30029729&form=6&db=m Dehydroepiandrosterone and Bone. diagnostic usage,therapeutic application,unassigned 2,1,0 3.1.6.2 Ovarian Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2956173&form=6&db=m Decreased serum level of dehydroepiandrosterone sulfate in postmenopausal women with ovarian cancer. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Ovarian Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3732908&form=6&db=m Peripheral and ovarian venous concentrations of steroid and gonadotropin hormones in postmenopausal women with epithelial ovarian tumors. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Ovarian Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12594820&form=6&db=m Circulating levels of sex steroid hormones and risk of ovarian cancer. diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Ovarian Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18947862&form=6&db=m Estrone sulfatase activity in patients with advanced ovarian cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,4 3.1.6.2 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597464&form=6&db=m The epidemiology of DHEAS and cardiovascular disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 1,4,2,1 3.1.6.2 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9023605&form=6&db=m Plasma lipids, dehydroepiandosterone sulphate and insulin concentrations in elderly overweight angina patients, and effect of weight loss. diagnostic usage,therapeutic application,unassigned 1,1,0 3.1.6.2 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27392706&form=6&db=m Maternal Pre-pregnancy BMI and Reproductive Health of Daughters in Young Adulthood. diagnostic usage,unassigned 3,0 3.1.6.2 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28099123&form=6&db=m Hyperandrogenemia in women with polycystic ovary syndrome: prevalence, characteristics and association with body mass index. diagnostic usage,unassigned 4,0 3.1.6.2 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30042733&form=6&db=m Trajectories of Growth and Serum DHEAS and IGF-1 Concentrations in Girls With a History of Premature Adrenarche: Attenuation of the Phenotype by Adulthood. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32435230&form=6&db=m Very High Dehydroepiandrosterone Sulfate (DHEAS) in Serum of an Overweight Female Adolescent Without a Tumor. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33112833&form=6&db=m From adrenarche to aging of adrenal zona reticularis: precocious female adrenopause onset. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,2,0 3.1.6.2 Pancreatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34386598&form=6&db=m Dehydroepiandrosterone modulates oxidative DNA damage in pancreatic cancer: A case-control study. causal interaction,diagnostic usage,unassigned 1,4,0 3.1.6.2 Paralysis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597462&form=6&db=m DHEAS as an effective vaccine adjuvant in elderly humans. Proof-of-principle studies. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Parasitemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11119497&form=6&db=m Human resistance to Plasmodium falciparum increases during puberty and is predicted by dehydroepiandrosterone sulfate levels. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Parkinsonian Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11909687&form=6&db=m Dehydroepiandrosterone-sulfate serum levels and common age-related diseases: results from a cross-sectional Italian study of a general elderly population. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Pemphigus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26910580&form=6&db=m Evaluating serum prolactin and serum dehydroepiandrosterone sulfate levels in patients with pemphigus. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Periodontitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18727654&form=6&db=m Association of cortisol and dehydroepiandrosterone sulphate levels in serum with periodontal status in older Japanese adults. causal interaction,diagnostic usage,unassigned 1,4,0 3.1.6.2 Periventricular Nodular Heterotopia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16504797&form=6&db=m Steroid Sulfatase Deficiency With Bilateral Periventricular Nodular Heterotopia. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 Pheochromocytoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8165486&form=6&db=m Laparoscopic adrenalectomy. The importance of a flank approach in the lateral decubitus position. unassigned - 3.1.6.2 Pheochromocytoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15148390&form=6&db=m Dehydroepiandrosterone and allopregnanolone protect sympathoadrenal medulla cells against apoptosis via antiapoptotic Bcl-2 proteins. ongoing research,unassigned 4,0 3.1.6.2 Pituitary ACTH Hypersecretion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2246674&form=6&db=m Cushing's syndrome: new variants and association with empty sella syndrome. causal interaction,unassigned 4,0 3.1.6.2 Pituitary ACTH Hypersecretion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704956&form=6&db=m Reduced serum levels of dehydroepiandrosterone sulphate in adrenal incidentalomas: a marker of adrenocortical tumour. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Pituitary ACTH Hypersecretion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7889616&form=6&db=m Dissociation of adrenal androgen and cortisol secretion in Cushing's syndrome. causal interaction,diagnostic usage,ongoing research,unassigned 2,4,1,0 3.1.6.2 Pituitary ACTH Hypersecretion http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12398228&form=6&db=m Prostate-specific antigen is increased in female patients with Cushing's disease. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Pityriasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17478393&form=6&db=m Pityriasis rotunda with low levels of steroid sulfatase. diagnostic usage,unassigned 2,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12413984&form=6&db=m Use of dexamethasone and clomiphene citrate in the treatment of clomiphene citrate-resistant patients with polycystic ovary syndrome and normal dehydroepiandrosterone sulfate levels: a prospective, double-blind, placebo-controlled trial. diagnostic usage,ongoing research,unassigned 2,2,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12749433&form=6&db=m Troglitazone decreases adrenal androgen levels in women with polycystic ovary syndrome. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,1 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15482786&form=6&db=m Role of a CYP17 polymorphism in the regulation of circulating dehydroepiandrosterone sulfate levels in women with polycystic ovary syndrome. causal interaction,ongoing research,unassigned 2,3,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16259799&form=6&db=m Development of a method for the measurement of dehydroepiandrosterone sulphate by liquid chromatography-tandem mass spectrometry. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,1,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17392600&form=6&db=m Prevalence and metabolic characteristics of adrenal androgen excess in hyperandrogenic women with different phenotypes. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18439591&form=6&db=m Dehydroepiandrosterone sulfate and insulin resistance in patients with polycystic ovary syndrome. diagnostic usage,ongoing research,therapeutic application,unassigned 4,2,2,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18684447&form=6&db=m Effect of insulin and testosterone on androgen production and transcription of SULT2A1 in the NCI-h295r adrenocortical cell line. causal interaction,ongoing research,unassigned 3,4,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19249030&form=6&db=m Prevalence of hyperandrogenemia in the polycystic ovary syndrome diagnosed by the National Institutes of Health 1990 criteria. diagnostic usage,ongoing research,therapeutic application,unassigned 4,2,1,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23122578&form=6&db=m Placental steroidogenesis in pregnant women with polycystic ovary syndrome. