3.5.3.18	Asthma	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24465497&form=6&db=m	Overexpression of dimethylarginine dimethylaminohydrolase 1 attenuates airway inflammation in a mouse model of asthma.	causal interaction,diagnostic usage,ongoing research,therapeutic application	4,1,4,1	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17293168&form=6&db=m	Taurine protects against low-density lipoprotein-induced endothelial dysfunction by the DDAH/ADMA pathway.	causal interaction,therapeutic application,unassigned	4,1,0	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19603041&form=6&db=m	Dimethylarginine dimethylaminohydrolase in rat penile tissue: reduced enzyme activity is responsible for erectile dysfunction in a rat model of atherosclerosis.	causal interaction,ongoing research,therapeutic application,unassigned	4,2,1,0	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20348244&form=6&db=m	Dimethylarginine dimethylaminohydrolase overexpression ameliorates atherosclerosis in apolipoprotein E-deficient mice by lowering asymmetric dimethylarginine.	causal interaction,therapeutic application,unassigned	3,4,0	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20462645&form=6&db=m	Improvement of endothelial dysfunction in atherosclerotic rabbit aortas by ex vivo gene transferring of dimethylarginine dimethylaminohydrolase-2.	causal interaction,diagnostic usage,ongoing research,therapeutic application	4,1,3,3	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24690995&form=6&db=m	Effect of Lowering Asymmetric Dimethylarginine (ADMA) on Vascular Pathology in Atherosclerotic ApoE-Deficient Mice with Reduced Renal Mass.	diagnostic usage,ongoing research,unassigned	1,4,0	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26226438&form=6&db=m	FoxO1 regulates asymmetric dimethylarginine via downregulation of dimethylaminohydrolase 1 in human endothelial cells and subjects with atherosclerosis.	ongoing research,unassigned	4,0	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33535073&form=6&db=m	Treatment of atherosclerosis through transplantation of endothelial progenitor cells overexpressing dimethylarginine dimethylaminohydrolase (DDAH) in rabbits.	causal interaction,ongoing research,therapeutic application,unassigned	4,2,4,0	
3.5.3.18	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34438260&form=6&db=m	Hyperuricemia induces endothelial dysfunction and accelerates atherosclerosis by disturbing the asymmetric dimethylarginine/dimethylarginine dimethylaminotransferase 2 pathway.	ongoing research,therapeutic application,unassigned	2,1,0	
3.5.3.18	Brain Ischemia	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33271854&form=6&db=m	Sequence Variation in the DDAH1 Gene Predisposes for Delayed Cerebral Ischemia in Subarachnoidal Hemorrhage.	causal interaction,ongoing research,unassigned	1,1,0