4.1.3.16	4-hydroxy-2-oxoglutarate aldolase deficiency	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22729392&form=6&db=m	Primary hyperoxaluria type III-a model for studying perturbations in glyoxylate metabolism.	causal interaction,unassigned	4,0	
4.1.3.16	4-hydroxy-2-oxoglutarate aldolase deficiency	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22781098&form=6&db=m	Novel findings in patients with primary hyperoxaluria type III and implications for advanced molecular testing strategies.	causal interaction,therapeutic application,unassigned	4,3,0	
4.1.3.16	Hyperoxaluria	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22781098&form=6&db=m	Novel findings in patients with primary hyperoxaluria type III and implications for advanced molecular testing strategies.	causal interaction,therapeutic application,unassigned	4,3,0	
4.1.3.16	Hyperoxaluria	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33350326&form=6&db=m	Mutations in HOGA1 do Not Confer a Dominant Phenotype Manifesting as Kidney Stone Disease.	causal interaction,ongoing research,unassigned	4,3,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21896830&form=6&db=m	Primary hyperoxaluria type III gene HOGA1 (formerly DHDPSL) as a possible risk factor for idiopathic calcium oxalate urolithiasis.	diagnostic usage,unassigned	3,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22115483&form=6&db=m	Re: Mutations in DHDPSL are Responsible for Primary Hyperoxaluria Type III.	causal interaction,unassigned	4,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22729392&form=6&db=m	Primary hyperoxaluria type III-a model for studying perturbations in glyoxylate metabolism.	causal interaction,unassigned	4,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25972204&form=6&db=m	Renal function can be impaired in children with primary hyperoxaluria type 3.	causal interaction,unassigned	2,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27096395&form=6&db=m	Cellular degradation of 4-hydroxy-2-oxoglutarate aldolase leads to absolute deficiency in primary hyperoxaluria type 3.	causal interaction,unassigned	4,0	
4.1.3.16	Hyperoxaluria, Primary	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31401635&form=6&db=m	Mutation Hot Spot Region in the HOGA1 Gene Associated with Primary Hyperoxaluria Type 3 in the Chinese Population.	unassigned	-