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23861462&form=6&db=m Variants in SULT2A1 Affect the DHEA Sulphate to DHEA Ratio in Patients With Polycystic Ovary Syndrome But Not the Hyperandrogenic Phenotype. diagnostic usage,unassigned 1,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25342852&form=6&db=m Visceral adiposity index and DHEAS are useful markers of diabetes risk in women with polycystic ovary syndrome. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25585504&form=6&db=m Influence of adrenal hyperandrogenism on the clinical and metabolic phenotype of women with polycystic ovary syndrome. diagnostic usage,ongoing research,unassigned 2,1,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25594860&form=6&db=m PAPSS2 deficiency causes androgen excess via impaired DHEA sulfation - in vitro and in vivo studies in a family harboring two novel PAPSS2 mutations. causal interaction,unassigned 4,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26523627&form=6&db=m AMH MEASUREMENT VERSUS OVARIAN ULTRASOUND IN THE DIAGNOSIS OF POLYCYSTIC OVARY SYNDROME IN DIFFERENT PHENOTYPES. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,2,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30958034&form=6&db=m Association of Luteinizing hormone and LH receptor gene polymorphism with susceptibility of Polycystic ovary syndrome. unassigned - 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31149308&form=6&db=m THE RARE CASE OF MIXED GONADAL DYSGENESIS, MOSAIC KARYOTYPE, PETROCLIVAL MENINGIOMA AND IDIOPATHIC HYPERDEHYDROEPIANDROSTERONISM. unassigned - 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33045962&form=6&db=m Premature Adrenarche and its Association with Cardiovascular Risk in Females. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Polycystic Ovary Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33775010&form=6&db=m Dehydroepiandrosterone Sulfate (DHEAS) Levels in Polycystic Ovarian Syndrome (PCOS). diagnostic usage,ongoing research,therapeutic application,unassigned 2,1,2,0 3.1.6.2 Polycythemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22274717&form=6&db=m Aromatase activity after a short-course of letrozole administration in adult men at sea level and at high altitude (with or without excessive erythrocytosis). causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 Polymicrogyria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21931015&form=6&db=m Banding pattern on polarized hair microscopic examination and unilateral polymicrogyria in a patient with steroid sulfatase deficiency. causal interaction,unassigned 3,0 3.1.6.2 Polymyalgia Rheumatica http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12508908&form=6&db=m Hypothalamic-pituitary-adrenal axis impairment in the pathogenesis of rheumatoid arthritis and polymyalgia rheumatica. causal interaction,unassigned 3,0 3.1.6.2 Polymyalgia Rheumatica http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16783861&form=6&db=m Low serum levels of DHEAS in untreated polymyalgia rheumatica/giant cell arteritis. diagnostic usage,ongoing research,unassigned 2,3,0 3.1.6.2 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2962026&form=6&db=m Progesterone and estradiol production by normal and preeclamptic placentas. causal interaction,unassigned 3,0 3.1.6.2 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6448002&form=6&db=m Stable prolactin level after enhanced estradiol production following dehydroepiandrosterone sulfate. diagnostic usage,ongoing research,therapeutic application,unassigned 1,2,1,0 3.1.6.2 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8848592&form=6&db=m Concentrations of corticotrophin-releasing hormone in the umbilical-cord blood of pregnancies complicated by pre-eclampsia. causal interaction,diagnostic usage,unassigned 2,1,0 3.1.6.2 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15683369&form=6&db=m Early second-trimester maternal hyperandrogenemia and subsequent preeclampsia: a prospective study. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,1,0 3.1.6.2 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27871476&form=6&db=m Steroid sulfatase is increased in the placentas and whole blood of women with early-onset preeclampsia. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27992610&form=6&db=m Maternal Preeclampsia and Androgens in the Offspring around Puberty: A Follow-Up Study. causal interaction,unassigned 1,0 3.1.6.2 Pre-Eclampsia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32070380&form=6&db=m Levels of sex steroid hormones and their receptors in women with preeclampsia. diagnostic usage,unassigned 4,0 3.1.6.2 Precursor Cell Lymphoblastic Leukemia-Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27907849&form=6&db=m Association between dehydroepiandrosterone-sulfate and attention in long-term survivors of childhood acute lymphoblastic leukemia treated with only chemotherapy. ongoing research,therapeutic application,unassigned 1,1,0 3.1.6.2 Primary Ovarian Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27112552&form=6&db=m The importance of adrenal hypoandrogenism in infertile women with low functional ovarian reserve: a case study of associated adrenal insufficiency. diagnostic usage,unassigned 4,0 3.1.6.2 Primary Ovarian Insufficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30658614&form=6&db=m Adrenarche and pubarche in girls with turner syndrome during growth-promoting therapy with human growth hormone. unassigned - 3.1.6.2 Prostatic Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27645128&form=6&db=m Role of OATP transporters in steroid uptake by prostate cancer cells in vivo. causal interaction,diagnostic usage,therapeutic application,unassigned 2,2,3,0 3.1.6.2 Prostatic Hyperplasia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27030299&form=6&db=m Cross-sectional analysis between prostate volumes and serum sex hormones in Japanese men attending a urological clinic. causal interaction,unassigned 1,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2838688&form=6&db=m Differential metabolism of dehydroepiandrosterone sulfate and estrogen conjugates by normal or malignant AXC/SSh rat prostate cells and effects of these steroid conjugates on cancer cell proliferation in vitro. unassigned - 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15544928&form=6&db=m 6-[2-(adamantylidene)-hydroxybenzoxazole]-O-sulfamate, a steroid sulfatase inhibitor for the treatment of androgen- and estrogen-dependent diseases. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16502050&form=6&db=m Prostate cancer risk: the significance of differences in age related changes in serum conjugated and unconjugated steroid hormone concentrations between arab and caucasian men. diagnostic usage,ongoing research,unassigned 4,2,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16541422&form=6&db=m Steroid sulfatase and estrogen sulfotransferase in human prostate cancer. diagnostic usage,ongoing research,therapeutic application,unassigned 4,1,2,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16734850&form=6&db=m Do differences in age specific androgenic steroid hormone levels account for differing prostate cancer rates between Arabs and Caucasians? causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,1,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17277727&form=6&db=m The endocrine profiles in men with localized and locally advanced prostate cancer treated with radical prostatectomy. causal interaction,diagnostic usage,unassigned 3,3,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18268111&form=6&db=m V89L polymorphism of the 5alpha-reductase Type II gene (SRD5A2), endogenous sex hormones, and prostate cancer risk. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,4,1 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20621111&form=6&db=m Cytochrome P450 3A gene variation, steroid hormone serum levels and prostate cancer--The Rotterdam Study. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,1,1 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21904110&form=6&db=m Induction of steroid sulfatase expression by tumor necrosis factor-? through phosphatidylinositol 3-kinase/Akt signaling pathway in PC-3 human prostate cancer cells. ongoing research,unassigned 3,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24009850&form=6&db=m Bacterial Lipopolysaccharides Induce Steroid Sulfatase Expression and Cell Migration through IL-6 Pathway in Human Prostate Cancer Cells. