6.2.1.1	3T3-L1 cell	marked induction of AceCS1 mRNA and protein during differentiation of 3T3-L1 cells, neither AceCS2 mRNA nor protein is detected in undifferentiated or differentiated 3T3-L1 cells	652101	
6.2.1.1	A-549 cell	-	744656	
6.2.1.1	ACHN cell	-	744665	
6.2.1.1	adipose tissue	-	563, 572	
6.2.1.1	AsPC-1 cell	-	744656	
6.2.1.1	B-16 cell	cells express higher levels of cytosolic acetyl-CoA synthetase ACSS2 under hypoxia than normoxia. Knockdown of ACSS2 by RNA interference in tumor cells enhances tumor cell death under long-term hypoxia in vitro. The ACSS2 suppression slows tumor growth in vivo. Tumor cells excrete acetate and the quantity increases under hypoxia, the pattern of acetate excretion follows the expression pattern of ACSS2. The ACSS2 knockdown leads to a corresponding reduction in the acetate excretion in tumor cells	703038	
6.2.1.1	bacteroid	from Glycine max L. cv. Williams inoculated with Bradyrhizobium japonicum	568	
6.2.1.1	brain	-	562, 563, 572	
6.2.1.1	brain	AceCS2, low activity	652101	
6.2.1.1	breast tumor cell	-	744620