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,3,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24055520&form=6&db=m Induction of steroid sulfatase expression in PC-3 human prostate cancer cells by insulin-like growth factor II. causal interaction,ongoing research,therapeutic application,unassigned 3,4,3,0 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26181260&form=6&db=m Statin Use at the Time of Initiation of Androgen Deprivation Therapy and Time to Progression in Patients With Hormone-Sensitive Prostate Cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 2,3,4,1 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27583544&form=6&db=m Influence of Statins on Survival Outcome in Patients with Metastatic Castration Resistant Prostate Cancer Treated with Abiraterone Acetate. unassigned - 3.1.6.2 Prostatic Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32928794&form=6&db=m Steroid sulfatase stimulates intracrine androgen synthesis and is a therapeutic target for advanced prostate cancer. causal interaction,diagnostic usage,therapeutic application,unassigned 4,1,4,0 3.1.6.2 Proteinuria http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16958298&form=6&db=m [High androgen plasmatic levels in women affected with pregnancy-induced hypertension] diagnostic usage,ongoing research,therapeutic application,unassigned 4,2,1,0 3.1.6.2 Psoriasis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7554562&form=6&db=m Blood dehydroepiandrosterone sulphate (DHEAS) levels in pemphigoid/pemphigus and psoriasis. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 3.1.6.2 Puberty, Delayed http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9499614&form=6&db=m X-linked ichthyosis with hypogonadism: not always Kallmann's syndrome. causal interaction,unassigned 3,0 3.1.6.2 Puberty, Precocious http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16059976&form=6&db=m [Relationship between serum dehydroepiandrosterone levels and female precocious puberty] causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 Puberty, Precocious http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18546533&form=6&db=m [Psychological behavior of girls with idiopathic central precocious puberty before and after treatment with gonadotropin-releasing hormone analogue] diagnostic usage,therapeutic application,unassigned 2,1,0 3.1.6.2 Pulmonary Disease, Chronic Obstructive http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11909687&form=6&db=m Dehydroepiandrosterone-sulfate serum levels and common age-related diseases: results from a cross-sectional Italian study of a general elderly population. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,1,0 3.1.6.2 Pulmonary Disease, Chronic Obstructive http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18021209&form=6&db=m Sex hormone alterations and systemic inflammation in chronic obstructive pulmonary disease. causal interaction,diagnostic usage,unassigned 2,1,0 3.1.6.2 Pulmonary Disease, Chronic Obstructive http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27940297&form=6&db=m Increased levels of enzymes involved in local estradiol synthesis in chronic obstructive pulmonary disease. causal interaction,diagnostic usage,ongoing research,unassigned 3,3,3,0 3.1.6.2 Pulmonary Disease, Chronic Obstructive http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28945888&form=6&db=m Estrogen Activation by Steroid Sulfatase increases Colorectal Cancer proliferation via GPER. causal interaction,ongoing research,therapeutic application,unassigned 3,4,1,0 3.1.6.2 Retroviridae Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9155639&form=6&db=m Modulation of immune dysfunction during murine leukaemia retrovirus infection of old mice by dehydroepiandrosterone sulphate (DHEAS). unassigned - 3.1.6.2 Retroviridae Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9648938&form=6&db=m Dehydroepiandrosterone (DHEA) sulfate prevents reduction in tissue vitamin E and increased lipid peroxidation due to murine retrovirus infection of aged mice. causal interaction,unassigned 4,0 3.1.6.2 Retroviridae Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10371406&form=6&db=m Immune dysfunction during alcohol consumption and murine AIDS: the protective role of dehydroepiandrosterone sulfate. ongoing research,unassigned 2,0 3.1.6.2 Rheumatic Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33592022&form=6&db=m Dehydroepiandrostenedione sulphate (DHEAS) levels predict high risk of rheumatoid arthritis (RA) in subclinical hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,4,0 3.1.6.2 Rhinitis, Allergic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9427150&form=6&db=m Allergic disease as an association of steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Sarcopenia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15123757&form=6&db=m Effect of DHEAS on skeletal muscle over the life span: the InCHIANTI study. unassigned - 3.1.6.2 Sarcopenia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23222704&form=6&db=m Sex hormones and sarcopenia in older persons. therapeutic application,unassigned 4,0 3.1.6.2 Schistosomiasis mansoni http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12716389&form=6&db=m The relationships between dehydroepiandrosterone sulphate (DHEAS), the intensity of Schistosoma mansoni infection and parasite-specific antibody responses. A cross-sectional study in residents of endemic communities in north-east Ethiopia. causal interaction,diagnostic usage,ongoing research,unassigned 3,1,2,0 3.1.6.2 Scleroderma, Systemic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9159534&form=6&db=m High prolactin and low dehydroepiandrosterone sulphate serum levels in patients with severe systemic sclerosis. diagnostic usage,ongoing research,unassigned 2,2,0 3.1.6.2 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11124400&form=6&db=m The effects of neurosteroids on picrotoxin-, bicuculline- and NMDA-induced seizures, and a hypnotic effect of ethanol. unassigned - 3.1.6.2 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15816945&form=6&db=m Seizure frequency and cortisol and dehydroepiandrosterone sulfate (DHEAS) levels in women with epilepsy receiving antiepileptic drug treatment. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,2,1,1 3.1.6.2 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17116037&form=6&db=m Differential effects of antiepileptic drugs on neuroactive steroids in men with epilepsy. causal interaction,diagnostic usage,therapeutic application,unassigned 1,1,1,0 3.1.6.2 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20205112&form=6&db=m Mean daily plasma concentrations of beta-endorphin, leu-enkephalin, ACTH, cortisol, and DHEAS in epileptic patients with complex partial seizures evolving to generalized tonic-clonic seizures. causal interaction,diagnostic usage,ongoing research,unassigned 2,2,1,0 3.1.6.2 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21931015&form=6&db=m Banding pattern on polarized hair microscopic examination and unilateral polymicrogyria in a patient with steroid sulfatase deficiency. causal interaction,unassigned 3,0 3.1.6.2 Seizures http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27051273&form=6&db=m Effects of acute and chronic administration of neurosteroid dehydroepiandrosterone sulfate on neuronal excitability in mice. ongoing research,therapeutic application,unassigned 4,3,0 3.1.6.2 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12398784&form=6&db=m Decreased levels of dehydroepiandrosterone sulphate in severe critical illness: a sign of exhausted adrenal reserve? causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,2,1 3.1.6.2 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15635504&form=6&db=m Adrenal androgens and the immune system. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16608898&form=6&db=m Dissociation of serum dehydroepiandrosterone and dehydroepiandrosterone sulfate in septic shock. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Sepsis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28810889&form=6&db=m Dehydroepiandrosterone sulfate and dehydroepiandrosterone sulfate/cortisol ratio in cirrhotic patients with septic shock: another sign of hepatoadrenal syndrome? causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 3.1.6.2 Sertoli Cell-Only Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33290605&form=6&db=m Testicular steroid sulfatase overexpression is associated with Leydig cell dysfunction in primary spermatogenic failure. diagnostic usage,ongoing research,unassigned 4,3,0 3.1.6.2 Shock, Septic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12398784&form=6&db=m Decreased levels of dehydroepiandrosterone sulphate in severe critical illness: a sign of exhausted adrenal reserve? causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,2,1 3.1.6.2 Shock, Septic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16608898&form=6&db=m Dissociation of serum dehydroepiandrosterone and dehydroepiandrosterone sulfate in septic shock. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,4,1 3.1.6.2 Shock, Septic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28810889&form=6&db=m Dehydroepiandrosterone sulfate and dehydroepiandrosterone sulfate/cortisol ratio in cirrhotic patients with septic shock: another sign of hepatoadrenal syndrome? causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 3.1.6.2 Skin Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6820437&form=6&db=m Review: genetics of steroid sulphatase deficiency and X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 Skin Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15544928&form=6&db=m 6-[2-(adamantylidene)-hydroxybenzoxazole]-O-sulfamate, a steroid sulfatase inhibitor for the treatment of androgen- and estrogen-dependent diseases. causal interaction,therapeutic application,unassigned 3,4,0 3.1.6.2 Skin Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11911290&form=6&db=m Serum dehydroepiandrosterone and dehydroepiandrosterone sulfate and risk of melanoma or squamous cell carcinoma of the skin. diagnostic usage,therapeutic application,unassigned 4,3,0 3.1.6.2 Smith-Lemli-Opitz Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12135803&form=6&db=m Advances in prenatal screening for Down syndrome: I. general principles and second trimester testing. unassigned - 3.1.6.2 Smith-Lemli-Opitz Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12850625&form=6&db=m Maternal serum unconjugated estriol as a predictor for Smith-Lemli-Opitz syndrome and other fetal conditions. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 Smith-Lemli-Opitz Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16390921&form=6&db=m Low estriol levels in the maternal triple-marker screen as a predictor of isolated adrenocorticotropic hormone deficiency caused by a new mutation in the TPIT gene. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 Smith-Lemli-Opitz Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20715120&form=6&db=m Prevalence of steroid sulfatase deficiency in California according to race and ethnicity. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 Smith-Lemli-Opitz Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24027193&form=6&db=m Are low maternal estriol levels a predictor for pro-opiomelanocortin (POMC) deficiency caused by POMC mutation during pregnancy? diagnostic usage,ongoing research,unassigned 3,1,0 3.1.6.2 Solitary Kidney http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7704955&form=6&db=m Hypogonadotrophic hypogonadism with hyposmia, X-linked ichthyosis, and renal malformation syndrome. diagnostic usage,unassigned 1,0 3.1.6.2 Spinal Cord Injuries http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30029726&form=6&db=m Astrocyte Neuroprotection and Dehydroepiandrosterone. therapeutic application,unassigned 4,0 3.1.6.2 Spinal Cord Ischemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10926963&form=6&db=m Dehydroepiandrosterone sulfate is neuroprotective in a reversible spinal cord ischemia model: possible involvement of GABA(A) receptors. ongoing research,unassigned 4,0 3.1.6.2 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15989553&form=6&db=m Aromatase inhibitors and other novel agents in breast cancer treatment. causal interaction,therapeutic application,unassigned 4,4,0 3.1.6.2 steroid 21-monooxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6219334&form=6&db=m Disproportionate suppression of dehydroepiandrosterone sulfate (DHEAS) in treated patients with congenital adrenal hyperplasia due to 21-hydroxylase deficiency. causal interaction,diagnostic usage,therapeutic application,unassigned 4,1,1,0 3.1.6.2 steroid 21-monooxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31627493&form=6&db=m [Screening for congenital adrenal hyperplasia in pregnancy: results and perspectives]. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=136200&form=6&db=m Prenatal diagnosis of placental steroid sulfatase deficiency. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=162557&form=6&db=m Four cases of pregnancy with low estrogen production due to placental enzymatic deficiency. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=294955&form=6&db=m Steroid sulfatase deficiency and X-linked ichthyosis. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=491629&form=6&db=m Steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=521732&form=6&db=m Urinary excretion of oestrone, oestradiol-17 beta and oestriol in pregnancies complicated by steroid sulphatase deficiency. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=887310&form=6&db=m Steroid sulfatase deficiency. causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1427599&form=6&db=m [Biochemical study on steroid sulfatase and its clinical application to the obstetrics and gynecology] causal interaction,diagnostic usage,ongoing research,unassigned 4,4,4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1496984&form=6&db=m Role of the pseudoautosomal region in sex-chromosome pairing during male meiosis: meiotic studies in a man with a deletion of distal Xp. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1549475&form=6&db=m Characterization of a low copy repetitive element S232 involved in the generation of frequent deletions of the distal short arm of the human X chromosome. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1639422&form=6&db=m Sequence analysis of a partial deletion of the human steroid sulfatase gene reveals 3 bp of homology at deletion breakpoints. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1690506&form=6&db=m The human arylsulfatase-C isoenzymes: two distinct genes that escape from X inactivation. causal interaction,unassigned 1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1785631&form=6&db=m Deletion of the distal short arm of the X chromosome (Xp) in a patient with short stature, chondrodysplasia punctata, and X-linked ichthyosis due to steroid sulfatase deficiency. causal interaction,ongoing research,unassigned 2,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1923309&form=6&db=m Ocular findings in X-linked ichthyosis: a survey on 38 cases. causal interaction,diagnostic usage,ongoing research,unassigned 2,1,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1951426&form=6&db=m Two cases of steroid sulfatase deficiency with complex phenotype due to contiguous gene deletions. causal interaction,unassigned 1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2253421&form=6&db=m [Two cases of X-linked ichthyosis associated with myopathies] causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2344613&form=6&db=m Frequent deletions of the human X chromosome distal short arm result from recombination between low copy repetitive elements. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2424217&form=6&db=m Lipoprotein electrophoresis in recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2424236&form=6&db=m Carrier identification in steroid sulphatase deficiency and recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2602357&form=6&db=m Contiguous gene syndromes due to deletions in the distal short arm of the human X chromosome. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2644167&form=6&db=m Molecular heterogeneity of steroid sulfatase deficiency: a multicenter study on 57 unrelated patients, at DNA and protein levels. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2722337&form=6&db=m Ichthyosis. Reliability of clinical signs in the differentiation between autosomal dominant and sex-linked forms. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2743959&form=6&db=m X-linked recessive ichthyosis. Enzymatic diagnosis of affected males and female carriers. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2859745&form=6&db=m Steroid sulfatase deficiency and the genetics of the short arm of the human X chromosome. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2866054&form=6&db=m Steroid sulphatase deficiency disease. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2931494&form=6&db=m Hydrolysis of deoxycorticosterone-21-yl sulphate and dehydroepiandrosterone sulphate by microsomal preparations of human placentae: evidence for a common enzyme. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2968067&form=6&db=m Antenatal detection of placental steroid sulphatase deficiency by measurement of urinary 16 alpha-hydroxydehydroepiandrosterone sulphate. causal interaction,diagnostic usage,unassigned 3,4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2977418&form=6&db=m Metabolism of 3H-dehydroepiandrosterone sulphate by subjects with steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3007328&form=6&db=m X-linked ichthyosis, due to steroid sulphatase deficiency, associated with Kallmann syndrome (hypogonadotropic hypogonadism and anosmia): linkage relationships with Xg and cloned DNA sequences from the distal short arm of the X chromosome. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3034252&form=6&db=m Genetic heterogeneity of steroid sulfatase deficiency revealed with cDNA for human steroid sulfatase. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3123779&form=6&db=m Studies on cross-reacting material to steroid sulphatase in fibroblasts from patients affected by different types of steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3162681&form=6&db=m Characterization of low-density lipoproteins from patients with recessive X-linked ichthyosis. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3163320&form=6&db=m X chromosome deletions detectable by flow cytometry in some patients with steroid sulphatase deficiency (X-linked ichthyosis). causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3165358&form=6&db=m Recurrent bilateral corneal erosions due to an association of epidermolysis bullosa simplex Köbner and X-linked ichthyosis with steroid sulfatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3422798&form=6&db=m More on X-linked ichthyosis, steroid sulfatase deficiency, and hypogonadism and anosmia. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3462435&form=6&db=m Regulation of serum testosterone in men with steroid sulfatase deficiency: response to human chorionic gonadotropin. causal interaction,diagnostic usage,ongoing research,unassigned 2,2,4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3464202&form=6&db=m Dosage of the Sts gene in the mouse. ongoing research,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3468016&form=6&db=m [Arylsulfatase C deficiency in leukocytes in patients and carrier of X-chromosome recessive ichthyosis] causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3469950&form=6&db=m Steroid sulfatase deficiency: enzymatic studies using placenta and leucocytes in one family. diagnostic usage,ongoing research,therapeutic application,unassigned 3,2,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3470015&form=6&db=m Human placental steryl-sulfatase. Enzyme purification, production of antisera, and immunoblotting reactions with normal and sulfatase-deficient placentas. causal interaction,unassigned 2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3470422&form=6&db=m Steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477126&form=6&db=m X-linked ichthyosis due to steroid sulfatase deficiency associated with hypogonadism and anosmia. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3477995&form=6&db=m [Steroid sulfatase and placental deficiency. Current data as instigators of new research] causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3478297&form=6&db=m Multipoint linkage analysis of steroid sulfatase (X-linked ichthyosis) and distal Xp markers. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3531932&form=6&db=m Placental sulfatase deficiency: maternal and fetal expression of steroid sulfatase deficiency and X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3556047&form=6&db=m Human steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3709904&form=6&db=m Ichthyosis of steroid sulphatase deficiency. Clinical study of 76 cases. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3855654&form=6&db=m Lactation in placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3857937&form=6&db=m Steroid sulphatase deficiency in patients initially diagnosed as ichthyosis vulgaris or recessive X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3860193&form=6&db=m Placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3863670&form=6&db=m Lactation in placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864079&form=6&db=m Placental steroid sulfatase deficiency. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864397&form=6&db=m Familial X-linked ichthyosis, steroid sulfatase deficiency, mental retardation, and nullisomy for Xp223-pter. causal interaction,therapeutic application,unassigned 2,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3864567&form=6&db=m Abnormal androgen and oestrogen metabolism in men with steroid sulphatase deficiency and recessive X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3865454&form=6&db=m [Placental steroid sulfatase deficiency. Reduced estriol production] unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3883479&form=6&db=m Determination of cholesteryl sulphate by isotope dilution-mass spectrometry for diagnosis of steroid sulphatase deficiency. causal interaction,diagnostic usage,unassigned 2,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3914597&form=6&db=m [Ichthyosis X. Clinical review and rapid identification using lipid electrophoresis] causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6102271&form=6&db=m Heterozygote detection in steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6123259&form=6&db=m Placental steroid deficiency: association with arylsulfatase A deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6135610&form=6&db=m Clinical spectrum of steroid sulfatase deficiency: X-linked recessive ichthyosis, birth complications and cryptorchidism. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6140547&form=6&db=m Steroid sulphatase deficiency associated with testis cancer. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6231946&form=6&db=m Antenatal detection of placental steroid sulphatase deficiency: use of a dehydroepiandrosterone sulphate loading test. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6234482&form=6&db=m Placental steroid sulfatase deficiency: biochemical diagnosis and clinical review. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,4,1 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6407677&form=6&db=m Recognising placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6419868&form=6&db=m Recognising placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6422158&form=6&db=m Hair root analysis in X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6434877&form=6&db=m Steroid sulphatase deficiency. Steroid sulphatase and arylsulphatase C determination in normal and affected fibroblasts. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6434878&form=6&db=m Steroid sulphatase deficiency is present in patients with the syndrome 'ichthyosis and male hypogonadism' and with 'Rud syndrome'. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6448625&form=6&db=m Serum steroid sulphates in ichthyosis. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6461439&form=6&db=m Prenatal diagnosis of X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6489382&form=6&db=m Steroid sulphatase deficiency and hypogonadism. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6572291&form=6&db=m Studies of the biochemical basis of steroid sulphatase deficiency--III. Phospholipid composition of microsomes from normal and steroid sulphatase deficient placentas. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6573909&form=6&db=m A new method for the determination of steroid sulphatase activity in leukocytes in X-linked recessive ichthyosis. causal interaction,diagnostic usage,unassigned 3,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6584688&form=6&db=m The nature of placental steroid sulphatase deficiency in man. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6596909&form=6&db=m Placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6598228&form=6&db=m Foetal steroid sulphatase deficiency. A case report. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6820437&form=6&db=m Review: genetics of steroid sulphatase deficiency and X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6929654&form=6&db=m X-linked ichthyosis and X-linked placental sulfatase deficiency: a disease entity. Histochemical observations. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6930526&form=6&db=m Studies of the biochemical basis of steroid sulphatase deficiency: preliminary evidence suggesting a defect in membrane-enzyme structure. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6930527&form=6&db=m 3 beta-Hydroxysteroid dehydrogenase-isomerase activity in placentae from pregnancies complicated by steroid sulphatase deficiency. causal interaction,unassigned 2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6940862&form=6&db=m Increased cholesterol sulfate in plasma and red blood cell membranes of steroid sulfatase deficient patients. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6958397&form=6&db=m The role of antenatal oestrogen in post-partum human lactogenesis: evidence from oestrogen-deficient pregnancies. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6958681&form=6&db=m Antenatal diagnosis of steroid sulphatase deficiency: case report and literature survey. causal interaction,diagnostic usage,therapeutic application,unassigned 4,2,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6985463&form=6&db=m Regional assignment of the gene locus for steroid sulfatase. causal interaction,unassigned 1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7426407&form=6&db=m Sex-linked ichthyosis and placental sulphatase C deficiency. causal interaction,ongoing research,unassigned 4,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7521964&form=6&db=m Second-trimester maternal serum screening using alpha-fetoprotein, human chorionic gonadotrophin, and unconjugated oestriol: experience of a regional programme. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7554562&form=6&db=m Blood dehydroepiandrosterone sulphate (DHEAS) levels in pemphigoid/pemphigus and psoriasis. causal interaction,diagnostic usage,ongoing research,unassigned 4,4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7726134&form=6&db=m Low maternal serum unconjugated estriol during prenatal screening as an indication of placental steroid sulfatase deficiency and X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7794518&form=6&db=m Very low maternal serum unconjugated estriol and prenatal diagnosis of steroid sulfatase deficiency. causal interaction,diagnostic usage,therapeutic application,unassigned 1,4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7943126&form=6&db=m Clinical pathologic correlation of superficial corneal opacities in X-linked ichthyosis. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7951263&form=6&db=m Characterization of the deletion breakpoints in a patient with steroid sulfatase deficiency. ongoing research,unassigned 2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8001874&form=6&db=m Steroid sulfatase activity in human leukocytes. causal interaction,diagnostic usage,ongoing research,unassigned 1,3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8052574&form=6&db=m Prenatal detection of X-linked ichthyosis by maternal serum screening for Down syndrome. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8059846&form=6&db=m Maternal steroid sulfatase deficiency--cause of high-risk pregnancy? unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8186420&form=6&db=m Steroid sulfatase deficiency in Japanese patients: characterization of X-linked ichthyosis by using polymerase chain reaction. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8262159&form=6&db=m Correction of steroid sulfatase deficiency by gene transfer into basal cells of tissue-cultured epidermis from patients with recessive X-linked ichthyosis. causal interaction,ongoing research,unassigned 1,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8330450&form=6&db=m Deletion analysis maps ocular albinism proximal to the steroid sulphatase locus. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8403433&form=6&db=m Enzymatic diagnosis of steroid sulfatase deficiency by high performance liquid chromatography. diagnostic usage,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8791985&form=6&db=m Steroid sulphatase deficiency: identification of heterozygotes using hydrolysis of dehydroepiandrosterone sulphate by peripheral leucocytes. causal interaction,diagnostic usage,therapeutic application,unassigned 3,2,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9101220&form=6&db=m Prenatal diagnosis of X-linked ichthyosis using molecular cytogenetics. diagnostic usage,unassigned 1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9336808&form=6&db=m Ichthyosis: the skin manifestation of multiple sulfatase deficiency. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,2 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9427150&form=6&db=m Allergic disease as an association of steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9499614&form=6&db=m X-linked ichthyosis with hypogonadism: not always Kallmann's syndrome. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9583203&form=6&db=m Frequency of steroid sulfatase deficiency in Hiroshima. diagnostic usage,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9640374&form=6&db=m X-linked ichthyosis: relation between cholesterol sulphate, dehydroepiandrosterone sulphate and patient's age. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9742566&form=6&db=m Steroid sulphatase deficiency is the major cause of extremely low oestriol production at mid-pregnancy: a urinary steroid assay for the discrimination of steroid sulphatase deficiency from other causes. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9764665&form=6&db=m Unconjugated estriol as an indication for prenatal diagnosis of steroid sulfatase deficiency by in situ hybridization. causal interaction,diagnostic usage,therapeutic application,unassigned 3,4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10092953&form=6&db=m Steroid sulfatase activity in leukocytes: a comparative study in 45,X; 46,Xi(Xq) and carriers of steroid sulfatase deficiency. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10533035&form=6&db=m Submicroscopic Xpter deletion in a boy with growth and mental retardation caused by a familial t(X;14). unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10692123&form=6&db=m Mutation report: a novel partial deletion of exons 2-10 of the STS gene in recessive X-linked ichthyosis. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10844566&form=6&db=m Novel point mutations in the steroid sulfatase gene in patients with X-linked ichthyosis: transfection analysis using the mutated genes. causal interaction,ongoing research,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10905086&form=6&db=m [Occurrence of X-linked ichthyosis along with atopy] causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11155790&form=6&db=m Replacement therapy with DHEA plus corticosteroids in patients with chronic inflammatory diseases--substitutes of adrenal and sex hormones. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11231321&form=6&db=m Deletion of exons 1-5 of the STS gene causing X-linked ichthyosis. causal interaction,diagnostic usage,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11676848&form=6&db=m Maternal transmission of the 3 bp deletion within exon 7 of the STS gene in steroid sulfatase deficiency. causal interaction,therapeutic application,unassigned 1,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11844872&form=6&db=m Deletion pattern of the STS gene in X-linked ichthyosis in a Mexican population. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11853283&form=6&db=m Serum dehydroepiandrosterone sulfate, testosterone, and biochemical markers of bone turnover in elderly Thai men. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,4 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12014422&form=6&db=m Association of dehydroepiandrosterone sulfate and testosterone deficiency with bone turnover in men with inflammatory bowel disease. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,1 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12135803&form=6&db=m Advances in prenatal screening for Down syndrome: I. general principles and second trimester testing. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12174111&form=6&db=m Mild ichthyosis in a 4-year-old boy with multiple sulphatase deficiency. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12364433&form=6&db=m Prenatal diagnosis of sex differentiation disorders: the role of fetal ultrasound. therapeutic application,unassigned 1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12406347&form=6&db=m Somatic and germinal mosaicism for the steroid sulfatase gene deletion in a steroid sulfatase deficiency carrier. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,4,2,1 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12424769&form=6&db=m Low or absent unconjugated estriol in pregnancy: an indicator for steroid sulfatase deficiency detectable by fluorescence in situ hybridization and biochemical analysis. diagnostic usage,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12850625&form=6&db=m Maternal serum unconjugated estriol as a predictor for Smith-Lemli-Opitz syndrome and other fetal conditions. causal interaction,diagnostic usage,unassigned 2,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12970278&form=6&db=m Congenital hypopituitarism as a cause of undetectable estriol levels in the maternal triple-marker screen. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14646417&form=6&db=m Association of undetectable unconjugated estriol on multiple marker screening with steroid sulfatase deficiency. causal interaction,diagnostic usage,unassigned 1,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16504797&form=6&db=m Steroid Sulfatase Deficiency With Bilateral Periventricular Nodular Heterotopia. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16720460&form=6&db=m X-chromosome inactivation: role in skin disease expression. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17022596&form=6&db=m [Steroid sulphatase deficiency] causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17157776&form=6&db=m DHEAS deficiency during consumption of sustained-action prescribed opioids: evidence for opioid-induced inhibition of adrenal androgen production. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19395096&form=6&db=m Gonadal and adrenal androgen deficiencies as independent predictors of increased cardiovascular mortality in men with type II diabetes mellitus and stable coronary artery disease. causal interaction,diagnostic usage,therapeutic application,unassigned 3,3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19418464&form=6&db=m Maternal urine and serum steroid measurements to identify steroid sulfatase deficiency (STSD) in second trimester pregnancies. diagnostic usage,unassigned 2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19609942&form=6&db=m Steroid sulfatase deficiency and contiguous gene deletion syndrome amongst pregnant patients with low serum unconjugated estriols. diagnostic usage,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20595194&form=6&db=m Deficiencies in circulating testosterone and dehydroepiandrosterone sulphate, and depression in men with systolic chronic heart failure. causal interaction,diagnostic usage,therapeutic application,unassigned 4,4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20715120&form=6&db=m Prevalence of steroid sulfatase deficiency in California according to race and ethnicity. diagnostic usage,ongoing research,unassigned 3,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21533175&form=6&db=m Eight common genetic variants associated with serum DHEAS levels suggest a key role in ageing mechanisms. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21723668&form=6&db=m Steroid sulfatase-deficient mice exhibit endophenotypes relevant to Attention Deficit Hyperactivity Disorder. ongoing research,unassigned 1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21931015&form=6&db=m Banding pattern on polarized hair microscopic examination and unilateral polymicrogyria in a patient with steroid sulfatase deficiency. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22419362&form=6&db=m Steroid-resistant nephrotic syndrome associated with steroid sulfatase deficiency-x-linked recessive ichthyosis: a case report and review of literature. causal interaction,therapeutic application,unassigned 4,3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22475435&form=6&db=m Does steroid sulfatase deficiency influence postpartum psychosis risk? unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23803163&form=6&db=m Anabolic deficiencies in men with systolic heart failure: do co-morbidities and therapies really contribute significantly? causal interaction,therapeutic application,unassigned 4,2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24027193&form=6&db=m Are low maternal estriol levels a predictor for pro-opiomelanocortin (POMC) deficiency caused by POMC mutation during pregnancy? diagnostic usage,ongoing research,unassigned 3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24119255&form=6&db=m Nonsyndromic types of ichthyoses - an update. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25502769&form=6&db=m High levels of oxysterol sulfates in serum of patients with steroid sulfatase deficiency. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26239050&form=6&db=m Simultaneous quantification of cholesterol sulfate, androgen sulfates, and progestagen sulfates in human serum by LC-MS/MS. diagnostic usage,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27264819&form=6&db=m [Genetic analysis of a rare case with Kallman syndrome and steroid sulfatase deficiency]. unassigned - 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27531568&form=6&db=m Role of steroid sulfatase in steroid homeostasis and characterization of the sulfated steroid pathway: Evidence from steroid sulfatase deficiency. causal interaction,unassigned 2,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27817780&form=6&db=m [Identification of gene mutation and prenatal diagnosis in a family with X-linked ichthyosis]. causal interaction,therapeutic application,unassigned 3,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27919139&form=6&db=m [A Case of Steroid Sulfatase Deficiency Complicated by Bilateral Undescended Testis]. causal interaction,unassigned 3,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29678582&form=6&db=m Reversion of aging-related DHEAS decline in mouse plasma alleviates aging-related glucose tolerance impairment by potentiation of glucose-stimulated insulin secretion of acute phase. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30531180&form=6&db=m Placental steroid sulphatase deficiency: an approach to antenatal care and delivery. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=100598359&form=6&db=m Recognising placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=100608511&form=6&db=m : Recognising placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=100608550&form=6&db=m : Recognising placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 steryl-sulfatase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=100608672&form=6&db=m : Recognising placental steroid sulphatase deficiency. causal interaction,unassigned 4,0 3.1.6.2 Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1423966&form=6&db=m Dehydroepiandrosterone sulfate, incidence of myocardial infarction, and extent of atherosclerosis in men. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,4 3.1.6.2 Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14609305&form=6&db=m Are low dehydroepiandrosterone sulphate levels predictive for cardiovascular diseases? A review of prospective and retrospective studies. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,2,0 3.1.6.2 sulfate adenylyltransferase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31461705&form=6&db=m Low DHEAS Concentration in a Girl Presenting with Short Stature and Premature Pubarche: A Novel PAPSS2 Gene Mutation. causal interaction,unassigned 4,0 3.1.6.2 Testicular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2865141&form=6&db=m Absence of testicular steroid sulphatase activity in a boy with recessive X-linked ichthyosis and testicular maldescent. causal interaction,unassigned 4,0 3.1.6.2 Testicular Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2863590&form=6&db=m Lack of an association between testicular cancers and low steroid sulphatase activity in peripheral leucocytes. diagnostic usage,ongoing research,unassigned 3,1,0 3.1.6.2 Testicular Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=6140547&form=6&db=m Steroid sulphatase deficiency associated with testis cancer. causal interaction,unassigned 4,0 3.1.6.2 Tetanus http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8597462&form=6&db=m DHEAS as an effective vaccine adjuvant in elderly humans. Proof-of-principle studies. causal interaction,ongoing research,therapeutic application,unassigned 1,4,2,0 3.1.6.2 Thalassemia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26839091&form=6&db=m Evaluation of the glucocorticoid, mineralocorticoid, and adrenal androgen secretion dynamics in a large cohort of patients aged 6-18 years with transfusion-dependent ?-thalassemia major, with an emphasis on the impact of cardiac iron load. causal interaction,diagnostic usage,ongoing research,unassigned 1,4,4,0 3.1.6.2 Thyroid Nodule http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9100552&form=6&db=m Decreased serum IGF-I and dehydroepiandrosterone sulphate may be risk factors for the development of reduced bone mass in postmenopausal women with endogenous subclinical hyperthyroidism. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,4,1,1 3.1.6.2 Thyroiditis, Autoimmune http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9678543&form=6&db=m Dehydroepiandrosterone sulfate does not prevent spontaneous and iodine-induced lymphocytic thyroiditis and diabetes mellitus in the BB/Wor rat. ongoing research,unassigned 3,0 3.1.6.2 Thyrotoxicosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11523906&form=6&db=m Serum concentration of androstenediol and androstenediol sulfate in patients with hyperthyroidism and hypothyroidism. causal interaction,diagnostic usage,ongoing research,unassigned 4,1,3,0 3.1.6.2 Toxoplasmosis, Ocular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22153766&form=6&db=m Serum levels of dehydroepiandrosterone sulfate (DHEAS) in ocular toxoplasmosis. ongoing research,therapeutic application,unassigned 3,1,0 3.1.6.2 Trisomy 13 Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7521964&form=6&db=m Second-trimester maternal serum screening using alpha-fetoprotein, human chorionic gonadotrophin, and unconjugated oestriol: experience of a regional programme. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,1,0 3.1.6.2 Trisomy 18 Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7521964&form=6&db=m Second-trimester maternal serum screening using alpha-fetoprotein, human chorionic gonadotrophin, and unconjugated oestriol: experience of a regional programme. causal interaction,diagnostic usage,ongoing research,unassigned 2,3,1,0 3.1.6.2 Trisomy 18 Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12135803&form=6&db=m Advances in prenatal screening for Down syndrome: I. general principles and second trimester testing. unassigned - 3.1.6.2 Tuberculosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15233561&form=6&db=m The endocrinology of adrenal tuberculosis: the effects of tuberculosis on the hypothalamo-pituitary-adrenal axis and adrenocortical function. causal interaction,unassigned 4,0 3.1.6.2 Tuberculosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17082909&form=6&db=m Dehydroepiandrosterone sulphate enhances IgG and Interferon-gamma production during immunization to tuberculosis in young but not aged mice. ongoing research,therapeutic application,unassigned 4,1,0 3.1.6.2 Turner Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10468952&form=6&db=m Reduced androgen levels in adult turner syndrome: influence of female sex steroids and growth hormone status. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,1,0 3.1.6.2 Turner Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12135803&form=6&db=m Advances in prenatal screening for Down syndrome: I. general principles and second trimester testing. unassigned - 3.1.6.2 Turner Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15001603&form=6&db=m The early dehydroepiandrosterone sulfate rise of adrenarche and the delay of pubarche indicate primary ovarian failure in Turner syndrome. causal interaction,diagnostic usage,unassigned 2,4,0 3.1.6.2 Turner Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31613320&form=6&db=m Effect of Dosage of 17ß-Estradiol on Uterine Growth in Turner Syndrome-A Randomized Controlled Clinical Pilot Trial. causal interaction,diagnostic usage,therapeutic application,unassigned 4,1,2,0 3.1.6.2 Urinary Bladder Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30542396&form=6&db=m Steroid sulfatase promotes invasion through epithelial-mesenchymal transition and predicts the progression of bladder cancer. diagnostic usage,therapeutic application,unassigned 3,1,0 3.1.6.2 Uterine Cervical Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18817841&form=6&db=m Estrogen formation in endometrial and cervix cancer cell lines: Involvement of aromatase, steroid sulfatase and 17beta-hydroxysteroid dehydrogenases (types 1, 5, 7 and 12). diagnostic usage,ongoing research,unassigned 1,4,0 3.1.6.2 Uterine Cervical Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27956712&form=6&db=m Induction of Integrin Signaling by Steroid Sulfatase in Human Cervical Cancer Cells. ongoing research,unassigned 3,0 3.1.6.2 Uterine Cervical Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28977889&form=6&db=m Human steroid sulfatase induces Wnt/?-catenin signaling and epithelial-mesenchymal transition by upregulating Twist1 and HIF-1? in human prostate and cervical cancer cells. ongoing research,unassigned 3,0 3.1.6.2 Vascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7498969&form=6&db=m Effects of dehydroepiandrosterone sulfate on cellular calcium responsiveness and vascular contractility. ongoing research,unassigned 4,0 3.1.6.2 Vascular Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10337439&form=6&db=m Dehydroepiandrosterone sulfate, diseases and mortality in a general aged population. causal interaction,diagnostic usage,unassigned 4,4,0 3.1.6.2 Vascular System Injuries http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19298964&form=6&db=m Adrenal androgen dehydroepiandrosterone sulfate inhibits vascular remodeling following arterial injury. causal interaction,therapeutic application,unassigned 4,1,0 3.1.6.2 Vertigo http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9583785&form=6&db=m Effects of the neurosteroid dehydroepiandrosterone sulfate on medial vestibular nucleus neurons. causal interaction,therapeutic application,unassigned 1,1,0 3.1.6.2 Vitiligo http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31203593&form=6&db=m Association of serum cortisol and dehydroepiandrosterone sulfate (DHEAS) levels with psychological stress in patients with vitiligo unassigned - 3.1.6.2 Whooping Cough http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11454934&form=6&db=m Metabotropic neurosteroid/sigma-receptor involved in stimulation of nociceptor endings of mice. unassigned - 3.1.6.2 Wilms Tumor http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33312738&form=6&db=m New frontiers on the molecular underpinnings of hypospadias according to severity. diagnostic usage,unassigned 1,0